Following is a list of causes or underlying conditions (see also Misdiagnosis of underlying causes of Herpes zoster oticus) that could possibly cause Herpes zoster oticus includes:
The follow list shows some of the possible medical causes of Herpes zoster oticus that are listed by the Diseases Database:
Source: Diseases DatabaseOther conditions that might have Herpes zoster oticus as a complication may, potentially, be an underlying cause of Herpes zoster oticus. Our database lists the following as having Herpes zoster oticus as a complication of that condition:
As with all medical conditions, there may be many causal factors. Further relevant information on causes of Herpes zoster oticus may be found in:
16 MEDICAL BOOKS ONLINE! Review excerpts from medical books online, free, without registration, for more information about the causes of Herpes zoster oticus.
Genital herpes is usually caused by infection with herpes simplex virus Type 2, but some studies report increasing incidence of infection with herpes simplex virus Type 1. This disease is typically transmitted through sexual intercourse, orogenital sexual activity, kissing, and hand-to-body contact. Pregnant women may transmit the infection to neonates during vaginal delivery if an active infection is present. Such transmitted infection may be localized (for instance, in the eyes) or disseminated and may be associated with central nervous system involvement.
An estimated 86 million people worldwide are thought to have genital herpes.
Source: Professional Guide to Diseases (Eighth Edition), 2005
Herpes zoster results from reactivation of varicella virus that has lain dormant in the cerebral ganglia (extramedullary ganglia of the cranial nerves) or the ganglia of posterior nerve roots since a previous episode of chickenpox. Exactly how or why this reactivation occurs isn’t clear. Some believe that the virus multiplies as it's reactivated and that antibodies remaining from the initial infection neutralize it. However, if effective antibodies aren't present, the virus continues to multiply in the ganglia, destroy the host neuron, and spread down the sensory nerves to the skin.
Herpes zoster occurs primarily in adults, especially those older than age 50. It seldom recurs. It's also seen in patients with human immunodeficiency virus and other immunodeficiency disorders.
Source: Professional Guide to Diseases (Eighth Edition), 2005
Herpes zoster results from reactivation of varicella virus that has lain dormant in the cerebral ganglia (extramedullary ganglia of the cranial nerves) or the ganglia of posterior nerve roots since a previous episode of chickenpox.
Exactly how or why this reactivation occurs isn’t clear. Some believe that the virus multiplies as it’s reactivated and that it’s neutralized by antibodies remaining from the initial infection. However, if effective antibodies aren’t present, the virus continues to multiply in the ganglia, destroy the host neuron, and spread down the sensory nerves to the skin.
Source: Handbook of Diseases, 2003
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