Glossary for Liver symptoms

Medical terms related to Liver symptoms or mentioned in this section include:

• $3-alpha-Hydroxyacyl-CoA Dehydrogenase Deficiency$: A rare inherited form of biochemical disorder characterized by the deficiency of a particular enzyme (3-Hydroxyacyl-CoA Dehydrogenase). The enzyme deficiency only affects certain body tissues, in particular the skeletal muscles. The lack of enzyme activity prevents some fats being converted into energy. Symptoms tend to be exacerbated during fasting as during fasting, the body tries to rely more heavily on fats for energy. Fatty acids that are not completely metabolized due to the enzyme deficiency may build up in various organs and cause serious complications.
• $3-alpha-hydroxyacyl-coenzyme A dehydrogenase deficiency$: A rare inherited form of biochemical disorder characterized by the deficiency of a particular enzyme (3-Hydroxyacyl-CoA Dehydrogenase). The enzyme deficiency only affects certain body tissues, in particular the skeletal muscles. The lack of enzyme activity prevents some fats being converted into energy. Symptoms tend to be exacerbated during fasting as during fasting, the body tries to rely more heavily on fats for energy. Fatty acids that are not completely metabolized due to the enzyme deficiency may build up in various organs and cause serious complications.
• $3-methylglutaconic aciduria, type 4$: A rare genetic disorder where the body's cells are unable to make sufficient energy resulting in an accumulation in the body of 3-methylglutaconic acid. Type 4 is characterized by symptoms which overlap type 1 and 3.
• $4-Alpha-hydroxyphenylpyruvate hydroxylase deficiency$: A very rare metabolic disorder where a deficiency of a particular enzyme results in the urinary excretion of a chemical called hawkinsin. Symptoms start once the infant is weaned off breast milk.
• ACAD9 deficiency: A metabolic disorder involving a deficiency of an enzyme (acyl-CoA dehydrogenase-9). The symptoms are variable and are usually triggered by a viral infection or ingestion of aspirin which can trigger a Reye-like episode which can cause death.
• ALL-Down syndrome: The presence of acute lymphoblastic leukemia in Down syndrome patients. These patients tend to have a poorer prognosis for the leukemia than patients without Down syndrome.
• AREDYLD: A rare condition characterized by abnormalities of the extremities, teeth, hair, nail and kidney as well as lipoatrophic diabetes.
• Aagenaes syndrome: A rare inherited birth abnormality involving underdeveloped lymph vessels which results in swollen legs and liver problems.
• Abdominal Cancer: Growth of abnormal cells (tumour) affecting the organs in the abdominal cavity; may be due to primary growth of a tumour or spread from another tumour (metastases, secondary tumour)
• Abdominal Neoplasms: A tumor that occurs in the abdomen.
• Abdominal pain: Pain in the abdominal area or stomach.
• Abdominal symptoms: Symptoms affecting the abdomen or digestive tract
• Abnormal liver function tests: Abnormal blood test results indicating that there may be a problem with ones liver
• Absence of septum pellucidum and septo-optic dysplasia: A rare birth defect where a thin membrane in the middle of the brain is missing. This brain abnormality is never present on it's own but is a characteristic of septo-optic dysplasia where the patient also has optic disk abnormalities and pituitary deficiencies.
• Absent alpha 1 band: An absence of alpha-1-antitrypsin the the body
• Acanthocytosis: A rare disorder where most of the red blood cells are abnormal with spiny projections due to lipid abnormalities. The blood abnormality is seen in conditions such as abetalipoproteinemia, severe liver disease and severe malnutrition. Symptoms and prognosis depend on the underlying disorder.
• Acetaminophen poisoning: Excessive ingestion of a drug called acetaminophen.
• Acetazolamide - Teratogenic Agent: Experimental studies on mice, rats and rabbits indicate that the use of Acetazolamide during pregnancy may cause various harmful effects on the fetus. The likelihood and severity of symptoms may be affected by the level of exposure and the stage of pregnancy that the exposure occurred at. The effect on human fetuses has not been conclusively determined.
• Achard-Thiers Syndrome: A rare hormonal disorder that occurs in diabetic postmenopausal women where body hair grows in a masculine manner. Hormonal therapy is used to correct the endocrine imbalance.
• Achrestic anemia: Achrestic anemia is a form of anemia similar to that caused by Vitamin B12 deficiency but it doesn't respond to treatment with Vitamin B12. The condition tends to progress slowly and can result in death if not treated. There are a variety of possible causes.
• Acidemia, methylmalonic: An inborn error of metabolism where amino acids in the body aren't metabolized properly resulting in high levels of the acid throughout the body.
• Acinic cell carcinoma: A usually slow-growing malignant tumor that that can occur in various parts of the body but is most often found in the pancreas, salivary glands, palate and upper lip. Symptoms are determined by the size and location of the growth.
• Acorn poisoning: Acorns contain tannic acid which affects the metabolism of proteins and causing serious symptoms if large amounts are consumed. The amount of tannin in the acorn varies amongst species - higher tannin content results in a more bitter tasting acorn.
• Acquired idiopathic sideroblastic anaemia: A rare disorder where iron is transported into a developing blood cells but because it is unable to be used, it builds up within the cell and tends to stop it from developing into a fully functioning red blood cell. Thus anemia can occur despite adequate or even high iron levels. Acquired cases can occur on exposure to excess alcohol, lead and drugs or can occur to nutritional problems involving a deficiency of folic acid or copper or an excess of zinc. The condition can also be caused by conditions such as kidney problems, endocrine dysfunction, metabolic disorders, rheumatoid arthritis and leukemia.
• Acquired total lipodystrophy: A rare acquired disorder that involves adipose tissue abnormalities and is characterized by loss of adipose tissue through the body. The disorder is very similar to the congenital form but the liver involvement is more severe.
• Acral dysostosis - dyserythropoiesis: A rare disorder characterized by hand and foot defects as well as a congenital form of anemia characterized by the production of abnormal red blood cells.
• Acromegaly: An abnormal enlargement of the limbs due to increased secretion of growth hormone after the cessation of puberty
• Actinomycetales infection: A bacterial infection from the order of Actinobacteria. The range of symptoms is variable depending on which bacteria from the order is involved.
• Acute Cholecystitis: Acute inflammation of the gall bladder, usually due to obstruction by a gall stone
• Acute Pesticide poisoning - Triforine: Triforine is an ingredient used in certain herbicides. Exposure to the chemical can cause a range of symptoms depending on the level and route of exposure. Exposure can occur through inhalation, ingestion, the skin or eyes. Acute exposure involves a exposure over a short period of time whereas chronic exposure occurs over a longer period of time.
• Acute adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The acute subtype tends to progress rapidly and is the most prevalent form of the condition.
• Acute basophilic leukaemia: A rare type of acute myeloid leukemia characterized by the presence of abnormal basophils.
• Acute biphenotypic leukemia: A rare form of leukemia that has myeloid and lymphoid features.
• Acute cholinergic dysautonomia: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The acute form has more severe symptoms than the chronic form.
• Acute fatty liver of pregnancy: A rare complication of pregnancy that can occur in the second half of the pregnancy. It is characterized by excessive fatty deposits in the liver which can be fatal without prompt diagnosis and treatment which involves delivering the baby as soon as possible.
• Acute leukaemia of ambiguous lineage: A term used to describe a type of leukemia (a blood cancer) where the leukemic cells cannot be determined as myeloid or lymphoid or where both types of cells are present.
• Acute lymphoblastic leukemia: A malignant disease that starts suddenly and progresses quickly. It is characterized by a high number of immature cells in the organs, bone marrow and blood. Symptoms include fever, pallor, anorexia, fatigue, anemia, hemorrhage, bone pain, splenomegaly and frequent infections. Also called acute lymphocytic leukemia.
• Acute lymphoblastic leukemia, adult: Cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets.
• Acute megacaryoblastic leukemia: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. More specifically, it involves the rapid proliferation of megakaryoblasts (premature form of megakaryocytes).
• Acute meningitis: Acute meningitis is a form of meningitis caused by bacteria that normally lives in the mouth and throat. When the immune system is unable to supress this bacteria, it travels to the cerebrospinal spinal fluid in the brain. From there it affects the membranes surrounding the brain.
• Acute myeloblastic leukemia type 1: A form of blood cancer resulting in the rapid proliferation of immature blood cells (blast cells).
• Acute myeloblastic leukemia type 2: A form of blood cancer resulting in the rapid proliferation of granulocytes and monocytes.
• Acute myeloblastic leukemia type 3: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 3 involves the proliferation of promyelocytes.
• Acute myeloblastic leukemia type 4: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 4 involves the rapid proliferation of myelocytes and monocytes.
• Acute myeloblastic leukemia type 5: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. Type 5 involves the rapid proliferation of monoblasts (immature precursors of monocytes) in particular.
• Acute myeloblastic leukemia type 6: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 6 involves the proliferation of the immature precursors of red blood cells called erythroblasts.
• Acute myeloblastic leukemia type 7: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. Type 7 involves the rapid proliferation of megakaryoblasts (premature form of megakaryocytes) in particular.
• Acute myelocytic leukemia: A malignant cancer of blood-forming tissues resulting in a high number of immature leukocytes. Symptoms include soft bleeding gums, anemia, fatigue, fever, dyspnea, moderate splenomegaly, joint and bone pains and frequent infections. Also called acute granulocytic leukemia, acute myelogenous leukemia, acute nonlymphocytic leukemia, myeloid leukemia, splenomedullary leukemia, splenomyelogenous leukemia.
• Acute myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
• Acute myeloid leukaemia and myelodysplastic syndromes related to alkylating agent: The use of alkylating agents to treat cancer can result in leukemia in some patients.
• Acute myeloid leukaemia and myelodysplastic syndromes related to topoisomerase type II inhibitor: The use of topoisomerase type II inhibitors to treat cancer can result in leukemia in some patients.
• Acute myeloid leukaemia and myelodysplastic syndromes, therapy related: Certain cancer therapies can result in the development of leukemia in some patients. These therapies includes topoisomerase type II inhibitors and alkylating agents.
• Acute myeloid leukemia: A form of rapidly progressing blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets.
• Acute myeloid leukemia, adult: A form of blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets.
• Acute myelosclerosis: A rare disorder where the bone marrow makes too many blood cells. The disease progresses rapidly with death usually occurring within 6 months of onset.
• Acute panmyelosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement. The disease often progresses rapidly and results in death.
• Acute vitamin A toxicity: Acute ingestion of vitamin A can cause symptoms. Symptoms usually only last for a day or two.
• Acyl-CoA dehydrogenase, very long chain, deficiency of: A rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form.
• Addison-Gull syndrome: A condition where scarring of the liver and bile ducts which results in chronic jaundice, enlarged spleen and liver and yellow skin plaques due to abnormal lipid metabolism.
• Adhesive abuse: Adhesive abuse is the use of various inhalants for the purpose of achieving a "high". They are often used as a cheap, readily available alternative to street drugs but they can cause serious damage to the body. Adhesives include household glues, rubber cement and model aeroplane glue. These adhesives can be abused by sniffing them, spraying directly into the mouth, heating them and then inhaling them or injecting them directly into the body.
• Adhesive addiction: Adhesive addiction refers to the compulsive need to abuse adhesives (e.g. sniffing them). Sufferers have withdrawal symptoms when attempting to stop the habit and feel unable to stop the habit despite knowing the harm it is causing their health. Aerosols are very damaging to the body and can readily result permanent brain damage and even death. Death can occur through chronic use and in rare cases can occur after one session of use. Children and teenagers are particular at risk for this type of addiction - it is readily available and users feel it gains them greater acceptance from their peers. Adhesives includes household glue, rubber cement and model airplane glue.
• Adrenal hemorrhage, neonatal: Hemorrhage of the adrenal gland after birth. The severity of the disorder is varies from a small hemorrhage to damage to the whole adrenal gland. Sometimes the condition is discovered incidentally during ultrasounds for other reasons. The hemorrhage may occur as the result of a variety of causes including adrenal tumor, neonatal stress, and blood coagulation disorder or for no apparent reason.
• Adrenoleukodystrophy: A rare hereditary metabolic disease that only occurs in male children and is characterized by adrenal atrophy and extensive cerebral demyelination causing progressive loss of mental functioning, aphasia, apraxia and sometimes blindness. The patient usually dies within 5 years.
• Adrenoleukodystrophy, autosomal, neonatal form: A rare inherited disorder involving the adrenal glands, testes and certain parts of the brain (white matter). It is a less severe form of leukodystrophy where an abnormality within the body cells prevents the metabolism of certain fats (long chain fatty acids).
• Adult Cystic Fibrosis: Cystic fibrosis is an inherited condition characterized by the production of thick sticky mucus by the mucus glands in the lungs, intestines, liver and pancreas. The condition is most often diagnosed in children or young adults but occasionally, relatively mild symptoms may lead to frequent misdiagnosis or no diagnosis at all unless the symptoms become worse. The condition may be misdiagnosed as emphysema, asthma or chronic bronchitis. It is usually females with a mild form of the disease who tend to be diagnosed at a later age.
• Adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis.
• Adult T-Cell lymphoma: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The lymphoma subtype is aggressive and tends to affect the lymph nodes more than the blood.
• Adult onset Still's disease: A form of Still's disease that has a later onset and involves arthralgia or arthritis and a characteristic rash that often appears during periods of temperature increase.
• Adverse reaction to chemical - 1,2-Dibromoethane: 1,2-Dibromoethane is a chemical used in gasoline, soil fumigants, fire extinguishers, flue gases and mechanical gauge fluid. Excessive exposure to this chemical can cause serious symptoms. The severity of symptoms varies amongst patients.
• Aerosol abuse: Aerosol abuse is the use of various inhalants for the purpose of achieving a "high". They are often used as a cheap, readily available alternative to street drugs but they can cause serious damage to the body. Aerosols include air fresheners, hair spray, spray pain and deodorants. These aerosols can be abused by sniffing them, spraying directly into the mouth, heating them and then inhaling them or injecting them directly into the body.
• Aerosol addiction: Aerosol addiction refers to the compulsive need to abuse aerosol (e.g. sniffing them). Sufferers have withdrawal symptoms when attempting to stop the habit and feel unable to stop the habit despite knowing the harm it is causing their health. Aerosols are very damaging to the body and can readily result permanent brain damage and even death. Death can occur through chronic use and in rare cases can occur after one session of use. Children and teenagers are particular at risk for this type of addiction - it is readily available and users feel it gains them greater acceptance from their peers. Aerosols includes spray pain, air freshener, deodorants and hair sprays.
• Aflatoxin B1 exposure: Aflatoxin B1 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• Aflatoxin B1- Teratogenic Agent: Reports indicate that the use of Aflatoxin B1 during pregnancy may cause various harmful effects on the fetus. The likelihood and severity of symptoms may be affected by the level of exposure and the stage of pregnancy that the exposure occurred at. The effect on human fetuses has not been conclusively determined.
• Aflatoxin B2 exposure: Aflatoxin B2 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• Aflatoxin G1 exposure: Aflatoxin G1 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• Aflatoxin G2 exposure: Aflatoxin G2 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• Aflatoxin exposure: Aflatoxins are toxins produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• African Sleeping sickness: A disease caused by parasites (Trypanosome brucei gamiense or T. brucei rodesiense) and transmitted to humans by the tsetse fly which is found only in Africa. Causes symptoms such as fever, chills, headache, anemia, edema of hands and feet, enlarged lymph glands, lethargy, sleepiness, convulsions and coma. Also called African trypanosomiasis and sleeping sickness.
• Aggressive NK-cell leukaemia: An aggressive form of blood cancer involving the rapid proliferation of natural killer (NK) cells.
• Aggressive systemic mastocytosis: The excessive proliferation of mast cells. Mast cells control the skin's response to minor injury and release a chemical called histamine which causes the skin to redden. In the aggressive form, mast cells accumulate in the liver, spleen and lymphatic system.
• Aicardi-Goutieres syndrome 1: A rare inherited progressive disease that affects the brain and immune system. Type 1 is caused by a defect on chromosome 3p21.3-p21.2.
• Aicardi-Goutieres syndrome 2: A rare inherited progressive disease that affects the brain and immune system. Type 2 is caused by a defect on chromosome 13q14-q21.
• Aicardi-Goutieres syndrome 3: A rare inherited progressive disease that affects the brain and immune system. Type 3 is caused by a defect on chromosome 11q13.2.
• Aicardi-Goutieres syndrome 4: A rare inherited progressive disease that affects the brain and immune system. Type 4 is caused by a defect on chromosome 19p13.13.
• Aicardi-Goutieres syndrome 5: A rare inherited progressive disease that affects the brain and immune system. Type 5 is caused by a defect on chromosome 3p21.3-p21.2.
• Alagille syndrome: A genetic disorder affecting the liver and characterized by the absence of some or all of the liver bile ducts that transport bile within the liver.
• Albers-Schonberg disease - malignant recessive form: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. The malignant infantile form is the most severe form of this disorder and death usually occurs in the first decade of life.
• Alcohol abuse: Excessive alcohol as a symptom of other conditions
• Alcohol-Induced Disorders: Disorders caused by excessive alcohol consumption. The symptoms are variable depending on the disorder involved. Some of the disorders are: alcohol abuse, alcohol dependence, alcohol intoxication, alcohol withdrawal, alcohol intoxication delirium, alcohol withdrawal delirium, alcohol-induced persisting dementia, alcohol-induced persisting amnestic disorder, alcohol-induced psychotic disorder, alcohol-induced mood disorder, alcohol-induced anxiety disorder, alcohol-induced sexual dysfunction, alcohol-induced sleep disorder, liver damage, liver cancer and esophageal cancer.
• Alcohol-induced pseudo-Cushing syndrome: The excessive consumption of alcohol can result in symptoms similar to a condition called Cushing's syndrome. When alcohol consumption is stopped, symptoms regress.
• Alcoholic liver disease: Alcoholic liver disease is the major cause of liver disease in Western countries, (in Asian countries, viral hepatitis is the major cause). It arises from the excessive ingestion of alcohol.
• Aldolase A deficiency: A rare condition where a deficiency of the enzyme called aldolase A causes muscle problems and anemia.
• Allopurinol hypersensitivity syndrome: An immune reaction to the ingestion of a drug called allopurinol. It is a potentially fatal disorder.
• Alpers Syndrome: A rare syndrome characterized by liver disease, seizures and progressive, episodic psychomotor retardation.
• Alpha thalassemia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. Alpha thalassemia involves defects in one or more of the four genes required to make each ? protein chain. The main symptom is anemia, the severity of which can vary amongst patients depending on how many defective genes are involved.
• Alpha thalassemia - Hemoglobin H disease: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. Hemoglobin H disease involves defects in three of the four genes required to make each ? protein chain. The main symptom is moderate to severe anemia.
• Alpha thalassemia major: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. Alpha thalassemia major is very rare involves defects in all of the four genes required to make each ? protein chain. The condition leads to infant death before or soon after birth.
• Alpha-mannosidosis type II: A rare inherited metabolic disorder involving a deficiency of an enzyme (alpha-mannosidosase) which results in the accumulation of certain chemicals in the body which leads to progressive damage. This form of the condition is less severe than type I (infantile form).
• Alstrom syndrome: A rare, hereditary genetic disorder which is progressive. Initial symptoms in infants are usually extreme light sensitivity, wobbling of the eyes and sometimes dilated dilated cardiomyopathy and congestive heart failure. Young children quickly gain weight and become obese and as they get older, multiple organ systems may be affected causing blindness, hearing loss, type 2 diabetes, heart failure, liver disease, urological dysfunction, pulmonary fibrosis and renal failure.
• Alveolar Hydatid Disease: Rare multi-organ tapeworm infection caught from animals.
• Alveolar echinococcosis: A rare parasitic infection caused by the larva of a miniscule tapeworm called Echinococcus multilocularis. Transmission occurs through contact with foxes, coyotes, dogs and cats. The condition results in the development of parasitic tumors in the liver (sometimes other organs such as brain and lungs) but it generally causes no symptom for 5 to 15 years after infection.
• Amebic dysentery: Intestinal inflammation caused by Entamoeba histolytica and often marked by symptoms such as frequent, loose bowel movements that contain blood and mucus. Also called intestinal amebiasis.
• Amyloidosis: A disease characterized by the accumulation of insoluble amyloid protein in tissues and organs which in turn affects the functioning of these tissues and organs.
• Amyloidosis AL: A disease involving the abnormal deposit of amyloid fibrils in virtually any part of the body - the heart, liver, kidney and peripheral and autonomic nerves are most commonly affected. The abnormal amyloid fibrils are produced abnormal plasma cells in the bone marrow. In some cases, the excess growth of abnormal plasma cells can result in a cancerous condition called myeloma resulting in bone pain and infections. A patient with myeloma may develop amyloidosis but it is rare for a patient with AL amyloidosis to go on to develop myeloma.
• Amyloidosis, familial visceral: A rare genetic disorder involving widespread amyloidosis (abnormal buildup of amyloid protein in tissues) which tends the affect the kidneys severely.
• Amyloidosis, inflammatory: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage and is potentially fatal. Symptoms depend on the organs involved. Secondary amyloidosis is caused by a chronic infection of inflammatory conditions such as rheumatoid arthritis, multiple myeloma, tuberculosis and osteomyelitis. The main organs affected in secondary amyloidosis are usually the kidneys, liver, spleen and lymph nodes. The peripheral and autonomic nerves and the heart are rarely affected.
• Andersen disease: An rare inborn error of metabolism involving glycogen storage and characterized by cirrhosis and sometimes liver failure. Lack of the amyl-transglucosidase enzyme and abnormal glycogen causes the condition.
• Anemia: Reduced red blood cells in the blood
• Anemia, Blackfan Diamond: Diamond-Blackfan anemia is a rare genetic condition where the bone marrow is unable to make sufficient red blood cells which leads low levels of red blood cells. There are eight subtypes of the condition which differ in the location of the genetic defect and the incidence of additional symptoms such as malformations. The severity of symptoms is variable but most cases are serious.
• Anemia, Hemolytic, Warm Antibody: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 37ºC or higher. The severity of the disorder is variable.
• Anemia, Neonatal: Insufficient red blood cells that can carry oxygen around the body. It is common in premature births or can occur as a result of blood loss before, during or just after the birth.
• Anemia, Sideroblastic: A rare blood disorder where abnormal utilization of iron results in the production of defective red blood cells which have excessive deposits of iron in them.
• Anemia, hypochromic microcytic: A blood disorder where red blood cells are too small and lack sufficient iron. It can be inherited or caused by insufficient iron in the diet or from a genetic disorder.
• Anemias, Sideroblastic: Sideroblastic anemias are a group of rare blood disorders where the bone marrow is unable to produce normal red blood cells. The body has enough iron but the red blood cells are unable to utilize it in a normal manner and anemia results. The red blood cells become overloaded with iron and are unable to carry out their normal functions. Some forms of sideroblastic anemia are inherited but most tend to be acquired due to such things as exposure to toxins and certain drugs, leukemia, inflammatory conditions such as rheumatoid arthritis and nutritional deficiencies (e.g. copper and pyridoxine deficiency). Inherited forms usually appear in childhood whereas acquired forms usually occur in adulthood.
• Anemic - hematuria syndrome: An epidemic disease in Argentina which has a prolonged recovery time but usually there are no complications. Symptoms vary between seasons so that affected patients suffer anorexia, vomiting, diarrhea and dehydration in summer but suffer reduced urination, excess blood, albumin and renal casts in the urine. Other symptoms occur irrespective of the season.
• Anesthetic agent-induced liver damage: Damage or injury to the liver caused by exposure to anesthetic agents. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Chloroform: Damage or injury to the liver caused by exposure to an anesthetic agent called chloroform. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Cyclopropane: Damage or injury to the liver caused by exposure to an anesthetic agent called cyclopropane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Ether: Damage or injury to the liver caused by exposure to an anesthetic agent called ether. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Halothane: Damage or injury to the liver caused by exposure to an anesthetic agent called halothane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Methoxyflurane: Damage or injury to the liver caused by exposure to an anesthetic agent called methoxyflurane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Nitrous Oxide: Damage or injury to the liver caused by exposure to an anesthetic agent called nitrous oxide. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Angioimmunoblastic with dysproteinemia lymphadenopathy: A rare immune system disorder which is similar to lymphoma. The condition is progressive but the course varies with some patients surviving a long time without treatment and others surviving only a short period of time.
• Angiosarcoma of the liver: A rare type of cancer that starts in the lining of blood vessels in the liver. It is generally an aggressive tumor which often metastasizes.
• Antepartum Eclampsia: Antepartum eclampsia is the development of seizures or coma in pregnant women suffering from high blood pressure. Antepartum means that it occurs before delivery. Eclampsia is a serious condition which requires urgent medical treatment. Eclampsia may be associated with moderate as well as significant increases in blood pressure. The blood pressure can return to normal after delivery or may persist for a period of time.
• Antibiotics-induced liver damage: Damage or injury to the liver caused by taking certain antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Cephalosporin: Damage or injury to the liver caused by exposure to Cephalosporin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Chloramphenicol: Damage or injury to the liver caused by exposure to an antibiotic called Chloramphenicol. Chloramphenicol a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Clindamycin: Damage or injury to the liver caused by exposure to Clindamycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Erythromycin Ethyl succinate: Damage or injury to the liver caused by exposure to Erythromycin Ethyl succinate antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Erythromycin estolate: Damage or injury to the liver caused by exposure Erythromycin estolate antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Novobiocin: Damage or injury to the liver caused by exposure to Novobiocin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Quinolone: Damage or injury to the liver caused by exposure to Quinolone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Spectinomycin: Damage or injury to the liver caused by exposure to Spectinomycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Sulfones: Damage or injury to the liver caused by exposure to sulfone antiboitics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Telithromycin: Damage or injury to the liver caused by exposure to Telithromycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Tetracycline: Damage or injury to the liver caused by exposure to Tetracycline antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Nitrofuran: Damage or injury to the liver caused by exposure to Nitrofuran antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Penicillin: Damage or injury to the liver caused by exposure to Penicillin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Rifampicin: Damage or injury to the liver caused by exposure to Rifampicin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage: Damage or injury to the liver caused by exposure to anticonvulsives. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage - Mephenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called mephenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage - Phenobarbital: Damage or injury to the liver caused by exposure to an anticonvulsive called phenobarbital. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage - Phenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called Phenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage - Valproic Acid: Damage or injury to the liver caused by exposure to an anticonvulsive called valproic acid. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage: Damage or injury to the liver caused by exposure to antifungal agents. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - 5-Fluorocytosine: Damage or injury to the liver caused by exposure to an antifungal agent called 5-Fluorocytosine. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - Amphotericin: Damage or injury to the liver caused by exposure to an antifungal agent called Amphotericin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - Griseofulvin: Damage or injury to the liver caused by exposure to an antifungal agent called Griseofulvin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - Ketoconazole: Damage or injury to the liver caused by exposure to an antifungal agent called Ketoconazole. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - Saramycetin: Damage or injury to the liver caused by exposure to an antifungal agent called Saramycetin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage: Damage or injury to the liver caused by exposure to Antimetazoal agents. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage - Amodiaquine: Damage or injury to the liver caused by exposure to an antimetazoal agent called amodiaquine. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage - Hycanthone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called hycanthone. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage: Damage or injury to the liver caused by exposure to antiprotozoal agents. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - 8-Hydroxyquinolone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called 8-Hydroxyquinolone. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Carbarsone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called carbarsone. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Emetine: Damage or injury to the liver caused by exposure to an antiprotozoal agent called emetine. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Mepacrine: Damage or injury to the liver caused by exposure to an antiprotozoal agent called mepacrine. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Metronidazole: Damage or injury to the liver caused by exposure to an antiprotozoal agent called Metronidazole. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Thiabendazole: Damage or injury to the liver caused by exposure to an antiprotozoal agent called Thiabendazole. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage: Damage or injury to the liver caused by exposure to antituberculous agents. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - Cycloserine: Damage or injury to the liver caused by exposure to an antituberculous agent called cycloserine. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - Ethionamide: Damage or injury to the liver caused by exposure to an antituberculous agent called ethionamide. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - Isoniazid: Damage or injury to the liver caused by exposure to an antituberculous agent called isoniazid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - Rifampicin: Damage or injury to the liver caused by exposure to an antituberculous agent called rifampicin. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - p-aminosalicylic acid: Damage or injury to the liver caused by exposure to an antituberculous agent called p-aminosalicylic acid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage: Damage or injury to the liver caused by exposure to antiviral agents. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage - Cytarabine: Damage or injury to the liver caused by exposure to an antiviral agent called cytarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage - Vidarabine: Damage or injury to the liver caused by exposure to an antiviral agent called vidarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage - idoxuridine: Damage or injury to the liver caused by exposure to an antiviral agent called idoxuridine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage - xenylamine: Damage or injury to the liver caused by exposure to an antiviral agent called xenylamine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Aplastic anemia: A blood disorder where the bone marrow produces insufficient new blood cells.
• Apo A-I deficiency: Low plasma HDL cholesterol that tends to run in families.
• Arakawa's syndrome 2: An inherited metabolic disorder where an enzyme deficiency (methionine synthase) causes mental and physical retardation, blood disorders, degeneration of brain tissue and various other symptoms.
• Arctic bearded seal poisoning: The Arctic Bearded seal is often used as a food source by the arctic inhabitants. Eating the liver and kidneys of the arctic bearded seal can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases. It is believed that eating more than 100-250 grams of the seal liver can result in human death.
• Arginase deficiency: A very rare urea cycle disorder caused by a deficiency of the enzyme (arginase) needed to convert ammonia to the urea which can then be removed in the urine. The condition leads to excess build-up of ammonia in the body which is toxic to the nervous system.
• Argininosuccinase lyase deficiency, neonatal: A rare inherited urea cycle disorder caused by lack of enzymes (argininosuccinase lyase) needed to turn ammonia into urea resulting in excess ammonia in the body. The neonatal form of the condition can result in death or severe complications if not treated early enough.
• Argininosuccinic aciduria: A rare inherited disorder of the urea cycle characterized by the lack of an enzyme (argininosuccinate lyase) which is needed to remove nitrogen from the body so a lack of the enzyme leads to a build-up of ammonia in the blood.
• Aromatase deficiency: A congenital deficiency of the enzyme called aromatase which is needed to convert androgens to estrogens.
• Arthralgia - purpura - weakness syndrome: A rare autoimmune disorder characterized mainly by weakness, joint pain, reddish or purplish skin discolorations and a blood abnormality where cryoglobulins in the blood precipitate at low temperatures.
• Arthrogryposis - renal dysfunction - cholestasis syndrome: A very rare syndrome characterized by joint contractures, kidney dysfunction and liver problems.
• Aspartylglucosaminidase deficiency: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
• Aspartylglucosaminuria: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
• Aspartylglycosaminuria: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
• Asrar-Facharzt-Haque syndrome: A form of sinus histiocytosis characterized by lymphadenopathy as well as involvement of other tissues such as the sinus cavity, skin, lungs, bone, eyes, kidneys, testes, head and neck area and the central nervous system. Symptoms vary according to the organ involved. The condition usually runs a prolonged course with spontaneous remission occurring in some cases. Rare cases may be fatal.
• Athabaskan severe combined immunodeficiency: A severe immunodeficiency disorder found in Navajo and Apache populations.
• Athyrotic hypothyroidism sequence: A rare congenital disorder characterized by a thyroid gland defect.
• Australian Sea Lion poisoning: The Australian Sea Lion is sometimes used as a food source and is found in the South-Southwest waters of Australia. Eating the liver of the Australian Sea Lion can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases.
• Autoimmune Hemolytic Anemia: Autoimmune hemolytic anemia is characterized by an abnormal immune system response which leads to the destruction of red blood cells and hence anemia. The severity of the condition varies depending on the underlying cause e.g. cytomegalovirus, hepatitis, HIV and lupus. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Autoimmune Hepatitis: Liver inflammation caused due to autoimmune processes where the body's immune system attacks the liver.
• Autoimmune Lymphoproliferative Syndrome: An inherited autoimmune condition characterized by proliferation of lymphocytes and autoimmunity against the body's own blood cells resulting in premature death of certain blood cells.
• Autoimmune Thrombocytopenia: Autoimmune disorder causing a lack of blood platelets.
• Autoimmune polyendocrinopathy syndrome, type 1: A very rare autoimmune disorder characterized by autoimmune polyendocrinopathy (APE), candidiasis (C) and ectodermal dysplasia (ED).
• Autosomal Recessive Polycystic Kidney Disease: Severe form of PKD, a genetic kidney disease.
• Autosomal dominant polycystic kidney disease: Genetic kidney disease causing kidney cysts.
• B-cell chronic lymphocytic leukemia: A slow progressing disease involving cancerous B-cell lymphocytes which take over the healthy cells in the lymph nodes. B-cells help the body to fight infections so when the disease becomes more advanced, the body is less able to fight infection as there are fewer healthy, functioning B-cells.
• B-cell prolymphocytic leukaemia: A rare type of leukemia involving a proliferation of immature white blood cells (prolymphocytes - B-cells).
• Baber's syndrome: A very rare syndrome characterized by the association of congenital liver cirrhosis with Fanconi syndrome.
• Babesiosis: A protozoal infection which is transmitted to human via the bite of certain ticks.
• Bacillus cereus type I food poisoning: Bacillus cereus is a bacterium that can cause food poisoning symptoms such as vomiting and diarrhea. There are two types: Type I causes mainly vomiting and is associated with fried rice whereas type II causes mainly diarrhea and is associated with meats, cereals, vegetables and milk.
• Bacterial endocarditis: Infection and inflammation of the inner layers of the heart, most commonly the valves cause by bacteria.
• Bacterial meningitis: Bacterial meningitis is a form of meningitis caused by bacteria that normally lives in the mouth and throat. When the immune system is unable to supress this bacteria, it travels to the cerebrospinal spinal fluid in the brain. From there it affects the membranes surrounding the brain.
• Baker-Winegrad disease: A very rare syndrome caused by a deficiency of the enzyme fructose-1-6-diphosphatase which impairs the body's ability to break down fructose that is consumed in the diet.
• Ballard syndrome: A rare condition characterized by enlarged liver and spleen and fractures that occur in areas of weakened bone.
• Banti's syndrome: A chronic, progressive condition marked by enlargement of the spleen which is associated with anemia, splenomegaly, ascites, jaundice, leucopenia, thrombocytopenia and gastrointestinal bleeding.
• Bantu siderosis: An iron overload disorder initially observed in South African people. The disorder involves abnormal iron deposits in the liver. It is believed that some African people are predisposed to an increased ability to absorb iron.
• Bardet-Biedl syndrome, type 1: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 1 is caused by a defect in chromosome 11q13.
• Bardet-Biedl syndrome, type 10: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 10 is caused by a defect in chromosome 12q.
• Bardet-Biedl syndrome, type 11: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 11 is caused by a defect in chromosome 9q33.1.
• Bardet-Biedl syndrome, type 12: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 12 is caused by a defect in chromosome 4q27.
• Bardet-Biedl syndrome, type 2: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 2 is caused by a defect in chromosome 16q21.
• Bardet-Biedl syndrome, type 3: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 3 is caused by a defect in chromosome 3p12-q13.
• Bardet-Biedl syndrome, type 4: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 4 is caused by a defect in chromosome 15q22.3.
• Bardet-Biedl syndrome, type 5: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 5 is caused by a defect in chromosome 2q31.
• Bardet-Biedl syndrome, type 6: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 6 is caused by a defect in chromosome 20p12.
• Bardet-Biedl syndrome, type 7: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 7 is caused by a defect in chromosome 4q27.
• Bardet-Biedl syndrome, type 8: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 8 is caused by a defect in chromosome 14q32.11.
• Bardet-Biedl syndrome, type 9: A rare genetic disorder characterized by mental retardation, obesity, polydactyly and retinal pigmentation as well as other abnormalities. Type 9 is caused by a defect in chromosome 7p14.
• Bartonella: A class of bacteria that can infect humans at a range of different sites. The most well known is Cat Scratch Disease, caused by B.henselae.
• Bartonella infections: Infection with bacteria from the Bartonella genus of bacteria. Specific bacteria from within this group are Bartonella bacilliforms (Oroya fever), Bartonella Heneslae (Cat-scratch disease). Other conditions caused by this bacteria are endocarditis, bacteremia and angiomatosis. Symptoms vary depending on the type of bacteria involved and the severity of the infection - immunocompromised patients face greater risk of severe infection.
• Bartonellosis: An infection by Bartonella bacilliformis which is transmitted through sandfly bites. Symptoms include fever, severe anemia, bone pain and skin lesions. Also called Carrion's disease, Oroya fever or verruga peruana.
• Bartonellosis due to Bartonella quintana infection: A disease caused by infection with Bartonella quintana which are transmitted by the body louse. It causes trench fever but may also result in septicemia and endocarditis in patients with a weakened immune system.
• Bearn-Kunkel syndrome: A type of autoimmune liver disease characterized by liver damage, very high blood gammaglobulin levels and increased plasma cells.
• Benign intrahepatic cholestasis type 1: A rare liver disorder characterized by intermittent episodes of cholestasis (impaired bile flow within the liver) that doesn't involve obstruction of bile ducts outside the liver. The jaundice tends to disappear in weeks or months without treatment and the condition does not lead to liver failure. Type 1 is caused by a defect on the ATP8B1 gene on chromosome 18q21.
• Benign intrahepatic cholestasis type 2: A rare liver disorder characterized by intermittent episodes of cholestasis (impaired bile flow within the liver) that doesn't involve obstruction of bile ducts outside the liver. The jaundice tends to disappear in weeks or months without treatment and the condition does not lead to liver failure. Type 2 is caused by a defect on the ABCB11 gene on chromosome 2q24.
• Berardinelli-Seip congenital lipodystrophy: A rare genetic disorder characterized by diabetes mellitus, loss of body fat, hepatomegaly, enlarged genitals, increased skeletal growth and other abnormalities.
• Berardinelli-Seip congenital lipodystrophy, type 1: A rare genetic disorder characterized by early-onset diabetes mellitus, loss of body fat, serious insulin resistance, high blood triglycerides and fatty liver. Type 1 is distinguished from type 2 by the origin of the genetic defect. Type 1 is caused by a defect on the AGPAT2 gene on chromosome 9q34.3. Type 1 seems to be less severe with some cases of type 2 resulting in premature death which can occur as early as the first year of life. Type 2 also involves mental retardation which is not seen in type 1.
• Berardinelli-Seip congenital lipodystrophy, type 2: A rare genetic disorder characterized by early-onset diabetes mellitus, loss of body fat, serious insulin resistance, high blood triglycerides and fatty liver. Type 2 is distinguished from type 2 by the origin of the genetic defect. Type 2 is caused by a defect on the BSCL2 gene on chromosome 11q13. Type 2 seems to be more severe with some cases resulting in premature death which can occur as early as the first year of life. Type 2 also involves mental retardation which is not seen in type 1.
• Bernard syndrome: A familial condition characterized by acute anemia, jaundice, hemoglobinuria and destruction of red blood cells. Acute symptoms may follow an episode of fever, abdominal pain, vomiting, malaise and joint pain.
• Bernheim's syndrome: Reduced size of right heart ventricle due to enlargement of the left ventricle which encroaches on the space in the right ventricle. Blood flow from the right atrium to the right ventricle is obstructed.
• Berry aneurysm, cirrhosis, pulmonary emphysema, and cerebral calcification: A very rare syndrome characterized primarily by brain aneurysms, liver cirrhosis, pulmonary emphysema and calcification of the brain (cerebrum).
• Besnier-Boeck-Schaumann disease: A rare disease where inflammatory granular nodules form in various organs.
• Beta Thalassemia intermedia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. There are two subtypes of the disorder (? and ?) depending on what portion of the hemoglobin is abnormally synthesized. Beta Thalassemia intermedia involves defects in both of the two genes required to make each ? protein chain. The condition causes varying degrees of moderate anemia.
• Beta thalassemia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. Beta thalassemia involves defects in one or more of the two genes required to make each ? protein chain. The main symptom is anemia, the severity of which can vary amongst patients depending on how many defective genes are involved.
• Bile Duct Cancer: A malignancy arising in the bile ducts of the liver
• Bile Duct Conditions: An inflammatory bacterial infection that affects the meninges
• Bile acid synthesis defect, congenital, 2: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (cholestasis with delta(4)-3-oxosteroid 5-beta-reductase) needed to make bile acid.
• Bile acid synthesis defect, congenital, 4: A defect which prevents the body from making bile acid which results in progressive liver disease.
• Bile acid synthesis defects: A defect which prevents the body from making bile acid which results in progressive liver disease.
• Bile acid synthesis defects, congenital, 1: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (3-beta-hydroxy-delta-5-C27-steroid oxidoreductase) needed to make bile acid.
• Bile acid synthesis defects, congenital, 2: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (cholestasis with delta(4)-3-oxosteroid 5-beta-reductase) needed to make bile acid.
• Bile acid synthesis defects, congenital, 3: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect involved a deficiency of 7-alpha-hydroxylase which is an enzyme needed to prevent the accumulation of 27-hydroxycholesterol which is toxic to the liver.
• Bile acid synthesis defects, congenital, 4: A defect which prevents the body from making bile acid which results in progressive liver disease.
• Bile duct cancer, extrahepatic: A rare cancer that develops in the part of the common bile duct that is outside the liver. The common bile duct channels bile from the gall bladder (which stores bile) and the liver (which makes bile) to the small intestine to assist digestion.
• Bile duct cysts: The formation of a cyst (dilation or swelling) in the bile duct which can obstruct the flow of bile and result in jaundice.
• Bile duct paucity in Graft-Versus-Host Disease: A reduced number of ducts that carry bile through and from the liver (bile duct) that results from graft-versus-host disease.
• Bile duct paucity in Liver Allograft Rejection: A reduced number of ducts that carry bile through and from the liver (bile duct) that results from liver allograft rejection.
• Bile duct paucity, non syndromic form: A reduced number of ducts that carry bile through and from the liver (bile duct) that is not associated with a syndrome. Paucity of bile ducts is associated with many liver conditions such as biliary cirrhosis and sclerosing cholangitis.
• Bile plug syndrome: A rare condition where thick bile in infants causes blockage of the bile ducts which can result in jaundice, enlarged liver and anemia.
• Biliary Atresia: A rare condition that can affect newborns where the bile duct that carries the bile from the liver to the small intestine is blocked or missing.
• Biliary atresia, intrahepatic, non syndromic form: Congenital obstruction of the passages in the liver that carry bile. The nonsyndromic form is not associated with any other abnormalities.
• Biliary atresia, intrahepatic, syndromic form: Congenital obstruction of the passages in the liver that carry bile. The syndromic form is associated with other congenital abnormalities such as heart and visceral defects.
• Biliary cirrhosis: Biliary cirrhosis is a condition where the bile ducts are unable to transport bile effectively due to blockage, inflammation, scarring or some other damage to the bile ducts. The condition may result from such things as congenital defect of the bile ducts (e.g. biliary atresia), cystic fibrosis, gallstones or a variety of other secondary conditions. The cause of primary biliary cirrhosis is not fully understood.
• Biliary disorder: Any condition affecting the bile ducts
• Biliary hypoplasia: An underdeveloped biliary duct system which is involved in transporting bile. The bile ductules may be completely absent or be fewer in number than normal.
• Biliary tract cancer: Cancer that develops in the gallbladder or bile ducts.
• Biotinidase deficiency, late onset: A metabolic disorder where the body lacks the enzyme biotinidase needed to process the vitamin called biotin (vitamin H) into carboxylase enzymes. The severity of symptoms may vary depending on the degree of deficiency. Severe cases can result in metabolic acidosis which can lead to death if treatment isn't given.
• Birth symptoms: Symptoms related to childbirth.
• Black nightshade poisoning: The Black Nightshade is a herb which bears small white or purple flowers and dull black berries. The plant originated in South America. The berries contain solanine alkaloid which can be toxic if eaten in large quantities. The leaves and unripe berries are considered toxic whereas the ripe fruit is possibly edible.
• Blastoma: A type of tumor that originates from precursor cells or blasts (immature or embryonic tissue). The symptoms can vary greatly and are determined by the part of the body that is affected. Blastomas can occur in parts of the body such as the brain, liver, kidneys, nervous system, bones and the retina.
• Blastomycosis: A fungal infection caused by Blastomyces dermatitidis and resulting in lung, skin, bone and genitourinary involvement.
• Blue devil poisoning: The Blue Devil is a hairy flowering herb which tends to grow in mountainous, forested areas. All parts of the plant contain pyrrolizidine alkaloids which are slightly toxic if eaten. Skin contact with the hairs on the plant can also cause minor skin irritation.
• Blueberry muffin syndrome: A manifestation of fetal rubella infection. The newborn has purplish or bluish skin lesions or bumps as well as thrombocytopenia. The skin usually clears within 6 weeks.
• Boichis syndrome: A rare syndrome involving nephronophthisis (progressive destruction of kidney tissue) from birth which leads to kidney failure and liver fibrosis.
• Bornholm disease: Contagious viral infection
• Boutonneuse fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Breast Milk Jaundice: Jaundice in an infant caused by the presence of a particular substance in the mother's breast milk which leads to high bilirubin levels in the infant. The condition tends to run in families. The jaundice tends to persist up to six weeks after birth.
• Breynia officinalis poisoning: Ingestion of the Breynia officinalis plant can cause irritation to mucosal linings and liver problems. The plant is often used as a herbal drug (Chi R Yun) to treat such things as poor growth, heart failure and venereal disease.
• Brown Recluse spider poisoning: The Brown Recluse spider is poisonous and is found mainly in southern and central areas of the US.
• Brucellosis: An infectious disease caused by the Brucella genus which is transmitted from animals to humans.
• Bruch's disease: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Brunzell syndrome: A rare recessively inherited syndrome characterized by a lack of skin fat (Seip syndrome), cystic angiomatosis of the long bones which can lead to fractures.
• Budd chiari syndrome: A disorder where the main vein leaving the liver becomes blocked leading to symptoms such as liver enlargement and fluid buildup in the abdomen. Also called Chiari's syndrome or Rokitansky's disease.
• CCF: When the heart is no longer able to pump enough blood to meet the needs of the body.
• CDG syndrome (generic term): Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. The main symptom in all the disorders is psychomotor retardation but other variable symptoms also occur depending on the subtype of the disorder.
• CDG syndrome type 1A: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
• CDG syndrome type 1B: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1B has a phosphomannose isomerase enzyme defect.
• CDG syndrome type 3: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 3 has variable symptoms.
• CDG syndrome type I: A rare genetic disorder where the body is unable to synthesize glycoproteins which results in multisystem problems.
• CML-Like Syndrome, Familial: A very rare condition characterized by symptom similar to myelocytic leukemia that develop during infancy. The condition can result in death during the first years of life.
• CMV antenatal infection: A rare condition where a fetus becomes infected with the cytomegalovirus through the mother.
• COACH syndrome: A very rare syndrome characterized by ataxia, gaps or holes in various eye structures, mental retardation, liver fibrosis and brain abnormalities.
• Camurati-Engelmann Disease: A very rare genetic disease thickening of areas of bone causing pain, weakness and wasting. Usually affects the legs during childhood. Also called diaphyseal dysplasia.
• Carbamoyl-phosphate synthase 1 deficiency: A very rare inherited urea cycle disorder where the lack of the enzyme carbamoyl phosphate synthetase prevents ammonia from being turned into urea and being excreted in the urine. Excess ammonia builds up in the body which can cause serious complications or even death if left untreated.
• Carcinoid syndrome: Carcinoid heart disease is a rare, metastatic disease that occurs predominantly in the right heart. The tricuspid and pulmonic valves are affected, leading to right heart failure, which results in increased morbidity and mortality.
• Cardiomyopathy - hypogonadism - metabolic anomalies: A rare syndrome characterized mainly by heart muscle disease, hypogonadism, blindness, deafness and metabolic anomalies. Puberty was normal despite the hypogonadism.
• Cardiomyopathy - spherocytosis: A rare disorder characterized by the association of spherocytosis with heart muscle disease. Spherocytosis is a red blood cell disorder where the red blood cells have abnormal membranes which gives them a spherical shape and makes them weak resulting in their premature death.
• Cardiomyopathy due to anthracyclines: Damage to the heart muscle caused by anthracycline drugs which are used in chemotherapy. The damage occurs more frequently with higher cumulative doses. Often the patients have no symptoms of the heart damage for many year.
• Carnitine palmitoyl transferase 1 deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase I) prevents fatty acids being transported to the part of the cell that converts it to energy.
• Carnitine palmitoyl transferase 2 deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase) which prevents fatty acids being transported to the part of the cell that converts it to energy. There are two main subtypes of the disorder with each involving a slightly different form of the enzyme. Type I can be readily managed through diet. Type II has three subtypes: the myopathic form affects mainly the muscles; the hepatocardiomuscular form affects the liver and heart muscle; and the lethal neonatal form affects muscles and organs and usually results in death during the first year of life.
• Carnitine palmitoyl transferase II deficiency, infantile hepatocardiomuscular type: A very rare metabolic disorder where deficiency of a particular enzyme (CPT II) prevents muscle fats being converted to energy. The infantile form of this disease affects the muscles and the liver and heart.
• Carnitine palmitoyl transferase II deficiency, lethal neonatal form: A very rare metabolic disorder where deficiency of a particular enzyme (CPT II) prevents muscle fats being converted to energy. The lethal neonatal form affects various organs as well as the muscles and death usually occurs during the first year of life.
• Carnitine palmitoyl transferase deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase) which prevents fatty acids being transported to the part of the cell that converts it to energy. There are two main subtypes of the disorder with each involving a slightly different form of the enzyme. Type I can be readily managed through diet. Type II has three subtypes: the myopathic form affects mainly the muscles; the hepatocardiomuscular form affects the liver and heart muscle; and the lethal neonatal form affects muscles and organs and usually results in death during the first year of life.
• Carnitine transporter deficiency: An inherited deficiency of carnitine caused by the impaired ability of the carnitine transporter protein to carry the carnitine to where it is needed. Instead the carnitine is excreted through the urine. Fasting or illness can trigger a severe attack.
• Carnitine-acylcarnitine translocase deficiency: A very rare inherited metabolic disorder where long-chain fatty acids can't be metabolized properly because the compound needed to transport it is faulty. Ultimately this prevents certain fats (long-chain acylcarnitine) being converted to energy and results in a build up of the fat which is harmful to body organs and tissues.
• Caroli Disease: A rare disorder where the bile ducts inside the liver become enlarged resulting in infection, irritation and gallstone formation.
• Carotenemia: High level of carotene in blood causing skin and plasma to become yellowish. Also called pseudojaundice or xanthemia.
• Carotenoid overuse: Excessive consumption of carotenoids through diet or supplementation can cause harmless symptoms. Carotenoids are pigments found in various colored fruit and veges such as carrots and pumpkin.
• Caspase-8 deficiency: A rare type of immunodeficiency disorder caused by a deficiency of caspase-8. Caspase-8 an important part of the immune system as it is involved in the activation of T lymphocytes, B lymphocytes and natural killer cells.
• Castellani syndrome: A rare syndrome characterized by fever, arthritis and enlarged kidney and liver. The fever may fluctuate or come and go. It is most often seen in middle aged males.
• Cephalosporin-induced Immune Hemolytic Anemia: Cephalosporin-induced immune hemolytic anemia is a condition where a use of a medication called Cephalosporin triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Cerebellar vermis hypoplasia - oligophrenia - congenital ataxia - coloboma - hepatic fibrosis: A very rare syndrome characterized by ataxia, gaps or holes in various eye structures, mental retardation, liver fibrosis and brain abnormalities.
• Cerebrorenodigital syndrome: A rare group of syndromes characterized mainly by brain, kidney, finger and toe abnormalities.
• Ceroid storage disease: A rare metabolic storage disease characterized by the abnormal deposits of a waxy substance called ceroid lipfuscin in various parts of the body such as the liver, spleen and intestinal lining.
• Chagas disease: A parasitic infection caused by the protozoa Trypanosoma cruzi and transmitted by insect bites or blood transfusions. The disease primarily involves the heart and gastrointestinal system.
• Chediak-Higashi like syndrome: A rare genetic disorder characterized mainly by albinism (lack of pigmentation). There are three different subtypes of the disorder (I, II and III) each with varying additional features such as immunodeficiency and neurological symptoms. Type 1 involves partial albinism and neurological symptoms, type II involves partial albinism, immunodeficiency and sometimes neurological symptoms and type III involves albinism only.
• Chemical poisoning - 1,1-Dimethylhydrazine: 1,1-Dimethylhydrazine is a chemical used mainly in jet fuel and rocket fuel, plant growth agent, photography and various other industrial uses. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - 1,2-Dibromo-3-Chloropropane: 1,2-Dibromo-3-Chloropropane is a chemical used in soil fumigants and as a nematocide for various field crops. Commercial examples include Fumagon, Nemagon, Fumazone, Nemapax, Nemafume. Excessive exposure to this chemical can cause relatively mild symptoms. The chemical may be absorbed through the skin. The severity of symptoms varies depending on the amount of chemical involved and the route of exposure.
• Chemical poisoning - 1,2-Dibromoethane: 1,2-Dibromoethane is a chemical used in gasoline, soil fumigants, fire extinguishers, flue gases and mechanical gauge fluid. Excessive exposure to this chemical can cause serious symptoms. Some people can suffer an adverse reaction to the chemical. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - 1,3-Dichloropropene: 1,3-Dichloropropene is a chemical used in solvents and soil fumigants for nematode control. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - 1,3-Dinitrobenzene: 1,3-Dinitrobenzene is a chemical used mainly in explosives. The chemical can be readily absorbed through the skin and cause systemic symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - 1,4-Dioxane: 1,4-Dioxane is a chemical used mainly as a reagent in laboratries and as a solvent in chemical processing. The chemical is readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - 2,4,6-Trinitrotoluene: 2,4,6-Trinitrotoluene is a chemical used mainly as an explosive agent and in the production of dyes and photographic chemicals. The chemical may be absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - 4-Aminopyridine: 4-Aminopyridine is a pesticide used mainly to control bird pests. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Acetylene Tetrabromide: Acetylene Tetrabromide is a chemical used mainly in mineral separation and as a solvent. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Acetylsalicylic Acid: Acetylsalicylic Acid is also known as aspirin and is primarily used to relieve pain, fever and inflammation. Excessive exposure to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Allyl chloride: Allyl chloride is a chemical used mainly in the manufacture of epichlorohydrin and glycerin but is also used in the production of products such as polyester, varnish plastic adhesive, insecticides, perfumes and pharmaceuticals. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Aniline: Aniline is a chemical used mainly in the manufacture of perfumes, varnishes, resins, dyes, paint removers, herbicides, fungicides, explosives, solvents and photographic chemicals. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Anti-rust products: Anti-rust products contain various chemicals which are toxic if ingested. The ingested chemicals can continue to cause damage to the organs and gastrointestinal lining for weeks after the ingestion and severe cases can result in death. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Arsine: Arsine is a chemical used in the making of semiconductors and in the metal refining industry. It is considered a possible chemical agent in chemical warfare. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Benzidine: Benzidine is a chemical used mainly in the production of azo dyes for such products as leather, paper and textiles. The chemical is readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Biphenyl: Biphenyl is a chemical used mainly as a fungicide for fruit packaging and in textile dyes. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Boric Acid: Boric Acid is a chemical used mainly in foods (preservative, emulsifier, neutralizer), antiseptics, pesticides and contact lens cleaners. Ingestion and other exposures to the chemical can cause various symptoms. Application of boric acid directly to damaged skin can cause the chemical to be absorbed rapidly into the body and lead to death. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Bromoform: Bromoform is a chemical with limited industrial uses but is used as a laboratory chemical and can be present in treated water. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Calcium Polysulfide: Calcium polysulfide is a chemical used mainly in antifungal treatments for trees. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Carbon Disulfide: Carbon Disulfide is a chemical used mainly in corrosion inhibitors, cold and nickel plating, photography applications and as a solvent in gums and resins. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Carbon Tetrachloride: Carbon tetrachloride is a chemical used mainly in grain fumigants, insecticides and in the production of fluorocarbons. Ingestion and other exposures to the chemical can cause various symptoms. The chemical is readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Cerium: Cerium is metallic element used mainly in carbon electrodes for arc lamps, flat screen televisions, aluminium and iron alloys and the manufacture of stainless steel and permanent magnets. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chloralose: Chloralose is a chemical used mainly in poisons for rodents and crows . Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chlorate salts: Chlorate salt is a chemical used mainly in herbicides and in the manufacture of matches and explosives. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chlordane: Chlordane is a poison use to control termites - is banned in the US and many other countries due to its harmful effects. Ingestion and other exposures to the chemical can cause various symptoms. The chemical is readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chlorinated naphthalene: Chlorinated naphthalene is a chemical used in a wide range of applications: plasticizers, rubber industries, manufacture of electrical equipment and the petroleum industry. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chlorodiphenyl: Chlorodiphenyl is a chemical used mainly in electrical cables and wires, electric condensers, lubricants and foundry coating and in the manufacture of paper, resins, rubbers, textiles, wood preservatives, electrical components, lacquers, herbicides and plasticizers. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chloroform: Chloroform is a chemical used mainly as a refrigerant but also as a solvent in various processing and industrial applications. It's use as an anesthetic is relatively uncommon these days. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chloromethane: Chloromethane is a chemical used mainly in the production of silicones as well as agricultural chemicals, butyl rubber and other products. Ingestion and other exposures to the chemical can cause various symptoms. The chemical is readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Cresols: Cresols are a group of chemicals that occur naturally in mammals and various plants. It is also manufactured and used in the production of disinfectants, deodorizers and pesticides. Ingestion and other exposures to the chemical can cause various symptoms. The chemical is readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Cresylic acid: Cresylic acids are a group of chemicals that are used as solvents and in the manufacture of various products such as deodorants, disinfectants, pesticides, glues, paints, herbicides, pharmaceuticals as well as others. Ingestion and other exposures to the chemical can cause various symptoms. The chemical is readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - DDD: DDD is a chemical used mainly as a pesticide. Ingestion and other exposures to the chemical can cause various symptoms. The chemical may be readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - DDT: DDT is a chemical used mainly as a pesticide. Ingestion and other exposures to the chemical can cause various symptoms. The chemical may be readily absorbed through the skin. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Diethylene Glycol: Diethylene Glycol is a chemical used mainly in coolants, manufacture of plastic products and resins as well as other uses. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Dimethylnitrosamine: Dimethylnitrosamine is a chemical used mainly as a solving in the manufacture of plastics, rubbers, lubricants and rocket fuel. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Dinitrocresol: Dinitrocresol is a chemical used mainly as a herbicide and fungicide. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Dinitrophenol: Dinitrophenol is a chemical that has various applications: herbicide, pesticide, fungicide, acaricide, manufacture of dyes and wood preservative. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Diquat Dibromide: Diquat Dibromide is a chemical used mainly in herbicides. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Disulfiram: Disulfiram is a drug used mainly to manage alcoholism. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Ethylamine: Ethylamine is a chemical used mainly in the manufacture of dyes, rayon, rocket propellant, as a fuel additive and in leather-tanning and cellulose treatment. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Ethylene Dichloride: Ethylene Dichloride is a chemical used mainly in fat solvents and as a fumigant. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Ethyleneamine: Ethyleneamine is a chemical which is widely used in the manufacture of products such as adhesive, paper, textiles, fuels, lubricants, varnishes, lacquers, coating resins, cosmetics, photographic chemicals and agricultural chemicals. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Ethylenediamine: Ethylenediamine is a chemical used mainly as a solvent in the manufacturing process for the production of things such as fungicides, waxes, gasoline additives and pharmaceuticals. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Gasoline: Gasoline is a chemical used as a fuel for combustion engines. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Glaze: Glazes are used to put a shiny finish on various surfaces such as pottery. Glazes contain chemicals such as lead and zinc oxide which can cause serious symptoms if sufficient quantities are eaten. The chemicals cause damage to the gastrointestinal lining and the damage may continue for weeks after the poison was ingested. Death can result in severe cases. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Hexabromobiphenyl: Hexabromobiphenyl is a chemical used mainly in fire retardant in thermoplastics found in such things as electrical equipment, car upholstery and motor housing.. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Hexachlorobenzene: Hexachlorobenzene is a chemical used mainly in seed treatments. The chemical may be absorbed through the skin. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Imazapyr: Imazapyr is a chemical used mainly in herbicides. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Jet Fuel-4: Jet Fuel-4 is an aviation turbine fuel used by the US military. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Methylene Dianiline: Methylene Dianiline is a chemical used mainly in corrosive inhibitors, epoxy resins and polyurethane. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Mirex: Mirex is a chemical used mainly to control fire ants but also other insecticides such as mealy bugs. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Morpholine: Morpholine is a chemical used in a variety of applications: rubber industry, corrosion inhibitor, pharmaceuticals, dyes, crop pesticides and as a solvent in various manufacturing processes. The chemical may be absorbed through the skin. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Naphthalene: Naphthalene is a chemical used mainly as a moth repellant, toilet deodorizer and the manufacture of other chemicals. The chemical may be absorbed through the skin. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Para-Dichlorobenzene: Para-Dichlorobenzene is a chemical used mainly as a pesticide, mold and mildew preventer, moth repellent and toilet deodorant. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Pentachlorophenol: Pentachlorophenol is a chemical used mainly in fungicides, herbicides, insecticides, molluscicides, algicides and bactericides. It is commonly used as a wood preservative. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Phenol: Phenol is a chemical used mainly in the production of fertilizer, explosives, rubber, paint, paint remover, perfumes, asbestos products, wood preservatives, resins, textiles, pharmaceuticals and drugs. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Phosphine: Phosphine is a chemical used mainly in pesticides and rodenticides. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Plutonium: Plutonium is metallic element used mainly in the production of nuclear weapons. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Polychlorinated Biphenyls: Polychlorinated Biphenyls are a group of chemicals which had a variety of applications but are now banned due to the fact that they don't degrade in the environment and tend to build up in the food chain where they can cause harmful effects. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Polychlorinated Dibenzofurans: Polychlorinated Dibenzofurans are a group of chemicals that are usually formed as a byproduct of various industrial processes. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Pyrimidifen: Pyrimidifen is a chemical used mainly as an insecticide and acaricide. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Sodium Monofluoroacetate: Sodium Monofluoroacetate is a chemical used mainly as a rodenticides, often to control mammal pests in crops. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Solder: Solder contains various chemicals and heavy metals which can cause serious symptoms if sufficient quantities are swallowed. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Tetrachloroethylene: Tetrachloroethylene is a chemical used mainly as a fabric dry cleaner, degreaser, worming treatment for animals and in the manufacture of freons. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Toluene: Toluene is a chemical used mainly in pesticides, degreasers, glues and pain removers. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Toxaphene: Toxaphene is a chemical used mainly as a livestock insecticide. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Trichloroethylene: Trichloroethylene is a chemical used mainly as an industrial solvent and in adhesives, lacquer, fire retardants and house cleaning solvents. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Vinyl Choride: Vinyl Choride is an intermediate chemical used in the manufacturing process of a variety of products: polyvinyl chloride, pipes, wire coverings, vehicle plastics, rubber, paper, furniture and glass. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - White Phosphorus: White Phosphorus is a chemical used mainly in fertilizers, water treatment, rodenticides and insecticides (for cockroaches). Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Childhood liver cancer, primary: Cancer that develops in the tissue of the liver in children.
• Chloroleukemia: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Chloroma: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Chloromyeloma: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Cholangitis: bile duct inflammation (cholangitis)
• Cholecystitis: inflammation of the gall bladder.
• Choledochal cyst, hand malformation: A rare disorder characterized by the dilation of a portion of the biliary system and hand malformation.
• Cholestasis: A condition where bile formation or flow is reduced.
• Cholestasis - lymphoedema, syndrome: A rare inherited birth abnormality involving underdeveloped lymph vessels which results in swollen legs and liver problems.
• Cholestasis - pigmentary retinopathy - cleft palate: A rare syndrome characterized by degeneration of retinal pigments, cleft lip, cleft palate, kidney problems and cholestasis (bile flow obstruction).
• Cholestasis, Intrahepatic: A liver disorder where the bile is unable to flow properly through the liver. The severity of the condition is variable depending on the cause of the condition.
• Cholestasis, progressive familial intrahepatic 1: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage.
• Cholestasis, progressive familial intrahepatic 2: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage. The condition has an early onset and usually leads to end-stage liver disease by the end of the second decade. The various types of this condition differ in the origin of the genetic defect (liver-specific ATP-binding cassette transporter on chromosome 2q24). Type 2 is also associated with an increased risk of liver cancer in the first few years of life.
• Cholestasis, progressive familial intrahepatic 3: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage. The condition has an early onset and usually leads to end-stage liver disease by the end of the second decade. The various types of this condition differ in the origin of the genetic defect (liver-specific ATP-binding cassette transporter on chromosome 2q24). Type 2 is also associated with an increased risk of liver cancer in the first few years of life.
• Cholestatic jaundice -renal tubular insufficiency: A very rare syndrome characterized by liver and kidney problems.
• Choriocarcinoma, infantile: A rare malignant cancer that originates in the placenta. It is an aggressive cancer which generally results in infant death.
• Chromosome 11, deletion 11p: A syndrome that is caused by the deletion of short arm (p) of chromosome 11. The type and severity of symptoms varies depending on the amount and exact location of the genetic material that is deleted.
• Chromosome 18, trisomy 18q: A rare chromosomal disorder involving an extra copy of genetic material from the long arm of chromosome 18. The type and severity of symptoms are determined by the amount and location of the duplicated genetic material.
• Chronic Granulomatous Disease: A very rare inherited blood disorder where certain cells involved with immunity (phagocytes) are unable to destroy bacteria and hence the patient suffers repeated bacterial infections.
• Chronic Idiopathic Myelofibrosis: The bone marrow is consists of tissues which make blood cells and fibrous tissue supports these tissues that make the blood cells. In chronic idiopathic myelofibrosis, abnormal cells and fibres build up inside the bone marrow resulting in the production of fewer normal blood cells.
• Chronic Kidney Disease: Long-term and generally irreversible disease of the kidneys due to infection, obstruction, congenital diseases or generalised diseases causing failure of the kidneys' normal functions.
• Chronic Myeloproliferative Disease, Unclassified: A form of blood disorder characterized by the abnormal proliferation of myeloid precursors in the bone marrow. This category refers to cases of myeloproliferative disease which don't fit into any of the other specific type of myelproliferative diseases.
• Chronic Myeloproliferative Disorders: A group of blood cancers where excessive numbers of blood cells are made by overactive or cancerous bone marrow. The number of excess blood cells tends to grow slowly. Examples of such disorders includes chronic myelogenous leukemia, polycythemia vera and essential thrombocythemia. The symptoms are determined by which particular blood cancer is involved.
• Chronic Neutrophilic Leukemia: A rare form of leukemia characterized by excessive levels of mature neutrophils.
• Chronic Pesticide poisoning - xylene: Xylene is an ingredient used in certain insecticides. Exposure to the chemical can cause a range of symptoms depending on the level and route of exposure. Exposure can occur through inhalation, ingestion, the skin or eyes. Acute exposure involves a exposure over a short period of time whereas chronic exposure occurs over a longer period of time.
• Chronic adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The acute form tends to progress relatively slowly and generally responds better to treatment than the other subtypes.
• Chronic fatigue syndrome: A persistent debilitating fatigue of recent onset
• Chronic lymphocytic leukemia: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body.
• Chronic myelogenous leukemia: A slow-growing cancer of the white blood cells where the bone marrow makes too many white blood cells which eventually invade various parts of the body.
• Chronic myelomonocytic leukemia: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of certain blood cells - myelocytes and monocytes. The proliferation is slower than in acute forms of the disease.
• Chronic vitamin A toxicity: Chronic excessive ingestion of vitamin A can cause symptoms.
• Chédiak-Higashi syndrome: An inherited immune system disorder characterized by reduced pigmentation, recurrent infection and neurological disorders.
• Cirrhosis, familial: Liver cirrhosis that is inherited in a familial pattern. The liver scarring (cirrhosis) is not caused by any discernable disease process. The liver becomes progressively scarred and its function is impaired.
• Classic galactosemia: Rare serious genetic defect in galactose metabolism.
• Cocaine overdose: Cocaine is an illegal and highly addictive recreational drug. Excessive doses of the drug can result in various symptoms and even death in severe cases.
• Coenzyme Q cytochrome c reductase deficiency of: A rare genetic defect where an enzyme deficiency (CoQ-Cytochrome C reductase) disrupts cellular processes. Any of a variety of the components of the enzyme may be missing or defective and hence the clinical presentation and severity may vary. The deficiency may result in a variety of symptoms and conditions of variable severity such as cardiomyopathy, fatal infant conditions and Leber's myopathy.
• Cold Autoimmune Hemolytic Anemia: Cold autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The red cells are destroyed at an abnormally rapid rate which leads to anemia. Cold haemolytic anemia is characterized by the fact that the abnormal destruction of red blood cells is more active when the patient is exposed to cold temperatures. The severity of the condition varies depending on the underlying cause. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Cold antibody hemolytic anemia: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 30ºC or lower.
• Colorectal cancer: Cancer of the colon (bowel) or rectum.
• Common Variable Immunodeficiency: An immunodeficiency disorder involving low blood gamma globulin levels which results in an increased susceptibility to infections. The condition may be inherited or can be caused by certain drugs (levamisole, hydantoin and carbamazepine).
• Complex 1 mitochondrial respiratory chain deficiency: A rare genetic defect where an enzyme deficiency (NADH CoQ) disrupts cellular processes and causes various organic acid disorders. Any of a variety of the components of the enzyme may be missing or defective and hence the clinical presentation and severity may vary. Presentation may range from infantile death to various disorders such as Leigh's disease, Parkinson's disease and cardiomyopathy.
• Complex 5 mitochondrial respiratory chain deficiency: A rare genetic defect where an enzyme deficiency (ATP synthetase) disrupts cellular processes. Any of a variety of the components of the enzyme may be missing or defective and hence the clinical presentation and severity may vary. The deficiency may result in a variety of symptoms and conditions of variable severity such as Leber's myopathy, Leigh syndrome, cardiomyopathy and NARP (neuropathy, ataxia, retinitis pigmentosa).
• Congenital Disorders of Glycosylation: Congenital disorders of glycosylation is a group of disorders involving abnormally synthesis of N-linked oligosaccharides. There is a long chain of events involved in the synthesis and defects may occur with any of the compounds or enzymes involved in the process. Progressive impairment and regression of skills often occurs after a period of normal development following birth.
• Congenital Disorders of Glycosylation Type Ia: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
• Congenital Toxoplasmosis: Fetal infection with toxoplasmosis.
• Congenital cytomegalovirus: Fetal infection with cytomegalovirus.
• Congenital disorder of glycosylation type 1/IIX: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type I/IIX refers to cases where the specific abnormality cannot be determined.
• Congenital disorder of glycosylation type 1A: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
• Congenital disorder of glycosylation type 1B: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1B has a phosphomannose isomerase enzyme defect.
• Congenital disorder of glycosylation type 1E: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1E has a Dol-P-Man synthase enzyme defect.
• Congenital disorder of glycosylation type 1H: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ih is caused by a defect on chromosome 11pter-p15.5 and involves the gene for a particular enzyme (dolichyl-P-glucose:Glc-1-Man-9-GlcNAc-2-PP-dolichyl-alpha-3-glucosyltransferase).
• Congenital disorder of glycosylation type 1I: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ii is caused by a defect on chromosome 9q22 and involves a defect on the ALG2 gene.
• Congenital disorder of glycosylation type 1K: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ik is caused by a defect on chromosome 16p13.3 and involves a defect in the gene for beta-1,4-mannosyltransferase. The disorder is generally fatal within a year or two of birth.
• Congenital disorder of glycosylation type 1L: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Il is caused by a defect on chromosome 11q23 and involves a defect in the ALG9 gene.
• Congenital disorder of glycosylation type 1X: Congenital disorder of glycosylation is a rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1X also involves thrombocytopenia with normal levels of phosphomannomutase and phosphomannose isomerase. This form of the condition is severe and results in death during infancy.
• Congenital disorder of glycosylation type 2A: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 2A has a GlcNAc transferase 2 enzyme defect.
• Congenital disorder of glycosylation type 2B: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 2B has glucosidase I enzyme defect.
• Congenital disorder of glycosylation type 2E: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 2e is caused by a defect on chromosome 16p and involves a defect in the gene for oligomeric complex-7.
• Congenital disorder of glycosylation type 2G: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type IIg is caused by a defect on chromosome 17q25.1 and involves a defect on the COG1 gene.
• Congenital disorder of glycosylation, type In: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1n involves a defect in the RFT1 gene and is characterized mainly by enlarged liver, seizures, developmental delay, reduced muscle tone and abnormal blood coagulation.
• Congenital hepatic fibrosis: A rare inherited birth disorder characterized by fibrosis (scarring) of the liver which affects its ability to function. The severity of the condition is variable with some patients being symptomatic during infancy while others may be asymptomatic for most of their life.
• Congenital hepatic porphyria: A rare congenital disorder where there is an excess of porphyrin (pigments) in the body. The liver is responsible for making porpyrins.
• Congenital herpes simplex: An infant born with a herpes simplex infection transmitted through the mother. The infection may be localized or involve various internal organs and even the central nervous system in which case death can occur.
• Congenital malaria: Fetal infection with malaria.
• Congenital nonhemolytic jaundice: A rare disorder where the liver is unable to remove bilirubin from the body which results in jaundice. Chronically high bilirubin levels can lead to neurological toxicity which manifests as progressive neurological symptoms.
• Congenital spherocytic hemolytic anemia: A blood disorder present at birth where the membranes of red blood cells are defective which results in them being spherical rather than doughnut-shaped. These abnormally shaped red blood cells are broken down prematurely by the body which results in anemia. The condition may range from mild to severe.
• Congenital tuberculosis: Fetal infection with tuberculosis
• Conor's disease: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Constrictive pericarditis: Inflammation, swelling and thickening of the pericardium (fibrous sac surrounding the heart) which causes it to tighten around the heart and affect its function. The condition may be misdiagnosed as a heart attack and vice versa.
• Constrictive tuberculous pericarditis: Inflammation and swelling of the pericardium (fibrous sac surrounding the heart) that occurs as a complication of tuberculosis. The condition may be misdiagnosed as a heart attack and vice versa.
• Cooley syndrome: An inherited blood disorder involving abnormal production of red blood cells which causes serious anemia. It is a severe form of thalassemia.
• Copper poisoning: A type of heavy metal poisoning caused by excessive exposure to copper.
• Copper toxicity: Excessive accumulation of copper in the body can cause symptoms.
• Cor pulmonale: Right ventricular enlargement and failure caused by pulmonary hypertension.
• Cortes-Lacassie syndrome: A rare syndrome characterized by nail, hair and teeth abnormalities, malformed hands and feet and seizures. The disorder has only been reported in one cause which resulted in death at 31 months
• Crigler-Najjar Syndrome: An autosomal recessive form of nonhemolytic jaundice due to the absence of the hepatic enzyme glucuronide transferase.
• Crigler-Najjar syndrome, type 1: A rare congenital condition involving a total absence of the liver enzyme called glucoronyl transferase which is needed to change bilirubin into a form that can be removed from the body. The bilirubin builds up in the body and causes damage and severe symptoms.
• Crigler-Najjar syndrome, type 2: A rare congenital condition involving a partial absence of the liver enzyme called glucoronyl transferase which is needed to change bilirubin into a form that can be removed from the body. The bilirubin builds up in the body and causes damage and symptoms but to a lesser extent than in type 1.
• Crohn's disease: Crohn's disease causes inflammation of the digestive system. It is one of a group of diseases called inflammatory bowel disease. The disease can affect any area from the mouth to the anus. It often affects the lower part of the small intestine called the ileum.
• Cryoglobulinemia: These are immune cells that precipitate in the cold and redissolve on warming.
• Crystal meth addiction: An uncontrollable desire to use crystal meth on a regular basis. Crystal meth is a powerful stimulant used illegally for its effects. It is highly addictive and known by street names such as ice, speed, glass, crank and chalk. Frequent use leads to an increased tolerance to the drug so higher and higher doses are required to achieve the desired euphoric feeling.
• Cycad nut poisoning: The cycad nut contains a toxic chemical called cyasin which can be leeched out by soaking in water. The nuts are often used as a food source but it is important to leech out the toxic chemicals first. Eating nuts that still contain the toxin can cause serious symptoms. Cycad nuts may also increase the risk of developing liver cancer.
• Cycad poisoning: Cycads are a green plant which has a thick trunk from the top of which sprouts palm-like leaves. Eating the seeds, leaves or unprocessed flour made from the trunk of the plant can cause various symptoms if large quantities are eaten. The harmful compounds in the plant are glycosides and BMAA
• Cyclic neutropenia: A rare blood disorder characterized by recurrent periods of extremely low blood levels of neutrophils which results in frequent infections. The low level usually occurs for about 7 days every 21 days. Levels of other blood components may also be affected.
• Cystic Fibrosis: Cystic fibrosis is a hereditary disease affecting the exocrine (mucus) glands of the lungs, liver, pancreas, and intestines, causing progressive disability due to multisystem failure.
• Cytochrome C Oxidase Deficiency: Cytochrome C oxidase deficiency is a rare inherited condition involving insufficient quantities of the cytochromc C oxidase enzyme. This enzyme plays a role in the functioning of the energy producing part of body cells (mitochondria) and its deficiency impairs the energy-producing functions of the cells. The type and severity of symptoms can vary considerably depending on which particular cells in the body are affected and the degree of the enzyme deficiency. In some cases only skeletal muscles are affected whereas in other cases organs such as the heart and brain are involved. In other cases, the whole body may be involved.
• Cytomegalic Inclusion Body Disease: An infection due to cytomegalovirus and marked by nuclear inclusion bodies in enlarged infected cells
• Cytomegalovirus: A easily transmissible viral infection that is common but generally causes no symptoms except in infants and people with weakened immune systems.
• DOC 12 (Neutral Lipid Storage Type): A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities. The body is unable to metabolize (break down) triglycerides so they build up in various tissues throughout the body. The severity of the symptoms is variable as the body tissues and organs affected varies amongst patients.
• Darvocet overdose: Darvocet is a prescription drug mainly used to treat pain. Excessive doses of the drug can result in various symptoms and even death in severe cases.
• Deafness - lymphoedema - leukemia: A rare syndrome characterized by deafness, early-onset leukemia and lymphoedema in the lower legs.
• Deafness peripheral - neuropathy - arterial disease: A very rare syndrome characterized mainly by artery disease, deafness and peripheral neuropathy.
• Deal-Barratt-Dillon syndrome: A very rare syndrome characterized mainly by scaly skin, jaundice, diarrhea and Fanconi syndrome (bone marrow fails to make sufficient new blood cells).
• Debler anemia: A familial type of anemia that starts during infancy and involves the premature destruction of red blood cells.
• Defect in synthesis of adenosylcobalamin: A rare genetic disorder characterized by the impaired ability to make a chemical called adenosylcobalamin. Adenosylcobalamin is a derivative of vitamin B12. The defect results a biochemical abnormality which affects the body's normal biochemical functioning.
• Deletion 11p: A syndrome that is caused by the deletion of short arm (p) of chromosome 11. The type and severity of symptoms varies depending on the amount and exact location of the genetic material that is deleted.
• Devergie syndrome: A chronic skin disorder involving the development of raised spots on the skin that grow and merge into each other to produce large red scaling plaques.
• Di Guglielmo I - acute: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The acute form has more severe symptoms than the chronic form.
• Di Guglielmo I - chronic: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The chronic form has more severe symptoms than the acute form.
• Diabetes, neonatal - congenital hypothyroidism - congenital glaucoma - hepatic fibrosis - polycystic kidneys: A rare syndrome characterized by the presence of diabetes, hypothyroidism, liver fibrosis, polycystic kidneys and glaucoma in infants.
• Diamond-Blackfan anemia: Diamond-Blackfan anemia is a rare genetic condition where the bone marrow is unable to make sufficient red blood cells which leads low levels of red blood cells. There are eight subtypes of the condition which differ in the location of the genetic defect and the incidence of additional symptoms such as malformations. The severity of symptoms is variable but most cases are serious.
• Dibasic aminoaciduria 2: A rare condition where protein intolerance occurs as a result of a defect in the transport of dibasic amino acids through the intestines and kidneys. The amino acids (component of protein) can't be broken down properly and used by the body so it builds up and causes damage.
• Diffuse neonatal hemangiomatosis: A very rare syndrome characterized mainly by multiple capillary hemangiomas as well as other abnormalities.
• Digestive symptoms: Any symptoms affecting the digestive tract.
• Disorder of Cornification 12 (Neutral Lipid Storage Type): A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities. The body is unable to metabolize (break down) triglycerides so they build up in various tissues throughout the body. The severity of the symptoms is variable as the body tissues and organs affected varies amongst patients.
• Distomatosis: Infection by parasitic flat worms. Infection can involve liver, lungs or intestines. Symptoms are determined by the location of the infection. Contamination usually occurs through ingesting contaminated food or water.
• Doxepine-induced Immune Hemolytic Anemia: Doxepine -induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Dressler (D.)syndrome: A rare autoimmune blood disorder where erythrocytes are destroyed suddenly after exposure to cold (usually 15ºC or lower).
• Drug-induced liver damage - Clindamycin: Damage or injury to the liver caused by exposure to Clindamycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Quinolone: Damage or injury to the liver caused by exposure to Quinolone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Spectinomycin: Damage or injury to the liver caused by exposure to Spectinomycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Sulfones: Damage or injury to the liver caused by exposure to sulfone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced Immune Hemolytic Anemia: Drug-induced immune hemolytic anemia is a condition where a medication triggers the body's immune system to destroy its own red blood cells which results in anemia. Certain drugs are more likely to trigger this abnormal immune response than others e.g. cephalosphorins and Quinidine.
• Drug-induced liver damage - 5-Fluorocytosine: Damage or injury to the liver caused by exposure to an antifungal agent called 5-Fluorocytosine. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Allopurinol: Damage or injury to the liver caused by exposure to allopurinol. Allopurinol is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Amphotericin: Damage or injury to the liver caused by exposure to an antifungal agent called Amphotericin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Anabolic C-17: Damage or injury to the liver caused by exposure to Anabolic C-17 which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Anesthetic agent: Damage or injury to the liver caused by exposure to anesthetic agents. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antianginal agents: Damage or injury to the liver caused by exposure to antianginal agents. Antianginal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antiarrhythmics: Damage or injury to the liver caused by exposure to antiarrhythmics. Antiarrhythmics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antibiotics: Damage or injury to the liver caused by exposure to certain antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Anticoagulants: Damage or injury to the liver caused by exposure to anticoagulants. Anticoagulants are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antifungals: Damage or injury to the liver caused by exposure to antifungal agents. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antihyperlipidemic agents: Damage or injury to the liver caused by exposure to antihyperlipidemic agents. Antihyperlipidemic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antihypertensives: Damage or injury to the liver caused by exposure to antihypertensives. Antihypertensives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antineoplastic agents: Damage or injury to the liver caused by exposure to antineoplastic agents. Antineoplastic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antithyroid drugs: Damage or injury to the liver caused by exposure to endocrine agents called antithyroid drugs. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Benzodiazepine: Damage or injury to the liver caused by exposure to a psychotropic agent called benzodiazepine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - British anti-Lewisite penicillamine: Damage or injury to the liver caused by exposure to British anti-Lewisite penicillamine. British anti-Lewisite penicillamine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Butyrophenone: Damage or injury to the liver caused by exposure to a psychotropic agent called butyrophenone. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cephalosporin: Damage or injury to the liver caused by exposure to Cephalosporin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Chloramphenicol: Damage or injury to the liver caused by exposure to Chloramphenicol. Chloramphenicol is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Chloroform: Damage or injury to the liver caused by exposure to an anesthetic agent called chloroform. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cimetidine: Damage or injury to the liver caused by exposure to Cimetidine. Cimetidine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Colchicine: Damage or injury to the liver caused by exposure to colchicine. Colchicine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cyclopropane: Damage or injury to the liver caused by exposure to an anesthetic agent called cyclopropane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cycloserine: Damage or injury to the liver caused by exposure to an antituberculous agent called cycloserine. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cytarabine: Damage or injury to the liver caused by exposure to an antiviral agent called cytarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Dantrolene: Damage or injury to the liver caused by exposure to Dantrolene. Dantrolene is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Diflunisal: Damage or injury to the liver caused by exposure to diflunisal. Diflunisal is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Disulfiram: Damage or injury to the liver caused by exposure to Disulfiram. Disulfiram is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Diuretic Agents: Damage or injury to the liver caused by exposure to diuretic agents. Diuretic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Erythromycin estolate: Damage or injury to the liver caused by exposure to an antibiotic called erythromycin estolate. Erythromycin estolate is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Erythromycin ethyl succinate: Damage or injury to the liver caused by exposure to an antibiotic called Erythromycin ethyl succinate. Erythromycin ethyl succinate is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Ethionamide: Damage or injury to the liver caused by exposure to an antituberculous agent called ethionamide. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Fenoprofen: Damage or injury to the liver caused by exposure to fenoprofen. Fenoprofen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Glucocorticoids: Damage or injury to the liver caused by exposure to endocrine agents called glucocorticoids. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Griseofulvin: Damage or injury to the liver caused by exposure to an antifungal agent called Griseofulvin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Halothane: Damage or injury to the liver caused by exposure to an anesthetic agent called halothane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Ibuprofen: Damage or injury to the liver caused by exposure to ibuprofen. Ibuprofen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Indomethacin: Damage or injury to the liver caused by exposure to indomethacin. Indomethacin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Iodide ion: Damage or injury to the liver caused by exposure to Iodide ion. Iodide ion is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Isoniazid: Damage or injury to the liver caused by exposure to an antituberculous agent called isoniazid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Ketoconazole: Damage or injury to the liver caused by exposure to an antifungal agent called Ketoconazole. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Mephenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called mephenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Methoxyflurane: Damage or injury to the liver caused by exposure to an anesthetic agent called methoxyflurane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Naproxen: Damage or injury to the liver caused by exposure to naproxen. Naproxen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Nitrofuran: Damage or injury to the liver caused by exposure to Nitrofuran antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Nitrous Oxide: Damage or injury to the liver caused by exposure to an anesthetic agent called nitrous oxide. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Novobiocin: Damage or injury to the liver caused by exposure to Novobiocin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Oral hypoglycemics: Damage or injury to the liver caused by exposure to oral hypoglycemics which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Penicillin: Damage or injury to the liver caused by exposure to an antibiotic called penicillin. Penicillin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Phenobarbital: Damage or injury to the liver caused by exposure to an anticonvulsive called phenobarbital. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Phenothiazines: Damage or injury to the liver caused by exposure to a psychotropic agent called phenothiazine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Phenylbutazone: Damage or injury to the liver caused by exposure to phenylbutazone. Phenylbutazone is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Phenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called Phenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Ranitidine: Damage or injury to the liver caused by exposure to Ranitidine. Ranitidine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Rifampicin: Damage or injury to the liver caused by exposure to an antituberculous agent called rifampicin. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Salicylate: Damage or injury to the liver caused by exposure to salicylates. Salicylates are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Saramycetin: Damage or injury to the liver caused by exposure to an antifungal agent called Saramycetin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Steroids: Damage or injury to the liver caused by exposure to steroids which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Sulfonamide: Damage or injury to the liver caused by exposure to Sulfonamide antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Sulindac: Damage or injury to the liver caused by exposure to sulindac. Sulindac is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Tamoxifen: Damage or injury to the liver caused by exposure to Tamoxifen which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Telithromycin: Damage or injury to the liver caused by exposure to an antibiotic called Telithromycin. Telithromycin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Tetracycline: Damage or injury to the liver caused by exposure to Tetracycline antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Thioxanthene: Damage or injury to the liver caused by exposure to a psychotropic agent called Thioxanthene. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Thorotrast: Damage or injury to the liver caused by exposure to Thorotrast. Thorotrast is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Valproic Acid: Damage or injury to the liver caused by exposure to an anticonvulsive called valproic acid. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Vidarabine: Damage or injury to the liver caused by exposure to an antiviral agent called vidarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Vitamin A: Damage or injury to the liver caused by exposure to Vitamin A. Vitamin A is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Zoxazolamine: Damage or injury to the liver caused by exposure to Zoxazolamine. Zoxazolamine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - anticonvulsives: Damage or injury to the liver caused by exposure to certain anticonvulsives. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - antituberculous agents: Damage or injury to the liver caused by exposure to antituberculous agents. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - antiviral medication: Damage or injury to the liver caused by exposure to antiviral agents. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - endocrine agent: Damage or injury to the liver caused by exposure to endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - idoxuridine: Damage or injury to the liver caused by exposure to an antiviral agent called idoxuridine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - monoamine oxidase inhibitors: Damage or injury to the liver caused by exposure to psychotropic agents called monoamine oxidase inhibitors. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - p-aminosalicylic acid: Damage or injury to the liver caused by exposure to an antituberculous agent called p-aminosalicylic acid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - psychotropic agents: Damage or injury to the liver caused by exposure to certain psychotropic agents. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - tricyclic antidepressant: Damage or injury to the liver caused by exposure to a psychotropic agent called tricyclic antidepressant. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - xenylamine: Damage or injury to the liver caused by exposure to an antiviral agent called xenylamine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver disease: Drug-induced liver diseases are diseases of the liver that are caused by physician-prescribed medications, over-the-counter medications, vitamins, hormones, herbs, illicit ("recreational") drugs, and environmental toxins.
• Drugs-induced liver damage - Ether: Damage or injury to the liver caused by exposure to an anesthetic agent called ether. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Dubin-Johnson Syndrome: Rare, chronic hyperbilirubinemia caused by the liver's inability to secrete certain organic compounds.
• Dupont-Sellier-Chocillon Syndrome: A rare condition characterized by lipomatosis of the pancreas and narrowing of the duodenum.
• Dyck Syndrome: A very rare condition observe in two brothers. It involves vision, hearing, muscle, sensory and adrenal system problems and an enlarged liver and spleen.
• Dykes-Markes-Harper syndrome: A very rare syndrome characterized mainly by dry, scaly skin, enlarged liver and spleen and a incoordination.
• Dyserythropoietic anemia, congenital: A congenital blood condition where the production of red blood cells is defective. The abnormal red blood cells are often destroyed prematurely within the body which can result in anemia. There are three subtypes of the disorder, each with varying severity of symptoms.
• Dyserythropoietic anemia, congenital type 1: A very rare genetic blood disorder where abnormal red blood cells are made resulting in anemia. Finger and toe abnormalities are also usually present.
• Dyserythropoietic anemia, congenital type 2: A very rare genetic blood disorder where abnormal red blood cells are made resulting in anemia.
• Dyserythropoietic anemia, congenital type 3: An extremely rare blood disorder where abnormal red blood cells are made resulting in anemia.
• Dysplastic cortical hyperostosis: A very rare syndrome characterized mainly by abnormal bone and brain development.
• Eccentrochondrodysplasia: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of certain enzymes needed to break it down.
• Eclampsia: serious complication of pregnancy and is characterised by high blood pressure and convulsions
• Ecstasy addiction: An uncontrollable desire to use ecstasy on a regular basis. Chronic ecstasy use can lead to dependency in as little as two weeks. Ecstasy is a synthetic psychoactive drug often used as a recreational drug. Street names for the drug includes: XTC, Adam, Clarity, Lover's Speed, Hug, Beans and Love Drug. Frequent use leads to an increased tolerance to the drug so higher and higher doses are required to achieve the desired euphoric feeling.
• Ectodermal dysplasia, anhidrotic with T-cell immunodeficiency, autosomal dominant: An inherited disorder characterized by dry, rough skin, sparse scalp hair, cone-shaped teeth and an immune system disorder.
• Elejalde syndrome: A rare congenital syndrome characterized excessive fetal growth, skull abnormalities, extra fingers or toes and other abnormalities.
• Emanuel syndrome: A rare chromosomal disorder where a portion of chromosome 11 is translocated with a portion of chromosome 22. The disorder involves a wide range of abnormalities such as kidney, genital and heart abnormalities, mental retardation, small head and failure to thrive.
• Encephalopathy due to GLUT1 deficiency: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
• End Stage Liver Failure: Late stage of liver failure characterised by the onset of mental and neurological symptoms, due to build up of toxic metabolites.
• Endocardial fibroelastosis: A rare heart disorder characterized by a thickening of the heart muscle lining causing heart enlargement and heart failure.
• Endocrine agent-induced liver damage: Damage or injury to the liver caused by exposure to endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Anabolic C-17: Damage or injury to the liver caused by exposure to Anabolic C-17 which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Antithyroid drugs: Damage or injury to the liver caused by exposure to endocrine agents called antithyroid drugs. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Glucocorticoids: Damage or injury to the liver caused by exposure to endocrine agents called glucocorticoids. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Oral contraceptives: Damage or injury to the liver caused by exposure to oral contraceptives which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Oral hypoglycemics: Damage or injury to the liver caused by exposure to oral hypoglycemics which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Steroids: Damage or injury to the liver caused by exposure to steroids which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Tamoxifen: Damage or injury to the liver caused by exposure to Tamoxifen which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine pancreatic cancer: Any disease that affects the endocrine pancreas
• Endometrial Cancer: Cancer of the endometrium (uterus lining).
• Endomyocardial fibrosis: Heart disease characterized by progressive fibrosis of the lining of one or both lower heart cavities. This results in constriction of the heart cavity and may involve the heart valves and other structures.
• Enlarged liver: Swelling of the liver.
• Enlarged liver and spleen: Enlargement of both liver and spleen (hepatosplenomegaly)
• Enterovirus antenatal infection: Fetal infection with enterovirus. The condition is extremely rare but infection around the time of birth often results in death or paralysis in survivors. The type and severity of symptoms is determined by the exact type of virus involved and at what stage of development the infection occurs.
• Epstein-Barr virus, chronic: A form of human herpes virus that produces persistent symptoms. Most people become infected with the virus at some stage in their life though they usually have few if any symptoms. However, some people develop severe symptoms as a result of an EBV infection.
• Erdheim-Chester syndrome: A very rare lipid storage disorder involving lipid deposits in various organs and hardening of the ends of long bones which affects the growth of the bone. The severity of the condition is variable.
• Escharonodulaire: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Esophagus symptoms: Symptoms affecting the esophagus (digestive throat passage)
• Essential mixed cryoglobulinemia: A rare autoimmune disorder characterized by the presence of a mixture of various types of antibodies (crylglobulins) in the blood.
• Essential thrombocytosis - same as essential thrombocythemia: A rare blood disorder where the blood contains too many platelets due to excessive megakaryocytes (platelet-producing cells). Platelets are essential for blood clotting but in essential thrombocythemia excessive platelets can cause the blood to form abnormal clots. If the platelets are defective as well then bleeding problems can occur. The severity of the condition is variable.
• Eugenol oil poisoning: Eugenol oil is used as a supplement or as a therapeutic ingredient in various medications and foods but excessive doses of undiluted oil can cause symptoms. Smoking undiluted cloves in cigarettes can also cause symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Extramedullary Myeloid Tumor: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Familial Mediterranean fever: A rare inherited condition characterized by recurrent fever and inflammation. The inflammation usually involves the stomach, lungs or joints.
• Familial hyperchylomicronemia: A rare inherited inborn error of metabolism involving the absence of the enzyme called lipoprotein lipase which results in increased blood triglyeride and chylomicron levels.
• Familial hyperlipoproteinemia: A group of genetic disorder characterized by abnormal breakdown of lipoproteins which causes abnormal lipoprotein and lipid levels in the blood. There are various types of this condition: hyperlipoproteinemia type I, II, III, IV and V. The type and severity of symptoms vary between types. The disorder tends to run in families (familial).
• Familial hyperlipoproteinemia type 1: A genetic disorder characterized by abnormal lipid (chylomicrons and high triglyceride lipids) breakdown which results in its accumulation in the blood. The disorder is caused by the reduced or absent activity of the enzyme lipoprotein lipase. The severity of the condition is determined by the degree of the deficiency and treatment. The disorder tends to run in families (familial).
• Familial myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
• Fanconi-Bickel syndrome: A rare inherited disorder where the impaired metabolism of carbohydrates results in a build-up of glycogen in the liver.
• Fanconi-ichthyosis-dysmorphism: A very rare syndrome characterized by scaly skin (ichthyosis), anemia, muscle anomalies and various other abnormalities. All six reported cases died within 6 months.
• Farber's disease: A rare inherited biochemical disorder involving the deficiency of an enzyme called ceramidase resulting in the harmful accumulation of certain chemicals in the body which causes damage and inflammation.
• Fascioliasis: A rare parastitic infectious disease caused by liver fluke Fasciola hepatica which can cause blockage of the bile ducts in the liver.
• Fatty liver: A condition which is characterized by the accumulation fatty deposits in the liver
• Fatty liver disease: Fatty liver disease can range from fatty liver alone (steatosis) to fatty liver associated with inflammation (steatohepatitis). This condition can occur with the use of alcohol (alcohol-related fatty liver) or in the absence of alcohol (nonalcoholic fatty liver disease [NAFLD]).
• Favism: Form of hemolytic anemia
• Felty syndrome: A rare complication of long-term rheumatoid arthritis which involves an enlarged spleen and blood abnormalities.
• Female reproductive toxicity - Acetaminophen: Taking excessive doses of acetaminophen during pregnancy can result in kidney and liver problems in the baby. Pregnant women should avoid taking more than the recommended amount of the drug.
• Fever: Raised body temperature usually with other symptoms.
• Fitz-Hugh-Curtis syndrome: A rare condition where the thin layer around the liver becomes infected as a result of a spread of infection from gonococcal or chlamydial infections in females.
• Fluke infections: An infection caused by flukes
• Forbes disease: A rare inherited glycogen storage disease caused by a deficiency of the enzyme amylo-1,6-glucosidase resulting in a build up of glycogen in the liver and muscles.
• Fowl Paralysis: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Fowl paralyses: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Free sialic Acid storage disease: A rare inherited biochemical disorder characterized by the accumulation of sialic acid in the tissues and excretion of sialic acid in the urine. There are mild and severe forms of the condition - the severe form result in death before birth or within a few years of birth.
• Fructose intolerance: An enzyme deficiency that causes toxic symptoms when fructose containing food is ingested.
• Fructose-1,6-bisphosphatase deficiency, hereditary: A rare inherited condition where an enzyme deficiency (fructose-1,6-bisphosphatase deficiency - FDPase) impairs the body's ability to metabolize fructose from the diet.
• Fructose-1-phosphate aldolase deficiency, hereditary: An inherited metabolic disorder where deficiency of the enzyme fructose-1-phsophate aldolase prevents fructose being metabolized resulting in fructose intolerance.
• Fucosidosis type 1: A rare biochemical disorder involving deficiency of an enzyme (alpha-fucosidase) which results in accumulation of certain chemicals (glycosphingolipids) in the central nervous system and other body tissues. It is an infantile form of fucosidosis which starts early and rapidly progresses to early death.
• Fucosidosis type II: A form of the biochemical disorder called fucosidosis where an enzyme deficiency (alpha-fucosidase) results in the accumulation of certain chemicals (glycosphingolipids) in the central nervous system and other body tissues. Symptoms start later and progress slower than in type I and is distinguished by warty skin growths.
• GM1 gangliosidosis: A rare biochemical disorder involving a deficiency of an enzyme (beta-galactosidase A) which results in the accumulation of harmful chemicals (GM1 gangliosides) in the central nervous system and other body tissues. Type 1 is a severe infantile form of the disorder and involves a greater degree of accumulation than type II or III.
• Galactokinase deficiency: A rare condition where an enzyme deficiency (galactokinase) impaires the body's ability to break down galactose consumed in the diet.
• Galactosemia: Accumulation of galactose in the blood leading to mental retardation and failure to thrive if not corrected.
• Galactosemia I: A rare inherited disorder where deficiency of a particular enzyme (galactose-1-phosphate uridyl transferase) prevents the metabolism of galactose which is a sugar component of milk. Ranges from milk intolerance in mild cases to death in severe untreated cases.
• Galactosemia III: A rare inherited disorder where deficiency of a particular enzyme (UDP-Galactose-4-epimerase) prevents the metabolism of galactose which is a sugar component of milk. The condition may vary from mild to severe.
• Gall Bladder Cancer: Cancer of the gall bladder.
• Gall bladder conditions: Any condition that affects the gallbladder
• Gallstones: A condition where biliary calculi form in the gallbladder
• Game-Friedman-Paradice syndrome: A rare condition characterized by retarded growth, hydrocephaly, underdeveloped lungs and various other anomalies. The condition was observed in four offspring from one family.
• Gangliosidosis generalized GM1, type 1: A rare biochemical disorder involving a deficiency of an enzyme (beta-galactosidase A) which results in the accumulation of harmful chemicals (GM1 gangliosides) in the central nervous system and other body tissues. Type 1 is a severe infantile form of the disorder and involves a greater degree of accumulation than type II or III.
• Garret-Tripp syndrome: A rare syndrome characterized mainly by extra digits, a lack of hair and scaly skin on the scalp, face and neck.
• Gaucher Disease: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3.
• Gaucher disease - perinatal lethal form: A rare syndrome characterized by the association of abnormally tight skin and Gaucher disease which is a lipid storage disease. This is the most severe form of Gaucher disease.
• Gaucher disease type 1: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 1 is the visceral, chronic form which usually starts during adulthood.
• Gaucher disease type 2: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 2 is acute neurological form apparent in infancy.
• Gaucher disease type 3: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 3 is a subacute neurological form which often first appears in childhood.
• Gaucher-like disease: A very rare inherited condition where the body's inability to bread down a certain fat (glucocerebroside) causes it to accumulate in body tissues and organs where it can cause damage. Gaucher-like disease is a sub-type of Gaucher disease which primarily affects the heart.
• Geleophysic dwarfism: A rare genetic disorder characterized by a happy facial expression, short stature and limb abnormalities.
• Ghosal syndrome: A very rare syndrome characterized mainly by difficult to treat anemia and skeletal abnormalities.
• Glory lily poisoning: The glory lily is a type of vine which bears unusual yellow and red flowers. It is often used as an indoor and outdoor ornamental plant. The plant contains various chemicals that can cause serious symptoms if eaten. The roots are the most toxic part of the plant. The plant is considered very toxic and serious cases can result in death.
• Glucose-6-Phosphate Dehydrogenase Deficiency: A rare enzyme abnormality involving a deficiency of the glucose-6-phosphate dehydrogenase which causes premature destruction of red blood cells. The excessive destruction of red blood cells can be triggered by certain infections or drugs or by eating fava beans.
• Glutaric Aciduria, neonatal form of type II A: A more serious neonatal form of glutaricaciduria where there is excessive blood and urine levels of glutaric acid and congenital anomalies may be present.
• Glutaric aciduria 1: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
• Glutaric aciduria 2: A metabolic disorder involving an enzyme deficiency - electron transfer flavoprotein ubiquinone oxydoreductase. The severity of symptoms depends on the level of deficiency. The infant onset form is the most severe and often results in death. Severe cases usually develop during childhood or infancy and usually involve metabolic acidosis and its associated symptoms. Milder cases may simply present with muscle weakness initially that develops in adulthood. Some cases may involve additional symptoms such as heart, liver and kidney problems, facial anomalies and genital abnormalities.
• Glutaricaciduria 2B: A milder, later-onset form of glutaricaciduria where there is excessive blood and urine levels of glutaric acid due to the body's impaired ability to metabolize protein and fat into energy.
• Glutaricaciduria I: A rare inherited enzyme deficiency disorder where deficiency of the glutaryl-CoA dehydrogenase enzyme results in dystonia, dyskinesia and sometimes mental retardation.
• Glutaricaciduria type 1: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
• Glutaryl-CoA dehydrogenase deficiency: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
• Glycogen Storage Disease Type I: An inherited metabolic disorder where a deficiency of the enzyme glucose-6-phosphatase prevents glycogen being turned into glucose leading to a buildup of glycogen in the liver and kidneys. Most problems tend to develop during adulthood.
• Glycogen branching deficiency: A rare metabolic disorder where an enzyme deficiency (glycogen branching enzyme) results in a harmful buildup of glycogen byproducts in the liver, muscle and even the heart in some cases. The severity of symptoms is variable depending on the degree of enzyme deficiency and how strictly treatment measures are adhered to.
• Glycogen debranching deficiency: A rare metabolic disorder where an enzyme deficiency (amylo-1,6-glucosidase) results in a harmful buildup of glycogen byproducts in the liver, muscle and even the heart in some cases. The severity of symptoms is variable depending on the degree of enzyme deficiency and how strictly treatment measures are adhered to.
• Glycogen storage disease type 1C: A genetic metabolic disorder involving a deficiency of the enzyme glucose-6-phosphatase (due to a defect in the microsomal phosphate) which results in the accumulation of glycogen in various tissues. G6P is stored as glycogen until the body needs to convert it to a sugar for energy. The enzyme deficiency prevents the conversion and hence low blood sugar levels result.
• Glycogen storage disease type 1D: A genetic metabolic disorder involving a deficiency of the enzyme glucose-6-phosphatase (due to a defect in the microsomal glucose transporter) which results in the accumulation of glycogen in various tissues. G6P is stored as glycogen until the body needs to convert it to a sugar for energy. The enzyme deficiency prevents the conversion and hence low blood sugar levels result.
• Glycogen storage disease type 2: A rare inherited biochemical disorder involving the harmful accumulation of certain chemicals (glycogen) in body tissues due to the deficiency of an enzyme (?-glucosidase or acid maltase) needed to break it down.
• Glycogen storage disease type 6: A rare, generally mild form of inherited glycogen storage disease where a deficiency of phosphorylase b kinase leads to hypoglycemia and accumulation of glycogen in the liver.
• Glycogen storage disease type 6A, due to phosphorylase kinase deficiency: A rare, generally mild form of inherited glycogen storage disease where a deficiency of phosphorylase kinase leads to hypoglycemia and accumulation of glycogen in the liver. Phosphorylase kinase deficiency can cause glycogen storage disease type VIa and/or IX.
• Glycogenosis type 2: A rare inherited biochemical disorder involving the harmful accumulation of certain chemicals (glycogen) in body tissues due to the deficiency of an enzyme (?-glucosidase or acid maltase) needed to break it down. The severity of the condition is variable and onset may occur during infancy, childhood or adulthood.
• Glycogenosis type 8: A mild glycogen storage disease which affects males only and involves a deficiency of the enzyme phosphorylase b-kinase.
• Glycoprotein storage disease: A rare inherited metabolic disorder involving an enlarged spleen and abnormal glycoprotein storage.
• Gold poisoning: A type of heavy metal poisoning caused by excessive exposure to gold.
• Goldberg syndrome: A rare lysosomal storage disorder characterized by an enzyme deficiency (neuraminidase and beta-galactosidase) which results in a build-up of glycoproteins in the urine. There are three main subtypes: infantile, juvenile and adult forms. The early infantile form is the most severe and often results in death during infancy.
• Goldstein-Hutt syndrome: A rare syndrome characterized mainly by cataracts, excessive eyelash growth and and a blood abnormality (the red blood cells are spherical instead of doughnut shaped which makes them fragile).
• Graft-versus-host disease: A disease characterised by an immune response as a result of a transplantation or transfusion resulting in a widespread systemic inflammatory response
• Griscelli disease: A rare genetic disorder characterized mainly by albinism (lack of pigmentation). There are three different subtypes of the disorder (I, II and III) each with varying additional features such as immunodeficiency and neurological symptoms. Type 1 involves partial albinism and neurological symptoms, type II involves partial albinism, immunodeficiency and sometimes neurological symptoms and type III involves albinism, only.
• Griscelli syndrome type II: A rare genetic condition characterized by a partial lack of pigmentation in the eye, skin and hair, clumps of pigmentation in hair shafts, immunodeficiency and neurological symptoms.
• Gräsbeck-Imerslund disease: A rare genetic blood disorder where a defect in the vitamin B12 receptor means that it can't be absorbed from food during digestion. As a result of the low vitamin B12 levels, the body produces increased numbers of abnormal enlarged red blood cells (megaloblasts).
• HADH deficiency: A rare inherited form of biochemical disorder characterized by the deficiency of a particular enzyme (3-Hydroxyacyl-CoA Dehydrogenase). The enzyme deficiency only affects certain body tissues, in particular the skeletal muscles. The lack of enzyme activity prevents some fats being converted into energy. Symptoms tend to be exacerbated during fasting as during fasting, the body tries to rely more heavily on fats for energy. Fatty acids that are not completely metabolized due to the enzyme deficiency may build up in various organs and cause serious complications.
• HELLP syndrome: A rare potentially fatal condition that occurs in pregnant women and is frequently associated with pre-eclampsia.
• HMG CoA synthetase deficiency: An inherited metabolic defect due to the deficiency of an enzyme (HMG CoA synthetase) needed to form ketone bodies. Symptoms are often preceded by periods of fasting or illness.
• Hairy cell leukemia: A chronic leukemia which causes an excess of abnormal mononuclear cells which appear hair like under microscopy
• Hanot-MacMahon-Thannhauser syndrome: A liver disorder where chronic inflammation of the small portal bile ducts leads to blockage of the smallest ducts. As a result, fatty deposits develop in the skin, the liver and kidneys become enlarged and jaundice occurs.
• Hashimoto-Pritzker syndrome: A rare harmless skin disorder characterized by the accumulation of histiocytes (infection-fighting cells) in the skin which forms nodules. The condition occurs at birth or soon after and resolves spontaneously within a month.
• Heart symptoms: Symptoms affecting the heart
• Heavy-chain diseases: A group of rare condition characterized by the production of the heavy chain portions of immunoglobulin molecules. Subtypes include y-chain disease, µ-chain disease and α-chain disease.
• Heliotrope poisoning: The Heliotrope is a herbaceous plant which bears small white, purple or blue flowers. The plant can be found growing in the wild and is also used indoors and outdoors as an ornamental plant. The leaves contain chemicals which can cause symptoms if eaten or skin irritation upon skin exposure. The plant is considered to have a low level of toxicity and large amounts would have to be eaten to cause symptoms. The leaves are sometimes used to make a tea.
• Helminth infections: The infection by a parasitic worm
• Helminthiasis: Infection of the human body with a parasitic worm such as roundworms and pinworms. The worms usually only involve the intestinal tract but sometimes they may invade other organs. The type and severity of symptoms is determined by the type of worm and the part of the body infected.
• Hemangioendothelioma: A rare type of blood vessel tumor that can occur anywhere in the body but is most often found in the skin, liver and spleen. Symptoms vary according to the exact location.
• Hematochromatosis: Hemochromatosis is the abnormal accumulation of iron in parenchymal organs, leading to organ toxicity. It is the most common inherited liver disease in whites and the most common autosomal recessive genetic disorder.
• Hemochromatosis: Excess of iron leading to problems with joints, liver, heart and pancreas.
• Hemochromatosis type 1: A genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. Type 1 is the most common form of the disorder, has an adult onset and is inherited recessively.
• Hemochromatosis type 2: A rare genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. Type 2 is a more severe type, has a juvenile onset and is inherited recessively.
• Hemochromatosis type 3: A rare genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. Type 3 has an onset that is between type 1 and type 2 and is inherited recessively.
• Hemochromatosis type 4: A rare genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. Type 4 has an adult onset but is inherited dominantly.
• Hemoglobin C disease: An inherited blood condition characterized by abnormal hemoglobin (hemoglobin C) which leads to premature break down of red blood cells and hence anemia. In homozygous patients (a hemoglobin C gene is inherited from both parents) mild hemolytic anemia may develop as nearly all of the hemoglobin is of the abnormal hemoglobin C form. In heterozygous patients (a hemoglobin C gene is inherited from one parent) more than half of the hemoglobin is normal and the patient is asymptomatic. Hemoglobin C occurs commonly in West African populations and is believed to provide some protection against severe malaria.
• Hemoglobin C homozygous (CC): A rare genetic blood disorder which tends to only cause mild hemolytic anemia.
• Hemoglobin E disease: A recessively inherited genetic blood disorder characterized by abnormally small red blood cells which generally causes no health problems. Some people may suffer mild hemolytic anemia or a slightly enlarged spleen.
• Hemoglobin E homozygous: A rare genetic blood anomaly which may cause anemia and occasionally an enlarged spleen.
• Hemoglobin H: A genetic blood anomaly that causes enlarged liver and spleen and hemolytic anemia.
• Hemoglobin, constant spring: A generally mild form of anemia caused by a defect in the hemoglobin which is the part of the blood that carries oxygen to the body tissues. The biggest risk of this condition is the fact that children born to carriers of this and other blood disorders may inherit more severe forms of blood disorders.
• Hemolytic anemia: A condition which is characterized by anaemia due to the destruction of red blood cells
• Hemolytic anemia, lethal - genital anomalies: A very rare syndrome characterized mainly by genital abnormalities and hemolytic anemia which often causes death.
• Hemolytic disease of the newborn: Blood disease in newborns mainly related to Rh incompatibility
• Hemolytic uremic syndrome: A rare condition characterized by acute kidney failure, hemolytic anemia and thrombocytopenia (reduced blood platelet count). The condition is often caused by upper respiratory infections or infectious diarrhea.
• Hemophagocytic lymphohistiocytosis, familial, 1: A rare recessively inherited disorder involving an overactive immune system. More specifically, the body becomes infiltrated by large numbers of histiocytes (macrophages) that accumulate in various organs such as the liver, spleen, bone marrow, skin and central nervous system. It usually only occurs in infants and young children and can be life-threatening.
• Hemophagocytic lymphohistiocytosis, familial, 2: A rare recessively inherited disorder involving an overactive immune system. More specifically, the body becomes infiltrated by large numbers of histiocytes (macrophages) that accumulate in various organs such as the liver, spleen, bone marrow, skin and central nervous system. It usually only occurs in infants and young children. Type 2 is caused by a defect on chromosome 10q22.
• Hemophagocytic lymphohistiocytosis, familial, 3: A rare recessively inherited disorder involving an overactive immune system. More specifically, the body becomes infiltrated by large numbers of histiocytes (macrophages) that accumulate in various organs such as the liver, spleen, bone marrow, skin and central nervous system. It usually only occurs in infants and young children. Type 3 is caused by a defect on chromosome 17q25.1.
• Hemophagocytic lymphohistiocytosis, familial, 4: A rare recessively inherited disorder involving an overactive immune system. More specifically, the body becomes infiltrated by large numbers of histiocytes (macrophages) that accumulate in various organs such as the liver, spleen, bone marrow, skin and central nervous system. It usually only occurs in infants and young children. Type 4 is caused by a defect on chromosome 6q24.
• Hemophagocytic reticulosis: The abnormal proliferation of reticulum cell (histiocytes) which infiltrate various organs and. Macrophages destroy blood cells causing blood abnormalities. Meningoencephalitis frequently occurs when the histiocytes infiltrate the mininges and cerebral tissue. Symptoms start at birth or soon after and become progressively worse without treatment. Medication can control the condition but a hematopoietic stem cell transplant is needed to achieve remission.
• Hemorrhagic thrombocythemia: A rare blood disorder characterized by increased number of platelets in the blood which often results in an enlarged spleen, bleeding and blood vessel blockages.
• Hepadnaviral infection: There are a number of viruses in the Hepadnavirdae family of viruses but only type B can cause infection in the human. It causes hepatitis B infection in humans. The other types of the virus can cause infection in other animals.
• Hepadnaviruses: There are a number of viruses in the Hepadnavirdae family of viruses but only type B can cause infection in the human. It causes hepatitis B infection in humans. The other types of the virus can cause infection in other animals.
• Hepadnoavirus infection: There are a number of viruses in the Hepadnavirdae family of viruses but only type B can cause infection in the human. It causes hepatitis B infection in humans. The other types of the virus can cause infection in other animals.
• Hepatic Venoocclusive Disease with immunodeficiency: An inherited disorder characterized by the association of immunodeficiency and liver disease involving the blockage of small veins in the liver due to swelling and fibrosis.
• Hepatic amyloidosis with intrahepatic cholestasis: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage. In hepatic amyloidosis, the amyloid deposits occur primarily in the liver which affects the flow of bile through the organ and results in intrahepatic cholestasis.
• Hepatic cystic hamartoma: A cystic liver tumor.
• Hepatic encephalopathy: Caused by marked liver disease that results in disturbances of consciousness
• Hepatic encephalopathy syndrome: A rare syndrome involving the association of advanced liver disease and neurological problems.
• Hepatic fibrosis, congenital: A rare condition characterized by scarring or fibrosis of the liver that impairs its function.
• Hepatic veno-occlusive disease - immunodeficiency: A rare form of liver disease involving the blockage of veins in the liver due to thickening of the blood vessel walls as well as immunodeficiency. The disease may be caused by a variety of factors including bone marrow transplant and genetic defects.
• Hepatitis: Any inflammation of the liver
• Hepatitis A: Contagious viral infection of the liver
• Hepatitis B: Viral liver infection spread by sex or body fluids.
• Hepatitis C: Viral liver infection spread by blood.
• Hepatitis D: Viral liver infection occurring in association with HepB.
• Hepatitis E: Viral liver infection.
• Hepatitis Virus - Teratogenic Agent: There is strong evidence to indicate that the development of the hepatitis virus during pregnancy may have a teratogenic effect on the fetus. A teratogen is a substance that can cause birth defects. The likelihood and severity of defects may be affected by the stage of pregnancy that the exposure occurred at.
• Hepatitis X: Hepatitis infection by an unknown virus not classified as HepA/B/C/D/E.
• Hepatitis X (non-A,-B,-C,-D,-E): Viral liver inflammation that cannot be determined to be one of the existing types of viral hepatitis - A,B,C,D and E.
• Hepatoma: Hepatocellular carcinoma (HCC) is a primary malignancy of the liver.
• Hepatorenal syndrome: A condition the occurs as a result of severe liver disease.
• Hepatorenal tyrosinemia: A rare genetic metabolic disorder characterized by a deficiency of particular enzymes which prevents the breakdown of tyrosine which then builds up in the liver. Type 1 involves a deficiency of the enzyme fumaril acetoacetate hydrolase.
• Hepatotoxicity: Damage or injury to the liver caused by a drug, chemical or other agent. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure.
• Heptaosplenic T-cell Lymphoma: A rare subtype of lymphoma which originates from T-cell lymphocytes in the liver and the spleen. This form of lymphoma is considered quite aggressive. People with solid organ transplants tend to be more susceptible to this form of cancer, possibility because of the need to take immune suppressing drugs to avoid rejection of the transplanted organ.
• Herbal Agent adverse reaction - Chaparral: Chaparral can be used as a herbal agent to treat abdominal cramps, pain and acne. The herbal agent contains a chemical called NDGA which can cause an adverse reaction in some people. Skin contact can also result in dermatitis. Patients with preexisting liver or kidney conditions are particularly susceptible to an adverse reaction to the herbal agent.
• Herbal Agent adverse reaction - Clove: Clove can be used as a herbal agent that can be used topically for tooth pain or as a local anesthetic in dentistry. The herbal agent can cause an adverse reaction or even anaphylaxis in some people.
• Herbal Agent adverse reaction - Germander: Germander can be used as a herbal agent to treat obesity. The herbal agent contains chemicals that can cause liver toxicity in some patients.
• Herbal Agent adverse reaction - Kombucha: Kombucha can be used as a herbal agent to treat insomnia, arthritis, aches, high blood pressure and to improve the immune system. Kombucha can cause an adverse reaction in some people.
• Herbal Agent adverse reaction - Margosa oil: Margosa oil can be used as a herbal agent to treat parasitic infestations. The herbal agent contains various chemicals which can cause an adverse reaction in some people.
• Herbal Agent adverse reaction - Pennyroyal Oil: Pennyroyal Oil can be used as a herbal agent to treat delayed menstruation and as an insect repellent. The herbal agent can cause an adverse reaction in some people.
• Herbal Agent adverse reaction - Polygonum multiflorum: The root from the Polygonum multiflorum can be used as a herbal agent to treat constipation, insomnia and vertigo. The herbal agent contains anthraquinones which can cause an adverse reaction in some people.
• Herbal Agent adverse reaction - Rue: Rue can be used to induce abortion, as a topical insect repellant or to treat spasms and delayed menstruation. The herbal agent contains chemicals (alkaloids, arborine, arborinine) which can cause an adverse reaction in some people.
• Herbal Agent adverse reaction - Sassafras Oil: Sassafras Oil can be used as a herbal agent to treat skin irritation such as insect bites. The herbal agent contains a chemical called safrole which can cause harmful effects if ingested .
• Herbal Agent overdose - Comfrey: Comfrey can be used as a herbal agent used topically to assist wound healing. The herbal agent contains pyrrolizidine alkaloid which is toxic to the liver. Ingesting sufficient quantities can result in severe liver problems.
• Herbal Agent overdose - Germanium: Germanium is used as a health food supplement mainly in Japan but it can cause various problems if too much is taken. Chronic use and the ingestion of a large amount at one time can result in overdose symptoms.
• Herbal Agent overdose - Kombucha: Kombucha can be used as a herbal agent to treat insomnia, arthritis, aches, high blood pressure and to improve the immune system. The ingestion of excessive amounts of Kombucha can result in overdose symptoms.
• Herbal Agent overdose - Psoralea Corylifolia: Psoralea Corylifolia can be used as a herbal agent to treat vitiligo and psoriasis. Excessive intake of the seeds can result in overdose symptoms.
• Herbal Agent overdose - Rue: Rue can be used to induce abortion, as a topical insect repellant or to treat spasms and delayed menstruation. The herbal agent contains chemicals (alkaloids, arborine, arborinine) which can cause various symptoms if excessive quantities are taken.
• Hereditary Hemochromatosis: A genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. There are 4 types of hemochromatosis and they are distinguished by age of onset, genetic cause and type of inheritance. Some sufferers may be asymptomatic.
• Hereditary carnitine deficiency: An inherited deficiency of carnitine resulting primarily in muscle problems. Severe symptoms can be triggered by periods of illness or fasting.
• Hereditary carnitine deficiency syndrome: An inherited deficiency of carnitine resulting primarily in muscle weakness. The carnitine deficiency may be due to excessive loss of insufficient production.
• Hereditary carnitine deficiency syndrome, systemic: An inherited deficiency of carnitine in tissues other than the muscles resulting primarily in muscle weakness.
• Hereditary elliptocytosis: An inherited blood disorder where a significant number of red blood cells (erythrocytes) are elliptical or oval shaped rather than doughnut shaped. The condition is often asymptomatic but anemia can result when the abnormal red blood cells are destroyed.
• Hereditary hemorrhagic telangiectasia: A rare genetic disorder characterized by epistaxes and multiple telangiectases.
• Hereditary non-spherocytic hemolytic anemia: A group of genetic blood disorders where red blood cells are prematurely destroyed resulting in anemia if they are not replaced fast enough. The blood cells are destroyed because they have abnormally weak membranes which gives them an irregular shape rather than normal doughnut shape.
• Hereditary primary Fanconi disease: A rare inherited disorder characterized by defective reabsorption of various substances such as phosphate, potassium, amino acids and glucose which manifests as a wide range of abnormalities and problems.
• Hereditary spherocytic hemolytic anemia: An inherited blood disorder where a metabolic defect causes defects in the red blood cells membranes which leads to their characteristic spherical shape (normal cells are doughnut shaped) and premature destruction.
• Hereditary spherocytosis: A rare genetic condition characterized by a defect of the red blood cell membrane.
• Hereditary thrombocythemia: An inherited disorder characterized by thrombocytosis which is a high blood platelet count that occurs for no apparent reason. It is usually only discovered incidentally during routine blood tests.
• Heroin dependence: The physical and psychological dependence to the recreational drug heroin
• Herpes, Neonatal: Neonatal herpes is the infection of a newborn with the herpes virus within the first six weeks of life. The virus may be transmitted from the mother to the baby while it is still in the uterus or during delivery. The risk of transmitting the virus is highest if genital herpes is contracted during the late stages of the pregnancy. A mother with long standing or recurring herpes infection usually has sufficient antibodies to the virus to prevent the infant becoming infected. Neonatal herpes can also be contracted when an infant comes into contact with an infected person e.g. being kissed by and adult with cold sores. A cesarean birth may be advised for mothers who have active genital lesions.
• Herpes, Neonatal - Disseminated: Disseminated neonatal herpes is a widespread infection of a newborn with the herpes virus within the first six weeks of life. The virus may be transmitted from the mother to the baby while it is still in the uterus or during delivery. The risk of transmitting the virus is highest if genital herpes is contracted during the late stages of the pregnancy. A mother with long standing or recurring herpes infection usually has sufficient antibodies to the virus to prevent the infant becoming infected. Neonatal herpes can also be contracted when an infant comes into contact with an infected person e.g. being kissed by and adult with cold sores. A cesarean birth may be advised for mothers who have active genital lesions. Brain infection will occur in over half of infants with the disseminated form.
• Histiocytosis, Non-Langerhans-Cell: A group of disorders involving the excessive proliferation of histiocytes (macrophages) which are not Langerhans cells. Subgroups of this disorder includes hemophagocytic lymphohistiocytosis, sinus histiocytosis, xanthogranuloma and sea-blue histiocyte syndrome. Sometimes only the skin is involved and in other cases there may be systemic involvement which can cause serious problems.
• Hodgkin's Disease: A form of cancer that affects the lymphatic system.
• Hodgkin's disease, adult: A type of cancer that affects the lymphatic system in adults. The lymphatic system forms part of the body's immune system. This type of cancer can also occur in children.
• Hodgkin's disease, childhood: A type of cancer that affects the lymphatic system in children. The lymphatic system forms part of the body's immune system. This type of cancer can also occur in children.
• Homozygotic Alpha 1 antitrypsin Defeciency: Alpha-1 antitrypsin deficiency is an inherited disorder that can cause lung disease in adults and liver disease in adults and children
• Homozygous hypobetalipoproteinemia: Inherited low blood betalipoprotein level. The type and severity of symptoms is determined by whether or not there are some betalipoproteins in the body.
• Horse nettle poisoning: Horse nettle is a herbaceous plant which has prickles and bears yellow berries. The berries contain solanin alkaloids which can cause symptoms if eaten in large quantities. It is often found growing in the wild in many parts of the world. Death is considered possible if large amounts are eaten, especially in children.
• Hunter-Carpenter-Macdonald syndrome: A very rare syndrome characterized by various abnormalities including small head, facial anomalies, mental retardation and hair, skin and nail anomalies. Fetal exposure to alcohol is believed to be a factor in the syndrome.
• Hydatidosis: A parasitic infection caused by a tapeworm larva called echinococci. The type and severity of symptoms is determined by the location of the infestation - the most common site for the larval cysts is the liver. The most common symptoms are due to compression of nearby organs or blood vessels due to increasing size of the cyst. The larva may incubate for months or even years.
• Hydrops ectrodactyly syndactyly: A very rare disorder characterized mainly be webbed fingers and toes, hand defect and hydrops (abnormal accumulation of fluid in the fetus).
• Hydrops fetalis: Abnormal accumulation of fluid in the fetus which can be fatal. Excessive fluid leaves the blood and enters the tissues. It can have non-immune and immune (mother's immune system destroys fetal red blood cells).
• Hyper IgM syndrome 1: Hyper IgM syndrome is a rare inherited immunodeficiency disorder which causes frequent infections involving the ears, eyes, sinuses, lungs, skin, respiratory tract and other areas of the body. Type 1 is caused by a defect on chromosome Xq26 and tends to affects males mostly.
• Hyper IgM syndrome 3: Hyper IgM syndrome is a rare inherited immunodeficiency disorder which causes frequent infections involving the ears, eyes, sinuses, lungs, skin, respiratory tract and other areas of the body. Type 3 is caused by a defect on chromosome 20q12-q13.2.
• Hyper-IgM Syndrome: A rare inherited immunodeficiency disorder which causes frequent infections involving the ears, eyes, sinuses, lungs, skin, respiratory tract and other areas of the body.
• Hyperbilirubinemia: Excessive bilirubin (a bile pigment) in the blood.
• Hyperchylomicronemia: A term used to describe the presence of too many chylomicrons in the blood.
• Hyperdibasic aminoaciduria type 2: A rare inborn urea cycle disorder characterized by an enzyme defect in the amino acid transporter gene SLC7A7 (positive amino acid transporter).
• Hypereosinophilic syndrome: A rare condition where too many eosinophils are produced over an extended period of time for no apparent reason. The eosinophils can infiltrate various organs and tissues and cause dysfunction or damage
• Hypergammaglobulinemia: An increase in the level of any of a number of gamma globulins. This can result from conditions such as chronic bacterial infections, multiple myeloma, lymphomas, dysproteinemias, liver disease and chronic granulomatous inflammations. The exact symptoms depend on the type of gamma globulins involved and the severity of the condition.
• Hyperimidodipeptiduria: A very rare genetic disorder characterized by an excessive level of imidodipeptides in the urine due to a deficiency of the enzyme prolidase.
• Hyperimmunoglobinemia D with recurrent fever: A very rare disorder involving a high immunoglobulin level associated with periods of fever which generally reoccur every month or two.
• Hyperlipoproteinemia type 3: A rare genetic disorder characterized by the body's impaired ability to break down certain lipids (triglycerides) which results in their buildup in the blood.
• Hyperlipoproteinemia type 5: An inherited disorder where the body's ability to break down lipids (triglycerides and cholesterol) is impaired resulting in increased blood lipid levels of chylomicrons and VLDL's.
• Hyperlysinemia, persistent: A rare genetic disorder where the body lacks enzymes (lysine ketoglutarate reductase and saccharopine dehydrogenase) to metabolize lysine which then causes a harmful builds up of lysine in the blood and urine and saccharopins in the urine. Some patients are asymptomatic while others have severe symptoms.
• Hyperparathyroidism, neonatal severe primary: A very rare disorder where high levels of parathyroid levels affects the body's use of calcium. The bones lack sufficiency calcification and become weak.
• Hyperpipecolatemia: A rare metabolic disorder characterized by high blood levels of pipecolic acid.
• Hypertension due to coarctation of the aorta: Hypertension due to coarctation of the aorta is high blood pressure resulting from a birth defect where a heart blood vessel called the aorta is abnormally narrowed. The high blood pressure tends to affect mainly the upper part of the body. Babies and very young children tend to have more severe symptoms as the degree of narrowing tends to be greater than in older children or adults.
• Hypoplasia hepatic ductular: A rare liver disorder where there are fewer than normal bile ducts in the liver - sometimes all the bile ducts are absent. Alagille syndrome is characterized by this liver abnormalitiy as well as a distinctive face, abnormal vertebrae and retarded physical, mental and sexual development.
• Hypoplastic Left Heart Syndrome: A rare condition where an infant is born with an underdeveloped left side of the heart which prevents the heart from pumping oxygenated blood efficiently to various parts of the body.
• I cell disease: A rare inherited biochemical disorder characterized by the harmful accumulation of chemicals (glycoproteins and glycoplipids) due to the deficiency of an enzyme (UDP-N-acetylglucosamine-I-phosphotransferase).
• Ichthyohepatotoxication: Ichthyohepatotoxication is a condition caused by eating the liver of certain fish. It is believed that the high vitamin A content of the liver leads to vitamin A overdose and the resulting symptoms. Tropical shark livers are associated with this condition.
• Ichythosiform Erythroderma with Leukocyte Vacuolation: A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities. The body is unable to metabolize (break down) triglycerides so they build up in various tissues throughout the body. The severity of the symptoms is variable as the body tissues and organs affected varies amongst patients.
• Idiopathic hypereosinophilic syndrome: A rare blood disorder where the bone marrow produces too many eosinophils over a long period of time which can cause organ or tissue damage. The disorder can affect and part of the body but most often affects the skin, heart and nervous system. The increased eosinophil production continues for a long period of time (at least 6 months) and there is no apparent cause.
• Idiopathic liver cirrhosis: Cirrhosis is defined histologically as a diffuse hepatic process characterized by fibrosis and the conversion of normal liver architecture into structurally abnormal nodules. The progression of liver injury to cirrhosis may occur over weeks to years.
• Imerslünd-Grasbeck Syndrome: A rare genetic blood disorder where a defect in the vitamin B12 receptor means that it can't be absorbed from food during digestion. As a result of the low vitamin B12 levels, the body produces increased numbers of abnormal enlarged red blood cells (megaloblasts).
• Immune defect due to absence of thymus: A congenital disorder where the absence of the thymus results in immune system problems (deficiency of T-Lymphocytes). The thymus is involved in the production of mature functioning T-cells which fight infection.
• Immunoglobulinic amyloidosis: A disease characterized by the abnormal deposit of amyloid in various parts of the body, especially organs such as the kidneys, heart, liver, gastrointestinal tract and peripheral nerves. It occurs when plasma cells in the bone marrow produce too much of a protein portion of an antibody called the light chain. The exact symptoms are determined by the extent of the organ involvement.
• Immunoproliferative diseases: Diseases characterised by proliferation of the lymphoid cells
• Inborn amino acid metabolism disorder: A group of inherited disorders where the body is not able to metabolize amino acids consumed in the diet. Amino acids are a part of carbohydrates, fats and proteins and are metabolized in order to provide energy or to make other needed compounds. There are many steps involved in metabolism and the severity can be greatly variable depending on the exact nature of the disorder.
• Inborn errors of thyroid hormone synthesis related to hypothyroidism: Congenital hypothyroidism is inadequate thyroid hormone production in newborn infants. This can occur because of an anatomic defect in the gland, an inborn error of thyroid metabolism, or iodine deficiency.
• India tick typhus: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Indian tick fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Infant Cytomegalic virus: A serious CMV viral infection in newborns.
• Infantile Refsum Disease: Genetic disease affecting nerve and muscle control.
• Infantile multisystem inflammatory disease: A rare autoinflammatory disease characterized by fever, rash, arthritic changes, eye problems and chronic meningitis.
• Infantile sialic acid storage disorder: A rare inherited biochemical disorder characterized by the accumulation of sialic acid in the tissues and excretion of sialic acid in the urine. The disorder results in death within the first few years of life - usually in infancy.
• Infantile spasms: A condition which is characterized by brief muscular spasms in infants
• Infectious myocarditis: Inflammation of the muscle of the heart (myocardium) due to an infection. It often occurs as a complication of various bacterial, viral or parasitic infections such as rubella, polio and rheumatic fever.
• Inhalant abuse: Inhalant abuse is the use of various inhalants for the purpose of achieving a "high". They are often used as a cheap, readily available alternative to street drugs but they can cause serious damage to the body. Inhalants include gasoline, adhesives, solvents, and aerosols. These inhalants can be abused by sniffing them, spraying directly into the mouth, heating them and then inhaling them or injecting them directly into the body.
• Inhalant addiction: Inhalant addiction refers to the compulsive need to abuse inhalants (e.g. inhaling them). Sufferers have withdrawal symptoms when attempting to stop the habit and feel unable to stop the habit despite knowing the harm it is causing their health. Inhalants are very damaging to the body and can readily result permanent brain damage and even death. Death can occur through chronic use and in rare cases can occur after one session of use. Children and teenagers are particular at risk for this type of addiction - it is readily available and users feel it gains them greater acceptance from their peers. Inhalants includes glues, shoe polish, household cleaners, room deodorizers and nail polish removers.
• Inherited spherocytic anemia: Inherited Spherocytic anemia is an inherited blood disorder characterized by red blood cells that are shaped like a sphere (spherocytes) rather than the normal doughnut shape. These abnormal cells are broken down prematurely resulting in a shortage of red blood cells (anemia).
• Interferon Beta - Teratogenic Agent: There is evidence to indicate that exposure to Interferon Beta during pregnancy may have a teratogenic effect on the fetus. A teratogen is a substance that can cause birth defects. The likelihood and severity of defects may be affected by the level of exposure and the stage of pregnancy that the exposure occurred at.
• Intrahepatic cholangiocarcinoma: A slow growing malignant cancer that occurs in the bile ducts in the liver.
• Intrapartum Eclampsia: Intrapartum eclampsia is the development of seizures or coma in pregnant women suffering from high blood pressure. Intrapartum means that it occurs during the delivery of the baby. Eclampsia is a serious condition which requires urgent medical treatment. Eclampsia may be associated with moderate as well as significant increases in blood pressure. The blood pressure can return to normal after delivery or may persist for a period of time.
• Invasive candidiasis: Severe fungal infection usually in immunocompromised persons
• Irish potato poisoning: The common potato is an edible root. However, the potato sprouts and green skin in old potatoes contain chemicals such as solanine which can cause symptoms if eaten. Severe cases can result in death but this is relatively rare.
• Israeli spotted fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Itraconazole toxicity: The toxic reaction of the body to the substance, possibly via allergic reaction or overdose.
• Ivemark II: A lethal syndrome characterized by lack of spleen function as well as kidney, liver, pancreatic and heart abnormalities. The condition generally results in death within days of birth.
• Jaundice: yellowish discoloration of the skin and mucous membrane
• Jaundice in pregnancy: Jaundice in pregnancy is a yellow discolouration of the skin and mucous membranes associated with liver malfunction.
• Jaundice-like symptoms: Yellowing symptoms of jaundice
• Jequier-Kozlowski-skeletal dysplasia: A very rare syndrome characterized mainly by various skeletal abnormalities and facial anomalies.
• Juvenile idiopathic arthritis: A group of chronic inflammatory joint disorders that affects children. The condition generally involves periods of time where the condition is active followed by periods of abatement of symptoms. In some cases, the condition can be systemic and can cause symptoms such as fever and rash with organ involvement. There are three main types of juvenile idiopathic arthritis - oligoarticular, polyarticular and systemic (Still's disease).
• Juvenile myelomonocytic leukemia: A rare form of malignant bone marrow cancer that occurs in children and involves the proliferation of immature precursors of certain blood cells - myelocytes and monocytes. The proliferation is slower than in acute forms of the disease.
• Kaposiform hemangio-endothelioma: A rare blood vessel tumor that occur primarily in children. It causes localized damage but is not metastatic. Main sites affected are the retroperitoneum and skin but can also occur in the head and neck area, mediastinum and deep soft tissue of the trunk and limbs. Symptoms and prognosis is determined by the size and location of the tumors.
• Katayama fever: An acute disease due to infection with Schistosoma parasites. Transmission can occur through contact with infected waters.
• Kawasaki disease: A childhood illness that generally affects the skin, mouth and lymph nodes.
• Kenya fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Kenya tick typhus: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Kenya tick-bite fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Kidney pain: The occurrence of pain that comes from the kidney
• Kidney symptoms: Symptoms affecting one or both kidneys.
• Korovnikov syndrome: A rare disorder characterized by enlarged spleen and increased blood platelets which leads to bleeding problems. It is considered to by a form of Banti's disease.
• L-3-alpha-hydroxyacyl-CoA dehydrogenase, short chain, deficiency: A rare inherited form of biochemical disorder characterized by the deficiency of a particular enzyme (3-Hydroxyacyl-CoA Dehydrogenase). The enzyme deficiency only affects certain body tissues, in particular the skeletal muscles. The lack of enzyme activity prevents some fats being converted into energy. Symptoms tend to be exacerbated during fasting as during fasting, the body tries to rely more heavily on fats for energy. Fatty acids that are not completely metabolized due to the enzyme deficiency may build up in various organs and cause serious complications.
• LADHSC deficiency: A rare inherited form of biochemical disorder characterized by the deficiency of a particular enzyme (3-Hydroxyacyl-CoA Dehydrogenase). The enzyme deficiency only affects certain body tissues, in particular the skeletal muscles. The lack of enzyme activity prevents some fats being converted into energy. Symptoms tend to be exacerbated during fasting as during fasting, the body tries to rely more heavily on fats for energy. Fatty acids that are not completely metabolized due to the enzyme deficiency may build up in various organs and cause serious complications.
• Lambert syndrome: A rare syndrome characterized by a club foot, inguinal hernia, biliar atresia and branchial dysplasia.
• Langerhans Cell Histiocytosis: A condition which is characterized by proliferation of Langerhans cells
• Langerhans cell sarcoma: A rare cancerous growth of Langerhans cells that can develop in the lymph nodes, skin, liver, spleen and bones.
• Large granular lymphocyte leukemia: A form of leukemia characterized by an increased number of circulating granular lymphocytes.
• Lathosterolosis: A very rare disorder where an enzyme (sterol C5-desaturase) deficiency prevents the normal synthesis of cholesterol in the body. The deficiency causes various malformations, mental retardation and liver disease.
• Lawrence-Seip syndrome: Lawrence-Seip syndrome is a relatively uncommon condition involving generalized loss of skin fat that is acquired rather than congenital as in Berardinelli-Seip Syndrome. Patients can also develop diabetes mellitus and other problems. The loss of skin fat tends to occur during childhood or adolescence and is often triggered by infections such as measles and hepatitis. The diabetes tends to occur after fat loss begins. The limbs and face tend to be the most affected.
• Leiomyosarcoma: A rare soft tissue cancer that arises from smooth muscle cells which form the involuntary muscles. Smooth muscle cells can occur in the skin, uterus, stomach, intestine and blood vessel walls. The main location for these tumors is in the uterus, retroperitoneum, pelvis and large blood vessels such as the inferior vena cava. Symptoms depend on the size and location of the tumor.
• Leishmaniasis: A rare infectious disease caused by any of a number of parasitic Leishmania species. Infection can cause any of three different manifestations: cutaneous leishmaniasis, mucosal leishmaniasis and visceral leishmaniasis.
• Leptospirosis: Bacterial infection usually caught from animal urine.
• Letterer-Siwe disease: A rare usually fatal condition characterized by skin lesions, bleeding tendency, enlarged liver and spleen, enlarged lymph nodes and progressive anemia. The condition is caused by excessive proliferation of histiocytes.
• Leukemia: Cancer of the blood cells, usually white blood cells.
• Leukemia, B-cell, chronic: A slow growing cancer of the immune system involving proliferation of B-cells.
• Leukemia, Lymphocytic, Acute, L1: Acute lymphocytic leukemia is a cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are three main subtypes (L1, L2 and L3) which are differentiated by the appearance of the cancerous cells: L1 is characterized by small, uniform cancer cells with a round nucleus and very little cytoplasm. L1 has a better prognosis than L2. L1 is the main form in children (about 85%) but is less common in adults (about 30%).
• Leukemia, Lymphocytic, Acute, L2: Acute lymphocytic leukemia is a cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are three main subtypes (L1, L2 and L3) which are differentiated by the appearance of the cancerous cells: L2 is characterized by larger cells, an irregular-shaped nucleus, more cytoplasm and significant variation between cells. L2 has a poorer prognosis than L1. L2 is the main form in adults (about 65%) but is less common in children (about 15%).
• Leukemia, Lymphocytic, Acute, L3: Acute lymphocytic leukemia is a relatively aggressive cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are three main subtypes (L1, L2 and L3) which are differentiated by the appearance of the cancerous cells. L3 is quite uncommon but is very similar to Burkitt's lymphoma - in fact, they may be considered the same disease with different manifestations.
• Leukemia, Monocytic, Acute: A cancer of the blood-forming tissues of the bone marrow involving the proliferation of a type of infection-fighting white blood cell called a monocyte. Acute leukemia involves a more rapid proliferation of cancer cells compared to chronic forms of leukemia.
• Leukemia, Myeloid: A form of blood cancer that causes a proliferation of the precursors or immature red blood cells, platelets and certain white blood cells such as granulocytes and monocytes.
• Leukemia, Myeloid, Aggressive-Phase: Myeloid leukemia is a form of cancer where the bone marrow makes too many myeloid cells (granulocytes and their precursors) in the bone marrow which accumulates in the blood and eventually invades various parts of the body. The aggressive phase of myeloid leukemia follows the chronic form and is a sign that the condition is progressing more rapidly to a blast crisis which is the final stage of leukemia.
• Leukemia, Myeloid, Chronic: A slow-growing cancer where the bone marrow makes too many myeloid cells (granulocytes and their precursors) in the bone marrow which accumulates in the blood and eventually invades various parts of the body.
• Leukemia, Myeloid, Chronic-Phase: Myeloid leukemia is a form of cancer where the bone marrow makes too many myeloid cells (granulocytes and their precursors) in the bone marrow which accumulates in the blood and eventually invades various parts of the body. The chronic phase of myeloid leukemia usually involves few if any symptoms. This is the early stage of the disease and without treatment it generally progresses to the accelerated phase of the condition. The majority of patients diagnosed with myeloid leukemia are diagnosed during the chronic phase.
• Leukemia, Myeloid, Philadelphia-Negative: Acute lymphocytic leukemia is a relatively aggressive cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. The Philadelphia negative form of the condition is not associated with a genetic mutation. It is distinguished from the positive form by the clinical course which is initially the same but progresses to eventual bone marrow failure without a distinct increase in blast cells. Other differences include poor response to chemotherapy, lower white blood cell counts, greater monocytosis, less basophilia, lower bone marrow myeloid to erythroid ratio and increased likelihood of developing thrombocytopenia. Philadelphia negative patients also tend to be older and median survival rates tend to be poorer.
• Leukemia, Myeloid, Philadelphia-Positive: Acute lymphocytic leukemia is a relatively aggressive cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. The Philadelphia form of myeloid leukemia carries the poorest prognosis of all the different types. It involves an acquired genetic mutation which results in the production tyrosine kinase which causes too many white blood cells to be produced which results in a shortage of other blood cell types. Treatment is aimed at inhibiting the production of tyrosine kinase.
• Leukemia, T-Cell: A form of blood cancer characterized by the proliferation of cancerous T-cells which make up part of the body's immune system. The exact symptoms and progression vary depending on the subtype involved.
• Leukemia, T-Cell, Acute: A form of blood cancer characterized by the proliferation of cancerous T-cells which make up part of the body's immune system. The exact symptoms and progression vary depending on the subtype involved. The acute form involves a rapid proliferation of cancerous T-cells and hence a more rapid disease progression and increased severity of symptoms.
• Leukemia, T-cell, chronic: Cancer of blood cells called T-cells which form part of the immune system.
• Leukemia, mast-cell: A very aggressive form of leukemia - a subtype of acute myeloid leukemia. The cancer can in rare cases develop from chronic myeloid leukemia or systemic mastocytosis but generally develops on its own.
• Lipidosis with triglyceride storage disease: A very disorder involving abnormal storage of fat (triglycerides in parts of the body). The condition causes blood abnormalities as well as skin, eye and hearing problems.
• Lipoamide dehydrogenase deficiency: A very rare enzyme deficiency (dihydrolipoamide dehydrogenase) which can cause lactic acidosis. The age of onset and symptoms are variable.
• Lipoatrophy with diabetes, hepatic steatosis, cardiomyopathy, and leukomelanodermic papules: A rare inherited disorder characterized by loss of fat under the skin, diabetes, heart muscle disease, fatty liver
• Lipodystrophy: A rare group of disorders that can be acquired or inherited and involves adipose tissue abnormalities. The disorder is characterized by varying degrees of loss of body fat. The three forms of the condition are: total lipodystrophy, partial lipodystrophy and localized lipodystrophy.
• Lipodystrophy, familial partial, type 1 (FPLD1): An inherited disorder characterized by the loss of fatty tissue from the extremities while fatty tissue on the face, neck and trunk is normal or increased.
• Lipodystrophy, familial partial, type 3 (FPLD3): A rare metabolic disorder involving abnormal fat gradually disappears from the limbs, trunk and buttocks but stays the same or accumulates on areas such as the face, shoulders, neck and genitals.
• Lipoproteine lipase deficiency: A rare inherited inborn error of metabolism involving the absence of the enzyme called lipoprotein lipase which results in increased blood triglyeride and chylomicron levels.
• Lissencephaly: A very rare disorder characterized by abnormal brain formation so that the brain surface appears smooth rather than convoluted.
• Lissencephaly syndrome type 1: A very rare form of the brain formation disorder called lissencephaly where the brain surface is abnormally smooth rather than convoluted resulting in mental retardation. Physical malformations are also present.
• Littoral cell angioma of the spleen: A usually benign type of tumor that develops in the spleen. The name arises from the fact that the tumor develops from the littoral cells which line the blood vessels in the spleen.
• Liver abnormalities: Any condition which affects the function or structure of the liver
• Liver abscess: Pus in the liver
• Liver cancer: The occurrence of malignancy that occurs in the liver
• Liver conditions: Any condition that affects the liver
• Liver damage: Any damage that occurs to the function or structure of the liver
• Liver disease - retinitis pigmentosa - polyneuropathy - epilepsy: An inherited disorder characterized by an enzyme (Alpha-methyl-acyl-CoA racemase) deficiency, liver disease, epilepsy, polyneuropathy and eye problems. The neuropathy tends to start during adulthood.
• Liver dysfunction: Where the liver does not function as it is required
• Liver enlargement: This is where there is an enlargement of the liver
• Liver failure: When the liver fails to function
• Liver inflammation: The occurrence of inflammation located in the liver
• Liver mass: A abnormal mass that is located in the liver.
• Liver pain: Pain or discomfort due to the liver
• Liver problems: Any problems that are associated with the livers structure and function
• Liver symptoms: Symptoms affecting the liver
• Liver symptoms in pregnancy: Liver symptoms in pregnancy are usually related to pain under the ribs on the right side of the upper abdomen, sometimes together with nausea and/or vomiting. This pain may or may not actually arise from the liver, so other potential causes are also listed.
• Liver tenderness: When there is tenderness located over the liver
• Liver vein outflow obstruction: Budd-Chiari syndrome is the clinical picture caused by occlusion of the hepatic veins. It presents with the classical triad of abdominal pain, ascites and hepatomegaly.
• Long-chain 3-hydroxyacyl-coenzyme A dehydrogenase deficiency: A rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme (3-hydroxyacyl-coenzyme A dehydrogenase) which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage.
• Lung symptoms: Symptoms affecting one or both lungs.
• Lupus: Autoimmune disease with numerous effects on various organs and linings.
• Lutz-Richner-Landolt syndrome: A rare disorder involving kidney and biliary abnormalities.
• Lyme disease: Lyme disease is an emerging infectious disease caused by at least three species of bacteria belonging to the genus Borrelia.
• Lymph node neoplasm: Lymph node tumor.
• Lymphadenopathy, angioimmunoblastic with dysproteinemia: A progressive immune system disorder that usually affects people over the age of 50 and is characterized by chills, sweating, weight loss, skin rashes.
• Lymphoblastic lymphoma: A cancer of the lymph system which usually involves immature T-lymphocytes and sometimes B-lymphocytes. The cancer usually affects the mediastinum (between the lungs), bone marrow, brain and spinal cord.
• Lymphoid interstitial pneumonia: A relatively rare form of lung disease characterized by the buildup of lymphocytes (a type of white blood cell) in the air spaces or alveoli of the lungs. The condition can affect children or adults and is frequently associated with conditions such as HIV and autoimmune conditions such as rheumatoid arthritis and Hashimoto's thyroiditis. Symptoms may develop gradually over a period of months or even years in some cases.
• Lymphoma: Any neoplastic disorder that occurs in lymphoid tissue
• M/SCHAD deficiency: A rare inherited form of biochemical disorder characterized by the deficiency of a particular enzyme (3-Hydroxyacyl-CoA Dehydrogenase). The enzyme deficiency only affects certain body tissues, in particular the skeletal muscles. The lack of enzyme activity prevents some fats being converted into energy. Symptoms tend to be exacerbated during fasting as during fasting, the body tries to rely more heavily on fats for energy. Fatty acids that are not completely metabolized due to the enzyme deficiency may build up in various organs and cause serious complications.
• MGA 4: MGA (methylglutaconic aciduria) is a rare genetic disorder where the body's cells are unable to make sufficient energy resulting in an accumulation in the body of 3-methylglutaconic acid. Type 4 is characterized by symptoms which overlap type 1 and 3.
• MPS 3 C: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) due to deficiency of an enzyme called acetyl-CoA:alpha-glucosamide N-acetyltransferase. Mucopolysaccharide levels build up and are then deposited in various tissues.
• Macrocytic anemia: Macrocytic anemia is a blood disorder where the red blood cells are larger than normal but have low levels of haemoglobin which is needed to carry oxygen throughout the body. The condition usually results from a deficiency of vitamin B12 or folate, digestive problems, malabsorption and certain medications which affect folic acid levels. Various rare inherited disorders may also result in macrocytic anemia e.g. Lesch-Nyhan syndrome. Severity and range of symptom may vary depending on the underlying condition.
• Macrosomia with lethal microphthalmia: A very rare syndrome characterized mainly by very small eyes and a large body size at birth.
• Majeed syndrome: A rare syndrome characterized by blood abnormality and recurring bone infections.
• Malaria: A parasitic disease transmitted through mosquito bites.
• Malignant Buotonneuse fever: A serious complication of Buotonneuse fever that tends to occur mainly in patients who are old or have other conditions such as heart disease.
• Malignant Jaundice: Jaundice due to an obstruction or blockage in one of the bile ducts by a tumor. Bile ducts are vessels which carry bile from the liver to the digestive system or gallbladder.
• Malignant obstructive biliary disease: Obstruction or blockage in one of the bile ducts by a tumor. Bile ducts are vessels which carry bile from the liver to the digestive system or gallbladder. Jaundice is usually one of the main symptoms.
• Mansonella ozzardi infection: Infection with a type of nematode (Mansonella ozzardi) found in Central America, South America and the Caribbean. Infection occurs through the bit of infected midges and blackflies.
• Marburg virus: Serious virus related to Ebola.
• Marchiafava-Micheli disease: A rare disorder involving episodes of hemoglobinuria that usually occur at night.
• Mareck's disease - visceral: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Marek disease: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Marek's disease: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Marine turtle poisoning: Marine turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Green Sea Turtle: Green Sea turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Hawksbill Turtle: Hawksbill turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Leatherback Turtle: Leatherback turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Loggerhead Turtle: Loggerhead turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Soft-shelled Turtle: Soft-shelled turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marseilles fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Mastocytosis: A disorder where excessive amounts of mast cells proliferate in organs such as the skin, liver, bone, spleen and gastrointestinal tract. Mast cells occur in connective tissue and defend the body against disease by releasing histamine to stimulate the immune system.
• Mauriac syndrome: A rare disorder characterized by diabetes, short stature and obesity.
• Mayapple poisoning: The Mayapple is a small flowering plant which is often found growing naturally. It bears small single flowers and apple-like fruit which turns yellow when ripe. The unripe fruit and leaves contain a chemical called podophyllin which can cause poisoning if eaten. The plant is considered highly toxic and death can occur if sufficient quantities are eaten. The leaves, roots and unripe fruit are toxic but the ripe fruit is edible. The plant has been used to treat venereal warts.
• Mc Leod neuroacanthocytosis syndrome: A rare syndrome characterized by neuromuscular, blood and central nervous system symptoms. The disease is slowly progressive.
• Meadows syndrome: A rare condition that affects pregnant women during the last trimester or within two months after birth. It is characterized by breathing difficult, chest pain, congestive heart failure, heart rhythm abnormalities, high blood pressure, gastrointestinal symptoms and embolisms.
• Meckel Syndrome: A rare genetic disorder involving numerous abnormalities and characterized by death within the first few weeks.
• Medium and long chan 3-hydroxyacyl-coenzyme A dehydrogenase deficiency: A metabolic disorder characterized by the deficiency of an enzyme (3-hydroxyacyl-coenzyme A dehydrogenase) which is needed to metabolise long and medum-chain fatty acids. The severity of symptoms may vary depending on the degree of the deficiency.
• Medium and short chain 3-hydroxyacyl-CoA dehydrogenase deficiency: A rare inherited form of biochemical disorder characterized by the deficiency of a particular enzyme (3-Hydroxyacyl-CoA Dehydrogenase). The enzyme deficiency only affects certain body tissues, in particular the skeletal muscles. The lack of enzyme activity prevents some fats being converted into energy. Symptoms tend to be exacerbated during fasting as during fasting, the body tries to rely more heavily on fats for energy. Fatty acids that are not completely metabolized due to the enzyme deficiency may build up in various organs and cause serious complications.
• Medium-Chain Acyl-CoA Dehydrogenase Deficiency: A rare disorder where the body lacks enzymes needed to convert some fats (medium-chain fatty acids) into energy and hence these fats build up in the body and cause damage.
• Megaloblastic Anemia 1: A rare genetic blood disorder where a defect in the vitamin B12 receptor means that it can't be absorbed from food during digestion. As a result of the low vitamin B12 levels, the body produces increased numbers of abnormal enlarged red blood cells (megaloblasts).
• Megaloblastic anemia: A rare blood disorder where insufficient vitamin B12 absorption results in reduced production of red blood cells and increased levels of abnormal, enlarged red blood cells (megaloblasts). Vitamin B12 insufficiency can result from absorption problems or lack of dietary intake of the vitamin or folic acid.
• Melanoma: Melanoma is the most dangerous type of skin cancer. It is the leading cause of death from skin disease. It involves cells called melanocytes, which produce a skin pigment called melanin. Melanin is responsible for skin and hair color.
• Melioidosis: Bacterial infection from soil or water.
• Meningitis: Infection of the membrane around the brain (as a symptom)
• Meningococcemia: A rare infectious disease whose main symptoms are upper respiratory tract infection, fever, rash and eye and ear problems.
• Menkes Disease: Genetic disease of copper deficiency.
• Mental retardation - macrocephaly - coarse facies - hypotonia: A very rare syndrome characterized mainly by mental retardation, large head, coarse face and reduced muscle tone.
• Metabolic disorder: occurs when abnormal chemical reactions occur in the body
• Metal-induced liver damage: Damage or injury to the liver caused by a exposure to a metal (usually ingestion). Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure.
• Methyldopa-induced Immune Hemolytic Anemia: Methyldopa-induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Methylmalonic acidemia, vitamin B12 responsive: A rare genetic disorder characterized by the impaired ability to make a chemical called adenosylcobalamin. Adenosylcobalamin is a derivative of vitamin B12. The defect results a biochemical abnormality which affects the body's normal biochemical functioning. The condition responds to the administration of vitamin B12.
• Methylmalonicaciduria, vitamin B12 unresponsive, mut 0: A metabolic disorder whose severity is somewhat variable - most patients die within months of birth with survivors having neurological problems. The condition involves abnormal metabolism of vitamin B12 which doesn't respond to treatment using vitamin B12 administration. This disorder is more severe than the mut (-) form
• Mevalonic aciduria: A rare disorder of amino acid metabolism characterized by a defect in the enzyme mevalonate kinase.
• Meyenburg-Altherr-Uehlinger syndrome: A rare disorder characterized by recurring inflammation of cartilage which results in deterioration of the cartilage. Any part of the body with cartilage may be affected - ears, nose bridge, larynx, trachea, aortic heart valve and around joints. Symptoms are determined by the part of the body that is affected. Breathing problems and even death can occur if the cartilage in the breathing airways or the aortic valve are affected.
• Microgastria short stature diabetes: A rare syndrome characterized by a very small stomach, diabetes and a growth hormone deficiency.
• Microsporidiosis: An infectious disease caused by a certain group of protozoa which form spores (microsporidia) e.g. Encephalitozoon, Enterocytozoon, Nosema, Pleistophora, Trachipleistophora, Vittaforma, Enterocytozoon bieneusi, Enterocytozoan )Septata) intestinalis). The protozoa invade and live inside the hosts cells. The release spores into the gastrointestinal tract where they are excreted and can infect other animals. The infection is often asymptomatic in healthy people but can cause serious symptoms affecting various parts of the body in immunocompromised people.
• Mild hereditary elliptocytosis in infancy: A rare inherited blood disorder characterized by the presence of oval-shaped red blood cells rather than the normal doughnut-shape as well as poikilocytosis. This form of hereditary elliptocytosis can cause hemolytic anemia.
• Mitochondrial DNA depletion syndrome: A group of conditions called mitochondrial disorders involving a reduced number of mitochondrial DNA in tissues rather than defective mitochondrial DNA. The disorders generally involve neurological symptoms which can occur during infancy or childhood. Symptoms will vary depending on the specific order involved.
• Mitochondrial trifunctional protein deficiency: A rare genetic condition where the body is unable to convert certain fats to energy. More specifically, there is insufficient levels of a particular enzyme needed to metabolize a type of fat called long-chain fatty acids.
• Molybdenum poisoning: A type of heavy metal poisoning caused by excessive exposure to molybdenum.
• Mononucleosis: Common infectious virus.
• Mosquito-borne diseases: Diseases that are carried by the mosquito
• Mosse syndrome: A condition involving the association of liver cirrhosis with polycythemia which is a chronic myeloproliferative disorder characterized by the excessive production of mainly red blood cells by the bone marrow.
• Mucopolysaccharidoses: A lysosomal storage disorder that is caused by a deficiency of the ability to metabolise glycosaminoglycans
• Mucopolysaccharidosis Type III: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans). This occurs due to there being not enough of the enzyme called N-acetyl-alpha-glucosaminidase (B) which is needed to break down the mucopolysacharides. The mucopolysaccharides are then deposited in various tissues.
• Mucopolysaccharidosis VII: Disorder of mucopolysaccharide metabolism.
• Mucopolysaccharidosis type 2 Hunter syndrome- mild form: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (iduronidate 2-sulfatase) needed to break it down.
• Mucopolysaccharidosis type 2 Hunter syndrome- severe form: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (iduronidate 2-sulfatase) needed to break it down.
• Mucopolysaccharidosis type 6: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (Arylsulfatase ?) needed to break it down.
• Mucopolysaccharidosis type 7 Sly syndrome: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (beta-glucuronidase) needed to break it down.
• Mucopolysaccharidosis type I Hurler syndrome: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (?-L-diuronidase) needed to break it down. There are three main sub-types of the disorder: Hurler - severe form, Scheie - mild form and Hurler/Scheie form which is intermediate.
• Mucopolysaccharidosis type I Hurler/Scheie syndrome: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (?-L-diuronidase) needed to break it down.
• Mulibrey Nanism syndrome: A very rare inherited malformation characterized by very small stature (dwarfism), pericardial constriction and yellow dots in fundus of the eye.
• Mullerian derivatives - lymphangiectasia - polydactyly: A rare syndrome characterized by extra fingers, Mullerian duct remnants and lymphangiectasia.
• Multi-centric Castleman's Disease: A rare disorder characterized by a generalized overgrowth of lymph node tissue which forms a tumor-like growth. This form of Castleman's disease is progressive and can be serious.
• Multifocal fibrosclerosis: A rare disorder characterized by the development of fibrous tissue that can occur in various parts of the body such as the retroperitoneum, mediastinum, eye area, bile ducts and thyroid gland. The severity and range of involvement is variable. There is no obvious cause for the condition.
• Multiple endocrine abnormalities - adenylyl cyclase dysfunction: A rare syndrome characterized by an enzyme defect (adenylyl cyclase) and a number of endocrine abnormalities.
• Multiple myeloma: A rare malignant cancer that occurs in the bone marrow. More common in skull, spine, rib cage, pelvis and legs.
• Muscle phosphoglycerate kinase deficiency: An inherited genetic muscle disease where an enzyme deficiency (phosphoglycerate kinase) affects the normal processes that convert carbohydrates from food into energy.
• Muscular fibrosis, multifocal - obstructed vessels: A very rare syndrome characterized mainly by
• Mycobacterial infections: Any infection that is caused by a mycobacterial organisms
• Mycosis fungoides: Mycosis fungoides is a rare form of T-cell lymphoma of the skin. The disease is typically slowly progressive and chronic.
• Mycosis fungoides, familial: A rare form of lymphatic cancer (T-cell lymphoma) that primarily affects the skin and tends to occur with higher than normal frequency within a family. The skin is affected first, then the lymph nodes become inflamed and usually cancerous. The cancer can then spread to organs such as the liver, lungs and bone marrow. Survival depends on how early treatment starts. Patients diagnosed in the early stages can survive more than 12 years whereas once the cancer has spread to other organs, death usually occurs within three years.
• Mycotoxin-induced liver damage - Aflatoxin: Damage or injury to the liver caused by exposure to a mycotoxin called Aflatoxin which is a toxin produced by some Aspergillus fungus. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Cyclochlorotine: Damage or injury to the liver caused by exposure to a Mycotoxin called Cyclochlorotine which is a toxin produced by a particular fungus (Penicillium islandicum). Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Luteoskyrins: Damage or injury to the liver caused by exposure to a Mycotoxin called Luteoskyrin which is a toxin produced by a particular fungus (Penicillium islandicum). Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Ochratoxin: Damage or injury to the liver caused by exposure to a Mycotoxin called Ochratoxin. Ochratoxin is a toxin produced by a particular fungus (Aspergillus ochraceus and Penicillium verrucosum). It is a relatively common food contaminant and can be found in mouldy grain, pork, coffee and dried grapes. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Rubratoxin: Damage or injury to the liver caused by exposure to a Mycotoxin called Rubratoxin. Rubratoxin is a toxin produced by a particular fungus (Penicillium rubrum and purpurogenum) which is most commonly found on cereal grains. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Sterigmatocystin: Damage or injury to the liver caused by exposure to a Mycotoxin called Sterigmatocystin. It is a toxin produced by a particular fungus (Aspergillus). Sterigmatocystin may also increase the risk of developing liver cancer. Most likely sources of exposure are foods such as wheat, maize, hard cheese and green coffee beans contaminated with mould. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Myelodysplastic syndromes: A group of syndromes characterized by a disruption in the production of blood cells. Often the bone marrow increases production of various blood cells but because many of them are defective, they are destroyed before the reach the blood stream.
• Myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
• Myelofibrosis-osteosclerosis: Formation of fibrous tissue in the bone marrow in response to bone marrow damage.
• Myeloid Sarcoma: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Myeloid splenomegaly: Spleen enlargement caused by myeloid cell (any leukocyte that isn't a lymphocyte) proliferation. The condition occurs with no obvious cause and is thus termed idiopathic.
• Myelpathic anemia: Myelopathic anemia is a form of anemia resulting from the development of abnormal tissue in the bone marrow - usually metastatic cancers. It is characterized by abnormal number of immature blood cells in the blood.
• Myxedema: The most severe form of hypothyroidism characterized by swelling of extremities and face.
• NADH CoQ reductase, deficiency of: A rare genetic defect where an enzyme deficiency (NADH CoQ) disrupts cellular processes and causes various organic acid disorders. Any of a variety of the components of the enzyme may be missing or defective and hence the clinical presentation and severity may vary. Presentation may range from infantile death to various disorders neurodegenerative disorders such as Leigh's disease, Parkinson's disease and cardiomyopathy.
• NASH syndrome: A form of fatty liver where fat and fibrous tissue accumulate in the liver. Fatty liver is normally seen in alcoholic patients but in NASH syndrome, alcohol is not a factor. The condition may be generally asymptomatic or in some cases may lead to progressive scarring (cirrhosis) of the liver. The condition has a variable progression.
• NISCH syndrome: A rare syndrome characterized by sparse scalp hair, scaly skin and inflammation and fibrosis of the bile ducts.
• NOMID syndrome: A rare autoinflammatory disease characterized by fever, rash, arthritic changes, eye problems and chronic meningitis.
• Nakajo-Nishimura syndrome: A rare disorder involving muscle degeneration, loss of skin fat and impaired immune functioning.
• Narcotic addiction: An uncontrollable desire to use narcotics on a regular basis. The drug may be used as a therapeutic medication for various conditions but it's use is also frequently abused. Examples of narcotic drugs include heroin, morphine, Demerol and codeine. Frequent use leads to an increased tolerance to the drug so higher and higher doses are required to achieve the desired euphoric feeling.
• Navajo neurohepatopathy: A rare genetic disease found in Navajo populations. It involves peripheral nerve degeneration, liver disease and corneal ulcers. The genetic disease is believed to be caused by maternal exposure to uranium from waters contaminated by old mines.
• Neonatal ALD: Progressive form of ALD in newborns.
• Neonatal bacterial meningitis: Bacterial meningitis that occurs in an infant under 3 months of age. Bacterial meningitis is a bacterial brain infection.
• Neonatal hepatitis: A rare life-threatening condition where iron accumulates in the liver of the fetus and produces severe symptoms after birth. Death is common during the fetal or infant stage.
• Neonatal jaundice: A yellow discolouration of the skin which sometimes occurs in newborns
• Neonatal sepsis: Bacterial blood infection in an infant under 3 months of age.
• Neoplastic porphyria tarda: A rare condition where a liver tumor causes a deficiency of the enzyme hepatic uroporphyrinogen decarboxylase. The deficiency causes a build up of porphyrins in the liver and the skin which causes damage to the skin and various other symptoms.
• Nephroblastomatosis - fetal ascites - macrosomia - wilms tumor: A rare condition characterized by kidney abnormalities, macroxomia, endocrine pancreas abnormalities, large fetus and mental retardation.
• Nephroblastomatosis, fetal ascites, macrosomia and Wilms tumor: A rare condition characterized by kidney abnormalities, macroxomia, endocrine pancreas abnormalities, large fetus and mental retardation.
• Nephronophthisis - hepatic fibrosis: A rare syndrome characterized by progressive destruction of kidney tissue (nephronophthisis) which occurs from birth and results in kidney failure and liver fibrosis.
• Neuraminidase deficiency: A rare inherited disorder involving an enzyme (neuraminidase and beta-galactosidase) deficiency which results in a build up of glycoproteins (sialyloligosaccharides) in the urine.
• Neuroblastoma: neuroblastoma is a malignant (cancerous) tumor of infants and children that develops from nerve tissue
• Neuroma biliary tract: A benign nerve tumor that occurs in the biliary tract. Often occurs after certain surgeries such as gall bladder removal.
• Niacin toxicity: Excessive consumption of niacin can cause symptoms of toxicity.
• Niemann-Pick disease: A rare inherited biochemical disorder involving the deficiency of an enzyme (acid sphingomyelinase) needed to break down certain lipids which results in an accumulation of these lipids in the body.
• Niemann-Pick disease, type A: Niemann-Pick disease is an inherited metabolic disorder where lipids are not able to be metabolized adequately and hence build up and cause damage in various parts of the body such as the spleen, liver, lungs, bone marrow and brain. The different types of the disease are due to different genetic mutations. Type A is a severe infantile form in which death usually occurs by early childhood.
• Niemann-Pick disease, type B: Niemann-Pick disease is an inherited metabolic disorder where lipids are not able to be metabolized adequately and hence build up and cause damage in various parts of the body such as the spleen, liver, lungs, bone marrow and brain. The different types of the disease are due to different genetic mutations. Type B is a chronic form of the condition with no neurological involvement and patients can survive into adulthood.
• Non functioning pancreatic endocrine tumor: A tumor of the pancreas that does not result in an increased hormone production but can cause symptoms when the tumor becomes big enough to push against other structures. The tumor may be malignant or benign.
• Non-Hodgkin's lymphoma: A condition which is any neoplastic disorder of the lymphoid tissue
• Non-Hodgkin's lymphoma, during pregnancy: A cancer that originates in the lymphatic system and occurs during pregnancy. The greatest problem is the fact that the cancer is usually quite aggressive and delays in delivery often results in delayed treatment and a poor prognosis.
• Non-alcoholic steatohepatitis (NASH): A form of fatty liver where fat and fibrous tissue accumulate in the liver. Fatty liver is normally seen in alcoholic patients but in NASH syndrome, alcohol is not a factor. The condition may be generally asymptomatic or in some cases may lead to progressive scarring (cirrhosis) of the liver. The condition has a variable progression.
• Non-hereditary spherocytic anemia: Non-inherited spherocytic anemia is an acquired blood disorder characterized by red blood cells that are shaped like a sphere (spherocytes) rather than the normal doughnut shape. These abnormal cells are broken down prematurely resulting in a shortage of red blood cells (anemia). The blood condition may result from such things as acute, widespread burns to the body, low blood phosphate levels, Zieve's syndrome and toxins from poisonous snakes, spiders and insects.
• Obliterative portal venopathy: A rare condition characterized by abnormal hardening of liver blood vessels (portal veins), portal hypertension and an enlarged spleen. The condition usually occurs in adults but can rarely occur in children. The exact cause of the condition is unknown but various factors have been implicated e.g. genetic predisposition, workplace exposure to inorganic arsenicals, thorium compounds and vinyl chloride monomer.
• Obstructive biliary disease: A disease process that causes obstruction or blockage in one of the bile ducts which are vessels that carry bile from the liver to the digestive system or gallbladder. Diseases that can cause such obstructions includes tumors, gallstones, parasites, bile duct inflammation, trauma to the bile duct and biliary strictures.
• Occupational Cancer - Liver cancer: Occupational exposure to vinyl chloride can increase the risk of developing liver cancer.
• Occupational liver damage - 1,1,1-Tetrachloroethane: Damage or injury to the liver caused by exposure to 1,1,1-Tetrachloroethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 1,1,2-Tetrachloroethane: Damage or injury to the liver caused by exposure to 1,1,2-Tetrachloroethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 1,2-Dibromoethane: Damage or injury to the liver caused by exposure to 1,2-Dibromoethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 1,2-Dichloroethane: Damage or injury to the liver caused by exposure to 1,2-Dichloroethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 2-Nitropropane: Damage or injury to the liver caused by exposure to 2-Nitropropane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 2-acetylamino-fluorene: Damage or injury to the liver caused by exposure to 2-acetylamino-fluorene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 3,3-Dichlorobenzidine: Damage or injury to the liver caused by exposure to 3,3-Dichlorobenzidine and all its salts in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 4-Dimethylaminoazobenzene: Damage or injury to the liver caused by exposure to 4-Dimethylaminoazobenzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Acetates: Damage or injury to the liver caused by exposure to various Acetates in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Acetonitrile: Damage or injury to the liver caused by exposure to Acetonitrile in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Acrylonitrile: Damage or injury to the liver caused by exposure to Acrylonitrile in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Alcohol: Damage or injury to the liver caused by exposure to alcohol in an occupational (industrial) setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Alicyclic Hydrocarbons: Damage or injury to the liver caused by exposure to alicyclic hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aliphatic Amines: Damage or injury to the liver caused by exposure to Aliphatic Amines in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aliphatic Hydrocarbons: Damage or injury to the liver caused by exposure to certain aliphatic hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aliphatic hydrogenated hydrocarbons: Damage or injury to the liver caused by exposure to aliphatic hydrogenated hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Allyl alcohol: Damage or injury to the liver caused by exposure to allyl alcohol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Amyl acetate: Damage or injury to the liver caused by exposure to Amyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aromatic Hydrocarbons: Damage or injury to the liver caused by exposure to certain Aromatic Hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aromatic amines: Damage or injury to the liver caused by exposure to certain aromatic amines (e.g. 2-acetylamino-fluorene) in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aromatic halogenated hydrocarbons: Damage or injury to the liver caused by exposure to certain aromatic halogenated hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Arsenic: Damage or injury to the liver caused by exposure to arsenic in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Arsine: Damage or injury to the liver caused by exposure to Arsine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Benzene: Damage or injury to the liver caused by exposure to Benzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Benzyl chloride: Damage or injury to the liver caused by exposure to Benzyl chloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Beryllium: Damage or injury to the liver caused by exposure to beryllium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Beta-Propiolactone: Damage or injury to the liver caused by exposure to Beta-Propiolactone in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Bipyridyl pesticides: Damage or injury to the liver caused by exposure to Bipyridyl pesticides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Bismuth: Damage or injury to the liver caused by exposure to bismuth and bismuth compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Boron: Damage or injury to the liver caused by exposure to boron and boron compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Boron hydrides: Damage or injury to the liver caused by exposure to Boron hydride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Bromide: Damage or injury to the liver caused by exposure to Bromide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Cadmium: Damage or injury to the liver caused by exposure to Cadmium and Cadmium compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Carbolic Acids and Anhydrides: Damage or injury to the liver caused by exposure to carbolic acids and anhydrides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Carbon Disulfide: Damage or injury to the liver caused by exposure to Carbon Disulfide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Carbon tetrachloride: Damage or injury to the liver caused by exposure to Carbon tetrachloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Carbonyls (metal): Damage or injury to the liver caused by exposure to Carbonyls (metal) in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chlorinated benzenes: Damage or injury to the liver caused by exposure to Chlorinated benzenes in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chlorinated naphthalenes: Damage or injury to the liver caused by exposure to Chlorinated naphthalenes in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chlorodiphenyls and derivatives: Damage or injury to the liver caused by exposure to Chlorodiphenyls and derivatives in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chloroform: Damage or injury to the liver caused by exposure to chloroform in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chloroprene: Damage or injury to the liver caused by exposure to chloroprene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chromium: Damage or injury to the liver caused by exposure to chromium and chromium compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Copper: Damage or injury to the liver caused by exposure to copper in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Cresol: Damage or injury to the liver caused by exposure to Cresol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Cyclopropane: Damage or injury to the liver caused by exposure to cyclopropane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dibromochloropropane: Damage or injury to the liver caused by exposure to Dibromochloropropane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dimethyl sulfate: Damage or injury to the liver caused by exposure to Dimethyl sulfate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dimethylnitrosamine: Damage or injury to the liver caused by exposure to Dimethylnitrosamine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dinitrobenzene: Damage or injury to the liver caused by exposure to Dinitrobenzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dinitrophenol: Damage or injury to the liver caused by exposure to Dinitrophenol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dinitrotoluene: Damage or injury to the liver caused by exposure to Dinitrotoluene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Diphenyl: Damage or injury to the liver caused by exposure to Diphenyl in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethanolamines: Damage or injury to the liver caused by exposure to Ethanolamines in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethyl Acetate: Damage or injury to the liver caused by exposure to Ethyl Acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethyl Ether: Damage or injury to the liver caused by exposure to ethyl ether in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethyl Salicylate: Damage or injury to the liver caused by exposure to Ethyl Salicylate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethyl alcohol: Damage or injury to the liver caused by exposure to ethyl alcohol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylene Dibromide: Damage or injury to the liver caused by exposure to Ethylene Dibromide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylene chlorohydrin: Damage or injury to the liver caused by exposure to ethylene chlorohydrin in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylene dichloride: Damage or injury to the liver caused by exposure to Ethylene dichloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylene oxide: Damage or injury to the liver caused by exposure to Ethylene oxide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylenediamine: Damage or injury to the liver caused by exposure to Ethylenediamine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Germanium: Damage or injury to the liver caused by exposure to Germanium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Hydrazine and derivatives: Damage or injury to the liver caused by exposure to Hydrazine and its derivatives in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Hydrogen Cyanide: Damage or injury to the liver caused by exposure to Hydrogen Cyanide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Hydrogen bromides: Damage or injury to the liver caused by exposure to Hydrogen bromides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ionizing radiation: Damage or injury to the liver caused by exposure to Ionizing radiation in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Iron: Damage or injury to the liver caused by exposure to Iron in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Isopropyl acetate: Damage or injury to the liver caused by exposure to Isopropyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Kepone pesticides: Damage or injury to the liver caused by exposure to Kepone pesticides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Mercaptans: Damage or injury to the liver caused by exposure to Mercaptans in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methyl Bromide: Damage or injury to the liver caused by exposure to Methyl Bromide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methyl Chloride: Damage or injury to the liver caused by exposure to Methyl Chloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methyl acetate: Damage or injury to the liver caused by exposure to Methyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methylene chloride: Damage or injury to the liver caused by exposure to Methylene chloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methylene dianiline: Damage or injury to the liver caused by exposure to Methylene dianiline in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N,N-Dimethylformamide: Damage or injury to the liver caused by exposure to N,N-Dimethylformamide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N-N-Dimethylacetamide: Damage or injury to the liver caused by exposure to N-N-Dimethylacetamide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N-Nitrosodimethylamine: Damage or injury to the liver caused by exposure to N-Nitrosodimethylamine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N-butyl acetate: Damage or injury to the liver caused by exposure to N-butyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N-propyl acetate: Damage or injury to the liver caused by exposure to N-propyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Naphthalene: Damage or injury to the liver caused by exposure to Naphthalene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Naphthol: Damage or injury to the liver caused by exposure to Naphthol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nickel: Damage or injury to the liver caused by exposure to nickel and nickel compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitriles: Damage or injury to the liver caused by exposure to certain nitriles in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitrobenzene: Damage or injury to the liver caused by exposure to Nitrobenzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitromethane: Damage or injury to the liver caused by exposure to Nitromethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitroparaffins: Damage or injury to the liver caused by exposure to Nitroparaffins in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitrophenol: Damage or injury to the liver caused by exposure to Nitrophenol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Phenol: Damage or injury to the liver caused by exposure to Phenol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Phosphine: Damage or injury to the liver caused by exposure to Phosphine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Phosphorus: Damage or injury to the liver caused by exposure to Phosphorus in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Phthalic Anhydride: Damage or injury to the liver caused by exposure to phthalic anhydride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Picric Acid: Damage or injury to the liver caused by exposure to Picric Acid in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Polybrominated biphenyls: Damage or injury to the liver caused by exposure to Polybrominated biphenyls in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Polychlorinated biphenyls: Damage or injury to the liver caused by exposure to Polychlorinated biphenyls in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Propylene dichloride: Damage or injury to the liver caused by exposure to Propylene dichloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Pyridine: Damage or injury to the liver caused by exposure to Pyridine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Pyrogallol: Damage or injury to the liver caused by exposure to Pyrogallol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Selenium: Damage or injury to the liver caused by exposure to selenium and selenium compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Stibine: Damage or injury to the liver caused by exposure to Stibine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Styrene/ethyl benzene: Damage or injury to the liver caused by exposure to Styrene/ethyl benzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tetrachloroethane: Damage or injury to the liver caused by exposure to Tetrachloroethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tetrachloroethylene: Damage or injury to the liver caused by exposure to Tetrachloroethylene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tetramethylthiuram disulfide: Damage or injury to the liver caused by exposure to Tetramethylthiuram disulfide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tetryl: Damage or injury to the liver caused by exposure to Tetryl in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Thallium: Damage or injury to the liver caused by exposure to Thallium and Thallium compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Thallium sulfate pesticides: Damage or injury to the liver caused by exposure to Thallium sulfate pesticides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Thorium dioxide: Damage or injury to the liver caused by exposure to Thorium dioxide (Thorotrast) in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tin: Damage or injury to the liver caused by exposure to tin and tin compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Toluene: Damage or injury to the liver caused by exposure to Toluene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Trichloroethylene: Damage or injury to the liver caused by exposure to Trichloroethylene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Trinitrotoluene: Damage or injury to the liver caused by exposure to Trinitrotoluene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Turpentine: Damage or injury to the liver caused by exposure to Turpentine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Uranium: Damage or injury to the liver caused by exposure to Uranium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Vinyl Chloride: Damage or injury to the liver caused by exposure to Vinyl Chloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Whole body vibration: Damage or injury to the liver caused by whole body vibration in an occupational setting. This often occurs when operating equipment which cause constant physical vibration such as occurs when driving off-road vehicles or forklifts. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Xylene: Damage or injury to the liver caused by exposure to Xylene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - n-Heptane: Damage or injury to the liver caused by exposure to n-Heptane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational metal-induced liver damage - Antimony: Damage or injury to the liver caused by a exposure to antimony in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Arsenic: Damage or injury to the liver caused by a exposure to arsenic in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Barium: Damage or injury to the liver caused by a exposure to barium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Beryllium: Damage or injury to the liver caused by a exposure to beryllium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Bismuth: Damage or injury to the liver caused by a exposure to bismuth in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Boranes: Damage or injury to the liver caused by a exposure to boranes in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Boron: Damage or injury to the liver caused by exposure to boron in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Cadmium: Damage or injury to the liver caused by exposure to Cadmium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Chromium: Damage or injury to the liver caused by a exposure to chromium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Cobalt: Damage or injury to the liver caused by exposure to Cobalt in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Copper: Damage or injury to the liver caused by exposure to copper in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Germanium: Damage or injury to the liver caused by a exposure to germanium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Gold: Damage or injury to the liver caused by exposure to gold in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Hafnium: Damage or injury to the liver caused by a exposure to hafnium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Halides: Damage or injury to the liver caused by a exposure to halides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Hydrazines: Damage or injury to the liver caused by a exposure to hydrazines in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Iron: Damage or injury to the liver caused by a exposure to iron in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Lanthanides: Damage or injury to the liver caused by a exposure to Lanthanides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Lead: Damage or injury to the liver caused by a exposure to lead in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Manganese: Damage or injury to the liver caused by a exposure to manganese in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Mercury: Damage or injury to the liver caused by exposure to mercury in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Molybdenum: Damage or injury to the liver caused by a exposure to molybdenum in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Nickel: Damage or injury to the liver caused by a exposure to nickel in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Niobium: Damage or injury to the liver caused by a exposure to niobium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Phosphorus: Damage or injury to the liver caused by a exposure to phosphorus in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Selenium: Damage or injury to the liver caused by a exposure to selenium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Tellurium: Damage or injury to the liver caused by a exposure to tellurium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Thallium: Damage or injury to the liver caused by a exposure to thallium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Tin: Damage or injury to the liver caused by a exposure to tin in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Oesophagostomiasis: A parasitic intestinal infection caused by a nematode called Oesophagostomum bifurcum. The parasite commonly infects monkeys, goats, cattle, sheep and pigs. The infection is relatively rare in humans but is most commonly found in northern Togo and Ghana. Transmission usually occurs through oral contact with contaminated soil.
• Okamuto Satomura syndrome: A very rare syndrome characterized mainly by kidney, heart and facial abnormalities as well as reduced muscle tone and mental retardation.
• Omenn syndrome: A rare inherited disorder of the immune system involving B and T lymphocytes and characterized by skin rash and frequent infections.
• Omsk hemorrhagic fever: A hemorrhagic fever caused by a virus. A serious outbreak occurred in Omsk and hence the name. Transmission occurs through tick bites. The infection has two phases: the first acute phase involves symptoms such as fever, rash and muscle pain and the second phase occurs after a week or two and involves the central nervous system (e.g. delirium, convulsions).
• Opioid addiction: An uncontrollable desire to use opioids on a regular basis. Opioids may may be prescribed by a physician for the purpose of pain relief but patients may become physically dependent on the drug and continue to obsessively use it even after the condition it was prescribed for has resolved. In other cases, opioid addiction results from the illicit use of the drug for recreational purposes. Frequent use leads to an increased tolerance to the drug so higher and higher doses are required to achieve the desired euphoric feeling. Examples of opioids includes morphine, heroin, oxycodone and fentanyl.
• Opisthorchiasis: Infection with a type of fluke (Southeast Asian liver fluke or cat liver fluke). Infection usually occurs by consuming infected undercooked fish. Acute infection may cause fever, joint pain, rash, eosinophilia and lymphadenopathy where as chronic infections may cause enlarged liver, malnutrition. Mild cases can cause constipation, diarrhea and abdominal pain. Occasionally, the infection may be asymptomatic and in the other extreme, severe cases may result in complications such as cholangiocarcinoma.
• Opium addiction: An uncontrollable desire to use opium on a regular basis. Opium may may be prescribed by a physician for the purpose of pain relief but patients may become physically dependent on the drug and continue to obsessively use it even after the condition it was prescribed for has resolved. In other cases, opium addiction results from the illicit use of the drug for recreational purposes. Frequent use leads to an increased tolerance to the drug so higher and higher doses are required to achieve the desired euphoric feeling.
• Organic acidemia: High blood levels of organic acids which is caused by abnormal protein metabolism. Maple syrup urine disease and propionic academia are examples of organic acidemias. Deficiency of certain metabolic enzymes one of the main causes of organic academia.
• Ornithine Transcarbamylase Deficiency: A rare metabolic disorder where a deficiency of the enzyme ornithine transcarbamylase means that nitrogen isn't removed from the body and builds up in the blood in the form of ammonia.
• Ornithine transcarbamylase (OTC) Deficiency: A rare metabolic disorder where a deficiency of the enzyme ornithine transcarbamylase means that nitrogen isn't removed from the body and builds up in the blood in the form of ammonia.
• Orotic aciduria hereditary: A rare metabolic disorder where an inherited enzyme deficiency (lack of orotidylic pyrophosphorylase and orotidylic decarboxylase or a lack of orotidylic decarboxylase only) impairs the body's ability to break down orotic acid which then builds up in the body and causes damage.
• Osler-Vaquez disease: An uncommon chronic blood disease involving an increased red blood cell count.
• Osteopetrosis lethal: A severe, lethal form of the brittle bone condition called osteopetrosis.
• Osteopetrosis, autosomal recessive 2: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. This form is relatively mild with survival possible for a couple of decades in some cases.
• Osteopetrosis, autosomal recessive 4: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. The malignant infantile form is the most severe form of this disorder and death usually occurs in the first decade of life.
• Osteopetrosis, intermediate form: A recessively inherited bone disease characterized bybrittle bones with increased density. The intermediate form is less severe than the infantile form but more severe than the adult form.
• Osteopetrosis, mild autosomal recessive form: A rare disorder characterized by abnormally increased bone density as old bone is not resorbed and replaced with new bone. This form is relatively mild.
• Overhydrated hereditary stomatocytosis: A rare disorder where abnormal red blood cell membranes fail to regulate movement of sodium and potassium into and out of the red blood cell. The red blood cell accumulates too much salt and water which causes it to burst. Destruction of red blood cells leads to anemia. The red blood cells are abnormally shaped and resemble a stoma or mouth.
• PEPCK 1 deficiency: A rare inherited disorder where deficiency of an enzyme (phosphoenolpyruvate carboxykinase-1) prevents fats and proteins being turned into glucose which causes various problems. In type 1, the enzyme deficiency occurs in the liquid part of the cells.
• PEPCK 2 deficiency: A rare inherited disorder where deficiency of an enzyme (phosphoenolpyruvate carboxykinase-2) prevents fats and proteins being turned into glucose which causes various problems. In type 2 the deficiency occurs in the mitochondrial part of the cell which is the energy conversion organelle in the cell.
• PFIC: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage. End-stage liver disease usually occurs before adulthood. There are three different subtypes of the disorder, each with a different genetic origin of the defect.
• POEMS syndrome: A very rare disorder that has widespread effects on the body: P - polyneuropathy, O - organopathy, E - endocrinopathy, M - monoclonal gammopathy, S - skin changes.
• Pancreas symptoms: Symptoms affecting the pancreas gland
• Pancreatic Acinar Cell Tumors: Pancreatic acinar cell tumors are a rare type of tumor that develops from acinar cells in the pancreas. Normal acinar cells secrete enzymes and hence acinar cells in the tumor will also usually secrete additional enzymes - mostly lipase. This type of tumor usually occurs in old age.
• Pancreatic Islet Cell Cancer: A malignant carcinoma that is located in the islet cells of the pancreas
• Pancreatic adenoma: A pancreatic tumor which may be benign or malignant. Symptoms may vary depending on the location and size of the tumor as well as whether the tumor secretes hormones or not. For example, the tumor may block the biliary duct.
• Pancreatic cancer: Pancreatic cancer is a malignant neoplasm of the pancreas
• Pancreatic cancer, adult: Cancer that develops in the tissues of the pancreas. The pancreas produces digestive juices and certain hormones such as insulin that regulate sugar levels in the body and tumors can in some cases causes excessive hormone secretion.
• Pancreatic carcinoma, familial: Cancer (malignant) that develops in the pancreas and tends to run in families. The pancreas produces digestive juices and certain hormones such as insulin that regulate sugar levels in the body.
• Pancreatic islet cell tumors (non-functioning tumor): A tumor that arises from the pancreatic islet cells and may involve cells that produce excess pancreatic hormones.
• Pancreatoblastoma: A very rare pancreatic tumor that occurs almost exclusively in children. The tumor can grow large enough to push against and hence obstruct the passage of material in the gastrointestinal tract.
• Pearson's anemia: Pearson's anemia is a rare progressive condition characterized by abnormal pancreatic functioning and sideroblastic anemia. Diagnosis usually occurs within the first 7 years of life and death in infancy is common.
• Penicillin-induced Immune Hemolytic Anemia: Penicillin-induced immune hemolytic anemia is a condition where use of a medication called Penicillin triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Percocet overdose: Percocet is a prescription drug used to treat pain. Excessive doses of the drug can result in various symptoms and even death in severe cases.
• Perinatal-lethal Gaucher disease: A disorder caused by a deficiency of an enzyme called glucocerebrosidase. The foetal form is one of several forms of Gaucher disease and is the rarest. The fetal form causes death before birth or soon after.
• Periodic hyperlysinemia: A rare genetic disorder where the body lacks enzymes to metabolize lysine which then causes a harmful builds up of lysine and ammonia in the body.
• Peripartum cardiomyopathy: A rare condition involving weakening of the heart in the final stages of pregnancy or within five months of giving birth. The cause of the condition is unknown and patients usually have no history of heart disease. The breathing difficulty associated with the condition is often ignored as a normal symptom in the late stages of pregnancy which increases the risk of potentially fatal complications developing.
• Pernicious anemia: A megaloblastic anaemia due to malabsorption of the vitamin B12
• Peroxisomal bifunctional enzyme deficiency: A rare disorder involving abnormal steroid metabolism due to an enzyme 17-beta-hydroxysteroid dehydrogenase 4) deficiency. The symptoms which make the condition appear very similar to another condition called neonatal adrenoleukodystrophy.
• Peroxisome biogenesis disorders: A group of inherited disorders involving the absence or dysfunction of one or more peroxisomal enzymes. Peroxisomes are numerous tiny organs within the cell which are involved in a large number of the body's biochemical reactions.
• Pfeiffer-Weber-Christian syndrome: A rare skin disorder characterized by recurring inflammation of the fatty layer of the skin and the development of nodules which can vary in size from 1 to 2 cm. The face and hands are not usually affected and the legs and feet tend to be the most commonly affected. The condition tends to reoccur within weeks or months.
• Pfiesteria piscicida poisoning: Pfiesteria piscicida is an estuarine microorganism (dinoflagellate) that can cause illness in humans as well as fish. The particular toxin involved has not yet been identified. The microorganism may release toxins into the water or it may be aerosolized which can result in skin, eye and respiratory exposure. The condition is not contagious and they symptoms may vary considerably amongst patients.
• Pfiesteria poisoning: Pfiesteria is an estuarine microorganism (dinoflagellate) that can cause illness in humans as well as fish. The particular toxin involved has not yet been identified. The microorganism may release toxins into the water or it may be aerosolized which can result in skin, eye and respiratory exposure. The condition is not contagious and they symptoms may vary considerably amongst patients.
• Pfiesteria shumwayae poisoning: Pfiesteria shumwayae is an estuarine microorganism (dinoflagellate) that can cause illness in humans as well as fish. The particular toxin involved has not yet been identified. The microorganism may release toxins into the water or it may be aerosolized which can result in skin, eye and respiratory exposure. The condition is not contagious and they symptoms may vary considerably amongst patients.
• Phenothiazine antenatal infection: Maternal use of a drug called phenothiazine has not been proven to cause problems in offspring. Animal studies show there is a risk but no definitive studies have been done on humans. Phenothiazine is used to treat mental and emotional disorders such as schizophrenia. The biggest risks are likely to occur during the first trimester (malformations) and towards the end of the pregnancy (poor muscle tone, poor reflexes and jaundice).
• Philadelphia-negative chronic myeloid leukemia: A form of leukemia.
• Phocomelia - thrombocytopenia - encephalocele - urogenital malformation: An early congenital condition that is characterized by multiple congenital anomalies. The severity and range of abnormalities may vary amongst patients.
• Phosphoenolpyruvate carboxykinase (PEPCK) deficiency: A rare inherited disorder where deficiency of an enzyme (phosphoenolpyruvate carboxykinase-1) prevents fats and proteins being turned into glucose which causes various problems. In type 1, the enzyme deficiency occurs in the liquid part of the cells and in type 2 the deficiency occurs in the mitochondrial part of the cell.
• Plant poisoning - Amygdalin: Amygdalin is a chemical found naturally in various plants e.g. stone fruit kernels and raw almonds. Eating these parts of the plant that contain the chemical can cause symptoms of cyanide poisoning as the amygdalin is converted to cyanide by the digestive process. Obviously, the concentration of the chemical varies amongst species of plant and often, significant quantities are needed to produce symptoms. Nevertheless, severe poisoning can result in death. Amygdalin is believed by some to inhibit cancers but there has been no conclusive proof of this.
• Plant poisoning - Calcium oxalate crystals: Calcium oxalate crystals is a chemical found naturally in plants such as dumb cane and rhubarb leaves. The amount of calcium oxalate crystals varies amongst species of plant. The crystals are quite sharp and abrasive and ingestion of plants containing them can cause abrasive and irritation injuries. Eating large amounts can cause kidney and liver damage and even death in serious cases.
• Plant poisoning - Comfrey (Senecio, Symphytum): Ingestion of parts of the Comfrey plant may cause liver problems.
• Plant poisoning - Crotalaria: Ingestion of parts of the Crotalaria plant may cause liver problems.
• Plant poisoning - Cyanogenic glycoside: Cyanogenic glycoside is a toxin found naturally in various plants e.g. cherries, plums, almonds, peaches, apricots, apples and cassava. The chemical is usually concentrated in the seeds, kernels or wilted leaves. Eating these parts of the plant that contain the chemical can cause symptoms of cyanide poisoning as the cyanogenic glycoside is converted to cyanide by the digestive process. Even chewing the leaves can result in conversion to cyanide due to the presence of digestive enzymes in the mouth. Obviously, the concentration of the chemical varies amongst species of plant and often, significant quantities are needed to produce symptoms. Nevertheless, severe poisoning can result in death.
• Plant poisoning - Heliatropium: Ingestion of parts of the Heliatropium plant may cause liver problems.
• Plant poisoning - Hydroquinone: Hydroquinone is a chemical metabolized by the human digestive system from a naturally occurring chemical called arbutin found in the leaves of plants such as blueberries, cranberries, bearberries and red whortleberries. The main symptoms are irritation of the gastrointestinal mucosa but severe poisoning can cause systemic symptoms.
• Plant poisoning - Pyrrolizidine alkaloids: Pyrrolizidine alkaloids is a toxin found naturally in plants such as comfrey. The main symptoms are gastrointestinal which can range in severity depending on the amount consumed.
• Plant poisoning - Tannin: Tannin is a bitter-tasting chemical found in many types of plants. Most plants contain insufficient quantities to cause any real problem. Acorns contain tannins and can cause symptoms if sufficient quantities of untreated nuts are eaten.
• Plant poisoning - Toxalbumin: Toxalbumin is a toxin found naturally in plants such as the castor bean. The severity of symptoms depends on the particular species of plant involved and the amount consumed. For example the toxalbumin called ricin found in castor beans is extremely toxic.
• Plant toxin-induced liver damage - Albitocin: Damage or injury to the liver caused by exposure to a toxin called albitocin found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Cycasin: Damage or injury to the liver caused by exposure to a toxin called cycasin found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Icterogenin: Damage or injury to the liver caused by exposure to a toxin called Icterogenin found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Indospicine: Damage or injury to the liver caused by exposure to a toxin called Indospicine found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Lanthana: Damage or injury to the liver caused by exposure to a toxin called Lanthana found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Ngaione: Damage or injury to the liver caused by exposure to a toxin called Ngaione found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Nutmeg: Damage or injury to the liver caused by exposure to a toxin called Nutmeg found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Pyrrolidizine: Damage or injury to the liver caused by exposure to a toxin called Pyrrolidizine found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Safrole: Damage or injury to the liver caused by exposure to a toxin called Safrole found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Tannic Acid: Damage or injury to the liver caused by exposure to a toxin called Tannic Acid found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plasma cell leukemia: A form of leukemia characterized by the proliferation of plasma cells in the peripheral blood system. The prognosis is generally poor for this usually aggressive condition.
• Polar bear poisoning: Polar bears are often used as a food source by the arctic inhabitants. Eating the liver and kidneys of the polar bear is believed to result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases. Eating more than 200 grams of Polar bear liver can result in human death however death is considered rare.
• Polyarteritis nodosa: A serious blood vessel disease where small and medium-sized arteries become swollen and damaged and are unable to adequately supply oxygenated blood to various tissues in the body. The disease can occur in a mild form or a serious, rapidly fatal form.
• Polyarthritis, systemic: A chronic inflammatory disease (usually autoimmune) that causes inflammation in multiple parts of the body and causes arthritis in five or more joints.
• Polycystic kidney disease: Genetic kidney disease causing kidney cysts.
• Polycystic kidney disease type 1: A rare condition where cysts in the kidney grow and cause the kidney to become larger and reduce it's ability to function. PKD 1 is an autosomal dominant form of the disease and differs from PKD 2 in that it is more severe and is caused by a mutation in a different gene.
• Polycystic kidney disease, adult type: A rare condition where cysts in the kidney grow and cause the kidney to become larger and reduce it's ability to function. PKD 2 is an autosomal dominant form of the disease and differs from PKD 1 in that it is less severe and is caused by a mutation in a different gene.
• Polycystic kidney disease, infantile type: Severe form of polycystic kidney disease which is a genetic kidney disease. Symptoms develop very early in life and may even be present during the fetal stage.
• Polycystic kidney disease, type 2: A rare condition where cysts in the kidney grow and cause the kidney to become larger and reduce it's ability to function. This adult form of the condition is milder than the infantile form.
• Polycystic liver disease: A rare inherited disorder involving the formation of numerous cysts in the liver.
• Polycythemia: increase in the total number of red blood cells in the circulation. It can be primary or secondary
• Polycythemia rubra: An uncommon chronic blood disease involving an increased production of red blood cells by the bone marrow. The production of platelets and white blood cells may also be increased.
• Polycythemia vera: A condition which is characterized by myeloproliferation of unknown etiology
• Polydactyly visceral anomalies cleft lip palate: A rare syndrome characterized mainly by the association of extra digits, cleft lip and/or palate and visceral anomalies. Various other abnormalities may also be present.
• Polyendocrine deficiency syndrome type 1: Multi-endocrine syndrome commonly affecting children
• Porphyria: A group of disorders characterized by excess production of porphyrin or its precursors which affects the skin and/or nervous system.
• Porphyria Cutanea Tarda: A rare metabolic condition involving a deficiency of the enzyme uroporphyrinogen decarboxylase. This results in a buildup of porphyrins in the skin which makes it sensitive to light. Most symptoms tend to primarily affected the areas of skin most exposed to sun e.g. back of hands and arms.
• Porphyria Cutanea Tarda - familial: A rare metabolic condition involving a deficiency of the enzyme uroporphyrinogen decarboxylase throughout the body - type I involves a deficiency of the enzyme only in the liver. This results in a buildup of porphyrins in the skin which makes it sensitive to light. Most symptoms tend to primarily affected the areas of skin most exposed to sun e.g. back of hands and arms.
• Porphyria Cutanea Tarda - sporadic: A rare metabolic condition involving a deficiency of the enzyme uroporphyrinogen decarboxylase in the liver - type II involves a deficiency of the enzyme throughout the whole body. This results in a buildup of porphyrins in the skin which makes it sensitive to light. Most symptoms tend to primarily affected the areas of skin most exposed to sun e.g. back of hands and arms.
• Porphyria cutanea tarda, familial type: A rare metabolic condition involving a deficiency of the enzyme uroporphyrinogen decarboxylase.
• Porphyria cutanea tarda, sporadic type: A rare metabolic condition involving a deficiency of the enzyme uroporphyrinogen decarboxylase in the liver only. Type II involves a deficiency of the same enzymes activity in various tissues throughout the body.
• Porphyria, congenital erythropoietic: An extremely rare inherited metabolic disorder involving a deficiency of the enzyme uroporphyrinogen III cosynthase which results in increased deposits of porphyrin in the bone marrow, blood, urine, feces and sometimes teeth and bones.
• Portal hypertension: Portal hypertension may be defined as a portal pressure gradient of 12 mm Hg or greater and is often associated with varices and ascites.
• Portal hypertension due to intrahepatic block: A blockage that forms in the portal vein (caries blood from the digestive tract to the liver) due to liver problems.
• Portal thrombosis: Clotting or obstruction of blood flow along the veins from the intestines and spleen and into the liver. This causes blood to back up and results various problems such as enlarged spleen and abdominal pain. The obstruction can occur acutely (over a short space of time) or chronically (over a longer period of time).
• Portal vein thrombosis: Clotting or obstruction of blood flow along the portal vein and into the liver. This causes blood to back up and results various problems such as enlarged spleen and abdominal pain. The obstruction can occur acutely (over a short space of time) or chronically (over a longer period of time). The portal vein collects blood from the intestines and spleen and carries it to the liver.
• Postoperative Jaundice: Some people can develop liver dysfunction after major surgery and this can result in jaundice. The liver dysfunction is usually poorly defined and can occur in people who have no history of liver problem as well as people with existing liver problems. It may result from such things as damage to the liver or from the anesthetic. However, patients with well controlled liver problems often undergo surgery without any resulting liver problems. The severity of the jaundice is variable and the condition usually resolves without any harmful effects. Heart and upper abdominal surgery are the most likely types of surgery to result in postoperative jaundice.
• Postpartum Eclampsia: Postpartum eclampsia is the development of seizures or coma in pregnant women suffering from high blood pressure. Postpartum means that it soon after the delivery. Eclampsia is a serious condition which requires urgent medical treatment. Eclampsia may be associated with moderate as well as significant increases in blood pressure.
• Postperfusion syndrome: A complication that can occasionally occur following blood transfusion. The condition is believed to be caused by Ebstein-Barr virus or cytomegalovirus present in the transfused blood.
• Pregnancy symptoms: Symptoms related to pregnancy.
• Pregnancy toxemia /hypertension: Preeclampsia is the development of high blood pressure, excess protein in the urine and swelling during pregnancy. Hypertension is a serious health condition due to the fact that it often causes no symptoms until it is severe. The blood pressure usually returns to normal after delivery.
• Prescribed medication addiction: An uncontrollable desire to use prescribed medication in a manner or frequency not prescribed. Drugs such as painkillers are prescribed to treat such things as pain but patients may become physically dependent on the drug and continue to obsessively use it even after the condition it was prescribed for has resolved. Frequent use leads to an increased tolerance to the drug so higher and higher doses are required to achieve the desired euphoric feeling.
• Primary Autoimmune Hemolytic Anemia: Primary autoimmune hemolytic anemia is characterized by an abnormal immune system response. The body's immune system produces antibodies against the body's own red blood cells which leads to the destruction of red blood cells and hence anemia. The secondary form of the condition is the result of an underlying condition such as lupus or hepatitis whereas the primary form has no underlying condition.
• Primary Cold Autoimmune Hemolytic Anemia: Primary cold autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The process occurs only when the patient is exposed to temperatures colder the average body temperature. The red cells are destroyed at an abnormally rapid rate which leads to anemia. The primary form of the condition means that there is no underlying condition. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Primary biliary cirrhosis: Primary biliary cirrhosis is a chronic and progressive cholestatic disease of the liver.
• Primary granulocytic sarcoma: A malignant tumor derived from immature white blood cells called myeloblasts. It can occur anywhere in the body but is most commonly found in bone, soft tissue, lymph nodes and skin. Symptoms will vary according to the location of the tumor.
• Primary lipodystrophies: A group of rare diseases which involve loss of body fat. The body fat loss may be localized to certain parts of the body or may be generalized depending on the particular subtype.
• Primary malignant lymphoma: The excessive proliferation of lymphocytes which forms part of the immune system. Primary cancers refer to the fact that the cancer originated in the lymph cells rather than having metastasized.
• Primary sclerosing cholangitis: Primary sclerosing cholangitis (PSC) is a chronic cholestatic liver disease of unknown etiology that is recognized increasingly in children.
• Progeria short stature pigmented nevi: A very rare inherited disorder characterized by premature aging, short stature, and immune system deficiency. The type and severity of symptoms is variable.
• Prolidase deficiency: A rare metabolic disorder where an enzyme (prolidase) deficiency impairs the breakdown of certain proteins which causes a harmful buildup in the body. The type and severity of symptoms are variable and may include skin disorders, retarded motor development, impaired cognitive development, frequent infections and skeletal abnormalities.
• Prolymphocytic leukemia: A very rare type of leukemia involving a proliferation of immature white blood cells (prolymphocytes - T-cells).
• Protoporphyria erythropoietic: An inherited metabolic disorder where a deficiency of ferrochelatase leads to a buildup of protoporphyrin in the bone marrow, red blood cells and occasionally the liver.
• Proximal tubulopathy - diabetes mellitus - cerebellar ataxia: A very rare syndrome characterized mainly by diabetes, movement disorder and kidney abnormality. Death often occurs during childhood.
• Pseudo-torch syndrome: A rare genetic syndrome characterized by a small head, eye disease and brain abnormalities. The symptoms are often very similar to another disorder caused by certain infections during pregnancy.
• Pseudoadrenoleukodystrophy: A rare disorder where an enzyme deficiency (Acyl-CoA Oxidase) results in symptoms such as seizures, apnea, delayed psychomotor retardation and neurological deterioration.
• Psittacosis: An infectious disease caused by Chlamydia psittaci and transmitted mainly by infected birds but also by some mammals.
• Psychotropic agent-induced liver damage: Damage or injury to the liver caused by exposure to psychotropic agents. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - Benzodiazepine: Damage or injury to the liver caused by exposure to a psychotropic agent called benzodiazepine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - Butyrophenone: Damage or injury to the liver caused by exposure to a psychotropic agent called butyrophenone. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - Phenothiazines: Damage or injury to the liver caused by exposure to a psychotropic agent called phenothiazine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - Thioxanthene: Damage or injury to the liver caused by exposure to a psychotropic agent called Thioxanthene. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - monoamine oxidase inhibitors: Damage or injury to the liver caused by exposure to psychotropic agents called monoamine oxidase inhibitors. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - tricyclic antidepressant: Damage or injury to the liver caused by exposure to a psychotropic agent called tricyclic antidepressant. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Pulmonary valve stenosis: Often a congenital defect but may be caused by such things as rheumatic fever or bacterial endocarditis. Severity depends on the degree of narrowing of the pulmonary valve.
• Pulmonary venous return anomaly: A rare disorder where one or more of the four veins that carry oxygenated blood from the lungs drain to the right atrium of the heart instead of the left atrium. Symptom severity is determined by the number of veins involved and the exact location of the heart that the veins drain into.
• Pure red cell aplasia, congenital: A rare genetic condition where the bone marrow is unable to make sufficient red blood cells which leads low levels of red blood cells. There are eight subtypes of the condition which differ in the location of the genetic defect and the incidence of additional symptoms such as malformations. The severity of symptoms is variable but most cases are serious.
• Pylephlebitis: A pus-producing inflammation of the wall of the portal vein that drains blood from the abdominal part of the gastrointestinal tract. The infection is often fatal. It usually occurs as a complication of abdominal or pelvic infections such as diverticulitis and appendicitis.
• Pyruvate Kinase Deficiency: A rare inherited blood disorder involving a deficiency of the enzyme pyruvate kinase.
• Pyruvate carboxylase deficiency, Group A: A rare inherited disorder characterized by a deficiency of pyruvate carboxylase which leads to accumulation of lactic acid and other compounds which can have a toxic effect on the nervous system and body organs. Type A has moderately severe symptoms that usually start during infancy.
• Pyruvate carboxylase deficiency, Group B: A rare inherited disorder characterized by a deficiency of pyruvate carboxylase which leads to accumulation of lactic acid and other compounds which can have a toxic effect on the nervous system and body organs. Type B has severe, life-threatening symptoms that can be noticed soon after birth.
• Pyruvate kinase deficiency, hemolytic anemia: A rare inherited blood disorder where the deficiency of an enzyme (pyruvate kinase) needed by red blood cells causes them to be destroyed prematurely and hence anemia occurs.
• Pyruvate kinase deficiency, liver type: An inherited deficiency of pyruvate kinase which causes varying degrees of hemolytic anemia.
• Pyruvate kinase deficiency, muscle type: An inherited deficiency of pyruvate kinase which causes varying degrees of hemolytic anemia.
• Q fever: A disease caused by Coxiella burnetti which causes fever, headache and muscle pain.
• Quinidine-induced Immune Hemolytic Anemia: Quinidine-induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Refsum disease, infantile form: A rare inherited biochemical disorder characterized by normal development initially followed by degeneration, facial anomalies and enlarged liver.
• Relapsing fever: Tick-borne disease with symptoms that resolve and then relapse
• Renal tubulopathy - diabetes mellitus - cerebellar ataxia: A very rare condition characterized by rapidly progressive diabetes, kidney disease and cerebellar ataxia. Symptoms develop during the first five years.
• Renal-hepatic-pancreatic dysplasia - Dandy Walker cyst: A very rare syndrome involving abnormal development of the kidneys, liver and pancreas as well as a brain cyst called a Dandy-Walker cyst.
• Reticuloendotheliosis: An inherited disorder of the reticuloendothelial system which is involved in destroying bacteria and other unwanted substances that enter the body.
• Retinal telangiectasia and hypogammaglobulinemia: A rare disorder characterized by the association of poor immunity with dilated retinal blood vessels.
• Reye's syndrome: is a potentially fatal disease that causes numerous detrimental effects to many organs, especially the brain and liver
• Reynolds syndrome: A very rare syndrome characterized mainly by scleroderma (hardened skin) and liver cirrhosis.
• Rh Disease: A disease that can occur when the mother's blood is not compatible with the fetal blood i.e. when an Rh-negative mother had a Rh-positive child. If the baby's blood is exposed to the mother's blood (through placenta, abortion, miscarriage, amniocentesis) the mother's body becomes sensitized and develops antibodies the Rh-positive blood. In future pregnancies, the mother's antibodies can attack the red blood cells of the unborn baby resulting in hemolytic disease. The severity of the disease is variable and can range from mild fetal anemia to severe anemia and even fetal death.
• Rh deficiency syndrome: A rare genetic blood disorder where the red blood cells have an abnormal appearance and cannot function properly. The condition causes hemolytic anemia and the severity varies from patient to patient.
• Rhabdomyosarcoma, embryonal: A type of cancer that arises from rhabdomyoblasts which are immature muscle cells. The tumors can occur arise from muscle tissue almost anywhere in the body but in the embryonal form, tends to occur primarily in the head, neck and genitourinary areas. Symptoms depend on size and location of the tumor.
• Rhabdomyosarcoma, embryonal 1: A type of cancer that arises from rhabdomyoblasts which are immature muscle cells. The tumors can occur arise from muscle tissue almost anywhere in the body but in the embryonal form, tends to occur primarily in the head, neck and genitourinary areas. Symptoms depend on size and location of the tumor. There are two subtypes of this cancer with main difference lying in the genetic origin of the disease. Type 1 is linked to a defect on chromosome 11p15.5.
• Rhabdomyosarcoma, embryonal 2: A type of cancer that arises from rhabdomyoblasts which are immature muscle cells. The tumors can occur arise from muscle tissue almost anywhere in the body but in the embryonal form, tends to occur primarily in the head, neck and genitourinary areas. Symptoms depend on size and location of the tumor. There are two subtypes of this cancer with main difference lying in the genetic origin of the disease. Type 1 is linked to a defect on chromosome 11p15.5.
• Rhesus isoimmunisation: The antibody mediated destruction of red blood cells by those sensitive
• Richter syndrome: A rare but serious form of acute adult leukemia. The disease occurs when chronic lymphocytic leukemia transforms into diffuse large cell lymphoma which is a fast-growing form of lymphoma. The condition tends to be more common in older adults.
• Rietti-Greppi-Micheli syndrome: A type of genetic blood disorder involving reduced hemoglobin production.
• Rift Valley Fever: Mosquito-borne viral infection affecting animals and humans
• Ringed seal poisoning: The Ringed seal is often used as a food source by the arctic inhabitants. Eating the liver of the ringed seal can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases.
• Rocky Mountain spotted fever: A bacterial disease caused by Rickettsia rickettsii and transmitted by ticks. The condition causes fever and a characteristic rash and may be fatal in severe or untreated cases.
• Roifman syndrome: A rare syndrome characterized by abnormal bone growth, immune deficiency and an eye disorder.
• Rosai-Dorfman disease: A rare condition characterized by excessive production and accumulation of a particular white blood cell (histiocyte). Accumulation primarily occurs in the lymph nodes, especially in the neck, but may also occur in the skin, central nervous system, digestive tract and kidneys.
• SCARF syndrome: A rare syndrome characterized mainly by skeletal abnormalities, loose skin, premature fusion of skull bones, ambiguous genitalia, psychomotor retardation and facial abnormalities.
• SCHAD Deficiency - formerly: A rare inherited form of biochemical disorder characterized by the deficiency of a particular enzyme (3-Hydroxyacyl-CoA Dehydrogenase). The enzyme deficiency only affects certain body tissues, in particular the skeletal muscles. The lack of enzyme activity prevents some fats being converted into energy. Symptoms tend to be exacerbated during fasting as during fasting, the body tries to rely more heavily on fats for energy. Fatty acids that are not completely metabolized due to the enzyme deficiency may build up in various organs and cause serious complications.
• SCID: Major failure of the immune system, usually genetic.
• Sandhoff Disease: An inherited biochemical disorder involving a deficiency of the enzyme called hexosaminidase A&B which results in the harmful accumulation of chemicals in the central nervous system and other body tissues.
• Sarcoidosis: Rare autoimmune disease usually affecting the lungs.
• Sarcoma botryoides: An aggressive form of cancer that arises from embryonic muscle cells. The tumor resembles a bunch of grapes and tends to occur in the genitourinary tract. Common locations are the cervix, vagina and bladder and very rare cases can occur in the bile duct or the soft tissues of the head and neck. It occurs most often in female infants and young children. Symptoms will vary depending on the exact location of the tumor.
• Sarrouy disease: A disease characterized mainly by anemia, poor growth and enlarged liver and spleen. The disease usually ends with death.
• Schistosomiasis: Parasitic fluke infection in developing countries
• Schnitzler syndrome: A rare syndrome characterized by the association of chronic urticaria as well as a blood abnormality called macroglobulinemia.
• Schridde syndrome: A rare disorder involving a fetal blood abnormality which usually results in death.
• Sea Hare poisoning: Certain species of sea hares can result in poisoning if eaten. Sea hares are marine mollusks. Two species with a potential for poisoning are found in Oceans near Fiji and Japan. Eating the eggs of these sea hares can also cause liver damage. Poisoning by sea hares is considered quite rare. The substance underlying the condition is organic bromine compounds.
• Sea-Blue histiocytosis: A rare condition involving the presence of too many histiocytes in the spleen and bone marrow. These histiocytes stain a sea-blue color during testing. These abnormal staining histiocytes have been noted in several diseases such as Niemann-Pick disease, lipid metabolism disorders and Norum disease.
• Secondary Autoimmune Hemolytic Anemia: Secondary autoimmune hemolytic anemia is characterized by an abnormal immune system response. The body's immune system produces antibodies against the body's own red blood cells which leads to the destruction of red blood cells and hence anemia. The secondary form of the condition is the result of an underlying condition such as lupus or hepatitis whereas the primary form has no underlying condition. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Secondary Biliary Cirrhosis: Secondary biliary cirrhosis is a condition where the bile ducts are unable to transport bile effectively due to a secondary cause which results in blockage, inflammation, scarring or some other damage to the bile ducts. The condition may result from such things as congenital defect of the bile ducts (e.g. biliary atresia), cystic fibrosis, gallstones or a variety of other secondary conditions.
• Secondary Cold Autoimmune Hemolytic Anemia: Secondary cold autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The process occurs only when the patient is exposed to temperatures colder the average body temperature. The red cells are destroyed at an abnormally rapid rate which leads to anemia. The secondary form of the condition means that it is the result of an underlying condition - it usually occurs after some sort of infection. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Secondary syphilis: A condition which is characterized by fever, multiform skin eruptions, iritis, alopecia, mucous patches and severe pain in the head and joints
• Selective Vitamin B12 malabsorption with Proteinuria: A rare genetic blood disorder where a defect in the vitamin B12 receptor means that it can't be absorbed from food during digestion. As a result of the low vitamin B12 levels, the body produces increased numbers of abnormal enlarged red blood cells (megaloblasts).
• Senecio poisoning: Senicio is a herbaceous plant which bears groups of small, usually yellow flowers. The plant can be found growing in the wild and is also used indoors and outdoors as an ornamental plant. The leaves contain chemicals which can cause symptoms if eaten or skin irritation upon skin exposure. The plant is considered to have a low level of toxicity.
• Sepsis: The presence of microorganisms in the blood circulation
• Septo-Optic Dysplasia: A rare birth defect characterized by impaired vision and pituitary deficiency.
• Shingles: Infectious viral infection occuring years after chickenpox infection.
• Short stature - cranial hyperostosis - hepatomegaly: A very rare syndrome characterized mainly by a thickened skull, short stature and an enlarged liver.
• Short stature cranial hyperostosis hepatomegaly diabetes: A very rare syndrome characterized mainly by enlarged liver, diabetes, short stature and a thickened skull.
• Short stature, cranial hyperostosis, hepatomegaly and diabetes: A very rare syndrome characterized mainly by enlarged liver, diabetes, short stature and a thickened skull.
• Shwachman syndrome: A rare disorder where a pancreatic defect impairs digestive enzyme production and malfunction of the bone marrow produces blood abnormalities, particularly blood cells involved with fighting infection. These defects produce a range of symptoms as a result of malabsorption and poor ability to fight infections. Characterized by a lack of digestive enzymes and low immunity due to low leukocyte level.
• Sialidosis type 2: A very rare inherited metabolic disorder where a defect in the enzyme alpha-neuraminidase prevents glycoproteins being metabolized - a severe form of Sialidosis type I.
• Sialidosis type II: A very rare inherited metabolic disorder where a defect in the enzyme alpha-neuraminidase prevents glycoproteins being metabolized - a severe form of Sialidosis type I.
• Sialidosis type II, infantile: A very rare inherited metabolic disorder where a defect in the enzyme alpha-neuraminidase prevents glycoproteins being metabolized. The infantile form is characterized by structural or functional kidney problems.
• Sialuria syndrome: A rare metabolic disorder characterized by excessive levels of sialic acid in the urine as well as liver and spleen enlargement and coarse facial features.
• Sialuria, French type: A rare disorder characterized by the excretion of large amounts of sialic acid in the urine due to a metabolic error.
• Siccardi syndrome: A rare disorder characterized by impaired immunity (due to defective bacterial destruction by neutrophils), lymphadenopathy and abnormal skin and hair color.
• Sickle cell anemia: Sickle cell anemia is an inherited blood disorder characterized by red blood cells which are crescent-shaped rather than the normal doughnut shape. These abnormally shaped red blood cells are unable to function normally and tend to undergo premature destruction which leads to anemia. If the genetic defect which causes the condition is inherited from both parents the condition can be quite severe whereas if it is inherited from only one parent, often there are no symptoms. The abnormally shaped red blood cells can cause problems when they clump together and block blood vessels.
• Sickle cell crisis: A condition which is characterized by either a hemolytic crisis or vaso-occlusive crisis
• Sideroblastic anemia, autosomal: An inherited form of anemia where an enzyme deficiency prevents red blood cells from using iron properly. The red blood cells become overloaded with iron which impairs the blood's ability to carry oxygen.
• Sideroblastic anemia, hereditary: An inherited form of anemia where an enzyme deficiency prevents red blood cells from using iron properly. The red blood cells become overloaded with iron which impairs the blood's ability to carry oxygen.
• Sideroblastic anemia, pyridoxine-responsive, autosomal recessive: Pyridoxine-responsive sideroblastic anemia is a rare inherited blood disorder characterized by abnormal deposits of iron in the red blood cells which makes them unable to function properly. The condition is inherited in an autosomal recessive manner and responds to treatment with pyridoxine.
• Siderosis: Deposits of iron oxide dust in the lungs usually occurs in an occupational setting.
• Simpson-Golabi-Behmel syndrome, type 1 (SGBS1): A rare genetic disorder characterized by accelerated growth, a peculiar face and other defects.
• Sitosterolemia: A very rare inherited disorder of lipid metabolism where excessive amounts of sterols are absorbed and not removed from the body resulting in a build up in the blood.
• Sjogren's Syndrome: Autoimmune disease damaging the eye tear ducts and other glands.
• Small bowel bacterial overgrowth syndrome: The small bowel is a part of the digestive system. A healthy small bowel contains some bacteria but in bacterial overgrowth syndrome, there are an excessive number of bacteria. Abnormal small bowel muscle function is often associated with conditions such as intestinal motility problems due to neurological diseases and muscular diseases, diabetes mellitus, small intestine obstruction and diverticulitis. It may also be caused by certain medications and abdominal surgeries.
• Smith disease: A harmless condition involving increased lymphocyte levels which may manifest as a variety of symptoms or may be asymptomatic. Diseases such as whooping cough and German measles are believed to be possible causes. The disease most commonly occurs in children and young adults.
• Smoking stools syndrome: Ingestion of yellow phosphorus (chemical found in many rodent poisons) which is toxic to the body. There is an initial phase involving symptoms such as vomiting and burning which is followed by an asymptomatic period (may last for weeks) and then symptoms caused by toxicity of various organs. Obviously symptoms and survival depend on the quantity of phosphorus involved.
• Smoldering adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The smoldering form tends to progress even slower than the chronic form and responds the best to treatment.
• Soft Tissue Sarcoma: A rare type of cancerous tumor that originates in soft tissues of the body which includes muscles, nerves, tendons, blood vessels and fat. They can occur anywhere in the body and are often asymptomatic during the early stages and symptoms in the later stages depend on the location and size of the tumor.
• Solvent abuse: Solvent abuse is the use of various inhalants for the purpose of achieving a "high". They are often used as a cheap, readily available alternative to street drugs but they can cause serious damage to the body. Solvents include nail polish removers, paint thinners, gasoline, typing correction fluid and toxic markers. These solvents can be abused by sniffing them, spraying directly into the mouth, heating them and then inhaling them or injecting them directly into the body.
• Solvent addiction: Solvent addiction refers to the compulsive need to abuse solvents (e.g. sniffing them). Sufferers have withdrawal symptoms when attempting to stop the habit and feel unable to stop the habit despite knowing the harm it is causing their health. Solvents are very damaging to the body and can readily result permanent brain damage and even death. Death can occur through chronic use and in rare cases can occur after one session of use. Children and teenagers are particular at risk for this type of addiction - it is readily available and users feel it gains them greater acceptance from their peers. Solvents includes paint thinner, toxic markers, gasoline, cigarette lighter fluid, typing correction fluid and nail polish removers.
• Soto's Syndrome: A rare hereditary disorder characterized by excessive growth during the first few years of life as well as various other mental and physical anomalies.
• Speech symptoms: Problems with speech or voice.
• Spherocytic anemia: Spherocytic anemia is a blood disorder characterized by red blood cells that are shaped like a sphere (spherocytes) rather than the normal doughnut shape. These abnormal cells are broken down prematurely resulting in a shortage of red blood cells (anemia). The condition may be inherited or acquired.
• Spherocytic hereditary elliptocytosis: A rare inherited blood disorder characterized by the presence of spherocytes and oval-shaped red blood cells rather than the normal doughnut-shape. This form of hereditary elliptocytosis can cause hemolytic anemia.
• Spherocytosis: A rare blood disorder characterized by a defect of the red blood cell membrane which gives it a spherical shape rather than the normal doughnut shape.
• Sphingolipidosis: A group of diseases involving the abnormal metabolism and storage of a substance called sphingolipid. Symptoms will vary depending on the disease. Examples of diseases from this group include gangliosidosis, Gaucher's disease and Niemann-Pick disease.
• Spirochetes disease: Infection with a type of bacteria which is often found in mud, sewage and polluted water. Symptoms are determined by the species involved. Diseases caused by this bacteria include Treponema infection and borreliosis.
• Spleen Cancer: Malignancy of white blood cells with tumour deposits in the spleen.
• Spleen conditions: Any condition that affects the spleen
• Spleen neoplasm: A tumor that originates in the spleen.
• Spondyloepimetaphyseal dysplasia congenita, Iraqi: A very rare syndrome characterized mainly by abnormal bone growth. Only a few cases have been reported.
• Spondyloepimetaphyseal dysplasia, hypotrichosis: An inherited bone growth disorder characterized by short stature, bone abnormalities and vision problems. An additional feature of this condition is the reduced amount of hair (hypotrichosis). The bone abnormalities are due to a genetic mutation that affects the development of bone and connective tissue.
• Spondylometaphyseal dysplasia with combined immunodeficiency: A rare syndrome characterized by skeletal abnormalities as well as a poor immune system.
• Spotted fevers: An acute condition caused by the bacteria Rickettsia rickettsii
• Spranger syndrome: A rare genetic disorder characterized by dwarfism and an unusually pleasant, happy face as well as other abnormalities involving the hands and feet. The condition involves localized accumulations of mucopolysaccharides in the liver, trachea and cardiovascular system cartilage.
• Stauffer syndrome: Spleen and liver problems associated with a kidney tumor.
• Steroid dehydrogenase deficiency - dental anomalies: A very rare liver disease involving an enzyme (steroid dehydrogenase) deficiency and dental anomalies.
• Still's Disease, Adult-Onset: A form of arthritic inflammation characterized by fever, rash and joint pain that occurs in adults. The cause is unknown.
• Stomach cancer: Stomach or gastric cancer can develop in any part of the stomach and may spread throughout the stomach and to other organs
• Strudwick syndrome: An inherited bone growth disorder characterized by short stature, bone abnormalities and vision problems. The bone abnormalities are due to a genetic mutation that affects the development of bone and connective tissue.
• Stuart-Bras disease: A type of liver disease that is seen in children (usually aged 1-6 years) in the West Indies and is possibly caused by ingestion of plant toxins from plants such as Senico.
• Subacute granulomatous thyroiditis: Subacute thyroiditis is a self-limited thyroid condition associated with a triphasic clinical course of hyperthyroidism, hypothyroidism, and return to normal thyroid function.
• Sulfatidosis juvenile, Austin type: A very rare metabolic disorder where all the sulfatase enzymes are impaired resulting in a variety of physical and developmental abnormalities.
• Sulfone syndrome: A hypersensitivity reaction to sulfone which is a component of a drug called dapsone which is often used to treat skin conditions such as leprosy, vasculitis, dermatitis herpetiformis and Sweet disease. The reaction can result in death in some cases.
• Sulfur tuft poisoning: Sulfur tuft is a type of small mushroom which can have yellow to orange to green caps. The mushroom has a very bitter taste and is poisonous - death can result if sufficient quantities are eaten.
• Sulphonamide -induced Immune Hemolytic Anemia: Sulphonamide-induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Summerskill-Walshe-Tygstrup syndrome: A rare liver disorder characterized by intermittent episodes of cholestasis (impaired bile flow within the liver) that doesn't involve obstruction of bile ducts outside the liver. The jaundice tends to disappear in weeks or months without treatment and the condition does not lead to liver failure. The two subtypes of the disorder have a different origin of the genetic mutation.
• Sump Syndrome: A complication that can sometimes occur after an operation done to treat recurring stone disease. The surgery involves forming a connection between the common bile duct and the intestines. Sometimes a pit-like (sump) portion develops along the bile duct and this allows debris such as food to build up in a pit-like (sump) portion of the bile duct. The buildup of debris can lead to infection.
• Swollen spleen: Swelling or enlargement of the spleen
• Syphilis: A sexually transmitted disease caused by a bacteria (Treponema pallidum). The condition is often asymptomatic in the early stages but one or more sores may be present in the early stages. Untreated syphilis usually results in remission of visible symptoms but further severe damage may occur to internal organs and other body tissues which can result in death.
• Systemic Juvenile Rheumatoid Arthritis: Onset of JRA with fevers and systemic symptoms
• Systemic mastocytosis: A condition which is characterized by an accumulation of mast cells in the tissues of the body
• Systemic monochloroacetate poisoning: Monochloracetate acid is a dangerous chemical which can cause systemic poisoning even if only skin exposure occurs. Exposure to the chemical can be life-threatening with serious symptoms starting within hours of the exposure.
• T-cell acute lymphoblastic leukemia: Cancer of particular white blood cells called T-cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets.
• T-cell lymphoma 1A: T-cell lymphoma 1A is a type of cancer that has genetic origins. The genetic defect is located on chromosome 14q32.1. The anomaly is usually implicated in t-cell prolymphocytic leukemia. The cancer is usually aggressive and tends to target the blood, bone marrow, lymph nodes, liver, spleen and skin. The cancer is generally quite rare and tends to occur in people over the age of 30.
• T-cell prolymphocytic leukemia: A form of cancer which causes proliferation of T-cells in the intermediate stage of development.
• TORCH Syndrome: Infection of a fetus by any of a group of infectious agents which have been transmitted from the mother through the placenta. The infections include toxoplasmosis, rubella, cytomegalovirus, herpes virus, hepatitis and syphilis. The severity and nature of symptoms is determined by the type of infection.
• TRAPS (TNF-receptor-associated periodic syndrome): A rare syndrome involving periods of fever and chills along with gastrointestinal symptoms and muscle pain. Symptoms last for two or three weeks.
• Tang Hsi Ryu syndrome: A rare syndrome characterized by enlarged liver and spleen, increased pigmentation and abnormal peripheral nerve functioning.
• Tangier disease: A rare disorder characterized by a very low level of HDL (high-density lipoprotein or "good cholesterol") in the blood. The condition occurs because the body lacks the gene to make a certain protein (Apolipoprotein A1) which normally transports fats from tissues to where it is needed.
• Thalassemia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. There are two subtypes of the disorder (? and ?) depending on what portion of the hemoglobin is abnormally synthesized. Each of these subtypes can be further classified depending on the number of thalassemia genes involved. A total of four genes is needed to make each ? protein chains and two genes are needed to make each ? chain. The main symptom is anemia, the severity of which can vary amongst patients depending on how many defective genes are involved.
• Thiamine responsive megaloblastic anemia syndrome: A rare genetic disorder characterized by megaloblastic anemia, hearing loss and diabetes. The condition is caused by a deficiency of a thiamine (vitamin B1) transporter protein which means that the body is unable to effectively utilize thiamine from the diet.
• Thompson-Baraitser syndrome: A very rare syndrome characterized mainly by liver fibrosis, kidney cysts and mental retardation.
• Thoraco abdominal enteric duplication: A very rare syndrome characterized by chest and abdominal abnormalities as well as intestinal duplication.
• Thoracopelvic dysostosis: A rare syndrome characterized mainly by abnormalities involving the chest, larynx and pelvis. The small chest cavity affects breathing and can affect survival, especially during infancy.
• Thrombocythemia: A haematological disorder in which there is excessive proliferation of platelet precursor cells, resulting in increased numbers of platelets in the blood.
• Thrombocytopenia: Decreased concentration of platelets in the blood.
• Thrombocytopenic purpura, autoimmune: A rare blood disorder where a low number of platelets impairs the bloods ability to clot and results in bleeding into the skin and mucous membranes.
• Thrombotic thrombocytopenic purpura, acquired: A rare blood condition where small blood clots form in blood vessels which reduces the number of blood platelets and results in kidney failure, neurological symptoms and anemia. The condition may be familial or acquired - symptoms tend to recur regularly in the familial form.
• Thrombotic thrombocytopenic purpura, congenital: A rare blood condition where small blood clots form in blood vessels which reduces the number of blood platelets and results in kidney failure, neurological symptoms and anemia.
• Thyroid agenesis: A rare disorder where the thyroid fails to develop resulting in hypothyroidism from birth.
• Tollner-Horst-Manzke syndrome: A very rare syndrome characterized by extra fingers, cleft lip, cleft palate and abdominal organ anomalies.
• Toni-Fanconi syndrome type 1:
• Toxic mushrooms - Monomethylhydrazine: Some mushrooms contain a toxic chemical called gyromitrin which is converted to monomethylhydrazine after digestion. Mushroom species from this group include certain species of Gyromitra, Helvella, Sarcosphaera and Peziza. Poisoning may occur from inhaling fumes from cooking mushrooms. The amount of toxin varies amongst and within species but some are toxic enough to cause death. Urgent medical attention should be sought if mushroom poisoning is suspected.
• Toxic mushrooms - Renal toxic (orelline): Some mushrooms (Amanita smithiana) contain chemicals (allenic norleucine, chlorocrotyglycine) which can cause kidney damage.
• Toxic mushrooms - cyclopeptides: Some mushrooms contain a toxic chemical called cyclopeptide which can cause primarily gastrointestinal symptoms if ingested. Most cases of mushroom poisoning in North America involve cyclopeptide-containing mushrooms. Mushroom species from this group include certain species of Amanita (bisporigera, ocreata, phalloides, suballiacea, tenufolia, verna, virosa), Galerina and Lepiota. One Amanita mushroom cap may result in death in an adult. Poisoning occurs in three phases: gastrointestinal symptoms (within 24 hours of ingestion); remission (up to 72 hours after ingestion); and liver and kidney symptoms (3 to 6 days after ingestion). Poisoning symptoms are more severe in children due to their smaller body size.
• Toxocariasis: A parasitic roundworm (Toxocara canis or Toxocara cati) infection that normally occurs in cats and dogs but can be transmitted to humans by ingesting the larvae or eggs. The infection may be asymptomatic or severe and symptoms depend on where the larvae travel to when they migrate through the body.
• Toxoplasmosis: Infection often caught from cats and their feces.
• Transfusion Reaction: The allergic reaction to the transfusion of blood products to a person
• Transposition of great arteries: A congenital malformation where the aorta and pulmonary artery are transposed which causes oxygenated blood from the lungs to be sent back to the lungs and de-oxygenated blood to be sent to body tissues. Often there is some other defect such as an opening in the heart chambers which allows mixing of the blood and hence survival is possible for a short while at least.
• Treponema infection: A rare infectious diseases which is transmitted through sexual contact and caused by Treponema pallidum (a spirochete bacterium). Untreated cases can result in severe complications and even death.
• Tricho-hepato-enteric syndrome: A very rare syndrome characterized mainly by hair and liver abnormalities as well as severe diarrhea that usually starts in early infancy and can lead to death.
• Trichomegaly cataract hereditary spherocytosis: A rare syndrome characterized mainly by cataracts, excessive eyelash growth and a blood abnormality (the red blood cells are spherical instead of doughnut shaped which makes them fragile).
• Tricuspid valve diseases: Any disease that affects the tricuspid valve
• Triglyceride storage disease with impaired long-chain fatty acid oxidation: A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities.
• Triosephosphate isomerase 1: A rare inherited blood disorder involving a deficiency of red cell triosephosphate isomerase which is involved in the glycolysis (glucose metabolism) process to produce energy. The disorder is characterized by anemia, recurring infection and often progressive neurological degeneration. The severity of the disorder is variable with some patients having central nervous system involvement while others don't.
• Trypanosomiasis, east-African: A rare infectious disease caused by a parasite called Trypanosoma brucei rhodesiense and is transmitted through the bite of an infected Tsetse fly. The infection causes an acute illness with symptoms occurring from days to weeks after infection. Death relatively common, especially in untreated cases.
• Tularemia: A rare infections disease caused by the bacterium Francisella tularensis (a gram-negative pleomorphic coccobacillus). Transmission occurs through contact with infected animals or there habitats e.g. bites from infected insects or other animals, eating infected wild animals, contact with contaminated water and soil. Symptoms can vary greatly depending on the method of infection. For example infection through inhalation can cause symptoms similar to pneumonia, eating infected animals can cause a sore throat and abdominal symptoms and transmission through the skin can cause result in a painful skin ulcer.
• Type I Hyperlipoproteinemia: Type I Hyperlipoproteinemia is a rare inherited condition characterized by high levels of chylomicrons (a type of lipoprotein) in the blood. This condition is the result of low levels of lipoprotein lipase which is responsible for lowering lipoprotein levels. Cholesterol levels are usually normal and the risk of heart disease is low.
• Type III Hyperlipoproteinemia: Type III hyperlipoproteinemia is a rare genetic condition characterized by increased blood levels of intermediate density lipoproteins (IDL) which in turn causes high cholesterol levels. The high IDL levels are caused by an abnormality in apolipoprotein E. Apolipoprotein E is a protein found on IDLs which allows the IDLs to bind to liver cells. Without this binding process, the level of IDLs in the blood increase. Lipoproteins are responsible for transporting cholesterol in the bloodstream and high levels of them in the bloodstream means that there are high levels of cholesterol in the bloodstream.
• Type Ia Hyperlipoproteinemia: Type Ia Hyperlipoproteinemia is a rare inherited condition characterized by high levels of chylomicrons (a type of lipoprotein) in the blood. This condition is the result of low levels of lipoprotein lipase which is responsible for lowering lipoprotein levels. Cholesterol levels are usually normal and the risk of heart disease is low.
• Type Ib Hyperlipoproteinemia: Type Ib Hyperlipoproteinemia is a rare inherited condition characterized by high levels of chylomicrons (a type of lipoprotein) in the blood. This condition is the result of low levels of apolipoprotein C-II which is responsible for lowering lipoprotein levels. Cholesterol levels are usually normal and the risk of heart disease is low.
• Type Ic Hyperlipoproteinemia: Type Ic Hyperlipoproteinemia is a rare inherited condition characterized by high levels of chylomicrons (a type of lipoprotein) in the blood. This condition is the result of inhibited lipoprotein lipase activity. Lipoprotein lipase is responsible for lowering lipoprotein levels. Cholesterol levels are usually normal and the risk of heart disease is low.
• Typhoid fever: Fever from bacterial food poisoning.
• Typhus: A general name for various arthropod-borne rickettsial infections
• Tyrosinemia: A rare genetic metabolic disorder characterized by a deficiency of particular enzymes which prevents the breakdown of tyrosine which then builds up in the liver.
• UDP-Galactose-4-epimerase deficiency: A condition which is characterised by a mutation in the UDP-galactose-4-epimerase gene.
• Ulcerative colitis: Ulcerative colitis (Colitis ulcerosa, UC) is a form of inflammatory bowel disease (IBD). Ulcerative colitis is a form of colitis, a disease of the intestine, specifically the large intestine or colon, that includes characteristic ulcers, or open sores, in the colon.
• Unusual facies, hepatic fibrosis, renal cysts and mental retardation: A rare syndrome characterized mainly by liver fibrosis, unusual facial appearance, kidney cysts and mental retardation.
• Uremic frost: The occurrence of powdery deposits of ureas and uric acid salts in the skin
• Uterine Cancer: Cancer of the uterus.
• VLCAD deficiency: A rare condition that is characterised by cardiomyopathy, fatty liver, skeletal myopathy, pericardial effusions, ventricular arrhythmias, and sudden death
• Valvular dysplasia of the child: A condition which is characterised by the dysplasia of one or more of the valves to a childs heart
• Vaquez disease: An uncommon chronic blood disease involving an increased red blood cell count.
• Venerupin shellfish poisoning: The venerupin shellfish can be eaten by humans. The shellfish can become toxic if they consume toxic algae. The shellfish is most commonly found and eaten in Japan but has been introduced into parts of the US. Interestingly, all cases of poisoning have occurred in very localized areas of Japan where they are harvested from lakes. This type of poisoning tends to have a high death rate - up to a third of patients die.
• Ventricular septal defect: An abnormal connection between the 2 lower chambers (ventricles) of the heart.
• Ventriculo-arterial discordance, isolated: A rare birth disorder where the right atrium is connected to the left ventricle and vice versa. The position of the heart ventricles is also inverted which allows normal blood oxygenation. Symptoms generally only occur later in life or if other heart defects are present which is usually the case.
• Very Long Chain Acyl CoA Dehydrogenase Deficiency - Early onset: Very Long Chain Acyl CoA Dehydrogenase Deficiency is a rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form. The early-onset form is the most severe and can readily lead to death if undiagnosed.
• Very Long Chain Acyl CoA Dehydrogenase Deficiency - adult-onset: Very Long Chain Acyl CoA Dehydrogenase Deficiency is a rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form. The adult-onset form is relatively mild and tends to be characterized mainly by muscle problems.
• Very Long Chain Acyl CoA Dehydrogenase Deficiency - intermediate: Very Long Chain Acyl CoA Dehydrogenase Deficiency is a rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form. The intermediate form is relatively mild and tends to be characterized by periods of low blood sugar - symptoms tend to develop during childhood.
• Very-Long-Chain Acyl-CoA Dehydrogenase Deficiency: A rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form.
• Viral Hepatitis: hepatitis describes inflammation of the liver. Hepatitis may be caused by alcohol, drugs, autoimmune diseases, metabolic diseases, and viruses. Viral infection accounts for more than half the cases of acute hepatitis.
• Virus associated hemophagocytic syndrome: A fulminant condition characterised by viral infection causing multiple organ infiltrates of haemophagocytic histiocytes.
• Visceral leishmaniasis (kala-azar): A rare infectious disease caused by any of a number of parasitic Leishmania species. Infection can cause any of three different manifestations: cutaneous leishmaniasis, mucosal leishmaniasis and visceral leishmaniasis. Visceral leishmaniasis infection involves the spleen, liver and bone marrow and can be fatal if untreated.
• Visceral steatosis: An inherited condition characterized by the deposition of small papules composing of granulomas in organs of the body.
• Vitamin A overdose: Overdose of Vitamin A usually due to Vitamin A supplement overuse or poisoning.
• Vitamin B12 Deficiency: A deficiency of Vitamin B12 primarily causes anemias the body is unable to make sufficient quantities of normal red blood cells. Severe cases can lead to permanent nervous system problems. The vitamin B12 deficiency can result from absorption problems, insufficient dietary intake, certain medications (e.g. metformin), inherited conditions (e.g. transcobalamin deficiency) and certain chronic parasitic intestinal infestations.
• Vitamin C Overdose: Symptoms occurring due to the ingestion of an amount of ascorbic acid (vitamin C) in excess of recommended doses or prolonged chewing of vitamin C supplement tablets; overdose is rare except in people with predisposing conditions as ascorbic acid is non-toxic.
• Von Gierke Disease: An inherited metabolic disorder where a deficiency of the enzyme glucose-6-phosphatase prevents glycogen being turned into glucose leading to a buildup of glycogen in the liver and kidneys.
• Von Gierke disease IA: A genetic metabolic disorder involving a deficiency of the enzyme glucose-6-phosphatase which results in the accumulation of glycogen in various tissues. G6P is stored as glycogen until the body needs to convert it to a sugar and use it to create energy. The enzyme deficiency prevents the conversion and hence low blood sugar levels result.
• Von Gierke disease IB: A metabolic disorder involving a deficiency of glucose-6-phosphatase (due to a G6P transporter defect) which results in the accumulation of glycogen in various tissues and a reduced blood sugar level. The condition is characterized primarily by an increased risk of bacterial infections due to impaired neutrophil activity which is needed to fight bacterial invasions.
• Von voss Cherstvoy syndrome: An early congenital condition that is characterised by multiple congenital anomalies.
• WAGR Syndrome: A syndrome that is due to the deletion of chromosome 11.
• WT limb blood syndrome: A rare genetic disorder characterized by blood and limb abnormalities.
• Waldenstrom macroglobulinemia: A plasma cell dyscrasia that resembles leukaemia
• Waldenström Macroglobulinemia: A rare disorder involving malignancy of the lymph and blood cells.
• Walrus poisoning: The walrus is used as a food source in some parts of the world. Eating the liver of the walrus can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases.
• Wandering spleen: A very rare birth defect where the structures (ligaments) that hold the spleen in position are absent or not developed properly which allows the spleen to move around in the abdominal cavity.
• Warm Autoimmune Hemolytic Anemia: Warm autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The red cells are destroyed at an abnormally rapid rate which leads to anemia. Warm haemolytic anemia is characterized by the fact that the abnormal destruction of red blood cells is more active when the patient is exposed to warm temperatures. The severity of the condition varies depending on the underlying cause e.g. cytomegalovirus, hepatitis, HIV and lupus. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Warm-reacting-antibody haemolytic anemia: An autoimmune disorder characterised by the premature destruction of red blood cells
• Warm-reacting-antibody hemolytic anemia: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 37ºC or lower. The severity of the disorder is variable.
• Weber-Christian disease: A form of panniculitis involving fever and liver and spleen enlargement.
• Weil syndrome: A rare infectious disorder which affects liver and kidney function and also causes hemorrhaging. It is a severe form of the second phase of leptospirosis which is an infection caused by the spiral shaped bacteria Leptospira interrogans which is transmitted from animals to humans.
• Weil's syndrome: Severe form of Leptospirosis
• Weinstein Kliman Scully syndrome: A syndrome that is characterised by cardiomyopathy, hypogonadism and metablic anomalies
• White Chameleon poisoning: The white chameleon is a type of thistle found mainly in dry areas of the Mediterranean. The rhizomes contains chemicals which can cause poisoning symptoms if eaten. The plant is often mistaken for a wild artichoke. The root extract is sometimes used in alternative medicine and excessive doses can also result in poisoning.
• Wilms tumor - aniridia - genitourinary anomalies - mental retardation: A syndrome resulting from deletion of genetic material from chromosome the short arm of chromosome 11 (11p13). The characteristic symptoms are partial or complete absence of iris, genitourinary anomalies, mental retardation and Wilms' tumor. The specific range and severity of symptoms is variable depending on the size and exact location of the genetic material that is missing.
• Wilson's disease: Wilson disease, or hepatolenticular degeneration, is a neurodegenerative disease of copper metabolism.
• Wolcott-Rallison syndrome: A very rare genetic condition characterized primarily by the onset of diabetes mellitus during infancy and epiphyseal dysplasia (abnormal joint development).
• Wolman disease: A rare inherited lipid storage disease. The condition usually results in death early in life.
• Wolman syndrome: A form of lipoidosis where acid cholesteryl ester hydrolase deficiency causes an accumulation of lipids (particularly cholesterol esters and triglycerides) in tissues and organs.
• Wyatt disease: A cytomegalus virus infection that occurs before birth.
• X-linked alpha thalassemia mental retardation syndrome (ATR-X): An x-linked condition that features mental retardation, dysmorphic features, and alpha thalassemia
• X-linked lymphoproliferative syndrome: A rare inherited immunodeficiency disorder where the body's immune systm is unable to respond appropriately to certain viral infections (Epstein Barr virus).
• X-linked sideroblastic anemia: X-linked sideroblastic anemia is a rare inherited blood disorder characterized by abnormal deposits of iron in the red blood cells which makes them unable to function properly. The body continues to absorb more iron from digesting food in order to try and correct the problem but this simply leads to excessive iron being deposited in body tissues as the it can't use the iron properly. The severity of the condition is variable.
• Xanthogranulomatous cholecystitis: A lipid laden foam cell tumour of the gallbladder resulting in inflammation
• Xerocytosis, heriditary: A form of hereditary stomatocytosis syndrome
• Yellow eyes: Yellow color of the white of the eyes
• Yellow fever: A viral infection transmitted by mosquito bites which can damage various organs such as the liver, heart, kidney and digestive tract.
• Yellow skin: Yellow coloring affecting the skin
• ZAP70 deficiency: A condition characterised by a deficiency of the Zap70 tyrosine kinase which is a component
• Zellweger Syndrome: Zellweger spectrum disorders are a group of rare, genetic, multisystem disorders that were once thought to be separate entities. These disorders are now classified as different expressions (variants) of one disease process. Collectively, they form a spectrum or continuum of disease. Zellweger syndrome is the most severe form; neonatal adrenoleukodystrophy is the intermediate form; and infantile Refsum disease is the mildest form.
• Zellweger-like syndrome, without peroxisomal anomalies: A rare syndrome characterized by unusual facial appearance, very low muscle tone, mental retardation and metabolic defects.
• Zieve syndrome: Liver and blood abnormalities caused by heavy alcohol consumption.
• Zimmerman-Laband syndrome: A rare inherited condition characterized by fibrosis of the gums and ear, nose, nail and finger abnormalities.
• Zinc poisoning: A type of heavy metal poisoning caused by excessive exposure to zinc.
• Zollinger-Ellison syndrome: A rare disorder where excessive levels of the hormone gastrin are released into the stomach which increases stomach acidity which results in peptic ulcer development. A hormone secreting pancreatic or duodenal tumor is usually the cause.

 » Next page: Surveys

Rate This Website

What do you think about the features of this website? Take our user survey and have your say:

Website User Survey

Medical Tools & Articles:

Next articles:

• Surveys
• Join in at the forums

Tools & Services:

• Bookmark this page
• Surveys relating to Liver symptoms
• Symptom Search
• Symptom Checker
• Medical Dictionary
• Give your feedback

Medical Articles:

• Disease & Treatments Search
• Misdiagnosis Center
• Full list of interesting articles

Forums & Message Boards

• Ask or answer a question at the Boards:
• I cannot get a diagnosis. Please help.
• Tell us your medical story.
• Share your misdiagnosis story.
• What is the best treatment for my condition?
• See all the Boards.

Enter your search terms Submit search form
Search The Web Search wrongdiagnosis.com

Common Health Mistakes

Symptom
Checker

Search Specialists by State and City