Diagnosis of Allergic bronchopulmonary aspergillosis
Diagnostic Test list for Allergic bronchopulmonary aspergillosis:
The list of medical tests
mentioned in various sources as
used in the diagnosis of Allergic bronchopulmonary aspergillosis
includes:
- Fungi skin testing, blood tests, X-rays, Fungi sputum tests
Allergic bronchopulmonary aspergillosis Diagnosis: Book Excerpts
Diagnostic Tests for Allergic bronchopulmonary aspergillosis: Online Medical Books
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AUSCULTATORY SIGNS OF PULMONARY DISEASE:
Approach to the Diagnosis
(Differential Diagnosis in Primary Care)
Clinically, the grouping together of signs provides the best way of narrowing the differential diagnosis.
Rales
- Bilateral crepitant rales, lack of dullness, and normal breath sounds suggest pulmonary edema or pneumonitis.
- Focal crepitant rales, reduced alveolar breathing, dullness to percussion, and increased tactile and vocal fremitus suggest lobar pneumonia or pulmonary infarction.
- Bilateral sibilant and sonorous rales without dullness and with increased bronchial breathing suggest asthma, chronic bronchitis and emphysema, acute bronchitis or bronchiolitis, and cardiac asthma.
- Focal crepitant rales and amphoric breathing with dullness below and hyperresonance above suggest a lung abscess or cavitation
Hyperresonance
- Hyperresonance bilaterally with diminished breath sounds bilaterally and sibilant rales suggests pulmonary emphysema or asthma.
- Focal hyperresonance with diminished or absent breath sounds and no rales suggests pneumothorax.
- Focal hyperresonance with normal or only diminished breath sounds suggests a large bulla.
Dullness or Flatness
- Dullness with diminished breath sounds and no rales suggests atelectasis or pleural effusion from empyema, CHF, or pulmonary infarct. In atelectasis, there is no hyperresonance or egophony above the dullness.
- Dullness with diminished breath sounds and crepitant rales suggests pneumonia or pulmonary infarct. If there is bronchophony as well, there is probably no associated effusion. If there is no bronchophony but hyperresonance and egophony above the dullness, then an associated pleural effusion should be considered.
» READ BOOK EXCERPT ONLINE »
Source: Differential Diagnosis in Primary Care, 2007
Aspergillosis:
Diagnosis
(Professional Guide to Diseases (Eighth Edition))
In patients with aspergilloma, a chest X-ray reveals a crescent-shaped radiolucency surrounding a circular mass, but this isn’t definitive for aspergillosis.
CONFIRMING DIAGNOSIS In aspergillosis endophthalmitis, a history of ocular trauma or surgery and a culture or exudate showing Aspergillus is diagnostic. In disseminated aspergillosis, culture and microscopic examination of affected tissue can confirm the diagnosis, but this form is usually diagnosed at autopsy.
In allergic aspergillosis, sputum examination shows eosinophils. Culture of mouth scrapings or sputum showing Aspergillus is inconclusive because even healthy people harbor this fungus.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Allergic purpuras:
Diagnosis
(Professional Guide to Diseases (Eighth Edition))
No laboratory test clearly identifies allergic purpura (although white blood cell count and erythrocyte sedimentation rate are elevated). Diagnosis therefore necessitates careful clinical observation, in many cases during the second or third attack. Except for a positive tourniquet test (a test to assess the capillaries’ability to withstand increased pressure), coagulation and platelet function tests are usually normal. Small-bowel X-rays may reveal areas of transient edema; in many cases, tests for blood in the urine and stool are positive. Increased blood urea nitrogen and creatinine levels may indicate renal involvement. Diagnosis must rule out other forms of nonthrombocytopenic purpura.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Solitary Pulmonary Nodule:
History
(The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter)
Obtain a complete history, including smoking, occupational exposure, immigration, and travel. Check previous chest x-ray studies to establish prior presence of a nodule, as well as growth on an existing nodule. An absence of growth over a period of 2 years is generally accepted as a sign of the benign nature of a SPN.
Physical examination
should include a search for evidence of weight loss, chronic obstructive pulmonary disease, and primary or metastatic disease of other organs.
» READ BOOK EXCERPT ONLINE »
Source: The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter, 2000
anaphylaxis:
Diagnosis
(Handbook of Diseases)
Anaphylaxis can be diagnosed by the rapid onset of severe respiratory or cardiovascular symptoms after ingestion or injection of a drug, vaccine, diagnostic agent, food, or food additive or after an insect sting. If these symptoms occur without a known allergic stimulus, rule out other possible causes of shock (such as acute myocardial infarction, status asthmaticus, and heart failure).
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Aspergillosis:
Diagnosis
(Handbook of Diseases)
In patients with aspergilloma, a chest X-ray reveals a crescent-shaped radiolucency surrounding a circular mass, but this isn’t definitive for aspergillosis. In patients with aspergillosis endophthalmitis, a history of ocular trauma or surgery and a culture or exudate showing Aspergillus supports the diagnosis. In patients with allergic aspergillosis, sputum examination shows eosinophils. Culture of mouth scrapings or sputum showing Aspergillus is inconclusive because even healthy persons harbor this fungus. In patients with disseminated aspergillosis, culture and microscopic examination of affected tissue can confirm the diagnosis, but this form is usually diagnosed at autopsy.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Allergic purpura:
Diagnosis
(Handbook of Diseases)
No laboratory test result clearly identifies allergic purpura (although the white blood cell count and erythrocyte sedimentation rate are elevated). Diagnosis therefore requires careful observation, usually during the second or third attack. Except for a positive tourniquet test result, coagulation and platelet function test results are usually negative. X-rays of the small bowel may reveal areas of transient edema; test results for blood in the urine and stool are often positive. Increased blood urea nitrogen and serum creatinine levels may indicate renal involvement. The diagnosis must rule out other forms of nonthrombocytopenic purpura.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Coccidioido-mycosis:
Diagnosis
(Handbook of Diseases)
Typical signs and symptoms and skin and serologic studies confirm the diagnosis. The primary form — and sometimes the disseminated form — produces an abnormal coccidioidin skin test result. In the first week of illness, complement fixation for immunoglobulin G antibodies or, in the first month, positive serum precipitins (immunoglobulins) also establish this diagnosis.
Examination or, more recently, immunodiffusion testing of sputum, pus from lesions, and a tissue biopsy may show C. immitis spores. The presence of antibodies in pleural and joint fluid and a rising serum or body fluid antibody titer indicate dissemination.
Other abnormal laboratory results include an increased white blood cell (WBC) count, increased eosinophil count, increased erythrocyte sedimentation rate, and a chest X-ray showing bilateral diffuse infiltrates.
In coccidioidal meningitis, examination of cerebrospinal fluid shows the WBC count increased to more than 500/µl (primarily because of mononuclear leukocytes) and increased protein and decreased glucose levels. Ventricular fluid obtained from the brain may contain complement fixation antibodies.
After the diagnosis has been reached, the results of serial skin tests, blood cultures, and serologic testing may document the effectiveness of therapy.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
AUSCULTATORY SIGNS OF PULMONARY DISEASE:
Approach to the Diagnosis
(Differential Diagnosis in Primary Care)
Clinically, the grouping together of signs provides the best way of
narrowing the differential diagnosis.
Rales
-
Bilateral crepitant rales, lack of dullness, and normal breath
sounds suggest pulmonary edema or pneumonitis.
-
Focal crepitant rales, reduced alveolar breathing, dullness to
percussion, and increased tactile and vocal fremitus suggest lobar pneumonia
or pulmonary infarction.
-
Bilateral sibilant and sonorous rales without dullness and with
increased bronchial breathing suggest asthma, chronic bronchitis and
emphysema, acute bronchitis or bronchiolitis, and cardiac asthma.
-
Focal crepitant rales and amphoric breathing with dullness
below and hyperresonance above suggest a lung abscess or cavitation.
Hyperresonance
-
Hyperresonance bilaterally with diminished breath sounds
bilaterally and sibilant rales suggests pulmonary emphysema or asthma.
-
Focal hyperresonance with diminished or absent breath sounds
and no rales suggests pneumothorax.
-
Focal hyperresonance with normal or only diminished breath
sounds suggests a large bulla.
Dullness or Flatness
-
Dullness with diminished breath sounds and no rales suggests
atelectasis or pleural effusion from empyema, CHF, or pulmonary infarct. In
atelectasis, there is no hyperresonance or egophony above the dullness.
-
Dullness with diminished breath sounds and crepitant rales
suggests pneumonia or pulmonary infarct. If there is bronchophony as well,
there is probably no associated effusion. If there is no bronchophony but
hyperresonance and egophony above the dullness, then an associated pleural
effusion should be considered.
» READ BOOK EXCERPT ONLINE »
Source: Differential Diagnosis in Primary Care, 2007
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