Decompression sickness
Decompression sickness: Excerpt from Professional Guide to Diseases (Eighth Edition)
Decompression sickness (the “bends”) is a painful condition that results from a too-rapid change from a high- to low-pressure environment (decompression). Most victims are scuba divers who ascend too quickly from water deeper than 30' (9.1 m) and pilots and passengers of unpressurized aircraft who ascend too quickly to high altitudes.
Causes
Decompression sickness results from an abrupt change in air or water pressure that causes nitrogen to spill out of tissues faster than it can be diffused through respiration. It causes gas bubbles to form in blood and body tissues, which produce excruciating joint and muscle pain, neurologic and respiratory distress, and skin changes.
Signs and symptoms
Symptoms usually appear during or within 30 minutes of rapid decompression, although they may be delayed up to 24 hours. Typically, decompression sickness results in:
❑ “the bends,” deep and usually constant joint and muscle pain so severe that it may be incapacitating
❑ transitory neurologic disturbances, such as difficult urination (from bladder paralysis), hemiplegia, deafness, visual disturbances, dizziness, aphasia, paresthesia and hyperesthesia of the legs, unsteady gait and, possibly, coma
❑ respiratory distress (known as the “chokes”), which includes chest pain, retrosternal burning, and a cough that may become paroxysmal and uncontrollable.
Such symptoms may persist for days and result in dyspnea, cyanosis, fainting and, occasionally, shock. Other symptoms include decreased temperature, pallor, itching, burning, mottled skin, fatigue and, in some patients, tachypnea.
Diagnosis
Confirming diagnosis A history of rapid decompression and a physical examination showing characteristic clinical features confirm the diagnosis.
Treatment
Treatment consists of recompression and oxygen administration, followed by gradual decompression. In recompression, which takes place in a hyperbaric chamber (not available in all hospitals), air pressure is increased to 2.8 absolute atmospheric pressure over 1 to 2 minutes. This rapid rise in pressure reduces the size of the circulating nitrogen bubbles and relieves pain and other clinical effects. During recompression, intermittent oxygen administration, with periodic maximal exhalations, promotes gas bubble diffusion. After symptoms subside and diffusion is complete, a slow decrease of air pressure in the chamber allows for gradual, safe decompression.
Supportive measures include fluid replacement in hypovolemic shock and, sometimes, corticosteroids to reduce the risk of spinal edema. Opioids are contraindicated because they further depress impaired respiration.
Special considerations
❑ To avoid oxygen toxicity during recompression, tell the patient to alternate breathing oxygen for 5 minutes with breathing air for 5 minutes.
❑ During oxygen administration, make sure all electrical equipment is grounded. Prohibit smoking, the use of electric appliances such as razors, and the use of blankets made of wool or other materials that produce static electricity in the patient’s room.
❑ If the patient with bladder paralysis needs catheterization, monitor intake and output.
❑ To prevent decompression sickness, advise divers and pilots to follow the U.S. Navy’s ascent guidelines.
Book Source Details
- Book Title: Professional Guide to Diseases (Eighth Edition)
- Author(s): Springhouse
- Year of Publication: 2005
- Copyright Details: Professional Guide to Diseases (Eighth Edition), Copyright © 2005 Lippincott Williams & Wilkins.
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Copyright notice for book excerpts: Copyright © 2008 Lippincott Williams & Wilkins. All rights reserved.
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