Treatments for Riboflavin deficiency
Treatments for Riboflavin deficiency
The list of treatments mentioned in various sources
for Riboflavin deficiency
includes the following list.
Always seek professional medical advice about any treatment
or change in treatment plans.
- Adequate riboflavin in diet - sources include yeasts, milk, diary, fortified breakfast cereals, liver, offal.
- Riboflavin supplementation
- Vitamin B2
Drugs and Medications used to treat Riboflavin deficiency:
Note:You must always seek professional medical advice about any prescription drug, OTC drug, medication, treatment
or change in treatment plans.
Some of the different medications used in the treatment of Riboflavin deficiency include:
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Vitamin B deficiencies:
Treatment
(Professional Guide to Diseases (Eighth Edition))
Diet and supplementary vitamins can correct or prevent vitamin B deficiencies, as follows:
❑ Thiamine deficiency: a high-protein diet, with adequate calorie intake, possibly supplemented by B-complex vitamins for early symptoms. Thiamine-rich foods include pork, peas, wheat bran, oatmeal, and liver. Alcoholic beriberi may require thiamine supplements or thiamine hydrochloride as part of a B-complex concentrate.
❑ Riboflavin deficiency: supplemental riboflavin in patients with intractable diarrhea or increased need for riboflavin related to growth, pregnancy, lactation, or wound healing. Good sources of riboflavin are meats, enriched flour, milk and dairy products, green, leafy vegetables, eggs, and cereal. Acute riboflavin deficiency requires daily oral doses of riboflavin alone or with other B-complex vitamins. Riboflavin phosphate can also be administered I.V. or I.M.
❑ Niacin deficiency: supplemental B-complex vitamins and dietary enrichment in patients at risk because of marginal diets or alcoholism. Meats, fish, peanuts, brewer’s yeast, enriched breads, and cereals are rich in niacin; milk and eggs, in tryptophan. Confirmed niacin deficiency requires daily doses of niacinamide orally or I.V.
❑ Pyridoxine deficiency: prophylactic pyridoxine therapy in infants and in children with a seizure disorder; supplemental B-complex vitamins in patients with anorexia, malabsorption, or those taking isoniazid or penicillamine. Some women who take hormonal contraceptives may have to supplement their diets with pyridoxine. Confirmed pyridoxine deficiencies require oral or parenteral pyridoxine. Children with convulsive seizures stemming from metabolic dysfunction may require daily doses of 200 to 600 mg pyridoxine.
❑ Cobalamin deficiency: parenteral cobalamin in patients with reduced gastric secretion of hydrochloric acid, lack of intrinsic factor, some malabsorption syndromes, or ileum resections. Strict vegetarians may have to supplement their diets with oral vitamin B12. Depending on the deficiency’s severity, supplementary cyanocobalamin is usually given parenterally for 5 to 10 days, followed by monthly or daily vitamin B12 supplements.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Vitamin A deficiency:
Treatment
(Professional Guide to Diseases (Eighth Edition))
Mild conjunctival changes or night blindness requires vitamin A replacement in the form of cod liver oil or halibut liver oil. Acute deficiency requires aqueous vitamin A solution I.M., especially when corneal changes have occurred. Therapy for underlying biliary obstruction consists of administration of bile salts; for pancreatic insufficiency, pancreatin. Dry skin responds well to cream-based or petroleum-based products.
In patients with chronic malabsorption of fat-soluble vitamins, and in those with low dietary intake, prevention of vitamin A deficiency requires aqueous I.V. supplements or an oral water-miscible preparation.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Vitamin C deficiency:
Treatment
(Professional Guide to Diseases (Eighth Edition))
Because scurvy is potentially fatal, treatment begins immediately to restore adequate vitamin C intake by daily doses of 100 to 200 mg vitamin C in synthetic form or in orange juice in mild disease and by doses as high as 500 mg/day in severe disease. Symptoms usually subside in 2 to 3 days; hemorrhages and bone disorders, in 2 to 3 weeks.
To prevent vitamin C deficiency, patients unable or unwilling to consume foods rich in vitamin C or those facing surgery should take daily supplements of ascorbic acid. The recommended daily allowance is 60 mg/day. Vitamin C supplementation may also prevent this deficiency in recently weaned infants or those drinking formula not fortified with vitamin C.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Vitamin D deficiency:
Treatment
(Professional Guide to Diseases (Eighth Edition))
For osteomalacia and rickets — except when caused by malabsorption — treatment consists of oral doses of vitamin D or sources such as fish, liver, and processed milk. Exposure to sunlight is encouraged. For rickets refractory to vitamin D or in rickets accompanied by hepatic or renal disease, treatment includes 25-hydroxycholecalciferol, 1, 25-dihydroxycholecalciferol, or a synthetic analogue of active vitamin D. Replacement of deficient calcium and phosphorus also helps to eliminate most symptoms of rickets. Positioning or bracing may be used to reduce or prevent deformities; some skeletal deformities may require corrective surgery.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Vitamin E deficiency:
Treatment
(Professional Guide to Diseases (Eighth Edition))
Replacement of vitamin E with a water-soluble supplement, either oral or parenteral, is the only appropriate treatment.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Vitamin K deficiency:
Treatment
(Professional Guide to Diseases (Eighth Edition))
Administration of vitamin K I.V. or I.M. corrects abnormal bleeding tendencies.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
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