16 MEDICAL BOOKS ONLINE! Review excerpts from medical books online, free, without registration, for more information about the prevalence and/or incidence of Erythroderma.
Wilson’s disease is inherited as an autosomal recessive trait only when both parents carry the abnormal gene. There is a 25% chance that carrier parents will transmit Wilson’s disease (and a 50% chance that they will transmit the carrier state) to each of their offspring. The disease usually occurs among eastern Europeans, Sicilians, and other southern Italians.
Wilson’s disease causes excessive intestinal absorption of copper and subsequent decreased excretion of copper in the stool. Copper accumulates first in the liver. As liver cells die, they release copper into the bloodstream, which carries it to other tissues. For example, in the kidneys, excretion of excessive amounts of unbound copper in urine (hypercupriuria) results from ceruloplasmin deficiency, a serum enzyme normally bound to copper. The deposit of copper in the tissue decreases serum copper (hypocupremia).
Source: Professional Guide to Diseases (Eighth Edition), 2005
Children usually present with hepatic manifestations; adolescents and young adults may present with neurologic symptoms.
Source: The 5-Minute Pediatric Consult, 2008
The term 'prevalence' of Erythroderma usually refers to the estimated population of people who are managing Erythroderma at any given time. The term 'incidence' of Erythroderma refers to the annual diagnosis rate, or the number of new cases of Erythroderma diagnosed each year. Hence, these two statistics types can differ: a short-lived disease like flu can have high annual incidence but low prevalence, but a life-long disease like diabetes has a low annual incidence but high prevalence. For more information see about prevalence and incidence statistics.
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