Symptoms of Mononucleosis
Symptoms of Mononucleosis
The list of signs and symptoms mentioned in various sources
for Mononucleosis includes the 16
symptoms listed below:
Research symptoms & diagnosis of Mononucleosis:
Mononucleosis: Complications
Review medical complications possibly associated with Mononucleosis:
Mononucleosis Symptoms: Book Excerpts
Diagnostic Testing
Diagnostic testing of medical conditions related to Mononucleosis:
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Do I have Mononucleosis?
Mononucleosis: Medical Mistakes
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Home medical tests related to Mononucleosis:
Wrongly Diagnosed with Mononucleosis?
The list of other diseases or medical conditions
that may be on the differential diagnosis list of alternative diagnoses
for Mononucleosis includes:
See the full list of 39
alternative diagnoses for Mononucleosis
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More about symptoms of Mononucleosis:
More information about symptoms of Mononucleosis and related conditions:
Other Possible Causes of these Symptoms
Click on any of the symptoms below to see a full list
of other causes including diseases, medical conditions, toxins, drug interactions,
or drug side effect causes of that symptom.
Medical Books Online about Mononucleosis
Medical Books Excerpts
Excerpts of published medical book chapters related to Mononucleosis
are available from published medical books
for more detailed information about Mononucleosis.
Medical Books Excerpts
- Fever
- "In a Page: Signs and Symptoms" (2004)
- [ read ]
- FEVER
- "Differential Diagnosis in Primary Care" (2007)
- [ read ]
- Fever
- "Handbook of Signs & Symptoms (Third Edition)" (2006)
- [ read ]
- Lassa fever
- "Professional Guide to Diseases (Eighth Edition)" (2005)
- [ read ]
- Fever
- "The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter" (2000)
- [ read ]
- Fever
- "Alarming Signs and Symptoms: Lippincott Manual of Nursing Practice Series" (2007)
- [ read ]
- Fever
- "Signs & Symptoms: A 2-in-1 Reference for Nurses" (2007)
- [ read ]
- Fever
- "The Diagnostic Approach to Symptoms and Signs in Pediatrics" (2006)
- [ read ]
- FEVER
- "Differential Diagnosis in Primary Care" (2007)
- [ read ]
- Fever
- "Pediatric Complaints and Diagnostic Dilemmas" (2003)
- [ read ]
Copyright notice for book excerpts: Copyright © 2008 Lippincott Williams & Wilkins. All rights reserved.
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Symptoms of Mononucleosis: Online Medical Books
16 MEDICAL BOOKS ONLINE!
Review excerpts from medical books online, free, without registration,
for more information about the symptoms of Mononucleosis.
Infectious mononucleosis:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
The symptoms of mononucleosis mimic those of many other infectious diseases, including hepatitis, rubella, and toxoplasmosis. Typically, after an incubation period of about 10 days in children and from 30 to 50 days in adults, infectious mononucleosis produces prodromal symptoms, such as headache, malaise, and fatigue. After 3 to 5 days, patients typically develop a triad of symptoms: sore throat, cervical lymphadenopathy, and temperature fluctuations, with an evening peak of 101° to 102° F (38.3° to 38.9° C). Splenomegaly, hepatomegaly, stomatitis, exudative tonsillitis, or pharyngitis may also develop.
Sometimes, early in the illness, a maculopapular rash that resembles rubella develops; also, jaundice occurs in about 5% of patients. Major complications are rare but may include splenic rupture, aseptic meningitis, encephalitis, hemolytic anemia, idiopathic thrombocytopenic purpura, and Guillain-Barré syndrome. Symptoms usually subside about 6 to 10 days after onset of the disease but may persist for weeks.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Colorado tick fever:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
After a 3- to 6-day incubation period, Colorado tick fever begins abruptly with chills; temperature of 104° F (40° C); severe aching of back, arms, and legs; lethargy; and headache with eye movement such as extraocular movement. Photophobia, abdominal pain, nausea, and vomiting may occur. Rare effects include petechial or maculopapular rashes and central nervous system involvement. Symptoms subside after several days but return within 2 to 3 days and continue for 3 more days before slowly disappearing. Complete recovery usually follows.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Lassa fever:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
After a 7- to 18-day incubation period, this disease produces a fever that persists for 2 to 3 weeks, exudative pharyngitis, oral ulcers, lymphadenopathy with swelling of the face and neck, purpura, conjunctivitis, and bradycardia. Severe infection may also cause hepatitis, myocarditis, pleural infection, encephalitis, and permanent unilateral or bilateral deafness.
Virus multiplication in reticuloendothelial cells causes capillary lesions that lead to erythrocyte and platelet loss; mild to moderate thrombocytopenia (with a tendency toward bleeding); and secondary bacterial infection. These capillary lesions may also cause focal hemorrhage in the stomach, small intestine, kidneys, lungs, and brain and, possibly, hemorrhagic shock and peripheral vascular collapse.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Relapsing fever:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
The incubation period for relapsing fever is 5 to 15 days (the average is 7 days). Clinically, tick- and louse-borne diseases are similar. Both begin suddenly, with a temperature approaching 105° F (40.6° C), prostration, headache, severe myalgia, arthralgia, diarrhea, vomiting, coughing, and eye or chest pains. Splenomegaly is common; hepatomegaly and lymphade-nopathy may occur. During febrile periods, the victim's pulse and respiratory rates rise, and a transient macular rash may develop over his torso.
The first attack usually lasts from 3 to 6 days; then the patient's temperature drops quickly and is accompanied by profuse sweating. A skin rash on the trunk lasting 1 to 2 days is common after the primary febrile episode. The rash may be petechiae, macular, or papular. About 5 to 10 days later, a second febrile, symptomatic period begins. In louse-borne infection, additional relapses are unusual; but, in tick-borne cases, a second or third relapse is common. As the afebrile intervals become longer, relapses become shorter and milder because of antibody accumulation. Relapses are possibly due to antigenic changes in the Borrelia organism.
Complications from relapsing fever include nephritis, bronchitis, pneumonia, endocarditis, seizures, cranial nerve lesions, paralysis, and coma. Death may occur from hyperpyrexia, massive bleeding, circulatory failure, splenic rupture, or a secondary infection.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Rheumatic fever and rheumatic heart disease:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
In 95% of patients, rheumatic fever characteristically follows a streptococcal infection that appeared a few days to 6 weeks earlier. A temperature of at least 100.4° F (38° C) occurs, and most patients complain of migratory joint pain or polyarthritis. Swelling, redness, and signs of effusion usually accompany such pain, which most commonly affects the knees, ankles, elbows, or hips. In 5% of patients (generally those with carditis), rheumatic fever causes skin lesions such as erythema marginatum, a nonpruritic, macular, transient rash that gives rise to red lesions with blanched centers. Rheumatic fever may also produce firm, movable, nontender, subcutaneous nodules about 3 mm to 2 cm in diameter, usually near tendons or bony prominences of joints (especially the elbows, knuckles, wrists, and knees) and less often on the scalp and backs of the hands. These nodules persist for a few days to several weeks and, like erythema marginatum, often accompany carditis.
Later, rheumatic fever may cause transient chorea, which develops up to 6 months after the original streptococcal infection. Mild chorea may produce hyperirritability, a deterioration in handwriting, or an inability to concentrate. Severe chorea (Sydenham’s chorea) causes purposeless, nonrepetitive, involuntary muscle spasms; poor muscle coordination; and weakness. Chorea always resolves without residual neurologic damage.
The most destructive effect of rheumatic fever is carditis, which develops in up to 50% of patients and may affect the endocardium, myocardium, pericardium, or the heart valves. Pericarditis causes a pericardial friction rub and, occasionally, pain and effusion. Myocarditis produces characteristic lesions called Aschoff bodies (in the acute stages) and cellular swelling and fragmentation of interstitial collagen, leading to formation of a progressively fibrotic nodule and interstitial scars. Endocarditis causes valve leaflet swelling, erosion along the lines of leaflet closure, and blood, platelet, and fibrin deposits, which form beadlike vegetations. Endocarditis affects the mitral valve most often in females; the aortic, most often in males. In both females and males, endocarditis affects the tricuspid valves occasionally and the pulmonic only rarely.
Severe rheumatic carditis may cause heart failure with dyspnea; right upper quadrant pain; tachycardia; tachypnea; a hacking, nonproductive cough; edema; and significant mitral and aortic murmurs. The most common of such murmurs include:
❑ a systolic murmur of mitral insufficiency (high-pitched, blowing, holosystolic, loudest at apex, possibly radiating to the anterior axillary line)
❑ a midsystolic murmur due to stiffening and swelling of the mitral leaflet
❑ occasionally, a diastolic murmur of aortic insufficiency (low-pitched, rumbling, almost inaudible). Valvular disease may eventually result in chronic valvular stenosis and insufficiency, including mitral stenosis and insufficiency, and aortic insufficiency. In children, mitral insufficiency remains the major sequela of rheumatic heart disease.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Rocky Mountain spotted fever:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
The incubation period is usually about 7 days, but it can range from 2 to 14 days. Generally, the shorter the incubation time, the more severe the infection. Signs and symptoms, which usually begin abruptly, include a persistent temperature of 102° to 104° F (38.9° to 40° C); a generalized, excruciating headache; nausea and vomiting; and aching in the bones, muscles, joints, and back. In addition, the tongue is covered with a thick white coating that gradually turns brown as the fever persists and rises.
Initially, the skin may simply appear flushed. Between days 2 and 5, eruptions begin around the wrists, ankles, or forehead; within 2 days, they cover the entire body, including the scalp, palms, and soles. The rash consists of erythematous macules 1 to 5 mm in diameter that blanch on pressure; if untreated, the rash may become petechial and maculopapular. By the third week, the skin peels off and may become gangrenous over the elbows, fingers, and toes.
The pulse is strong initially, but it gradually becomes rapid (possibly reaching 150 beats/minute) and thready.
Alert A rapid pulse rate and hypotension (systolic pressure less than 90 mm Hg) herald imminent death from complete vascular collapse.
Other signs and symptoms include a bronchial cough, a rapid respiratory rate (as high as 60 breaths/minute), anorexia, constipation, abdominal pain, hepatomegaly, splenomegaly, insomnia, restlessness and, in extreme cases, delirium. Urine output falls to half of the normal level or less, is dark in color, and contains albumin. Complications, although uncommon, include lobar pneumonia, otitis media, pa-rotitis, disseminated intravascular coagulation (DIC) and, possibly, renal failure. In rare cases, RMSF leads to death.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Rheumatic fever and rheumatic heart disease:
Signs and symptoms
(Handbook of Diseases)
In 95% of patients, rheumatic fever characteristically follows a streptococcal infection that appeared a few days to 6 weeks earlier. A temperature of at least 100.4° F (38° C) occurs.
Joint pain
Most patients complain of migratory joint pain or polyarthritis. Swelling, redness, and signs of effusion usually accompany such pain, which most commonly affects the knees, ankles, elbows, or hips.
Skin lesions and nodules
In 5% of patients (generally those with carditis), rheumatic fever causes skin lesions, such as erythema marginatum. This nonpruritic, macular, transient rash gives rise to red lesions with blanched centers.
Rheumatic fever may also produce firm, movable, nontender, subcutaneous nodules ⅛" to ¾" (0.5 to 2 cm) in diameter, usually near tendons or bony prominences of joints (especially the elbows, knuckles, wrists, and knees) and less commonly on the scalp and backs of the hands. These nodules persist for a few days to several weeks and, like erythema marginatum, often accompany carditis.
Chorea
Later, rheumatic fever may cause transient chorea, which develops up to 6 months after the original streptococcal infection.
Mild chorea may produce hyperirritability, a deterioration in handwriting, or an inability to concentrate. Severe chorea causes purposeless, nonrepetitive, involuntary muscle spasms; poor muscle coordination; and weakness. Chorea always resolves without residual neurologic damage.
Carditis
The most destructive effect of rheumatic fever is carditis, which develops in up to 50% of patients. It may affect the endocardium, myocardium, pericardium, or the heart valves.
Pericarditis causes a pericardial friction rub and, occasionally, pain and effusion. Myocarditis produces characteristic lesions called Aschoff bodies (in the acute stages) and cellular swelling and fragmentation of interstitial collagen, leading to formation of a progressively fibrotic nodule and interstitial scars.
Endocarditis causes valve leaflet swelling, erosion along the lines of leaflet closure, and blood, platelet, and fibrin deposits, which form beadlike vegetations. Endocarditis usually affects the mitral valve in females and the aortic valve in males. In both sexes, endocarditis affects the tricuspid valves occasionally and the pulmonic valve only rarely.
Severe rheumatic carditis may cause heart failure with dyspnea, right-upper-quadrant pain, tachycardia, tachypnea, significant mitral and aortic murmurs, and a hacking, nonproductive cough.
The most common murmurs include:
❑ a systolic murmur of mitral insufficiency (high-pitched, blowing, holo-systolic, loudest at apex, possibly radiating to the anterior axillary line)
❑ a midsystolic murmur caused by stiffening and swelling of the mitral leaflet
❑ occasionally, a diastolic murmur of aortic insufficiency. Valvular disease may eventually result in chronic valvular stenosis and insufficiency, including mitral stenosis and insufficiency and aortic insufficiency. In children, mitral insufficiency remains the major after-effect of rheumatic heart disease.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Fever - Case 11-1: 18-Month-Old Girl:
IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)
Despite advances in our understanding of the molecular basis for NF1, the
diagnosis remains one that is largely based on clinical criteria. Clinical
diagnosis of NF1 requires the presence of at least two of the seven consensus
criteria stipulated by the National Institutes of Health (NIH) (Table 11-3).
Children with sporadic rather than inherited cases may not meet the NIH
diagnostic criteria until later in life. At 1 year of age, approximately 50% of
individuals with sporadic disease lack two or more of the cardinal clinical
features permitting diagnosis, but by age 8 years, 95% meet NIH criteria.
The most visible features of NF1 are flat, evenly pigmented macules known as café-au-lait spots. These macules, often present at birth, increase in both number
and size over the first few years of life. One or two caf
é-au-lait macules are present in up to 25% of the normal population, but the
presence of six or more macules should raise suspicion for NF1. These macules
are easier to visualize with the use of a Wood
's lamp. Skinfold freckling, another pigmentary change associated with NF1,
usually occurs in the axillae, groin, nape of the neck, or under the chin. By 6
years of age, approximately 80% of children with NF1 demonstrate axillary or
inguinal freckling.
Lisch nodules are benign pigmented hamartomas of the iris that occur in patients
with NF1. These nodules do not interfere with vision. Lisch nodules may not be
apparent in young children, but they are present in more than 95% of adolescent
and adult patients. Detection of Lisch nodules on bedside examination is
challenging, and diagnosis frequently requires a slit-lamp examination by an
experienced ophthalmologist. In contrast to Lisch nodules, optic nerve tumors,
such as optic nerve gliomas, occur primarily in younger children. They are
often associated with asymmetric, noncorrectable vision loss, diminished
peripheral vision and color discrimination, and proptosis.
Subcutaneous or cutaneous (dermal) neurofibromas are rarely seen in young
children but appear during or just before adolescence. Neurofibromas are
present in 48% of 10-year-old patients and 84% of 20-year-old patients.
Cutaneous lesions frequently begin as small papules on the face, scalp, trunk,
and extremities. Deep lesions may be detected only through palpation. These
lesions represent a major cosmetic problem but do not transform into malignant
tumors. In contrast, plexiform neurofibromas surround soft tissue and bone,
causing aberrant growth. Plexiform neurofibromas, present in 30% of patients,
are locally invasive and may undergo malignant transformation. They may be
accompanied by overlying hyperpigmentation or hypertrichosis. Other tumors that
occur with higher frequency in patients with NF1 include pheochromocytomas,
juvenile chronic myeloid leukemia, and rhabdomyosarcomas.
Seizures occur in approximately 4% to 5% of patients with NF1. Seizures may be
generalized or partial. In a study by Korf et al., 22 of 359 NF1 patients
developed seizures. The seizures were most often characterized as
complex-partial (9 patients), febrile (6 patients), or generalized epilepsy (3
patients). Other manifestations of NF1 include learning disabilities, pain,
scoliosis, headaches, stroke, and bowel or bladder complications (secondary to
pelvic plexiform neurofibromas).
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Complaints and Diagnostic Dilemmas, 2003
Fever - Case 11-4: 7-Month-Old Girl:
IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)
Most patients with AIN in infancy suffer from mild infections such as otitis
media, gastroenteritis, lymphadenitis, superficial skin infections, or upper
respiratory tract infections. In one series, 6 (23%) of 26 girls developed
cellulitis of the labia majora, and 3 of these 6 infections were caused by
P. aeruginosa. Approximately 10% to 15% of patients have serious infections, including
pneumonia, sepsis, or meningitis. In approximately 10% of children, the
diagnosis is suspected only after a routine complete blood count reveals
neutropenia.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Complaints and Diagnostic Dilemmas, 2003
Fever and Neutropenia:
Presentation
(Pediatric Infectious Disease)
Fever in the neutropenic patient is usually defined as a single temperature
greater than 38.3
°C, (101.3°F) or a sustained temperature of 38°C (100.4°F) temperature for more than 1 hour.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Infectious Disease, 2004
Outpatient Evaluation of Fever:
Presentation
(Pediatric Infectious Disease)
The history, physical exam, and even complete laboratory evaluations do not
separate these infants into
“high-risk” (high statistical likelihood to have a serious bacterial infection) and “low-risk” categories.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Infectious Disease, 2004
Mononucleosis as a Cause of Symptoms or Medical Conditions
When considering symptoms of Mononucleosis, it is also important to consider Mononucleosis as a possible cause of other medical conditions.
The Disease Database lists the following medical conditions that Mononucleosis may cause:
- (Source - Diseases Database)
Medical articles and books on symptoms:
These general reference articles may be of interest
in relation to medical signs and symptoms of disease in general:
Full list of premium articles on symptoms and diagnosis
About signs and symptoms of Mononucleosis:
The symptom information on this page
attempts to provide a list of some possible signs and symptoms of Mononucleosis.
This signs and symptoms information for Mononucleosis has been gathered from various sources,
may not be fully accurate,
and may not be the full list of Mononucleosis signs or Mononucleosis symptoms.
Furthermore, signs and symptoms of Mononucleosis may vary on an individual basis for each patient.
Only your doctor can provide adequate diagnosis of any signs or symptoms and whether they
are indeed Mononucleosis symptoms.
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» Next page: Diagnostic Tests for Mononucleosis
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