Causes of Cerebral Aneurysm
List of causes of Cerebral Aneurysm
Following is a list of causes or underlying conditions
(see also Misdiagnosis of underlying causes of Cerebral Aneurysm)
that could possibly cause Cerebral Aneurysm includes:
Longer list of causes of Cerebrovascular symptoms:
see full list of causes for Cerebrovascular symptoms
Cerebral Aneurysm Causes: Book Excerpts
Cerebral Aneurysm as a complication of other conditions:
Other conditions that might have
Cerebral Aneurysm as a complication may,
potentially, be an underlying cause of Cerebral Aneurysm.
Our database lists the following as having
Cerebral Aneurysm as a complication of that condition:
Cerebral Aneurysm as a symptom:
Conditions listing Cerebral Aneurysm
as a symptom may also be potential underlying causes of Cerebral Aneurysm.
Our database lists the following as having
Cerebral Aneurysm as a symptom of that condition:
Related information on causes of Cerebral Aneurysm:
As with all medical conditions,
there may be many causal factors.
Further relevant information on causes of Cerebral Aneurysm may be found in:
Causes of Cerebral Aneurysm: Online Medical Books
16 MEDICAL BOOKS ONLINE!
Review excerpts from medical books online, free, without registration,
for more information about the causes of Cerebral Aneurysm.
Femoral and popliteal aneurysms:
Causes
(Professional Guide to Diseases (Eighth Edition))
Femoral and popliteal aneurysms are usually secondary to atherosclerosis. Rarely, they result from congenital weakness in the arterial wall. They may also result from trauma (blunt or penetrating), bacterial infection, or peripheral vascular reconstructive surgery (which causes “suture line” aneurysms, or false aneurysms, in which a blood clot forms a second lumen).
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Aneurysm, cerebral:
Causes
(Handbook of Diseases)
Cerebral aneurysms may result from a congenital defect, a degenerative process, or a combination of both. For example, hypertension and atherosclerosis may disrupt blood flow and exert pressure against a congenitally weak arterial wall, stretching it like an overblown balloon and making it likely to rupture. Such a rupture is followed by subarachnoid hemorrhage, in which blood spills into the space normally occupied by cerebrospinal fluid (CSF). Sometimes, blood also spills into brain tissue and subsequently forms a clot. This may result in potentially fatal increased intracranial pressure (ICP) and brain tissue damage.
These aneurysms are slightly more common in women than in men, especially those in their late 40s or early to middle 50s, but a cerebral aneurysm may occur at any age, in women and men.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Aneurysms, femoral and popliteal:
Causes
(Handbook of Diseases)
Femoral and popliteal aneurysms are usually secondary to atherosclerosis. Rarely, they result from congenital weakness in the arterial wall. They may also result from trauma (blunt or penetrating), bacterial infection, or peripheral vascular reconstructive surgery (which causes “suture line” aneurysms, whereby a blood clot forms a second lumen, also called false aneurysms).
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
aneurysm,ventricular:
Causes
(Handbook of Diseases)
When MI destroys a large muscular section of the left ventricle, necrosis reduces the ventricular wall to a thin sheath of fibrous tissue. Under intracardiac pressure, this thin layer stretches and forms a separate noncontractile sac (aneurysm).
Abnormal muscle wall movement
Accompanying ventricular aneurysm, abnormal muscle wall movement includes akinesia (lack of movement), dyskinesia (paradoxical movement), asynergia (decreased and inadequate movement), and asynchrony (uncoordinated movement).
During systolic ejection, the abnormal muscle wall movements associated with the aneurysm cause the remaining normally functioning myocardial fibers to increase the force of contraction in order to maintain stroke volume and cardiac output. At the same time, a portion of the stroke volume is lost to passive distention of the noncontractile sac.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
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