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Symptoms of Iron deficiency anemia
Symptoms of Iron deficiency anemia
The list of signs and symptoms mentioned in various sources for Iron deficiency anemia includes the 36 symptoms listed below:
- See also symptoms of anemia
- Weakness
- Fatigue
- Tiredness
- Mild early symptoms
- Pallor
- Anemia
- Concave nails
- Brittle nails
- Husky voice
- Tongue inflammation
- Difficulty swallowing
- Low levels of iron in blood
- Low levels of haemoglobin
- Reduced red blood cell count
- Pica
- Persistent urge to consume ice
- Persistent urge to consume clay
- Asymptomatic in mild cases
- Loss of appetite
- Pale lips
- Sore mouth
- Pale eyelids
- Lightheadedness
- Headache
- Early symptoms are mild
- Shortness of breath after exercise
- Concentration problems
- Sleeping problems
- Rapid heartbeat
- Irregular heartbeat
- Chest pain
- Dizziness
- Impaired cognitive ability
- Cold skin
- Fingernail abnormalities
- more information...»
Research symptoms & diagnosis of Iron deficiency anemia:
- Overview -- Iron deficiency anemia
- Diagnostic Tests for Iron deficiency anemia
- Home Diagnostic Testing
- Complications -- Iron deficiency anemia
- Doctors & Specialists
- Misdiagnosis and Alternative Diagnoses
- Hidden Causes of Iron deficiency anemia
- Other Causes -- causes of these or similar symptoms
Iron deficiency anemia: Complications
Review medical complications possibly associated with Iron deficiency anemia:
Diagnostic Testing
Diagnostic testing of medical conditions related to Iron deficiency anemia:
- Iron blood tests
- Fingerstick iron test
- more tests...»
Research More About Iron deficiency anemia
Do I have Iron deficiency anemia?
- Iron deficiency anemia: Introduction
- Iron deficiency anemia: Diagnostic Testing to confirm diagnosis
- Home Diagnostic Testing
- Alternative diagnoses and misdiagnosis for Iron deficiency anemia
- Failure to Diagnose Iron deficiency anemia
- Hidden Causes of Iron deficiency anemia
- How serious is it?
- Treatments for Iron deficiency anemia
- More about Iron deficiency anemia
Iron deficiency anemia: Medical Mistakes
- Women's Health Mistakes:
- more mistakes...»
Iron deficiency anemia: Undiagnosed Conditions
Diseases that may be commonly undiagnosed in related medical areas:
- Women's Reproductive Health: diseases that are commonly undiagnosed:
- more undiagnosed conditions...»
Home Diagnostic Testing
Home medical tests related to Iron deficiency anemia:
- Fatigue: Related Home Tests:
- Thyroid: Home Testing:
- Home Anemia Diagnostic Tests
- Menopause: Related Home Testing:
- Vaginal Health: Home Testing:
- Adrenal Gland Health: Home Testing:
- Breast Cancer: Related Home Tests:
- more home tests...»
Wrongly Diagnosed with Iron deficiency anemia?
The list of other diseases or medical conditions that may be on the differential diagnosis list of alternative diagnoses for Iron deficiency anemia includes:
- Anemia - other types than iron deficiency anemia.
- Sickle Cell Anemia
- Autoimmune Hemolytic Anemia
- Pernicious anemia
- Thalassemia
- more diagnoses...»
See the full list of 12 alternative diagnoses for Iron deficiency anemia
Iron deficiency anemia: Research Doctors & Specialists
- Pregnancy & Fertility Health Specialists:
- Womens Health Specialists:
- Blood Health Specialists (Hematology):
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More about symptoms of Iron deficiency anemia:
More information about symptoms of Iron deficiency anemia and related conditions:
- Other diseases with similar symptoms and common misdiagnoses
- Tests to determine if these are the symptoms of Iron deficiency anemia
- Symptoms that may be caused by complications of Iron deficiency anemia
- Underlying causes of Iron deficiency anemia
- Risk factors for Iron deficiency anemia
Other Possible Causes of these Symptoms
Click on any of the symptoms below to see a full list of other causes including diseases, medical conditions, toxins, drug interactions, or drug side effect causes of that symptom.
- Anemia - see all causes of Anemia
- Asymptomatic in mild cases - see all causes of No symptoms
- Brittle nails - see all causes of Brittle nails
- Chest pain - see all causes of Chest pain
- Cold skin - see all causes of Cold skin
- Concave nails - see all causes of Nail symptoms
- Concentration problems - see all causes of Concentration difficulty
- Difficulty swallowing - see all causes of Swallowing difficulty
- Dizziness - see all causes of Dizziness
- Fatigue - see all causes of Fatigue
- Headache - see all causes of Headache
- Husky voice - see all causes of Hoarse
- Irregular heartbeat - see all causes of Irregular heartbeat
- Lightheadedness - see all causes of Dizziness
- Loss of appetite - see all causes of Poor appetite
- Mild early symptoms - see all causes of Vague symptoms
- Pale lips - see all causes of Lip symptoms
- Pallor - see all causes of Paleness
- Pica - see all causes of Pica
- Rapid heartbeat - see all causes of Rapid heartbeat
- Shortness of breath after exercise - see all causes of Shortness of breath
- Sleeping problems - see all causes of Sleeping problems
- Sore mouth - see all causes of Sore mouth
- Tiredness - see all causes of Tiredness
- Tongue inflammation - see all causes of Sore tongue
- Weakness - see all causes of Weakness
Medical Books Online about Iron deficiency anemia
Medical Books Excerpts Excerpts of published medical book chapters related to Iron deficiency anemia are available from published medical books for more detailed information about Iron deficiency anemia.
Copyright notice for book excerpts: Copyright © 2008 Lippincott Williams & Wilkins. All rights reserved.
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Symptoms of Iron deficiency anemia: Online Medical Books
16 MEDICAL BOOKS ONLINE! Review excerpts from medical books online, free, without registration, for more information about the symptoms of Iron deficiency anemia.
Folic acid deficiency anemia:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
Folic acid deficiency anemia gradually produces clinical features characteristic of other megaloblastic anemias, without the neurologic manifestations: progressive fatigue, shortness of breath, palpitations, weakness, glossitis, nausea, anorexia, headache, fainting, irritability, forgetfulness, pallor, and slight jaundice. Folic acid deficiency anemia doesn’t cause neurologic impairment unless it’s associated with vitamin B12 deficiency, as in pernicious anemia.
Source: Professional Guide to Diseases (Eighth Edition), 2005
Pernicious anemia:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
Characteristically, pernicious anemia has an insidious onset but eventually causes an unmistakable triad of symptoms: weakness, sore tongue, and numbness and tingling in the extremities. The lips, gums, and tongue appear markedly bloodless. Hemolysis-induced hyperbilirubinemia may cause faintly jaundiced sclera and pale to bright yellow skin. In addition, the patient may become highly susceptible to infection, especially of the genitourinary tract.
Other systemic symptoms of pernicious anemia include the following:
❑ GI: Gastric mucosal atrophy and decreased hydrochloric acid production disturb digestion and lead to nausea, vomiting, anorexia, weight loss, flatulence, diarrhea, and constipation. Gingival bleeding and tongue inflammation may hinder eating and intensify anorexia.
❑ Central nervous system (CNS): Demyelination caused by vitamin B12 deficiency initially affects the peripheral nerves but gradually extends to the spinal cord. Consequently, the neurologic effects of pernicious anemia may include neuritis; weakness in extremities; peripheral numbness and paresthesia; disturbed position sense; lack of coordination; ataxia; impaired fine finger movement; positive Babinski’s and Romberg’s signs; light-headedness; altered vision (diplopia and blurred vision), taste, and hearing (tinnitus); optic muscle atrophy; loss of bowel and bladder control; and, in males, impotence. Its effects on the nervous system may also produce irritability, poor memory, headache, depression, and delirium. Although some of these symptoms are temporary, irreversible CNS changes may have occurred before treatment.
❑ Cardiovascular: Increasingly fragile cell membranes induce widespread destruction of RBCs, resulting in low Hb levels. The impaired oxygen-carrying capacity of the blood secondary to lowered Hb leads to weakness, fatigue, and light-headedness. Compensatory increased cardiac output results in palpitations, wide pulse pressure, dyspnea, orthopnea, tachycardia, premature beats and, eventually, heart failure.
❑ Musculoskeletal: Scissors gait can also occur as a late sign of untreated anemia.
Source: Professional Guide to Diseases (Eighth Edition), 2005
Sickle cell anemia:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
Characteristically, sickle cell anemia produces tachycardia, cardiomegaly, systolic and diastolic murmurs, pulmonary infarctions (which may result in cor pulmonale), chronic fatigue, unexplained dyspnea or dyspnea on exertion, hepatomegaly, jaundice, pallor, joint swelling, aching bones, chest pains, ischemic leg ulcers (especially around the ankles), and increased susceptibility to infection. Such symptoms usually don't develop until after age 6 months because large amounts of fetal Hb protect infants for the first few months after birth. Low socioeconomic status and related problems, such as poor nutrition and education, may delay diagnosis and supportive treatment.
Infection, stress, dehydration, and conditions that provoke hypoxia — strenuous exercise, high altitude, unpressurized aircraft, cold, and vasoconstrictive drugs —may all provoke periodic crises. A painful crisis (vasoocclusive crisis, infarctive crisis), the most common crisis and the hallmark of the disease, usually appears periodically after age 5. It results from blood vessel obstruction by rigid, tangled sickle cells, which causes tissue anoxia and possible necrosis. This type of crisis is characterized by severe abdominal, thoracic, muscular, or bone pain and possibly worsening jaundice, dark urine, and a low-grade fever.
Autosplenectomy, in which splenic damage and scarring is so extensive that the spleen shrinks and becomes impalpable, occurs in patients with long-term disease. This can lead to increased susceptibility to Streptococcus pneumoniae sepsis, which can be fatal without prompt treatment. Infection may develop after the crisis subsides (in 4 days to several weeks), so watch for lethargy, sleepiness, fever, or apathy.
An aplastic crisis (megaloblastic crisis) results from bone marrow depression and is associated with infection, usually viral. It's characterized by pallor, lethargy, sleepiness, dyspnea, possible coma, markedly decreased bone marrow activity, and RBC hemolysis.
In infants between ages 8 months and 2 years, an acute sequestration crisis may cause sudden massive entrapment of RBCs in the spleen and liver. This rare crisis causes lethargy and pallor and, if untreated, commonly progresses to hypovolemic shock and death.
A hemolytic crisis is quite rare and usually occurs in patients who also have glucose-6-phosphate dehydrogenase deficiency. It probably results from complications of sickle cell anemia, such as infection, rather than from the disorder itself. Hemolytic crisis causes liver congestion and hepatomegaly as a result of degenerative changes. It worsens chronic jaundice, although increased jaundice doesn't always point to a hemolytic crisis.
Suspect any of these crises in a sickle cell anemia patient with pale lips, tongue, palms, or nail beds; lethargy; listlessness; sleepiness with difficulty awakening; irritability; severe pain; a fever over 104° F (40° C); or a fever of 100° F (37.8° C) that persists for 2 days.
Sickle cell anemia also causes long-term complications. Typically, the child is small for his age and has delayed puberty. (However, fertility isn't impaired.) If he reaches adulthood, his body build tends to be spiderlike — narrow shoulders and hips, long extremities, curved spine, barrel chest, and elongated skull. An adult usually has complications from organ infarction, such as retinopathy and nephropathy. Premature death commonly results from infection or from repeated occlusion of small blood vessels and consequent infarction or necrosis of major organs (such as cerebral blood vessel occlusion causing stroke).
Source: Professional Guide to Diseases (Eighth Edition), 2005
Sideroblastic anemias:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
Sideroblastic anemias usually produce nonspecific clinical effects, which may exist for several years before being identified. Such effects include anorexia, fatigue, weakness, dizziness, pale skin and mucous membranes and, occasionally, enlarged lymph nodes. Heart and liver failure may develop due to excessive iron accumulation in these organs, causing dyspnea, exertional angina, slight jaundice, and hepatosplenomegaly. Hereditary sideroblastic anemia is associated with increased GI absorption of iron, causing signs of hemosiderosis. Additional symptoms in secondary sideroblastic anemia depend upon the underlying cause.
Source: Professional Guide to Diseases (Eighth Edition), 2005
Iron deficiency anemia:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
Because of the gradual progression of iron deficiency anemia, many patients are initially asymptomatic except for symptoms of any underlying condition. They tend not to seek medical treatment until anemia is severe. At advanced stages, decreased Hb levels and the consequent decrease in the blood’s oxygen-carrying capacity cause the patient to develop dyspnea on exertion, fatigue, listlessness, pallor, inability to concentrate, irritability, headache, and a susceptibility to infection. Decreased oxygen perfusion causes the heart to compensate with increased cardiac output and tachycardia.
In chronic iron deficiency anemia, nails become spoon-shaped and brittle, the mouth’s corners crack, the tongue turns smooth, and the patient complains of dysphagia or may develop pica. Associated neuromuscular effects include vasomotor disturbances, numbness and tingling of the extremities, and neuralgic pain.
Source: Professional Guide to Diseases (Eighth Edition), 2005
Aplastic anemias:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
Clinical features of aplastic anemias vary with the severity of pancytopenia but develop insidiously in many cases. Anemic symptoms include progressive weakness and fatigue, shortness of breath, headache, pallor and, ultimately, tachycardia and heart failure. Thrombocytopenia leads to ecchymosis, petechiae, and hemorrhage, especially from the mucous membranes (nose, gums, rectum, and vagina) or into the retina or central nervous system. Neutropenia may lead to infection (fever, oral and rectal ulcers, and sore throat) but without characteristic inflammation.
Source: Professional Guide to Diseases (Eighth Edition), 2005
Chronic fatigue syndrome:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
CFS has specific symptoms and signs, based on the exclusion of other possible causes. Its characteristic symptom is prolonged, often overwhelming fatigue that’s commonly associated with a varying complex of other symptoms that are similar to those of many infections, including myalgia and cephalgia. It may develop within a few hours and can last for 6 months or more. Fatigue isn’t relieved by rest and is severe enough to restrict activities of daily living by at least 50%. To aid in disease identification, the Centers for Disease Control and Prevention (CDC) uses a “working case definition” to group symptoms and severity.
Source: Professional Guide to Diseases (Eighth Edition), 2005
Folic acid deficiency anemia:
Signs and symptoms
(Handbook of Diseases)
Folic acid deficiency anemia gradually produces clinical features that are characteristic of other megaloblastic anemias without the neurologic manifestations. These include progressive fatigue, dyspnea, palpitations, weakness, glossitis, nausea, anorexia, headache, fainting, irritability, forgetfulness, pallor, and slight jaundice.
Folic acid deficiency anemia doesn’t cause neurologic impairment unless it’s associated with vitamin B12 deficiency, as in pernicious anemia.
Source: Handbook of Diseases, 2003
Pernicious anemia:
Signs and symptoms
(Handbook of Diseases)
Characteristically, pernicious anemia has an insidious onset but eventually causes an unmistakable triad of symptoms: weakness, sore tongue, and numbness and tingling in the extremities. The lips, gums, and tongue appear markedly bloodless. Hemolysis-induced hyperbilirubinemia may cause faintly jaundiced sclera and pale to bright yellow skin. The patient may also become highly susceptible to infection, especially of the genitourinary tract.
GI signs and symptoms
Gastric mucosal atrophy and decreased hydrochloric acid production disturb digestion and lead to nausea, vomiting, anorexia, weight loss, flatulence, diarrhea, and constipation. Gingival bleeding and tongue inflammation may hinder eating and intensify anorexia.
Central nervous system signs and symptoms
Nerve demyelination caused by vitamin B12 deficiency initially affects the peripheral nerves but gradually extends to the spinal cord. Consequently, the neurologic effects of pernicious anemia include neuritis, weakness in the extremities, peripheral numbness and paresthesia, disturbed position sense, lack of coordination, ataxia, impaired fine finger movement, positive Babinski’s and Romberg’s signs, light-headedness, optic muscle atrophy, loss of bowel and bladder control, impotence (in males), and altered vision (diplopia, blurred vision), taste, and hearing (tinnitus).
The effects of pernicious anemia on the nervous system may also produce irritability, poor memory, headache, depression, and delirium. Although some of these symptoms are temporary, irreversible central nervous system changes may have occurred before treatment is initiated.
Cardiovascular signs and symptoms
Increasingly fragile cell membranes induce widespread destruction of RBCs, resulting in low hemoglobin (Hb) levels. The impaired oxygen-carrying capacity of the blood secondary to lowered Hb leads to weakness, fatigue, and light-headedness. Compensatory increased cardiac output results in palpitations, wide pulse pressure, dyspnea, orthopnea, tachycardia, premature beats and, eventually, heart failure.
Source: Handbook of Diseases, 2003
Sickle cell anemia:
Signs and symptoms
(Handbook of Diseases)
Characteristically, sickle cell anemia produces tachycardia, cardiomegaly, systolic and diastolic murmurs, pulmonary infarctions (which may result in cor pulmonale), chronic fatigue, unexplained dyspnea or dyspnea on exertion, hepatomegaly, jaundice, pallor, joint swelling, aching bones, chest pains, ischemic leg ulcers (especially around the ankles), and increased susceptibility to infection.
Such symptoms usually don’t develop until after age 6 months, because large amounts of fetal Hb protect infants for the first few months after birth. Low socioeconomic status and related problems, such as poor nutrition and education, may delay diagnosis and supportive treatment.
Infection, stress, dehydration, and conditions that provoke hypoxia — strenuous exercise, high altitude, unpressurized aircraft, cold, and vasoconstrictive drugs — may all provoke periodic crisis. Four types of crises can occur: painful, aplastic, acute sequestration, or hemolytic.
Painful crisis
Also called a vaso-occlusive crisis or infarctive crisis, painful crisis is the most common crisis and the hallmark of this disease. It usually appears periodically after age 5.
A painful crisis results from blood vessel obstruction by rigid, tangled sickle cells, which causes tissue anoxia and possible necrosis. It’s characterized by severe abdominal, thoracic, muscular, or bone pain and, possibly, increased jaundice, dark urine, or a low-grade fever.
UNDER STUDY: In pediatric patients with vaso-occlusive crisis, 1-arginine, a precursor to nitric oxide, has been found to be diminished. In adults, it has been significantly diminished in 50% of patients administered to the emergency department with vaso-occlusive crisis.
Autosplenectomy, in which splenic damage and scarring is so extensive that the spleen shrinks and becomes impalpable, occurs in patients with long-term disease. This can lead to increased susceptibility to Streptococcus pneumoniae sepsis, which can be fatal without prompt treatment.
After the crisis subsides (in 4 days to several weeks), infection may develop, causing such signs as lethargy, sleepiness, fever, and apathy.
Aplastic crisis
Also called megaloblastic crisis, aplastic crisis results from bone marrow depression and is associated with infection, usually viral. It’s characterized by pallor, lethargy, sleepiness, dyspnea, possible coma, markedly decreased bone marrow activity, and RBC hemolysis.
Acute sequestration crisis
In infants between 8 months and 2 years old, an acute sequestration crisis may cause sudden massive entrapment of RBCs in the spleen and liver. This rare crisis causes lethargy and pallor; if untreated, it commonly progresses to hypovolemic shock and death.
Hemolytic crisis
Quite rare, hemolytic crisis usually occurs in patients who have glucose-6-phosphate dehydrogenase deficiency with sickle cell anemia. It probably results from complications of sickle cell anemia, such as infection, rather than from the disorder itself.
Hemolytic crisis causes liver congestion and hepatomegaly as a result of degenerative changes. It worsens chronic jaundice, although increased jaundice doesn’t always point to a hemolytic crisis.
Indicators of crisis
Suspect any of these crises in a sickle cell anemia patient with pale lips, tongue, palms, or nail beds; lethargy; listlessness; sleepiness, with difficulty awakening; irritability; severe pain; temperature over 104° F (40° C); or a fever of 100° F (37.8° C) that persists for 2 days.
Long-term complications
Sickle cell anemia also causes long-term complications. Typically, such a child is small for his age, and puberty is delayed. (However, fertility isn’t impaired). If he reaches adulthood, his body build tends to be spiderlike — narrow shoulders and hips, long extremities, curved spine, barrel chest, and elongated skull.
An adult usually has complications with organ infarction, such as retinopathy and nephropathy. Premature death commonly results from infection or repeated occlusion of small blood vessels and consequent infarction or necrosis of major organs. For example, cerebral blood vessel occlusion causes cerebrovascular accident.
Source: Handbook of Diseases, 2003
Sideroblastic anemias:
Signs and symptoms
(Handbook of Diseases)
Sideroblastic anemias usually produce nonspecific clinical effects, which may exist for several years before being identified. Such effects include anorexia, fatigue, weakness, dizziness, pale skin and mucous membranes and, occasionally, enlarged lymph nodes.
Heart and liver failure may develop from excessive iron accumulation in these organs, causing dyspnea, exertional angina, slight jaundice, and hepatosplenomegaly. Hereditary sideroblastic anemia is associated with increased GI absorption of iron, causing signs of hemosiderosis. Additional symptoms in secondary sideroblastic anemia depend on the underlying cause.
Source: Handbook of Diseases, 2003
Iron deficiency anemia:
Signs and symptoms
(Handbook of Diseases)
Because of the gradual progression of iron deficiency anemia, many patients are initially asymptomatic. They tend not to seek medical treatment until anemia is severe.
At advanced stages, a decreased Hb level and the consequent decrease in the blood’s oxygen-carrying capacity cause the patient to develop exertional dyspnea, fatigue, listlessness, pallor, inability to concentrate, irritability, headache, and a susceptibility to infection. Decreased oxygen perfusion causes the heart to compensate with increased cardiac output and tachycardia.
With chronic iron deficiency anemia, nails become spoon shaped and brittle, the corners of the mouth crack, the tongue turns smooth, and the patient complains of dysphagia or may develop pica. Associated neuromuscular effects include vasomotor disturbances, numbness and tingling of the extremities, and neuralgic pain.
Source: Handbook of Diseases, 2003
Aplastic and hypoplastic anemias:
Signs and symptoms
(Handbook of Diseases)
Signs and symptoms of aplastic anemias vary with the severity of pancytopenia but usually develop insidiously. These include progressive weakness and fatigue, shortness of breath, headache, pallor and, ultimately, tachycardia and heart failure. Thrombocytopenia leads to ecchymosis, petechiae, and hemorrhage, especially from the mucous membranes (nose, gums, rectum, and vagina) or into the retina or central nervous system. Neutropenia may lead to infection (with fever, oral and rectal ulcers, and sore throat) but without characteristic inflammation.
Source: Handbook of Diseases, 2003
Chronic fatigue and immune dysfunction syndrome:
Signs and symptoms
(Handbook of Diseases)
The characteristic symptom of CFIDS is prolonged, often overwhelming fatigue that’s commonly associated with a varying complex of other symptoms. To aid identification of the disease, the Centers for Disease Control and Prevention (CDC) uses a “working case definition” to group symptoms and severity. (See CDC criteria for diagnosing CFIDS.)
Source: Handbook of Diseases, 2003
Iron Deficiency Anemia:
Iron Deficiency Anemia - signs & symptoms
(The 5-Minute Pediatric Consult)
- Iron deficiency anemia often develops slowly, and no symptoms may be present.
- When present, signs and symptoms include:
- Irritability and behavioral disturbances
- Fatigue, exercise intolerance
- Pallor
- Headache
- Pica
Source: The 5-Minute Pediatric Consult, 2008
Medical articles and books on symptoms:
These general reference articles may be of interest in relation to medical signs and symptoms of disease in general:
- Diagnostic Testing for a Diagnosis of Iron deficiency anemia
- Research Alternative Diagnoses for Iron deficiency anemia
- How serious is Iron deficiency anemia?
- More about Iron deficiency anemia
- Online Diagnosis
- Self Diagnosis Pitfalls
- Pitfalls of Online Diagnosis
- Symptoms of the Silent Killer Diseases
- Lesser known silent killer diseases
- Books on signs and symptoms
Full list of premium articles on symptoms and diagnosis
About signs and symptoms of Iron deficiency anemia:
The symptom information on this page attempts to provide a list of some possible signs and symptoms of Iron deficiency anemia. This signs and symptoms information for Iron deficiency anemia has been gathered from various sources, may not be fully accurate, and may not be the full list of Iron deficiency anemia signs or Iron deficiency anemia symptoms. Furthermore, signs and symptoms of Iron deficiency anemia may vary on an individual basis for each patient. Only your doctor can provide adequate diagnosis of any signs or symptoms and whether they are indeed Iron deficiency anemia symptoms.
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