Causes of Diabetic Gastroparesis
List of causes of Diabetic Gastroparesis
Following is a list of causes or underlying conditions
(see also Misdiagnosis of underlying causes of Diabetic Gastroparesis)
that could possibly cause Diabetic Gastroparesis includes:
Longer list of causes of Digestive symptoms:
see full list of causes for Digestive symptoms
Diabetic Gastroparesis Causes: Book Excerpts
Diabetic Gastroparesis as a complication of other conditions:
Other conditions that might have
Diabetic Gastroparesis as a complication may,
potentially, be an underlying cause of Diabetic Gastroparesis.
Our database lists the following as having
Diabetic Gastroparesis as a complication of that condition:
Diabetic Gastroparesis as a symptom:
Conditions listing Diabetic Gastroparesis
as a symptom may also be potential underlying causes of Diabetic Gastroparesis.
Our database lists the following as having
Diabetic Gastroparesis as a symptom of that condition:
Medications or substances causing Diabetic Gastroparesis:
The following drugs, medications, substances or toxins are some of the possible
causes of Diabetic Gastroparesis as a symptom.
This list is incomplete and various other drugs or substances
may cause your symptoms.
Always advise your doctor of any medications or treatments you are using,
including prescription, over-the-counter, supplements, herbal or alternative treatments.
Read more about medication causes of Diabetic Gastroparesis
What causes Diabetic Gastroparesis?
Causes: Diabetic Gastroparesis:
Diabetic sugar levels damage the stomach nerve.
Gastroparesis is the result of damage to the vagus nerve, which
controls the movement of food through the digestive system. Instead of
the food moving through the digestive tract normally, it is retained in
the stomach.
(Source: excerpt from Gastroparesis and Diabetes: NIDDK)
Related information on causes of Diabetic Gastroparesis:
As with all medical conditions,
there may be many causal factors.
Further relevant information on causes of Diabetic Gastroparesis may be found in:
Causes of Diabetic Gastroparesis: Online Medical Books
16 MEDICAL BOOKS ONLINE!
Review excerpts from medical books online, free, without registration,
for more information about the causes of Diabetic Gastroparesis.
Diabetes mellitus:
Causes and incidence
(Professional Guide to Diseases (Eighth Edition))
DM affects an estimated 6% of the population of the United States, about half of whom are undiagnosed. Incidence is greater in females and rises with age. Type 2 accounts for 90% of cases.
In type 1 diabetes, pancreatic beta-cell destruction or a primary defect in beta-cell function results in failure to release insulin and ineffective glucose transport. Type 1 immune-mediated diabetes is caused by cell-mediated destruction of pancreatic beta cells. The rate of beta-cell destruction is usually higher in children than in adults. The idiopathic form of type 1 diabetes has no known cause. Patients with this form have no evidence of autoimmunity and don’t produce insulin.
In type 2 diabetes, beta cells release insulin, but receptors are insulin-resistant and glucose transport is variable and ineffective. Risk factors for type 2 diabetes include:
❑ obesity (even an increased percentage of body fat primarily in the abdominal region); risk decreases with weight and drug therapy
❑ lack of physical activity
❑ history of GDM
❑ hypertension
❑ Black, Hispanic, Pacific Islander, Asian American, Native American origin
❑ strong family history of diabetes
❑ older than age 45
❑ high-density lipoprotein cholesterol of less than 35 or triglyceride of greater than 250
❑ Seriously impaired glucose tolerance (IGT) test.
ELDER TIP As the body ages, the cells become more resistant to insulin, thus reducing the older adult’s ability to metabolize glucose. In addition, the release of insulin from the pancreatic beta cells is reduced and delayed. These combined processes result in hyperglycemia. In the older patient, sudden concentrations of glucose cause increased and more prolonged hyperglycemia.
The “other specific types” of DM result from various conditions (such as a genetic defect of the beta cells or endocrinopathies) or from use of or exposure to certain drugs or chemicals. GDM is considered present whenever a patient has any degree of abnormal glucose during pregnancy. This form may result from weight gain and increased levels of estrogen and placental hormones, which antagonize insulin.
Insulin transports glucose into the cell for use as energy and storage as glycogen. It also stimulates protein synthesis and free fatty acid storage in the fat deposits. Insulin deficiency compromises the body tissues’access to essential nutrients for fuel and storage.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Gastrointestinal Bleeding:
Differential Overview
(Field Guide to Bedside Diagnosis)
Upper GI
❑ Peptic ulcer disease
❑ Gastritis
❑ Mallory-Weiss tear
❑ Esophageal varices
❑ Esophagitis
❑ Epistaxis
❑ Esophageal cancer
❑ Gastric cancer
Lower GI
❑ Infectious diarrhea
❑ Diverticular bleeding
❑ Hemorrhoids
❑ Anal fissure
❑ Inflammatory bowel disease
❑ Angiodysplasia
❑ Colon cancer
❑ Mesenteric ischemia
❑ Aortoenteric fistula
» READ BOOK EXCERPT ONLINE »
Source: Field Guide to Bedside Diagnosis, 2007
Diabetes mellitus:
Causes
(Handbook of Diseases)
The effects of diabetes mellitus result from insulin deficiency. Insulin transports glucose into the cell for use as energy and storage as glycogen. It also stimulates protein synthesis and free fatty acid storage. Insulin deficiency or resistance compromises the body tissues’access to essential nutrients for fuel and storage.
Type 1A results from autoimmune beta-cell destruction, resulting in insulin deficiency. Type 1B leaves these immunologic markers but results in insulin deficiency and kerosis.
Other risk factors include the following:
❑ Obesity contributes to the resistance to endogenous insulin.
❑ Physiologic or emotional stress can cause prolonged elevation of stress hormone levels (cortisol, epinephrine, glucagon, and growth hormone). This raises blood glucose levels, which, in turn, places increased demands on the pancreas.
❑ Pregnancy causes weight gain and increases levels of estrogen and placental hormones, which antagonize insulin.
❑ Some medications can antagonize the effects of insulin, including thiazide diuretics, adrenal corticosteroids, and hormonal contraceptives.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Intestinal obstruction:
Causes
(Handbook of Diseases)
Adhesions and strangulated hernias usually cause small-bowel obstructions; carcinomas usually cause large-bowel obstructions. A mechanical intestinal obstruction results from a foreign body (fruit pits, gallstones, or worms) or compression of the bowel wall due to stenosis, intussusception, volvulus of the sigmoid or cecum, tumors, or atresia.
A nonmechanical obstruction results from a physiologic disturbance, such as paralytic ileus (see Paralytic ileus), electrolyte imbalance, toxicity (uremia or generalized infection), a neurogenic abnormality (spinal cord lesions), or thrombosis or embolism of mesenteric vessels.
The three forms of intestinal obstruction are:
❑ simple — blockage prevents intestinal contents from passing with no other complications
❑ strangulated — blood supply to part or all of the obstructed section is cut off in addition to blockage of the lumen
❑ close looped — both ends of a bowel section are occluded, isolating it from the rest of the intestine.
In all three forms, the physiologic effects are similar. When intestinal obstruction occurs, fluid, air, and gas collect near the site. Peristalsis increases temporarily as the bowel tries to force its contents through the obstruction, injuring intestinal mucosa and causing distention at and above the site of the obstruction. This distention blocks the flow of venous blood and halts normal absorptive processes. As a result, the bowel begins to secrete water, sodium, and potassium into the fluid pooled in the lumen. This results in distention and enormous amounts of fluid in the gut.
An obstruction in the upper intestine results in metabolic alkalosis from dehydration and loss of gastric hydrochloric acid; a lower obstruction causes slower dehydration and loss of intestinal alkaline fluids, resulting in metabolic acidosis. Ultimately, an intestinal obstruction may lead to ischemia, necrosis, and death.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Gastrointestinal Bleeding:
Principal Causes of Gastrointestinal Bleeding
(The Diagnostic Approach to Symptoms and Signs in Pediatrics)
- Uppergastrointestinal bleeding
- Nose
- Mouth and pharynx
- Esophagus
- Esophagitis
- Gastroesophageal reflux
- Caustic ingestion
- Infection
- Foreign body
- Varices
- Duplication
- Gastroesophageal junction
- Mallory-Weisstear
- Stomach
- Gastritis
- Ulcer
- Duplication
- Vascular malformation
- Neoplasm
- Duodenum
- Ulcer
- Foreign body
- Varices
- Duplication
- Vascular malformation
- Hemobilia
- Other
- Swallowed blood
- Coagulopathy
- Hemorrhagic disease of the newborn(vitamin K deficiency)
- Disseminated intravascular coagulation
- Drugs
- Lower gastrointestinal bleeding
- Intestine
- Cow milk/soyprotein sensitivity
- Necrotizing enterocolitis
- Infectious colitis
- Henoch-Schönlein purpura
- Intussusception
- Congenital aganglionic megacolon (Hirschsprungdisease)
- Meckel diverticulum
- Volvulus with malrotation
- Inflammatory bowel disease
- Intestinal polyps
- Solitaryjuvenile polyps/juvenile intestinal polyposis
- Adenomatous polyposis of the colon
- Peutz-Jeghers syndrome
- Benign lymphoid hyperplasia
- Duplication
- Vascular malformation
- Neoplasm
- Rectum and anus
- Anal fissure
- Trauma
- Sexual abuse
- Hemorrhoids
- Other
- Swallowed blood
- Coagulopathy
- Drugs
- Factitious bleeding
» READ BOOK EXCERPT ONLINE »
Source: The Diagnostic Approach to Symptoms and Signs in Pediatrics, 2006
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