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Diseases » Rubella » Symptoms
 

Symptoms of Rubella

Symptoms of Rubella

The list of signs and symptoms mentioned in various sources for Rubella includes the 23 symptoms listed below:

Research symptoms & diagnosis of Rubella:

Rubella: Complications

Review medical complications possibly associated with Rubella:

Rubella Symptoms: Book Excerpts

Diagnostic Testing

Diagnostic testing of medical conditions related to Rubella:

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Do I have Rubella?

Rubella: Undiagnosed Conditions

Diseases that may be commonly undiagnosed in related medical areas:

Home Diagnostic Testing

Home medical tests related to Rubella:

Wrongly Diagnosed with Rubella?

The list of other diseases or medical conditions that may be on the differential diagnosis list of alternative diagnoses for Rubella includes:

See the full list of 13 alternative diagnoses for Rubella

Rubella: Research Doctors & Specialists

Research all specialists including ratings, affiliations, and sanctions.

More about symptoms of Rubella:

More information about symptoms of Rubella and related conditions:

Other Possible Causes of these Symptoms

Click on any of the symptoms below to see a full list of other causes including diseases, medical conditions, toxins, drug interactions, or drug side effect causes of that symptom.

Medical Books Online about Rubella

Medical Books Excerpts Excerpts of published medical book chapters related to Rubella are available from published medical books for more detailed information about Rubella.

Medical Books Excerpts
  • Rubella
  • "Professional Guide to Diseases (Eighth Edition)" (2005)

Copyright notice for book excerpts: Copyright © 2008 Lippincott Williams & Wilkins. All rights reserved.

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Patient Surveys for Rubella

Symptoms of Rubella: Online Medical Books

16 MEDICAL BOOKS ONLINE! Review excerpts from medical books online, free, without registration, for more information about the symptoms of Rubella.


Rubella: Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))

In children, after an incubation period of from 14 to 21 days, an exanthematous, maculopapular rash erupts abruptly. (See Incubation and duration of common rash-producing infections, page 232.) In adolescents and adults, prodromal signs and symptoms — headache, malaise, anorexia, low-grade fever, coryza, lymphadenopathy and, sometimes, conjunctivitis — are the first to appear. Suboccipital, postauricular, and postcervical lymph node enlargement is a hallmark of this disease and precedes the rash.

Typically, the rubella rash begins on the face and spreads rapidly, in many cases covering the trunk and extremities within hours. Small, red, petechial macules on the soft palate (Forschheimer spots) may precede or accompany the rash but are not diagnostic of rubella. By the end of the second day, the facial rash begins to fade, but the rash on the trunk may become confluent and be mistaken for scarlet fever. The rash continues to fade in the downward order in which it appeared. It generally disappears on the third day, but it may persist for 4 or 5 days — sometimes accompanied by mild coryza and conjunctivitis. The rapid appearance and disappearance of the rubella rash distinguishes it from rubeola. In rare cases, rubella can occur without a rash. Low-grade fever may accompany the rash (99° to 101° F [37.2° to 38.3° C]), but it usually doesn’t persist after the first day of the rash; rarely, temperature may reach 104° F (40° C).

Complications seldom occur in children with rubella, but when they do, they commonly appear as hemorrhagic problems such as thrombocytopenia. Many young women, however, experience transient joint pain or arthritis, usually just as the rash is fading. Fever may then recur. These complications usually subside spontaneously within 5 to 30 days.

A significant number of cases, 20% to 50%, are asymptomatic.

» READ BOOK EXCERPT ONLINE »

Source: Professional Guide to Diseases (Eighth Edition), 2005

Rubella: Signs and symptoms
(Handbook of Diseases)

In children, after an incubation period of 16 to 18 days, an exanthematous, maculopapular rash erupts abruptly. In adolescents and adults, prodromal signs and symptoms — headache, anorexia, malaise, low-grade fever, coryza, lymphadenopathy and, sometimes, conjunctivitis — are the first signs and symptoms. Suboccipital, postauricular, and postcervical lymph node enlargement is a hallmark of rubella.

Short-term rash

Typically, the rubella rash begins on the face. This maculopapular eruption spreads rapidly, sometimes covering the trunk and extremities within hours. Small, red, petechial macules on the soft palate (Forschheimer spots) may precede or accompany the rash.

By the end of the 2nd day, the facial rash begins to fade, but the rash on the trunk may be confluent and may be mistaken for scarlet fever. The rash continues to fade in the downward order in which it appeared. The rash generally disappears on the 3rd day, but it may persist for 4 to 5 days — sometimes accompanied by mild coryza and conjunctivitis.

The rapid appearance and disappearance of the rubella rash distinguishes it from rubeola. Rubella can appear without a rash, but this occurrence is rare. Low-grade fever (99° to 101° F [37.2° to 38.3° C]) may accompany the rash, but it usually doesn’t persist after the 1st day of the rash; rarely, temperature may reach 104° F (40° C).

AGE ALERT: Complications seldom occur in children with rubella, but when they do, they typically appear as hemorrhagic problems such as thrombocytopenia. Many young women, however, experience transient joint pain or arthritis, usually just as the rash is fading. Fever may then recur. These complications usually subside spontaneously within 5 to 30 days.

GENDER INFLUENCE: Congenital rubella syndrome occurs in 25% of infants born to women who acquire rubella during the first trimester of pregnancy. However, defects are rare if the infection occurs after the 20th week of pregnancy. Complications in the fetus may include deafness, cataracts, microcephaly, mental retardation, congenital heart defects, and other problems. Miscarriage or stillbirth may also occur.

» READ BOOK EXCERPT ONLINE »

Source: Handbook of Diseases, 2003

Jaundice - Case 15-1: 14-Day-Old Boy: IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)

Classic galactosemia is an autosomal recessive disease. If not recognized and treated, it can be fatal in the neonatal period. Some of the more common presenting clinical signs have already been discussed (jaundice, hepatomegaly, vomiting, and encephalopathy); more fulminant clinical courses may represent superimposed bacterial sepsis. Among the laboratory findings seen with classic galactosemia are conjugated (or combined) hyperbilirubinemia, liver function test and coagulation study abnormalities, elevations of serum and urine amino acids, and a renal tubulopathy with galactosuria, glycosuria, proteinuria, and metabolic acidosis. Not surprisingly, plasma galactose and erythrocyte galactose-1-phosphate levels are also elevated.
Disappointingly, even galactosemic children whose diets were restricted very early are at increased risk for developmental delays and learning disabilities, compared with their healthy counterparts. Although many children have IQs in the normal range, cognitive, speech, and motor impairments are, nevertheless, more common. Longer delays before initial diagnosis and treatment of galactosemia correlate with worse neurodevelopmental sequelae.
Hypergonadotropic hypogonadism is often observed in girls with galactosemia, and most are infertile as adults. Galactosemic males demonstrate normal puberty and fertility.
The gene for the galactose-1-phosphate uridyl transferase enzyme has been localized to chromosome 9, and multiple variations at that locus have been described. Some African-Americans with galactosemia have a milder clinical course because of a different transferase variant. Still another variant, known as the Duarte variant, is usually clinically insignificant. Prenatal diagnosis of galactosemia is available.

» READ BOOK EXCERPT ONLINE »

Source: Pediatric Complaints and Diagnostic Dilemmas, 2003

Seizures - Case 19-1: 8-Day-Old Girl: IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)

Neonatal L. monocytogenes infection, like group B streptococcal infection, manifests in both an early- and a late-onset form. Clinical manifestations of L. monocytogenes infection are similar to those of other neonatal bacterial infections. Signs of infection include temperature instability, respiratory distress, irritability, lethargy, and poor feeding. In early-onset disease, transplacental transmission after maternal bacteremia or ascending spread from vaginal colonization leads to intrauterine infection with L. monocytogenes. Preterm labor is common among infants with early-onset L. monocytogenes infection; length of gestation is less than 35 weeks in approximately 70% of cases. There is often evidence of an acute febrile maternal illness, with symptoms of fatigue, arthralgias, and myalgias preceding delivery by 2 to 14 days. Blood cultures are positive for L. monocytogenes in 35% of mothers of infants with early-onset listeriosis.
Early-onset infection classically develops within the first or second day of life. Bacteremia (75%) and pneumonia (50%) are usually seen with early-onset infection. Meningitis is seen in 25% of early-onset cases. In severe infection, a granulomatous rash is associated with disseminated disease (granulomatosis infantisepticum). The mortality rate, including stillbirths, is 40% for early-onset infection. In late-onset infection, modes of transmission unrelated to maternal carriage may be involved. Late-onset infection develops during the second to eighth week of life. The most common form of L. monocytogenes infection over this period is meningitis, which is present in approximately 95% of cases. Bacteremia (20%) and pneumonia (10%) are less common. Mortality of late-onset infection is generally low (15%) if the infection is diagnosed early and treated appropriately.
A nosocomial outbreak occurred when nine newborn infants were bathed in mineral oil contaminated with L. monocytogenes. The affected infants developed bacteremia (two cases), meningitis (two cases), or both (five cases); one infant died. Signs of infection developed within 1 week after exposure to the mineral oil.

» READ BOOK EXCERPT ONLINE »

Source: Pediatric Complaints and Diagnostic Dilemmas, 2003

Seizures - Case 19-2: 10-Day-Old Boy: IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)

Signs of hypocalcemia usually develop within the first 3 weeks of life. Signs of neonatal hypocalcemia are often nonspecific and may be seen in a variety of other conditions. Tremors and jitteriness are most commonly seen. Other signs include irritability, hyperreflexia, facial twitching, carpopedal spasm, seizures, cyanosis, and, rarely, laryngospasm. More importantly, other disorders that can manifest with hypocalcemia should be considered. Features of 22q11 deletion syndromes include cleft palate, micrognathia, ear anomalies, bulbous nasal tip, and conotruncal heart defects. Findings associated with Albright hereditary osteodystrophy (pseudohypoparathyroidism type Ia) include round face, short distal phalanges of the thumbs, subcutaneous calcifications, and a family history of developmental delay and dental hypoplasia. Sensorineural deafness, renal dysplasia, and mental retardation are also associated with syndromes that include hypoparathyroidism.

» READ BOOK EXCERPT ONLINE »

Source: Pediatric Complaints and Diagnostic Dilemmas, 2003

German Measles (Third Disease, Rubella): German Measles - signs & symptoms
(The 5-Minute Pediatric Consult)

  • Rash:
    • Begins on face, then progresses to trunk and extremities
    • Does not usually coalesce
    • Lasts for 3 days
  • Adenopathies, especially postauricular, posterior cervical, and suboccipital, are commonly noted, along with conjunctivitis.
  • Arthralgia/arthritis may be seen in adolescents and adults.

» READ BOOK EXCERPT ONLINE »

Source: The 5-Minute Pediatric Consult, 2008

Measles (Rubeola, First Disease): Measles - signs & symptoms
(The 5-Minute Pediatric Consult)

  • The disease involves fever, cough, conjunctivitis, or coryza with an erythematous rash, which has a characteristic progression.
    • The rash appears on the face (often the nape of the neck, initially) and abdomen 14 days after exposure. The rash is erythematous and maculopapular and spreads from the head to the feet often becoming confluent at the more proximal sites.
  • Pharyngitis, cervical lymphadenopathy, and splenomegaly may accompany the rash.
  • Atypical measles:
    • This group of young adults (2nd and 3rd decades of life) may become quite ill, with sudden onset of fever from 103–105°F associated with headache. The rash, unlike typical measles, appears 1st on the distal extremities and progresses in a cephalad direction.
    • Virtually all patients with atypical measles have respiratory distress with clinical and radiographic signs of pneumonia, often with pleural effusions.
    • Diagnosis depends on recognition and on acute and convalescent measles antibody titers.

» READ BOOK EXCERPT ONLINE »

Source: The 5-Minute Pediatric Consult, 2008

Rubella as a Cause of Symptoms or Medical Conditions

When considering symptoms of Rubella, it is also important to consider Rubella as a possible cause of other medical conditions. The Disease Database lists the following medical conditions that Rubella may cause:

- (Source - Diseases Database)

Rubella: Onset and Incubation

Incubation period for Rubella: about 2 to 3 weeks

Medical articles and books on symptoms:

These general reference articles may be of interest in relation to medical signs and symptoms of disease in general:

Full list of premium articles on symptoms and diagnosis

About signs and symptoms of Rubella:

The symptom information on this page attempts to provide a list of some possible signs and symptoms of Rubella. This signs and symptoms information for Rubella has been gathered from various sources, may not be fully accurate, and may not be the full list of Rubella signs or Rubella symptoms. Furthermore, signs and symptoms of Rubella may vary on an individual basis for each patient. Only your doctor can provide adequate diagnosis of any signs or symptoms and whether they are indeed Rubella symptoms.


 » Next page: Diagnostic Tests for Rubella

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