Causes of Bronchiolitis obliterans with obstructive pulmonary disease
Bronchiolitis obliterans with obstructive pulmonary disease Causes: Book Excerpts
Related information on causes of Bronchiolitis obliterans with obstructive pulmonary disease:
As with all medical conditions,
there may be many causal factors.
Further relevant information on causes of Bronchiolitis obliterans with obstructive pulmonary disease may be found in:
Causes of Bronchiolitis obliterans with obstructive pulmonary disease: Online Medical Books
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Chronic obstructive pulmonary disease:
Causes and incidence
(Professional Guide to Diseases (Eighth Edition))
Predisposing factors include cigarette smoking, recurrent or chronic respiratory infections, air pollution, occupational exposure to chemicals, and allergies. Smoking is by far the most important of these factors — it impairs ciliary action and macrophage function, inflames airways, increases mucus production, destroys alveolar septae, and causes peribronchiolar fibrosis. Early inflammatory changes may reverse if the patient stops smoking before lung destruction is extensive. Familial and hereditary factors (such as deficiency of alpha1-antitrypsin) may also predispose a person to COPD.
The most common chronic lung disease, COPD (also known as chronic obstructive lung disease) affects an estimated 17 million Americans, and its incidence is rising. It affects more males than females, probably because until recently men were more likely to smoke heavily. COPD occurs mostly in people older than age 40.
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Source: Professional Guide to Diseases (Eighth Edition), 2005
Chronic obstructive pulmonary disease:
Causes
(Handbook of Diseases)
Predisposing factors include cigarette smoking, recurrent or chronic respiratory tract infections, air pollution, occupations involving exposure to dusts or noxious gases, and allergies. Familial and hereditary factors (for example, deficiency of alpha1-antitrypsin) may also predispose a person to COPD.
Smoking is by far the most important of these factors; it impairs ciliary action and macrophage function and causes airway inflammation, increased mucus production, destruction of alveolar septae, and peribronchiolar fibrosis. Early inflammatory changes may reverse if the patient stops smoking before lung destruction is extensive.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
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