Glossary for Swollen spleen

Medical terms related to Swollen spleen or mentioned in this section include:

• $4-Alpha-hydroxyphenylpyruvate hydroxylase deficiency$: A very rare metabolic disorder where a deficiency of a particular enzyme results in the urinary excretion of a chemical called hawkinsin. Symptoms start once the infant is weaned off breast milk.
• ALL-Down syndrome: The presence of acute lymphoblastic leukemia in Down syndrome patients. These patients tend to have a poorer prognosis for the leukemia than patients without Down syndrome.
• AREDYLD: A rare condition characterized by abnormalities of the extremities, teeth, hair, nail and kidney as well as lipoatrophic diabetes.
• Aagenaes syndrome: A rare inherited birth abnormality involving underdeveloped lymph vessels which results in swollen legs and liver problems.
• Acanthocheilonemiasis: A rare tropical infection caused by a particular threadworm usually found in Africa. It may cause skin rashes, muscle and joint pains, neurologic disorders and skin lumps. The cerebrospinal fluid may also contain the larvae.
• Acanthocytosis: A rare disorder where most of the red blood cells are abnormal with spiny projections due to lipid abnormalities. The blood abnormality is seen in conditions such as abetalipoproteinemia, severe liver disease and severe malnutrition. Symptoms and prognosis depend on the underlying disorder.
• Achrestic anemia: Achrestic anemia is a form of anemia similar to that caused by Vitamin B12 deficiency but it doesn't respond to treatment with Vitamin B12. The condition tends to progress slowly and can result in death if not treated. There are a variety of possible causes.
• Acidemia, methylmalonic: An inborn error of metabolism where amino acids in the body aren't metabolized properly resulting in high levels of the acid throughout the body.
• Acquired idiopathic sideroblastic anaemia: A rare disorder where iron is transported into a developing blood cells but because it is unable to be used, it builds up within the cell and tends to stop it from developing into a fully functioning red blood cell. Thus anemia can occur despite adequate or even high iron levels. Acquired cases can occur on exposure to excess alcohol, lead and drugs or can occur to nutritional problems involving a deficiency of folic acid or copper or an excess of zinc. The condition can also be caused by conditions such as kidney problems, endocrine dysfunction, metabolic disorders, rheumatoid arthritis and leukemia.
• Acquired total lipodystrophy: A rare acquired disorder that involves adipose tissue abnormalities and is characterized by loss of adipose tissue through the body. The disorder is very similar to the congenital form but the liver involvement is more severe.
• Acral dysostosis - dyserythropoiesis: A rare disorder characterized by hand and foot defects as well as a congenital form of anemia characterized by the production of abnormal red blood cells.
• Acromegaly: An abnormal enlargement of the limbs due to increased secretion of growth hormone after the cessation of puberty
• Actinomycetales infection: A bacterial infection from the order of Actinobacteria. The range of symptoms is variable depending on which bacteria from the order is involved.
• Acute adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The acute subtype tends to progress rapidly and is the most prevalent form of the condition.
• Acute basophilic leukaemia: A rare type of acute myeloid leukemia characterized by the presence of abnormal basophils.
• Acute biphenotypic leukemia: A rare form of leukemia that has myeloid and lymphoid features.
• Acute cholinergic dysautonomia: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The acute form has more severe symptoms than the chronic form.
• Acute leukaemia of ambiguous lineage: A term used to describe a type of leukemia (a blood cancer) where the leukemic cells cannot be determined as myeloid or lymphoid or where both types of cells are present.
• Acute leukemia: An acute condition which affects a cell line of the blood which shows little or no differentiation
• Acute lymphoblastic leukemia: A malignant disease that starts suddenly and progresses quickly. It is characterized by a high number of immature cells in the organs, bone marrow and blood. Symptoms include fever, pallor, anorexia, fatigue, anemia, hemorrhage, bone pain, splenomegaly and frequent infections. Also called acute lymphocytic leukemia.
• Acute lymphoblastic leukemia, adult: Cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets.
• Acute megacaryoblastic leukemia: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. More specifically, it involves the rapid proliferation of megakaryoblasts (premature form of megakaryocytes).
• Acute myeloblastic leukemia type 1: A form of blood cancer resulting in the rapid proliferation of immature blood cells (blast cells).
• Acute myeloblastic leukemia type 2: A form of blood cancer resulting in the rapid proliferation of granulocytes and monocytes.
• Acute myeloblastic leukemia type 3: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 3 involves the proliferation of promyelocytes.
• Acute myeloblastic leukemia type 4: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 4 involves the rapid proliferation of myelocytes and monocytes.
• Acute myeloblastic leukemia type 5: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. Type 5 involves the rapid proliferation of monoblasts (immature precursors of monocytes) in particular.
• Acute myeloblastic leukemia type 6: A rare form of malignant bone marrow cancer involving the rapid proliferation of immature precursors of blood cells. Type 6 involves the proliferation of the immature precursors of red blood cells called erythroblasts.
• Acute myeloblastic leukemia type 7: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of blood cells. Type 7 involves the rapid proliferation of megakaryoblasts (premature form of megakaryocytes) in particular.
• Acute myelocytic leukemia: A malignant cancer of blood-forming tissues resulting in a high number of immature leukocytes. Symptoms include soft bleeding gums, anemia, fatigue, fever, dyspnea, moderate splenomegaly, joint and bone pains and frequent infections. Also called acute granulocytic leukemia, acute myelogenous leukemia, acute nonlymphocytic leukemia, myeloid leukemia, splenomedullary leukemia, splenomyelogenous leukemia.
• Acute myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
• Acute myeloid leukaemia and myelodysplastic syndromes related to alkylating agent: The use of alkylating agents to treat cancer can result in leukemia in some patients.
• Acute myeloid leukaemia and myelodysplastic syndromes related to topoisomerase type II inhibitor: The use of topoisomerase type II inhibitors to treat cancer can result in leukemia in some patients.
• Acute myeloid leukaemia and myelodysplastic syndromes, therapy related: Certain cancer therapies can result in the development of leukemia in some patients. These therapies includes topoisomerase type II inhibitors and alkylating agents.
• Acute myeloid leukemia: A form of rapidly progressing blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets.
• Acute myeloid leukemia, adult: A form of blood cancer resulting in the rapid proliferation of granulocytes and monocytes, red blood cells and platelets.
• Acute myelosclerosis: A rare disorder where the bone marrow makes too many blood cells. The disease progresses rapidly with death usually occurring within 6 months of onset.
• Acute panmyelosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement. The disease often progresses rapidly and results in death.
• Acute vitamin A toxicity: Acute ingestion of vitamin A can cause symptoms. Symptoms usually only last for a day or two.
• Acyl-CoA dehydrogenase, very long chain, deficiency of: A rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form.
• Addison-Gull syndrome: A condition where scarring of the liver and bile ducts which results in chronic jaundice, enlarged spleen and liver and yellow skin plaques due to abnormal lipid metabolism.
• Adrenoleukodystrophy: A rare hereditary metabolic disease that only occurs in male children and is characterized by adrenal atrophy and extensive cerebral demyelination causing progressive loss of mental functioning, aphasia, apraxia and sometimes blindness. The patient usually dies within 5 years.
• Adrenoleukodystrophy, autosomal, neonatal form: A rare inherited disorder involving the adrenal glands, testes and certain parts of the brain (white matter). It is a less severe form of leukodystrophy where an abnormality within the body cells prevents the metabolism of certain fats (long chain fatty acids).
• Adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis.
• Adult T-Cell lymphoma: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The lymphoma subtype is aggressive and tends to affect the lymph nodes more than the blood.
• Adult onset Still's disease: A form of Still's disease that has a later onset and involves arthralgia or arthritis and a characteristic rash that often appears during periods of temperature increase.
• Aflatoxin B1 exposure: Aflatoxin B1 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• Aflatoxin B2 exposure: Aflatoxin B2 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• Aflatoxin G1 exposure: Aflatoxin G1 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• Aflatoxin G2 exposure: Aflatoxin G2 is a toxin produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• Aflatoxin exposure: Aflatoxins are toxins produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. The fungus are found frequently in nature and can readily contaminate crops before harvest or during storage. It is common in moist soils and decaying vegetation. There are more than 13 different subtypes of aflatoxin with B1 being the most toxic. The condition is most common in poorly developed countries where there are insufficient controls on the presence of aflatoxin in food.
• African Sleeping sickness: A disease caused by parasites (Trypanosome brucei gamiense or T. brucei rodesiense) and transmitted to humans by the tsetse fly which is found only in Africa. Causes symptoms such as fever, chills, headache, anemia, edema of hands and feet, enlarged lymph glands, lethargy, sleepiness, convulsions and coma. Also called African trypanosomiasis and sleeping sickness.
• Aggressive NK-cell leukaemia: An aggressive form of blood cancer involving the rapid proliferation of natural killer (NK) cells.
• Aggressive systemic mastocytosis: The excessive proliferation of mast cells. Mast cells control the skin's response to minor injury and release a chemical called histamine which causes the skin to redden. In the aggressive form, mast cells accumulate in the liver, spleen and lymphatic system.
• Aicardi-Goutieres syndrome 1: A rare inherited progressive disease that affects the brain and immune system. Type 1 is caused by a defect on chromosome 3p21.3-p21.2.
• Aicardi-Goutieres syndrome 2: A rare inherited progressive disease that affects the brain and immune system. Type 2 is caused by a defect on chromosome 13q14-q21.
• Aicardi-Goutieres syndrome 3: A rare inherited progressive disease that affects the brain and immune system. Type 3 is caused by a defect on chromosome 11q13.2.
• Aicardi-Goutieres syndrome 4: A rare inherited progressive disease that affects the brain and immune system. Type 4 is caused by a defect on chromosome 19p13.13.
• Aicardi-Goutieres syndrome 5: A rare inherited progressive disease that affects the brain and immune system. Type 5 is caused by a defect on chromosome 3p21.3-p21.2.
• Alagille syndrome: A genetic disorder affecting the liver and characterized by the absence of some or all of the liver bile ducts that transport bile within the liver.
• Albers-Schonberg disease - malignant recessive form: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. The malignant infantile form is the most severe form of this disorder and death usually occurs in the first decade of life.
• Alcohol-induced pseudo-Cushing syndrome: The excessive consumption of alcohol can result in symptoms similar to a condition called Cushing's syndrome. When alcohol consumption is stopped, symptoms regress.
• Aldolase A deficiency: A rare condition where a deficiency of the enzyme called aldolase A causes muscle problems and anemia.
• Alpha thalassemia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. Alpha thalassemia involves defects in one or more of the four genes required to make each ? protein chain. The main symptom is anemia, the severity of which can vary amongst patients depending on how many defective genes are involved.
• Alpha thalassemia - Hemoglobin H disease: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. Hemoglobin H disease involves defects in three of the four genes required to make each ? protein chain. The main symptom is moderate to severe anemia.
• Alpha thalassemia major: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. Alpha thalassemia major is very rare involves defects in all of the four genes required to make each ? protein chain. The condition leads to infant death before or soon after birth.
• Alpha-mannosidosis type II: A rare inherited metabolic disorder involving a deficiency of an enzyme (alpha-mannosidosase) which results in the accumulation of certain chemicals in the body which leads to progressive damage. This form of the condition is less severe than type I (infantile form).
• Amyloidosis: A disease characterized by the accumulation of insoluble amyloid protein in tissues and organs which in turn affects the functioning of these tissues and organs.
• Amyloidosis AL: A disease involving the abnormal deposit of amyloid fibrils in virtually any part of the body - the heart, liver, kidney and peripheral and autonomic nerves are most commonly affected. The abnormal amyloid fibrils are produced abnormal plasma cells in the bone marrow. In some cases, the excess growth of abnormal plasma cells can result in a cancerous condition called myeloma resulting in bone pain and infections. A patient with myeloma may develop amyloidosis but it is rare for a patient with AL amyloidosis to go on to develop myeloma.
• Amyloidosis, familial visceral: A rare genetic disorder involving widespread amyloidosis (abnormal buildup of amyloid protein in tissues) which tends the affect the kidneys severely.
• Amyloidosis, inflammatory: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage and is potentially fatal. Symptoms depend on the organs involved. Secondary amyloidosis is caused by a chronic infection of inflammatory conditions such as rheumatoid arthritis, multiple myeloma, tuberculosis and osteomyelitis. The main organs affected in secondary amyloidosis are usually the kidneys, liver, spleen and lymph nodes. The peripheral and autonomic nerves and the heart are rarely affected.
• Andersen disease: An rare inborn error of metabolism involving glycogen storage and characterized by cirrhosis and sometimes liver failure. Lack of the amyl-transglucosidase enzyme and abnormal glycogen causes the condition.
• Anemia: Reduced red blood cells in the blood
• Anemia, Blackfan Diamond: Diamond-Blackfan anemia is a rare genetic condition where the bone marrow is unable to make sufficient red blood cells which leads low levels of red blood cells. There are eight subtypes of the condition which differ in the location of the genetic defect and the incidence of additional symptoms such as malformations. The severity of symptoms is variable but most cases are serious.
• Anemia, Hemolytic, Warm Antibody: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 37ºC or higher. The severity of the disorder is variable.
• Anemia, Neonatal: Insufficient red blood cells that can carry oxygen around the body. It is common in premature births or can occur as a result of blood loss before, during or just after the birth.
• Anemia, Sideroblastic: A rare blood disorder where abnormal utilization of iron results in the production of defective red blood cells which have excessive deposits of iron in them.
• Anemia, hypochromic microcytic: A blood disorder where red blood cells are too small and lack sufficient iron. It can be inherited or caused by insufficient iron in the diet or from a genetic disorder.
• Anemias, Sideroblastic: Sideroblastic anemias are a group of rare blood disorders where the bone marrow is unable to produce normal red blood cells. The body has enough iron but the red blood cells are unable to utilize it in a normal manner and anemia results. The red blood cells become overloaded with iron and are unable to carry out their normal functions. Some forms of sideroblastic anemia are inherited but most tend to be acquired due to such things as exposure to toxins and certain drugs, leukemia, inflammatory conditions such as rheumatoid arthritis and nutritional deficiencies (e.g. copper and pyridoxine deficiency). Inherited forms usually appear in childhood whereas acquired forms usually occur in adulthood.
• Anesthetic agent-induced liver damage: Damage or injury to the liver caused by exposure to anesthetic agents. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Chloroform: Damage or injury to the liver caused by exposure to an anesthetic agent called chloroform. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Cyclopropane: Damage or injury to the liver caused by exposure to an anesthetic agent called cyclopropane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Ether: Damage or injury to the liver caused by exposure to an anesthetic agent called ether. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Halothane: Damage or injury to the liver caused by exposure to an anesthetic agent called halothane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Methoxyflurane: Damage or injury to the liver caused by exposure to an anesthetic agent called methoxyflurane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anesthetic agent-induced liver damage - Nitrous Oxide: Damage or injury to the liver caused by exposure to an anesthetic agent called nitrous oxide. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Angiofollicular lymph hyperplasia: A rare disorder of the lymph system characterized by the development of benign tumors in lymph tissue anywhere in the body.
• Angioimmunoblastic with dysproteinemia lymphadenopathy: A rare immune system disorder which is similar to lymphoma. The condition is progressive but the course varies with some patients surviving a long time without treatment and others surviving only a short period of time.
• Angiosarcoma of the liver: A rare type of cancer that starts in the lining of blood vessels in the liver. It is generally an aggressive tumor which often metastasizes.
• Antibiotics-induced liver damage: Damage or injury to the liver caused by taking certain antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Cephalosporin: Damage or injury to the liver caused by exposure to Cephalosporin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Chloramphenicol: Damage or injury to the liver caused by exposure to an antibiotic called Chloramphenicol. Chloramphenicol a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Clindamycin: Damage or injury to the liver caused by exposure to Clindamycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Erythromycin Ethyl succinate: Damage or injury to the liver caused by exposure to Erythromycin Ethyl succinate antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Erythromycin estolate: Damage or injury to the liver caused by exposure Erythromycin estolate antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Novobiocin: Damage or injury to the liver caused by exposure to Novobiocin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Quinolone: Damage or injury to the liver caused by exposure to Quinolone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Spectinomycin: Damage or injury to the liver caused by exposure to Spectinomycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Sulfones: Damage or injury to the liver caused by exposure to sulfone antiboitics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Telithromycin: Damage or injury to the liver caused by exposure to Telithromycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Tetracycline: Damage or injury to the liver caused by exposure to Tetracycline antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Nitrofuran: Damage or injury to the liver caused by exposure to Nitrofuran antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Penicillin: Damage or injury to the liver caused by exposure to Penicillin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antibiotics-induced liver damage - Rifampicin: Damage or injury to the liver caused by exposure to Rifampicin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage: Damage or injury to the liver caused by exposure to anticonvulsives. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage - Mephenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called mephenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage - Phenobarbital: Damage or injury to the liver caused by exposure to an anticonvulsive called phenobarbital. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage - Phenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called Phenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Anticonvulsive-induced liver damage - Valproic Acid: Damage or injury to the liver caused by exposure to an anticonvulsive called valproic acid. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage: Damage or injury to the liver caused by exposure to antifungal agents. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - 5-Fluorocytosine: Damage or injury to the liver caused by exposure to an antifungal agent called 5-Fluorocytosine. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - Amphotericin: Damage or injury to the liver caused by exposure to an antifungal agent called Amphotericin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - Griseofulvin: Damage or injury to the liver caused by exposure to an antifungal agent called Griseofulvin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - Ketoconazole: Damage or injury to the liver caused by exposure to an antifungal agent called Ketoconazole. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antifungal agent-induced liver damage - Saramycetin: Damage or injury to the liver caused by exposure to an antifungal agent called Saramycetin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage: Damage or injury to the liver caused by exposure to Antimetazoal agents. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage - Amodiaquine: Damage or injury to the liver caused by exposure to an antimetazoal agent called amodiaquine. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antimetazoal agent-induced liver damage - Hycanthone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called hycanthone. Antimetazoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage: Damage or injury to the liver caused by exposure to antiprotozoal agents. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - 8-Hydroxyquinolone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called 8-Hydroxyquinolone. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Carbarsone: Damage or injury to the liver caused by exposure to an antiprotozoal agent called carbarsone. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Emetine: Damage or injury to the liver caused by exposure to an antiprotozoal agent called emetine. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Mepacrine: Damage or injury to the liver caused by exposure to an antiprotozoal agent called mepacrine. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Metronidazole: Damage or injury to the liver caused by exposure to an antiprotozoal agent called Metronidazole. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiprotozoal agent-induced liver damage - Thiabendazole: Damage or injury to the liver caused by exposure to an antiprotozoal agent called Thiabendazole. Antiprotozoal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage: Damage or injury to the liver caused by exposure to antituberculous agents. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - Cycloserine: Damage or injury to the liver caused by exposure to an antituberculous agent called cycloserine. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - Ethionamide: Damage or injury to the liver caused by exposure to an antituberculous agent called ethionamide. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - Isoniazid: Damage or injury to the liver caused by exposure to an antituberculous agent called isoniazid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - Rifampicin: Damage or injury to the liver caused by exposure to an antituberculous agent called rifampicin. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antituberculous agent-induced liver damage - p-aminosalicylic acid: Damage or injury to the liver caused by exposure to an antituberculous agent called p-aminosalicylic acid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage: Damage or injury to the liver caused by exposure to antiviral agents. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage - Cytarabine: Damage or injury to the liver caused by exposure to an antiviral agent called cytarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage - Vidarabine: Damage or injury to the liver caused by exposure to an antiviral agent called vidarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage - idoxuridine: Damage or injury to the liver caused by exposure to an antiviral agent called idoxuridine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Antiviral agent-induced liver damage - xenylamine: Damage or injury to the liver caused by exposure to an antiviral agent called xenylamine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Aplastic anemia: A blood disorder where the bone marrow produces insufficient new blood cells.
• Apo A-I deficiency: Low plasma HDL cholesterol that tends to run in families.
• Arakawa's syndrome 2: An inherited metabolic disorder where an enzyme deficiency (methionine synthase) causes mental and physical retardation, blood disorders, degeneration of brain tissue and various other symptoms.
• Arctic bearded seal poisoning: The Arctic Bearded seal is often used as a food source by the arctic inhabitants. Eating the liver and kidneys of the arctic bearded seal can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases. It is believed that eating more than 100-250 grams of the seal liver can result in human death.
• Arginase deficiency: A very rare urea cycle disorder caused by a deficiency of the enzyme (arginase) needed to convert ammonia to the urea which can then be removed in the urine. The condition leads to excess build-up of ammonia in the body which is toxic to the nervous system.
• Argininosuccinase lyase deficiency, neonatal: A rare inherited urea cycle disorder caused by lack of enzymes (argininosuccinase lyase) needed to turn ammonia into urea resulting in excess ammonia in the body. The neonatal form of the condition can result in death or severe complications if not treated early enough.
• Argininosuccinic aciduria: A rare inherited disorder of the urea cycle characterized by the lack of an enzyme (argininosuccinate lyase) which is needed to remove nitrogen from the body so a lack of the enzyme leads to a build-up of ammonia in the blood.
• Arthralgia - purpura - weakness syndrome: A rare autoimmune disorder characterized mainly by weakness, joint pain, reddish or purplish skin discolorations and a blood abnormality where cryoglobulins in the blood precipitate at low temperatures.
• Aspartylglucosaminidase deficiency: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
• Aspartylglucosaminuria: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
• Aspartylglycosaminuria: A rare glycoprotein metabolism disorder caused by a deficiency of an enzyme called aspartylglucosaminidase. Patients tend to develop normally during the first few years of life and development continues slowly until adolescence when mental retardation becomes progressively worse.
• Asrar-Facharzt-Haque syndrome: A form of sinus histiocytosis characterized by lymphadenopathy as well as involvement of other tissues such as the sinus cavity, skin, lungs, bone, eyes, kidneys, testes, head and neck area and the central nervous system. Symptoms vary according to the organ involved. The condition usually runs a prolonged course with spontaneous remission occurring in some cases. Rare cases may be fatal.
• Athabaskan severe combined immunodeficiency: A severe immunodeficiency disorder found in Navajo and Apache populations.
• Australian Sea Lion poisoning: The Australian Sea Lion is sometimes used as a food source and is found in the South-Southwest waters of Australia. Eating the liver of the Australian Sea Lion can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases.
• Autoimmune Hemolytic Anemia: Autoimmune hemolytic anemia is characterized by an abnormal immune system response which leads to the destruction of red blood cells and hence anemia. The severity of the condition varies depending on the underlying cause e.g. cytomegalovirus, hepatitis, HIV and lupus. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Autoimmune Hepatitis: Liver inflammation caused due to autoimmune processes where the body's immune system attacks the liver.
• Autoimmune Lymphoproliferative Syndrome: An inherited autoimmune condition characterized by proliferation of lymphocytes and autoimmunity against the body's own blood cells resulting in premature death of certain blood cells.
• Autoimmune Thrombocytopenia: Autoimmune disorder causing a lack of blood platelets.
• B-cell chronic lymphocytic leukemia: A slow progressing disease involving cancerous B-cell lymphocytes which take over the healthy cells in the lymph nodes. B-cells help the body to fight infections so when the disease becomes more advanced, the body is less able to fight infection as there are fewer healthy, functioning B-cells.
• B-cell prolymphocytic leukaemia: A rare type of leukemia involving a proliferation of immature white blood cells (prolymphocytes - B-cells).
• Babesiosis: A protozoal infection which is transmitted to human via the bite of certain ticks.
• Baker-Winegrad disease: A very rare syndrome caused by a deficiency of the enzyme fructose-1-6-diphosphatase which impairs the body's ability to break down fructose that is consumed in the diet.
• Ballard syndrome: A rare condition characterized by enlarged liver and spleen and fractures that occur in areas of weakened bone.
• Banti's syndrome: A chronic, progressive condition marked by enlargement of the spleen which is associated with anemia, splenomegaly, ascites, jaundice, leucopenia, thrombocytopenia and gastrointestinal bleeding.
• Bantu siderosis: An iron overload disorder initially observed in South African people. The disorder involves abnormal iron deposits in the liver. It is believed that some African people are predisposed to an increased ability to absorb iron.
• Bartonella: A class of bacteria that can infect humans at a range of different sites. The most well known is Cat Scratch Disease, caused by B.henselae.
• Bartonella infections: Infection with bacteria from the Bartonella genus of bacteria. Specific bacteria from within this group are Bartonella bacilliforms (Oroya fever), Bartonella Heneslae (Cat-scratch disease). Other conditions caused by this bacteria are endocarditis, bacteremia and angiomatosis. Symptoms vary depending on the type of bacteria involved and the severity of the infection - immunocompromised patients face greater risk of severe infection.
• Bartonellosis: An infection by Bartonella bacilliformis which is transmitted through sandfly bites. Symptoms include fever, severe anemia, bone pain and skin lesions. Also called Carrion's disease, Oroya fever or verruga peruana.
• Bartonellosis due to Bartonella quintana infection: A disease caused by infection with Bartonella quintana which are transmitted by the body louse. It causes trench fever but may also result in septicemia and endocarditis in patients with a weakened immune system.
• Bearn-Kunkel syndrome: A type of autoimmune liver disease characterized by liver damage, very high blood gammaglobulin levels and increased plasma cells.
• Bejel: An infectious disease related to syphilis but is transmitted through nonsexual skin contact. Often starts with a sore in the mouth and then progresses to affect the skin and bones.
• Benign intrahepatic cholestasis type 1: A rare liver disorder characterized by intermittent episodes of cholestasis (impaired bile flow within the liver) that doesn't involve obstruction of bile ducts outside the liver. The jaundice tends to disappear in weeks or months without treatment and the condition does not lead to liver failure. Type 1 is caused by a defect on the ATP8B1 gene on chromosome 18q21.
• Benign intrahepatic cholestasis type 2: A rare liver disorder characterized by intermittent episodes of cholestasis (impaired bile flow within the liver) that doesn't involve obstruction of bile ducts outside the liver. The jaundice tends to disappear in weeks or months without treatment and the condition does not lead to liver failure. Type 2 is caused by a defect on the ABCB11 gene on chromosome 2q24.
• Berardinelli-Seip congenital lipodystrophy: A rare genetic disorder characterized by diabetes mellitus, loss of body fat, hepatomegaly, enlarged genitals, increased skeletal growth and other abnormalities.
• Berardinelli-Seip congenital lipodystrophy, type 1: A rare genetic disorder characterized by early-onset diabetes mellitus, loss of body fat, serious insulin resistance, high blood triglycerides and fatty liver. Type 1 is distinguished from type 2 by the origin of the genetic defect. Type 1 is caused by a defect on the AGPAT2 gene on chromosome 9q34.3. Type 1 seems to be less severe with some cases of type 2 resulting in premature death which can occur as early as the first year of life. Type 2 also involves mental retardation which is not seen in type 1.
• Berardinelli-Seip congenital lipodystrophy, type 2: A rare genetic disorder characterized by early-onset diabetes mellitus, loss of body fat, serious insulin resistance, high blood triglycerides and fatty liver. Type 2 is distinguished from type 2 by the origin of the genetic defect. Type 2 is caused by a defect on the BSCL2 gene on chromosome 11q13. Type 2 seems to be more severe with some cases resulting in premature death which can occur as early as the first year of life. Type 2 also involves mental retardation which is not seen in type 1.
• Bernheim's syndrome: Reduced size of right heart ventricle due to enlargement of the left ventricle which encroaches on the space in the right ventricle. Blood flow from the right atrium to the right ventricle is obstructed.
• Besnier-Boeck-Schaumann disease: A rare disease where inflammatory granular nodules form in various organs.
• Beta Thalassemia intermedia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. There are two subtypes of the disorder (? and ?) depending on what portion of the hemoglobin is abnormally synthesized. Beta Thalassemia intermedia involves defects in both of the two genes required to make each ? protein chain. The condition causes varying degrees of moderate anemia.
• Beta thalassemia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. Beta thalassemia involves defects in one or more of the two genes required to make each ? protein chain. The main symptom is anemia, the severity of which can vary amongst patients depending on how many defective genes are involved.
• Bile Duct Cancer: A malignancy arising in the bile ducts of the liver
• Bile Duct Conditions: An inflammatory bacterial infection that affects the meninges
• Bile acid synthesis defect, congenital, 2: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (cholestasis with delta(4)-3-oxosteroid 5-beta-reductase) needed to make bile acid.
• Bile acid synthesis defect, congenital, 4: A defect which prevents the body from making bile acid which results in progressive liver disease.
• Bile acid synthesis defects: A defect which prevents the body from making bile acid which results in progressive liver disease.
• Bile acid synthesis defects, congenital, 1: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (3-beta-hydroxy-delta-5-C27-steroid oxidoreductase) needed to make bile acid.
• Bile acid synthesis defects, congenital, 2: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect is a deficiency of a particular enzyme (cholestasis with delta(4)-3-oxosteroid 5-beta-reductase) needed to make bile acid.
• Bile acid synthesis defects, congenital, 3: A defect which prevents the body from making bile acid which results in progressive liver disease. The defect involved a deficiency of 7-alpha-hydroxylase which is an enzyme needed to prevent the accumulation of 27-hydroxycholesterol which is toxic to the liver.
• Bile acid synthesis defects, congenital, 4: A defect which prevents the body from making bile acid which results in progressive liver disease.
• Bile plug syndrome: A rare condition where thick bile in infants causes blockage of the bile ducts which can result in jaundice, enlarged liver and anemia.
• Biliary Atresia: A rare condition that can affect newborns where the bile duct that carries the bile from the liver to the small intestine is blocked or missing.
• Biliary atresia, intrahepatic, non syndromic form: Congenital obstruction of the passages in the liver that carry bile. The nonsyndromic form is not associated with any other abnormalities.
• Biliary atresia, intrahepatic, syndromic form: Congenital obstruction of the passages in the liver that carry bile. The syndromic form is associated with other congenital abnormalities such as heart and visceral defects.
• Biliary disorder: Any condition affecting the bile ducts
• Biliary hypoplasia: An underdeveloped biliary duct system which is involved in transporting bile. The bile ductules may be completely absent or be fewer in number than normal.
• Biotinidase deficiency, late onset: A metabolic disorder where the body lacks the enzyme biotinidase needed to process the vitamin called biotin (vitamin H) into carboxylase enzymes. The severity of symptoms may vary depending on the degree of deficiency. Severe cases can result in metabolic acidosis which can lead to death if treatment isn't given.
• Birth symptoms: Symptoms related to childbirth.
• Blastoma: A type of tumor that originates from precursor cells or blasts (immature or embryonic tissue). The symptoms can vary greatly and are determined by the part of the body that is affected. Blastomas can occur in parts of the body such as the brain, liver, kidneys, nervous system, bones and the retina.
• Boutonneuse fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Breynia officinalis poisoning: Ingestion of the Breynia officinalis plant can cause irritation to mucosal linings and liver problems. The plant is often used as a herbal drug (Chi R Yun) to treat such things as poor growth, heart failure and venereal disease.
• Brucellosis: An infectious disease caused by the Brucella genus which is transmitted from animals to humans.
• Bruch's disease: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Brunzell syndrome: A rare recessively inherited syndrome characterized by a lack of skin fat (Seip syndrome), cystic angiomatosis of the long bones which can lead to fractures.
• Budd chiari syndrome: A disorder where the main vein leaving the liver becomes blocked leading to symptoms such as liver enlargement and fluid buildup in the abdomen. Also called Chiari's syndrome or Rokitansky's disease.
• Budd-Chiari syndrome: A disorder where the main vein leaving the liver becomes blocked leading to symptoms such as liver enlargement and fluid buildup in the abdomen. Also called Chiari's syndrome or Rokitansky's disease.
• CCF: When the heart is no longer able to pump enough blood to meet the needs of the body.
• CDG syndrome (generic term): Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. The main symptom in all the disorders is psychomotor retardation but other variable symptoms also occur depending on the subtype of the disorder.
• CDG syndrome type 1A: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
• CDG syndrome type 1B: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1B has a phosphomannose isomerase enzyme defect.
• CDG syndrome type 3: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 3 has variable symptoms.
• CDG syndrome type I: A rare genetic disorder where the body is unable to synthesize glycoproteins which results in multisystem problems.
• CML-Like Syndrome, Familial: A very rare condition characterized by symptom similar to myelocytic leukemia that develop during infancy. The condition can result in death during the first years of life.
• CMV antenatal infection: A rare condition where a fetus becomes infected with the cytomegalovirus through the mother.
• Camurati-Engelmann Disease: A very rare genetic disease thickening of areas of bone causing pain, weakness and wasting. Usually affects the legs during childhood. Also called diaphyseal dysplasia.
• Carbamoyl-phosphate synthase 1 deficiency: A very rare inherited urea cycle disorder where the lack of the enzyme carbamoyl phosphate synthetase prevents ammonia from being turned into urea and being excreted in the urine. Excess ammonia builds up in the body which can cause serious complications or even death if left untreated.
• Carcinoid syndrome: Carcinoid heart disease is a rare, metastatic disease that occurs predominantly in the right heart. The tricuspid and pulmonic valves are affected, leading to right heart failure, which results in increased morbidity and mortality.
• Cardiomyopathy - hypogonadism - metabolic anomalies: A rare syndrome characterized mainly by heart muscle disease, hypogonadism, blindness, deafness and metabolic anomalies. Puberty was normal despite the hypogonadism.
• Cardiomyopathy - spherocytosis: A rare disorder characterized by the association of spherocytosis with heart muscle disease. Spherocytosis is a red blood cell disorder where the red blood cells have abnormal membranes which gives them a spherical shape and makes them weak resulting in their premature death.
• Cardiomyopathy due to anthracyclines: Damage to the heart muscle caused by anthracycline drugs which are used in chemotherapy. The damage occurs more frequently with higher cumulative doses. Often the patients have no symptoms of the heart damage for many year.
• Carnitine palmitoyl transferase 1 deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase I) prevents fatty acids being transported to the part of the cell that converts it to energy.
• Carnitine palmitoyl transferase 2 deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase) which prevents fatty acids being transported to the part of the cell that converts it to energy. There are two main subtypes of the disorder with each involving a slightly different form of the enzyme. Type I can be readily managed through diet. Type II has three subtypes: the myopathic form affects mainly the muscles; the hepatocardiomuscular form affects the liver and heart muscle; and the lethal neonatal form affects muscles and organs and usually results in death during the first year of life.
• Carnitine palmitoyl transferase II deficiency, infantile hepatocardiomuscular type: A very rare metabolic disorder where deficiency of a particular enzyme (CPT II) prevents muscle fats being converted to energy. The infantile form of this disease affects the muscles and the liver and heart.
• Carnitine palmitoyl transferase deficiency: A very rare inherited deficiency of a particular enzyme (Carnitine palmitoyl transferase) which prevents fatty acids being transported to the part of the cell that converts it to energy. There are two main subtypes of the disorder with each involving a slightly different form of the enzyme. Type I can be readily managed through diet. Type II has three subtypes: the myopathic form affects mainly the muscles; the hepatocardiomuscular form affects the liver and heart muscle; and the lethal neonatal form affects muscles and organs and usually results in death during the first year of life.
• Carnitine transporter deficiency: An inherited deficiency of carnitine caused by the impaired ability of the carnitine transporter protein to carry the carnitine to where it is needed. Instead the carnitine is excreted through the urine. Fasting or illness can trigger a severe attack.
• Carnitine-acylcarnitine translocase deficiency: A very rare inherited metabolic disorder where long-chain fatty acids can't be metabolized properly because the compound needed to transport it is faulty. Ultimately this prevents certain fats (long-chain acylcarnitine) being converted to energy and results in a build up of the fat which is harmful to body organs and tissues.
• Caroli Disease: A rare disorder where the bile ducts inside the liver become enlarged resulting in infection, irritation and gallstone formation.
• Caspase-8 deficiency: A rare type of immunodeficiency disorder caused by a deficiency of caspase-8. Caspase-8 an important part of the immune system as it is involved in the activation of T lymphocytes, B lymphocytes and natural killer cells.
• Castellani syndrome: A rare syndrome characterized by fever, arthritis and enlarged kidney and liver. The fever may fluctuate or come and go. It is most often seen in middle aged males.
• Cat scratch disease: An infectious disease transmitted through a cat's bite, scratch or lick and resulting primarily in lymph node pain and swelling. The condition can be mild or severe.
• Cephalosporin-induced Immune Hemolytic Anemia: Cephalosporin-induced immune hemolytic anemia is a condition where a use of a medication called Cephalosporin triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Cerebrorenodigital syndrome: A rare group of syndromes characterized mainly by brain, kidney, finger and toe abnormalities.
• Chagas disease: A parasitic infection caused by the protozoa Trypanosoma cruzi and transmitted by insect bites or blood transfusions. The disease primarily involves the heart and gastrointestinal system.
• Chediak-Higashi Syndrome: An inherited immune system disorder resulting in frequent infections, lack of skin and eye pigmentation, neurological diseases and early death.
• Chediak-Higashi like syndrome: A rare genetic disorder characterized mainly by albinism (lack of pigmentation). There are three different subtypes of the disorder (I, II and III) each with varying additional features such as immunodeficiency and neurological symptoms. Type 1 involves partial albinism and neurological symptoms, type II involves partial albinism, immunodeficiency and sometimes neurological symptoms and type III involves albinism only.
• Chemical poisoning - 1,2-Dibromoethane: 1,2-Dibromoethane is a chemical used in gasoline, soil fumigants, fire extinguishers, flue gases and mechanical gauge fluid. Excessive exposure to this chemical can cause serious symptoms. Some people can suffer an adverse reaction to the chemical. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chlorate salts: Chlorate salt is a chemical used mainly in herbicides and in the manufacture of matches and explosives. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Chloroform: Chloroform is a chemical used mainly as a refrigerant but also as a solvent in various processing and industrial applications. It's use as an anesthetic is relatively uncommon these days. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Dimethylnitrosamine: Dimethylnitrosamine is a chemical used mainly as a solving in the manufacture of plastics, rubbers, lubricants and rocket fuel. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Methylene Dianiline: Methylene Dianiline is a chemical used mainly in corrosive inhibitors, epoxy resins and polyurethane. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Phosphine: Phosphine is a chemical used mainly in pesticides and rodenticides. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Tetrachloroethylene: Tetrachloroethylene is a chemical used mainly as a fabric dry cleaner, degreaser, worming treatment for animals and in the manufacture of freons. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chemical poisoning - Vinyl Choride: Vinyl Choride is an intermediate chemical used in the manufacturing process of a variety of products: polyvinyl chloride, pipes, wire coverings, vehicle plastics, rubber, paper, furniture and glass. Ingestion and other exposures to the chemical can cause various symptoms. The type and severity of symptoms varies depending on the amount of chemical involved and the nature of the exposure.
• Chloroleukemia: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Chloroma: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Chloromyeloma: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Cholestasis - lymphoedema, syndrome: A rare inherited birth abnormality involving underdeveloped lymph vessels which results in swollen legs and liver problems.
• Cholestasis, progressive familial intrahepatic 1: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage.
• Cholestasis, progressive familial intrahepatic 2: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage. The condition has an early onset and usually leads to end-stage liver disease by the end of the second decade. The various types of this condition differ in the origin of the genetic defect (liver-specific ATP-binding cassette transporter on chromosome 2q24). Type 2 is also associated with an increased risk of liver cancer in the first few years of life.
• Cholestasis, progressive familial intrahepatic 3: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage. The condition has an early onset and usually leads to end-stage liver disease by the end of the second decade. The various types of this condition differ in the origin of the genetic defect (liver-specific ATP-binding cassette transporter on chromosome 2q24). Type 2 is also associated with an increased risk of liver cancer in the first few years of life.
• Choriocarcinoma, infantile: A rare malignant cancer that originates in the placenta. It is an aggressive cancer which generally results in infant death.
• Chromosome 11, deletion 11p: A syndrome that is caused by the deletion of short arm (p) of chromosome 11. The type and severity of symptoms varies depending on the amount and exact location of the genetic material that is deleted.
• Chronic Granulomatous Disease: A very rare inherited blood disorder where certain cells involved with immunity (phagocytes) are unable to destroy bacteria and hence the patient suffers repeated bacterial infections.
• Chronic Idiopathic Myelofibrosis: The bone marrow is consists of tissues which make blood cells and fibrous tissue supports these tissues that make the blood cells. In chronic idiopathic myelofibrosis, abnormal cells and fibres build up inside the bone marrow resulting in the production of fewer normal blood cells.
• Chronic Myeloproliferative Disease, Unclassified: A form of blood disorder characterized by the abnormal proliferation of myeloid precursors in the bone marrow. This category refers to cases of myeloproliferative disease which don't fit into any of the other specific type of myelproliferative diseases.
• Chronic Myeloproliferative Disorders: A group of blood cancers where excessive numbers of blood cells are made by overactive or cancerous bone marrow. The number of excess blood cells tends to grow slowly. Examples of such disorders includes chronic myelogenous leukemia, polycythemia vera and essential thrombocythemia. The symptoms are determined by which particular blood cancer is involved.
• Chronic Neutrophilic Leukemia: A rare form of leukemia characterized by excessive levels of mature neutrophils.
• Chronic Pesticide poisoning - xylene: Xylene is an ingredient used in certain insecticides. Exposure to the chemical can cause a range of symptoms depending on the level and route of exposure. Exposure can occur through inhalation, ingestion, the skin or eyes. Acute exposure involves a exposure over a short period of time whereas chronic exposure occurs over a longer period of time.
• Chronic adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The acute form tends to progress relatively slowly and generally responds better to treatment than the other subtypes.
• Chronic fatigue syndrome: A persistent debilitating fatigue of recent onset
• Chronic lymphocytic leukemia: A cancer of the blood and bone marrow where too many abnormal lymphocytes (white blood cells) are produced which eventually crowds out healthy blood cells in the body.
• Chronic myelogenous leukemia: A slow-growing cancer of the white blood cells where the bone marrow makes too many white blood cells which eventually invade various parts of the body.
• Chronic myeloid leukemia: Type of leukemia mostly in adults; rarely in children.
• Chronic myelomonocytic leukemia: A rare form of malignant bone marrow cancer involving the proliferation of immature precursors of certain blood cells - myelocytes and monocytes. The proliferation is slower than in acute forms of the disease.
• Chronic vitamin A toxicity: Chronic excessive ingestion of vitamin A can cause symptoms.
• Chédiak-Higashi syndrome: An inherited immune system disorder characterized by reduced pigmentation, recurrent infection and neurological disorders.
• Cirrhosis, familial: Liver cirrhosis that is inherited in a familial pattern. The liver scarring (cirrhosis) is not caused by any discernable disease process. The liver becomes progressively scarred and its function is impaired.
• Classic galactosemia: Rare serious genetic defect in galactose metabolism.
• Cold Autoimmune Hemolytic Anemia: Cold autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The red cells are destroyed at an abnormally rapid rate which leads to anemia. Cold haemolytic anemia is characterized by the fact that the abnormal destruction of red blood cells is more active when the patient is exposed to cold temperatures. The severity of the condition varies depending on the underlying cause. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Cold antibody hemolytic anemia: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 30ºC or lower.
• Common Variable Immunodeficiency: An immunodeficiency disorder involving low blood gamma globulin levels which results in an increased susceptibility to infections. The condition may be inherited or can be caused by certain drugs (levamisole, hydantoin and carbamazepine).
• Complex 1 mitochondrial respiratory chain deficiency: A rare genetic defect where an enzyme deficiency (NADH CoQ) disrupts cellular processes and causes various organic acid disorders. Any of a variety of the components of the enzyme may be missing or defective and hence the clinical presentation and severity may vary. Presentation may range from infantile death to various disorders such as Leigh's disease, Parkinson's disease and cardiomyopathy.
• Complex 5 mitochondrial respiratory chain deficiency: A rare genetic defect where an enzyme deficiency (ATP synthetase) disrupts cellular processes. Any of a variety of the components of the enzyme may be missing or defective and hence the clinical presentation and severity may vary. The deficiency may result in a variety of symptoms and conditions of variable severity such as Leber's myopathy, Leigh syndrome, cardiomyopathy and NARP (neuropathy, ataxia, retinitis pigmentosa).
• Congenital Disorders of Glycosylation Type Ia: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
• Congenital Toxoplasmosis: Fetal infection with toxoplasmosis.
• Congenital cytomegalovirus: Fetal infection with cytomegalovirus.
• Congenital disorder of glycosylation type 1/IIX: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type I/IIX refers to cases where the specific abnormality cannot be determined.
• Congenital disorder of glycosylation type 1A: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1A involves a phosphomannomutase enzyme defect and affects most body systems especially the nervous system and liver function.
• Congenital disorder of glycosylation type 1B: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1B has a phosphomannose isomerase enzyme defect.
• Congenital disorder of glycosylation type 1H: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ih is caused by a defect on chromosome 11pter-p15.5 and involves the gene for a particular enzyme (dolichyl-P-glucose:Glc-1-Man-9-GlcNAc-2-PP-dolichyl-alpha-3-glucosyltransferase).
• Congenital disorder of glycosylation type 1I: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ii is caused by a defect on chromosome 9q22 and involves a defect on the ALG2 gene.
• Congenital disorder of glycosylation type 1K: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Ik is caused by a defect on chromosome 16p13.3 and involves a defect in the gene for beta-1,4-mannosyltransferase. The disorder is generally fatal within a year or two of birth.
• Congenital disorder of glycosylation type 1L: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type Il is caused by a defect on chromosome 11q23 and involves a defect in the ALG9 gene.
• Congenital disorder of glycosylation type 1X: Congenital disorder of glycosylation is a rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1X also involves thrombocytopenia with normal levels of phosphomannomutase and phosphomannose isomerase. This form of the condition is severe and results in death during infancy.
• Congenital disorder of glycosylation type 2E: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 2e is caused by a defect on chromosome 16p and involves a defect in the gene for oligomeric complex-7.
• Congenital disorder of glycosylation type 2G: Congenital disorders of glycosylation is a group of very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type IIg is caused by a defect on chromosome 17q25.1 and involves a defect on the COG1 gene.
• Congenital disorder of glycosylation, type In: A very rare inherited metabolic disorder where defective carbohydrate compounds are attached to glycoproteins and thus impairing glycoprotein function. Type 1n involves a defect in the RFT1 gene and is characterized mainly by enlarged liver, seizures, developmental delay, reduced muscle tone and abnormal blood coagulation.
• Congenital hepatic fibrosis: A rare inherited birth disorder characterized by fibrosis (scarring) of the liver which affects its ability to function. The severity of the condition is variable with some patients being symptomatic during infancy while others may be asymptomatic for most of their life.
• Congenital hepatic porphyria: A rare congenital disorder where there is an excess of porphyrin (pigments) in the body. The liver is responsible for making porpyrins.
• Congenital herpes simplex: An infant born with a herpes simplex infection transmitted through the mother. The infection may be localized or involve various internal organs and even the central nervous system in which case death can occur.
• Congenital malaria: Fetal infection with malaria.
• Congenital spherocytic hemolytic anemia: A blood disorder present at birth where the membranes of red blood cells are defective which results in them being spherical rather than doughnut-shaped. These abnormally shaped red blood cells are broken down prematurely by the body which results in anemia. The condition may range from mild to severe.
• Congenital syphilis: Syphilis inherited from mother during pregnancy.
• Congenital tuberculosis: Fetal infection with tuberculosis
• Congestive heart failure: A condition which is characterized by breathlessness due to oedema and congestion of the lungs
• Conor's disease: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Constrictive pericarditis: Inflammation, swelling and thickening of the pericardium (fibrous sac surrounding the heart) which causes it to tighten around the heart and affect its function. The condition may be misdiagnosed as a heart attack and vice versa.
• Constrictive tuberculous pericarditis: Inflammation and swelling of the pericardium (fibrous sac surrounding the heart) that occurs as a complication of tuberculosis. The condition may be misdiagnosed as a heart attack and vice versa.
• Cooley syndrome: An inherited blood disorder involving abnormal production of red blood cells which causes serious anemia. It is a severe form of thalassemia.
• Cryoglobulinemia: These are immune cells that precipitate in the cold and redissolve on warming.
• Cyclic neutropenia: A rare blood disorder characterized by recurrent periods of extremely low blood levels of neutrophils which results in frequent infections. The low level usually occurs for about 7 days every 21 days. Levels of other blood components may also be affected.
• Cystic Fibrosis: Cystic fibrosis is a hereditary disease affecting the exocrine (mucus) glands of the lungs, liver, pancreas, and intestines, causing progressive disability due to multisystem failure.
• Cytomegalic Inclusion Body Disease: An infection due to cytomegalovirus and marked by nuclear inclusion bodies in enlarged infected cells
• Cytomegalovirus: A easily transmissible viral infection that is common but generally causes no symptoms except in infants and people with weakened immune systems.
• DOC 12 (Neutral Lipid Storage Type): A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities. The body is unable to metabolize (break down) triglycerides so they build up in various tissues throughout the body. The severity of the symptoms is variable as the body tissues and organs affected varies amongst patients.
• Deafness - lymphoedema - leukemia: A rare syndrome characterized by deafness, early-onset leukemia and lymphoedema in the lower legs.
• Deafness peripheral - neuropathy - arterial disease: A very rare syndrome characterized mainly by artery disease, deafness and peripheral neuropathy.
• Debler anemia: A familial type of anemia that starts during infancy and involves the premature destruction of red blood cells.
• Defect in synthesis of adenosylcobalamin: A rare genetic disorder characterized by the impaired ability to make a chemical called adenosylcobalamin. Adenosylcobalamin is a derivative of vitamin B12. The defect results a biochemical abnormality which affects the body's normal biochemical functioning.
• Deletion 11p: A syndrome that is caused by the deletion of short arm (p) of chromosome 11. The type and severity of symptoms varies depending on the amount and exact location of the genetic material that is deleted.
• Dengue fever: An acute viral disease characterized by fever, rash and myalgia and caused by a flavivirus which is transmitted by mosquitoes.
• Di Guglielmo I - acute: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The acute form has more severe symptoms than the chronic form.
• Di Guglielmo I - chronic: A rare condition characterized by the presence of abnormal red blood cells in the bone marrow and blood. The condition is characterized by anemia and generally leads to the development of acute myelogenous leukemia. The chronic form has more severe symptoms than the acute form.
• Dibasic aminoaciduria 2: A rare condition where protein intolerance occurs as a result of a defect in the transport of dibasic amino acids through the intestines and kidneys. The amino acids (component of protein) can't be broken down properly and used by the body so it builds up and causes damage.
• Diffuse neonatal hemangiomatosis: A very rare syndrome characterized mainly by multiple capillary hemangiomas as well as other abnormalities.
• Disorder of Cornification 12 (Neutral Lipid Storage Type): A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities. The body is unable to metabolize (break down) triglycerides so they build up in various tissues throughout the body. The severity of the symptoms is variable as the body tissues and organs affected varies amongst patients.
• Distomatosis: Infection by parasitic flat worms. Infection can involve liver, lungs or intestines. Symptoms are determined by the location of the infection. Contamination usually occurs through ingesting contaminated food or water.
• Dobriner syndrome: An inherited metabolic disorder involving a deficiency of coproporphyrinogen oxidase. The condition is similar to but milder than intermittent porphyria and sometimes includes photosensitivity.
• Doxepine-induced Immune Hemolytic Anemia: Doxepine -induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Dressler (D.)syndrome: A rare autoimmune blood disorder where erythrocytes are destroyed suddenly after exposure to cold (usually 15ºC or lower).
• Drug-induced liver damage - Clindamycin: Damage or injury to the liver caused by exposure to Clindamycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Quinolone: Damage or injury to the liver caused by exposure to Quinolone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Spectinomycin: Damage or injury to the liver caused by exposure to Spectinomycin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Sulfones: Damage or injury to the liver caused by exposure to sulfone antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced Immune Hemolytic Anemia: Drug-induced immune hemolytic anemia is a condition where a medication triggers the body's immune system to destroy its own red blood cells which results in anemia. Certain drugs are more likely to trigger this abnormal immune response than others e.g. cephalosphorins and Quinidine.
• Drug-induced liver damage - 5-Fluorocytosine: Damage or injury to the liver caused by exposure to an antifungal agent called 5-Fluorocytosine. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Allopurinol: Damage or injury to the liver caused by exposure to allopurinol. Allopurinol is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Amphotericin: Damage or injury to the liver caused by exposure to an antifungal agent called Amphotericin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Anabolic C-17: Damage or injury to the liver caused by exposure to Anabolic C-17 which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Anesthetic agent: Damage or injury to the liver caused by exposure to anesthetic agents. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antianginal agents: Damage or injury to the liver caused by exposure to antianginal agents. Antianginal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antiarrhythmics: Damage or injury to the liver caused by exposure to antiarrhythmics. Antiarrhythmics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antibiotics: Damage or injury to the liver caused by exposure to certain antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Anticoagulants: Damage or injury to the liver caused by exposure to anticoagulants. Anticoagulants are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antifungals: Damage or injury to the liver caused by exposure to antifungal agents. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antihyperlipidemic agents: Damage or injury to the liver caused by exposure to antihyperlipidemic agents. Antihyperlipidemic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antihypertensives: Damage or injury to the liver caused by exposure to antihypertensives. Antihypertensives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antineoplastic agents: Damage or injury to the liver caused by exposure to antineoplastic agents. Antineoplastic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Antithyroid drugs: Damage or injury to the liver caused by exposure to endocrine agents called antithyroid drugs. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Benzodiazepine: Damage or injury to the liver caused by exposure to a psychotropic agent called benzodiazepine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - British anti-Lewisite penicillamine: Damage or injury to the liver caused by exposure to British anti-Lewisite penicillamine. British anti-Lewisite penicillamine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Butyrophenone: Damage or injury to the liver caused by exposure to a psychotropic agent called butyrophenone. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cephalosporin: Damage or injury to the liver caused by exposure to Cephalosporin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Chloramphenicol: Damage or injury to the liver caused by exposure to Chloramphenicol. Chloramphenicol is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Chloroform: Damage or injury to the liver caused by exposure to an anesthetic agent called chloroform. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cimetidine: Damage or injury to the liver caused by exposure to Cimetidine. Cimetidine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Colchicine: Damage or injury to the liver caused by exposure to colchicine. Colchicine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cyclopropane: Damage or injury to the liver caused by exposure to an anesthetic agent called cyclopropane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cycloserine: Damage or injury to the liver caused by exposure to an antituberculous agent called cycloserine. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Cytarabine: Damage or injury to the liver caused by exposure to an antiviral agent called cytarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Dantrolene: Damage or injury to the liver caused by exposure to Dantrolene. Dantrolene is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Diflunisal: Damage or injury to the liver caused by exposure to diflunisal. Diflunisal is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Disulfiram: Damage or injury to the liver caused by exposure to Disulfiram. Disulfiram is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Diuretic Agents: Damage or injury to the liver caused by exposure to diuretic agents. Diuretic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Erythromycin estolate: Damage or injury to the liver caused by exposure to an antibiotic called erythromycin estolate. Erythromycin estolate is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Erythromycin ethyl succinate: Damage or injury to the liver caused by exposure to an antibiotic called Erythromycin ethyl succinate. Erythromycin ethyl succinate is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Ethionamide: Damage or injury to the liver caused by exposure to an antituberculous agent called ethionamide. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Fenoprofen: Damage or injury to the liver caused by exposure to fenoprofen. Fenoprofen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Glucocorticoids: Damage or injury to the liver caused by exposure to endocrine agents called glucocorticoids. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Griseofulvin: Damage or injury to the liver caused by exposure to an antifungal agent called Griseofulvin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Halothane: Damage or injury to the liver caused by exposure to an anesthetic agent called halothane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Ibuprofen: Damage or injury to the liver caused by exposure to ibuprofen. Ibuprofen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Indomethacin: Damage or injury to the liver caused by exposure to indomethacin. Indomethacin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Iodide ion: Damage or injury to the liver caused by exposure to Iodide ion. Iodide ion is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Isoniazid: Damage or injury to the liver caused by exposure to an antituberculous agent called isoniazid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Ketoconazole: Damage or injury to the liver caused by exposure to an antifungal agent called Ketoconazole. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Mephenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called mephenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Methoxyflurane: Damage or injury to the liver caused by exposure to an anesthetic agent called methoxyflurane. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Naproxen: Damage or injury to the liver caused by exposure to naproxen. Naproxen is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Nitrofuran: Damage or injury to the liver caused by exposure to Nitrofuran antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Nitrous Oxide: Damage or injury to the liver caused by exposure to an anesthetic agent called nitrous oxide. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Novobiocin: Damage or injury to the liver caused by exposure to Novobiocin antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Oral hypoglycemics: Damage or injury to the liver caused by exposure to oral hypoglycemics which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Penicillin: Damage or injury to the liver caused by exposure to an antibiotic called penicillin. Penicillin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Phenobarbital: Damage or injury to the liver caused by exposure to an anticonvulsive called phenobarbital. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Phenothiazines: Damage or injury to the liver caused by exposure to a psychotropic agent called phenothiazine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Phenylbutazone: Damage or injury to the liver caused by exposure to phenylbutazone. Phenylbutazone is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Phenytoin: Damage or injury to the liver caused by exposure to an anticonvulsive called Phenytoin. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Ranitidine: Damage or injury to the liver caused by exposure to Ranitidine. Ranitidine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Rifampicin: Damage or injury to the liver caused by exposure to an antituberculous agent called rifampicin. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Salicylate: Damage or injury to the liver caused by exposure to salicylates. Salicylates are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Saramycetin: Damage or injury to the liver caused by exposure to an antifungal agent called Saramycetin. Antifungal agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Steroids: Damage or injury to the liver caused by exposure to steroids which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Sulfonamide: Damage or injury to the liver caused by exposure to Sulfonamide antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Sulindac: Damage or injury to the liver caused by exposure to sulindac. Sulindac is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Tamoxifen: Damage or injury to the liver caused by exposure to Tamoxifen which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Telithromycin: Damage or injury to the liver caused by exposure to an antibiotic called Telithromycin. Telithromycin is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Tetracycline: Damage or injury to the liver caused by exposure to Tetracycline antibiotics. Antibiotics are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Thioxanthene: Damage or injury to the liver caused by exposure to a psychotropic agent called Thioxanthene. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Thorotrast: Damage or injury to the liver caused by exposure to Thorotrast. Thorotrast is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Valproic Acid: Damage or injury to the liver caused by exposure to an anticonvulsive called valproic acid. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Vidarabine: Damage or injury to the liver caused by exposure to an antiviral agent called vidarabine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Vitamin A: Damage or injury to the liver caused by exposure to Vitamin A. Vitamin A is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - Zoxazolamine: Damage or injury to the liver caused by exposure to Zoxazolamine. Zoxazolamine is a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - anticonvulsives: Damage or injury to the liver caused by exposure to certain anticonvulsives. Anticonvulsives are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - antituberculous agents: Damage or injury to the liver caused by exposure to antituberculous agents. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - antiviral medication: Damage or injury to the liver caused by exposure to antiviral agents. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - endocrine agent: Damage or injury to the liver caused by exposure to endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - idoxuridine: Damage or injury to the liver caused by exposure to an antiviral agent called idoxuridine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - monoamine oxidase inhibitors: Damage or injury to the liver caused by exposure to psychotropic agents called monoamine oxidase inhibitors. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - p-aminosalicylic acid: Damage or injury to the liver caused by exposure to an antituberculous agent called p-aminosalicylic acid. Antituberculous agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - psychotropic agents: Damage or injury to the liver caused by exposure to certain psychotropic agents. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - tricyclic antidepressant: Damage or injury to the liver caused by exposure to a psychotropic agent called tricyclic antidepressant. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drug-induced liver damage - xenylamine: Damage or injury to the liver caused by exposure to an antiviral agent called xenylamine. Antiviral agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Drugs-induced liver damage - Ether: Damage or injury to the liver caused by exposure to an anesthetic agent called ether. Anesthetic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Dyck Syndrome: A very rare condition observe in two brothers. It involves vision, hearing, muscle, sensory and adrenal system problems and an enlarged liver and spleen.
• Dykes-Markes-Harper syndrome: A very rare syndrome characterized mainly by dry, scaly skin, enlarged liver and spleen and a incoordination.
• Dyserythropoietic anemia, congenital: A congenital blood condition where the production of red blood cells is defective. The abnormal red blood cells are often destroyed prematurely within the body which can result in anemia. There are three subtypes of the disorder, each with varying severity of symptoms.
• Dyserythropoietic anemia, congenital type 1: A very rare genetic blood disorder where abnormal red blood cells are made resulting in anemia. Finger and toe abnormalities are also usually present.
• Dyserythropoietic anemia, congenital type 2: A very rare genetic blood disorder where abnormal red blood cells are made resulting in anemia.
• Dyserythropoietic anemia, congenital type 3: An extremely rare blood disorder where abnormal red blood cells are made resulting in anemia.
• Dysplastic cortical hyperostosis: A very rare syndrome characterized mainly by abnormal bone and brain development.
• Eccentrochondrodysplasia: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of certain enzymes needed to break it down.
• Ectodermal dysplasia, anhidrotic with T-cell immunodeficiency, autosomal dominant: An inherited disorder characterized by dry, rough skin, sparse scalp hair, cone-shaped teeth and an immune system disorder.
• Elejalde syndrome: A rare congenital syndrome characterized excessive fetal growth, skull abnormalities, extra fingers or toes and other abnormalities.
• Emanuel syndrome: A rare chromosomal disorder where a portion of chromosome 11 is translocated with a portion of chromosome 22. The disorder involves a wide range of abnormalities such as kidney, genital and heart abnormalities, mental retardation, small head and failure to thrive.
• Encephalopathy due to GLUT1 deficiency: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
• End Stage Liver Failure: Late stage of liver failure characterised by the onset of mental and neurological symptoms, due to build up of toxic metabolites.
• Endocardial fibroelastosis: A rare heart disorder characterized by a thickening of the heart muscle lining causing heart enlargement and heart failure.
• Endocarditis: Inflammatory alterations of the endocardium of ones heart
• Endocrine agent-induced liver damage: Damage or injury to the liver caused by exposure to endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Anabolic C-17: Damage or injury to the liver caused by exposure to Anabolic C-17 which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Antithyroid drugs: Damage or injury to the liver caused by exposure to endocrine agents called antithyroid drugs. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Glucocorticoids: Damage or injury to the liver caused by exposure to endocrine agents called glucocorticoids. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Oral contraceptives: Damage or injury to the liver caused by exposure to oral contraceptives which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Oral hypoglycemics: Damage or injury to the liver caused by exposure to oral hypoglycemics which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Steroids: Damage or injury to the liver caused by exposure to steroids which are endocrine agents. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endocrine agent-induced liver damage - Tamoxifen: Damage or injury to the liver caused by exposure to Tamoxifen which is an endocrine agent. Endocrine agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Endomyocardial fibrosis: Heart disease characterized by progressive fibrosis of the lining of one or both lower heart cavities. This results in constriction of the heart cavity and may involve the heart valves and other structures.
• Enlarged liver: Swelling of the liver.
• Enlarged liver and spleen: Enlargement of both liver and spleen (hepatosplenomegaly)
• Enterovirus antenatal infection: Fetal infection with enterovirus. The condition is extremely rare but infection around the time of birth often results in death or paralysis in survivors. The type and severity of symptoms is determined by the exact type of virus involved and at what stage of development the infection occurs.
• Epstein-Barr virus: Common virus causing mononucleosis
• Epstein-Barr virus, chronic: A form of human herpes virus that produces persistent symptoms. Most people become infected with the virus at some stage in their life though they usually have few if any symptoms. However, some people develop severe symptoms as a result of an EBV infection.
• Erdheim-Chester syndrome: A very rare lipid storage disorder involving lipid deposits in various organs and hardening of the ends of long bones which affects the growth of the bone. The severity of the condition is variable.
• Escharonodulaire: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Essential mixed cryoglobulinemia: A rare autoimmune disorder characterized by the presence of a mixture of various types of antibodies (crylglobulins) in the blood.
• Essential thrombocytosis - same as essential thrombocythemia: A rare blood disorder where the blood contains too many platelets due to excessive megakaryocytes (platelet-producing cells). Platelets are essential for blood clotting but in essential thrombocythemia excessive platelets can cause the blood to form abnormal clots. If the platelets are defective as well then bleeding problems can occur. The severity of the condition is variable.
• Extramedullary Myeloid Tumor: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Familial Mediterranean fever: A rare inherited condition characterized by recurrent fever and inflammation. The inflammation usually involves the stomach, lungs or joints.
• Familial hyperchylomicronemia: A rare inherited inborn error of metabolism involving the absence of the enzyme called lipoprotein lipase which results in increased blood triglyeride and chylomicron levels.
• Familial hyperlipoproteinemia: A group of genetic disorder characterized by abnormal breakdown of lipoproteins which causes abnormal lipoprotein and lipid levels in the blood. There are various types of this condition: hyperlipoproteinemia type I, II, III, IV and V. The type and severity of symptoms vary between types. The disorder tends to run in families (familial).
• Familial hyperlipoproteinemia type 1: A genetic disorder characterized by abnormal lipid (chylomicrons and high triglyceride lipids) breakdown which results in its accumulation in the blood. The disorder is caused by the reduced or absent activity of the enzyme lipoprotein lipase. The severity of the condition is determined by the degree of the deficiency and treatment. The disorder tends to run in families (familial).
• Familial myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
• Fanconi-Bickel syndrome: A rare inherited disorder where the impaired metabolism of carbohydrates results in a build-up of glycogen in the liver.
• Farber's disease: A rare inherited biochemical disorder involving the deficiency of an enzyme called ceramidase resulting in the harmful accumulation of certain chemicals in the body which causes damage and inflammation.
• Fascioliasis: A rare parastitic infectious disease caused by liver fluke Fasciola hepatica which can cause blockage of the bile ducts in the liver.
• Felty Syndrome: A rare complication of long-term rheumatoid arthritis which involves an enlarged spleen and blood abnormalities.
• Felty syndrome: A rare complication of long-term rheumatoid arthritis which involves an enlarged spleen and blood abnormalities.
• Fluke infections: An infection caused by flukes
• Forbes disease: A rare inherited glycogen storage disease caused by a deficiency of the enzyme amylo-1,6-glucosidase resulting in a build up of glycogen in the liver and muscles.
• Fowl Paralysis: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Fowl paralyses: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Free sialic Acid storage disease: A rare inherited biochemical disorder characterized by the accumulation of sialic acid in the tissues and excretion of sialic acid in the urine. There are mild and severe forms of the condition - the severe form result in death before birth or within a few years of birth.
• Fructose intolerance: An enzyme deficiency that causes toxic symptoms when fructose containing food is ingested.
• Fructose-1,6-bisphosphatase deficiency, hereditary: A rare inherited condition where an enzyme deficiency (fructose-1,6-bisphosphatase deficiency - FDPase) impairs the body's ability to metabolize fructose from the diet.
• Fructose-1-phosphate aldolase deficiency, hereditary: An inherited metabolic disorder where deficiency of the enzyme fructose-1-phsophate aldolase prevents fructose being metabolized resulting in fructose intolerance.
• Fucosidosis: A rare progressive biochemical disorder involving deficiency of an enzyme (alpha-fucosidase) which results in accumulation of certain chemicals (glycosphingolipids) in the central nervous system and other body tissues.
• Fucosidosis type 1: A rare biochemical disorder involving deficiency of an enzyme (alpha-fucosidase) which results in accumulation of certain chemicals (glycosphingolipids) in the central nervous system and other body tissues. It is an infantile form of fucosidosis which starts early and rapidly progresses to early death.
• Fucosidosis type II: A form of the biochemical disorder called fucosidosis where an enzyme deficiency (alpha-fucosidase) results in the accumulation of certain chemicals (glycosphingolipids) in the central nervous system and other body tissues. Symptoms start later and progress slower than in type I and is distinguished by warty skin growths.
• GM1 gangliosidosis: A rare biochemical disorder involving a deficiency of an enzyme (beta-galactosidase A) which results in the accumulation of harmful chemicals (GM1 gangliosides) in the central nervous system and other body tissues. Type 1 is a severe infantile form of the disorder and involves a greater degree of accumulation than type II or III.
• Galactokinase deficiency: A rare condition where an enzyme deficiency (galactokinase) impaires the body's ability to break down galactose consumed in the diet.
• Galactosemia I: A rare inherited disorder where deficiency of a particular enzyme (galactose-1-phosphate uridyl transferase) prevents the metabolism of galactose which is a sugar component of milk. Ranges from milk intolerance in mild cases to death in severe untreated cases.
• Game-Friedman-Paradice syndrome: A rare condition characterized by retarded growth, hydrocephaly, underdeveloped lungs and various other anomalies. The condition was observed in four offspring from one family.
• Gangliosidosis GM1 type 3: A rare biochemical disorder involving a deficiency of an enzyme (beta-galactosidase A) which results in the accumulation of harmful chemicals (GM1 gangliosides) in the central nervous system and other body tissues. Type III involves a lesser degree of accumulation than type II or I.
• Gangliosidosis generalized GM1, type 1: A rare biochemical disorder involving a deficiency of an enzyme (beta-galactosidase A) which results in the accumulation of harmful chemicals (GM1 gangliosides) in the central nervous system and other body tissues. Type 1 is a severe infantile form of the disorder and involves a greater degree of accumulation than type II or III.
• Garret-Tripp syndrome: A rare syndrome characterized mainly by extra digits, a lack of hair and scaly skin on the scalp, face and neck.
• Gaucher Disease: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3.
• Gaucher disease - perinatal lethal form: A rare syndrome characterized by the association of abnormally tight skin and Gaucher disease which is a lipid storage disease. This is the most severe form of Gaucher disease.
• Gaucher disease type 1: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 1 is the visceral, chronic form which usually starts during adulthood.
• Gaucher disease type 2: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 2 is acute neurological form apparent in infancy.
• Gaucher disease type 3: A rare inherited biochemical disorder characterized by the deficiency of the enzyme called glucocerebrosidase and accumulation of glycosylceramide (glucocerebroside). There are three forms of this disease: type 1, 2 and 3. Type 3 is a subacute neurological form which often first appears in childhood.
• Gaucher-like disease: A very rare inherited condition where the body's inability to bread down a certain fat (glucocerebroside) causes it to accumulate in body tissues and organs where it can cause damage. Gaucher-like disease is a sub-type of Gaucher disease which primarily affects the heart.
• Geleophysic dwarfism: A rare genetic disorder characterized by a happy facial expression, short stature and limb abnormalities.
• Ghosal syndrome: A very rare syndrome characterized mainly by difficult to treat anemia and skeletal abnormalities.
• Glucosephosphate isomerase deficiency: A rare inherited condition where a lack of the enzyme glucosephosphate isomerase causes red blood cells to be destroyed prematurely (hemolytic anemia).
• Glutaric Aciduria, neonatal form of type II A: A more serious neonatal form of glutaricaciduria where there is excessive blood and urine levels of glutaric acid and congenital anomalies may be present.
• Glutaric aciduria 1: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
• Glutaric aciduria 2: A metabolic disorder involving an enzyme deficiency - electron transfer flavoprotein ubiquinone oxydoreductase. The severity of symptoms depends on the level of deficiency. The infant onset form is the most severe and often results in death. Severe cases usually develop during childhood or infancy and usually involve metabolic acidosis and its associated symptoms. Milder cases may simply present with muscle weakness initially that develops in adulthood. Some cases may involve additional symptoms such as heart, liver and kidney problems, facial anomalies and genital abnormalities.
• Glutaricaciduria 2B: A milder, later-onset form of glutaricaciduria where there is excessive blood and urine levels of glutaric acid due to the body's impaired ability to metabolize protein and fat into energy.
• Glutaricaciduria I: A rare inherited enzyme deficiency disorder where deficiency of the glutaryl-CoA dehydrogenase enzyme results in dystonia, dyskinesia and sometimes mental retardation.
• Glutaricaciduria type 1: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
• Glutaryl-CoA dehydrogenase deficiency: A rare inherited metabolic disorder where a genetic mutation results in the deficiency of an enzyme called glutaryl-CoA dehydrogenase which is required to metabolise certain amino acids (lysine, hydroxylysine and tryptophan). Problems occur when these metabolites build up in the body and cause neurological problems. Symptoms often develop following an acute infection or fasting. The severity of the condition is highly variable from development of neurological symptoms during infancy to asymptomatic adults. The degree of enzyme deficiency will usually determine the severity.
• Glycogen Storage Disease Type I: An inherited metabolic disorder where a deficiency of the enzyme glucose-6-phosphatase prevents glycogen being turned into glucose leading to a buildup of glycogen in the liver and kidneys. Most problems tend to develop during adulthood.
• Glycogen branching deficiency: A rare metabolic disorder where an enzyme deficiency (glycogen branching enzyme) results in a harmful buildup of glycogen byproducts in the liver, muscle and even the heart in some cases. The severity of symptoms is variable depending on the degree of enzyme deficiency and how strictly treatment measures are adhered to.
• Glycogen debranching deficiency: A rare metabolic disorder where an enzyme deficiency (amylo-1,6-glucosidase) results in a harmful buildup of glycogen byproducts in the liver, muscle and even the heart in some cases. The severity of symptoms is variable depending on the degree of enzyme deficiency and how strictly treatment measures are adhered to.
• Glycogen storage disease type 1C: A genetic metabolic disorder involving a deficiency of the enzyme glucose-6-phosphatase (due to a defect in the microsomal phosphate) which results in the accumulation of glycogen in various tissues. G6P is stored as glycogen until the body needs to convert it to a sugar for energy. The enzyme deficiency prevents the conversion and hence low blood sugar levels result.
• Glycogen storage disease type 1D: A genetic metabolic disorder involving a deficiency of the enzyme glucose-6-phosphatase (due to a defect in the microsomal glucose transporter) which results in the accumulation of glycogen in various tissues. G6P is stored as glycogen until the body needs to convert it to a sugar for energy. The enzyme deficiency prevents the conversion and hence low blood sugar levels result.
• Glycogen storage disease type 2: A rare inherited biochemical disorder involving the harmful accumulation of certain chemicals (glycogen) in body tissues due to the deficiency of an enzyme (?-glucosidase or acid maltase) needed to break it down.
• Glycogen storage disease type 6: A rare, generally mild form of inherited glycogen storage disease where a deficiency of phosphorylase b kinase leads to hypoglycemia and accumulation of glycogen in the liver.
• Glycogen storage disease type 6A, due to phosphorylase kinase deficiency: A rare, generally mild form of inherited glycogen storage disease where a deficiency of phosphorylase kinase leads to hypoglycemia and accumulation of glycogen in the liver. Phosphorylase kinase deficiency can cause glycogen storage disease type VIa and/or IX.
• Glycogenosis type 2: A rare inherited biochemical disorder involving the harmful accumulation of certain chemicals (glycogen) in body tissues due to the deficiency of an enzyme (?-glucosidase or acid maltase) needed to break it down. The severity of the condition is variable and onset may occur during infancy, childhood or adulthood.
• Glycogenosis type 8: A mild glycogen storage disease which affects males only and involves a deficiency of the enzyme phosphorylase b-kinase.
• Glycoprotein storage disease: A rare inherited metabolic disorder involving an enlarged spleen and abnormal glycoprotein storage.
• Goldberg syndrome: A rare lysosomal storage disorder characterized by an enzyme deficiency (neuraminidase and beta-galactosidase) which results in a build-up of glycoproteins in the urine. There are three main subtypes: infantile, juvenile and adult forms. The early infantile form is the most severe and often results in death during infancy.
• Goldstein-Hutt syndrome: A rare syndrome characterized mainly by cataracts, excessive eyelash growth and and a blood abnormality (the red blood cells are spherical instead of doughnut shaped which makes them fragile).
• Granuloma inguinale: Granulomous disease spread sexually.
• Griscelli disease: A rare genetic disorder characterized mainly by albinism (lack of pigmentation). There are three different subtypes of the disorder (I, II and III) each with varying additional features such as immunodeficiency and neurological symptoms. Type 1 involves partial albinism and neurological symptoms, type II involves partial albinism, immunodeficiency and sometimes neurological symptoms and type III involves albinism, only.
• Griscelli syndrome type II: A rare genetic condition characterized by a partial lack of pigmentation in the eye, skin and hair, clumps of pigmentation in hair shafts, immunodeficiency and neurological symptoms.
• Gräsbeck-Imerslund disease: A rare genetic blood disorder where a defect in the vitamin B12 receptor means that it can't be absorbed from food during digestion. As a result of the low vitamin B12 levels, the body produces increased numbers of abnormal enlarged red blood cells (megaloblasts).
• HIV/AIDS: HIV is a sexually transmitted virus and AIDS is the progressive immune failure that HIV causes.
• HMG CoA synthetase deficiency: An inherited metabolic defect due to the deficiency of an enzyme (HMG CoA synthetase) needed to form ketone bodies. Symptoms are often preceded by periods of fasting or illness.
• Hairy cell leukemia: A chronic leukemia which causes an excess of abnormal mononuclear cells which appear hair like under microscopy
• Hanot-MacMahon-Thannhauser syndrome: A liver disorder where chronic inflammation of the small portal bile ducts leads to blockage of the smallest ducts. As a result, fatty deposits develop in the skin, the liver and kidneys become enlarged and jaundice occurs.
• Hashimoto-Pritzker syndrome: A rare harmless skin disorder characterized by the accumulation of histiocytes (infection-fighting cells) in the skin which forms nodules. The condition occurs at birth or soon after and resolves spontaneously within a month.
• Heart symptoms: Symptoms affecting the heart
• Heavy-chain diseases: A group of rare condition characterized by the production of the heavy chain portions of immunoglobulin molecules. Subtypes include y-chain disease, µ-chain disease and α-chain disease.
• Helminth infections: The infection by a parasitic worm
• Helminthiasis: Infection of the human body with a parasitic worm such as roundworms and pinworms. The worms usually only involve the intestinal tract but sometimes they may invade other organs. The type and severity of symptoms is determined by the type of worm and the part of the body infected.
• Hemangioendothelioma: A rare type of blood vessel tumor that can occur anywhere in the body but is most often found in the skin, liver and spleen. Symptoms vary according to the exact location.
• Hemochromatosis: Excess of iron leading to problems with joints, liver, heart and pancreas.
• Hemochromatosis type 1: A genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. Type 1 is the most common form of the disorder, has an adult onset and is inherited recessively.
• Hemochromatosis type 2: A rare genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. Type 2 is a more severe type, has a juvenile onset and is inherited recessively.
• Hemochromatosis type 3: A rare genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. Type 3 has an onset that is between type 1 and type 2 and is inherited recessively.
• Hemochromatosis type 4: A rare genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. Type 4 has an adult onset but is inherited dominantly.
• Hemoglobin C disease: An inherited blood condition characterized by abnormal hemoglobin (hemoglobin C) which leads to premature break down of red blood cells and hence anemia. In homozygous patients (a hemoglobin C gene is inherited from both parents) mild hemolytic anemia may develop as nearly all of the hemoglobin is of the abnormal hemoglobin C form. In heterozygous patients (a hemoglobin C gene is inherited from one parent) more than half of the hemoglobin is normal and the patient is asymptomatic. Hemoglobin C occurs commonly in West African populations and is believed to provide some protection against severe malaria.
• Hemoglobin E disease: A recessively inherited genetic blood disorder characterized by abnormally small red blood cells which generally causes no health problems. Some people may suffer mild hemolytic anemia or a slightly enlarged spleen.
• Hemoglobin E homozygous: A rare genetic blood anomaly which may cause anemia and occasionally an enlarged spleen.
• Hemoglobin H: A genetic blood anomaly that causes enlarged liver and spleen and hemolytic anemia.
• Hemoglobin, constant spring: A generally mild form of anemia caused by a defect in the hemoglobin which is the part of the blood that carries oxygen to the body tissues. The biggest risk of this condition is the fact that children born to carriers of this and other blood disorders may inherit more severe forms of blood disorders.
• Hemolytic anemia: A condition which is characterized by anaemia due to the destruction of red blood cells
• Hemolytic anemia, lethal - genital anomalies: A very rare syndrome characterized mainly by genital abnormalities and hemolytic anemia which often causes death.
• Hemolytic uremic syndrome: A rare condition characterized by acute kidney failure, hemolytic anemia and thrombocytopenia (reduced blood platelet count). The condition is often caused by upper respiratory infections or infectious diarrhea.
• Hemophagocytic Lymphohistiocytosis: A condition which is characterized by an abnormal appearance of histiocytes in the blood
• Hemophagocytic lymphohistiocytosis, familial, 1: A rare recessively inherited disorder involving an overactive immune system. More specifically, the body becomes infiltrated by large numbers of histiocytes (macrophages) that accumulate in various organs such as the liver, spleen, bone marrow, skin and central nervous system. It usually only occurs in infants and young children and can be life-threatening.
• Hemophagocytic lymphohistiocytosis, familial, 2: A rare recessively inherited disorder involving an overactive immune system. More specifically, the body becomes infiltrated by large numbers of histiocytes (macrophages) that accumulate in various organs such as the liver, spleen, bone marrow, skin and central nervous system. It usually only occurs in infants and young children. Type 2 is caused by a defect on chromosome 10q22.
• Hemophagocytic lymphohistiocytosis, familial, 3: A rare recessively inherited disorder involving an overactive immune system. More specifically, the body becomes infiltrated by large numbers of histiocytes (macrophages) that accumulate in various organs such as the liver, spleen, bone marrow, skin and central nervous system. It usually only occurs in infants and young children. Type 3 is caused by a defect on chromosome 17q25.1.
• Hemophagocytic lymphohistiocytosis, familial, 4: A rare recessively inherited disorder involving an overactive immune system. More specifically, the body becomes infiltrated by large numbers of histiocytes (macrophages) that accumulate in various organs such as the liver, spleen, bone marrow, skin and central nervous system. It usually only occurs in infants and young children. Type 4 is caused by a defect on chromosome 6q24.
• Hemophagocytic reticulosis: The abnormal proliferation of reticulum cell (histiocytes) which infiltrate various organs and. Macrophages destroy blood cells causing blood abnormalities. Meningoencephalitis frequently occurs when the histiocytes infiltrate the mininges and cerebral tissue. Symptoms start at birth or soon after and become progressively worse without treatment. Medication can control the condition but a hematopoietic stem cell transplant is needed to achieve remission.
• Hemorrhagic thrombocythemia: A rare blood disorder characterized by increased number of platelets in the blood which often results in an enlarged spleen, bleeding and blood vessel blockages.
• Hepatic Venoocclusive Disease with immunodeficiency: An inherited disorder characterized by the association of immunodeficiency and liver disease involving the blockage of small veins in the liver due to swelling and fibrosis.
• Hepatic amyloidosis with intrahepatic cholestasis: Amyloidosis is a rare group of metabolic disorders where a protein called amyloid accumulates in body organs and tissues where it can cause damage. In hepatic amyloidosis, the amyloid deposits occur primarily in the liver which affects the flow of bile through the organ and results in intrahepatic cholestasis.
• Hepatic cystic hamartoma: A cystic liver tumor.
• Hepatic encephalopathy syndrome: A rare syndrome involving the association of advanced liver disease and neurological problems.
• Hepatic fibrosis, congenital: A rare condition characterized by scarring or fibrosis of the liver that impairs its function.
• Hepatic veno-occlusive disease - immunodeficiency: A rare form of liver disease involving the blockage of veins in the liver due to thickening of the blood vessel walls as well as immunodeficiency. The disease may be caused by a variety of factors including bone marrow transplant and genetic defects.
• Hepatitis B: Viral liver infection spread by sex or body fluids.
• Hepatitis X (non-A,-B,-C,-D,-E): Viral liver inflammation that cannot be determined to be one of the existing types of viral hepatitis - A,B,C,D and E.
• Hepatoma: Hepatocellular carcinoma (HCC) is a primary malignancy of the liver.
• Hepatorenal tyrosinemia: A rare genetic metabolic disorder characterized by a deficiency of particular enzymes which prevents the breakdown of tyrosine which then builds up in the liver. Type 1 involves a deficiency of the enzyme fumaril acetoacetate hydrolase.
• Hepatotoxicity: Damage or injury to the liver caused by a drug, chemical or other agent. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure.
• Heptaosplenic T-cell Lymphoma: A rare subtype of lymphoma which originates from T-cell lymphocytes in the liver and the spleen. This form of lymphoma is considered quite aggressive. People with solid organ transplants tend to be more susceptible to this form of cancer, possibility because of the need to take immune suppressing drugs to avoid rejection of the transplanted organ.
• Herbal Agent adverse reaction - Kombucha: Kombucha can be used as a herbal agent to treat insomnia, arthritis, aches, high blood pressure and to improve the immune system. Kombucha can cause an adverse reaction in some people.
• Herbal Agent overdose - Kombucha: Kombucha can be used as a herbal agent to treat insomnia, arthritis, aches, high blood pressure and to improve the immune system. The ingestion of excessive amounts of Kombucha can result in overdose symptoms.
• Hereditary Hemochromatosis: A genetic disorder where too much iron is absorbed from food and it is stored in various parts of the body which can cause damage. There are 4 types of hemochromatosis and they are distinguished by age of onset, genetic cause and type of inheritance. Some sufferers may be asymptomatic.
• Hereditary hemorrhagic telangiectasia: A rare genetic disorder characterized by epistaxes and multiple telangiectases.
• Hereditary non-spherocytic hemolytic anemia: A group of genetic blood disorders where red blood cells are prematurely destroyed resulting in anemia if they are not replaced fast enough. The blood cells are destroyed because they have abnormally weak membranes which gives them an irregular shape rather than normal doughnut shape.
• Hereditary spherocytic hemolytic anemia: An inherited blood disorder where a metabolic defect causes defects in the red blood cells membranes which leads to their characteristic spherical shape (normal cells are doughnut shaped) and premature destruction.
• Hereditary spherocytosis: A rare genetic condition characterized by a defect of the red blood cell membrane.
• Hereditary thrombocythemia: An inherited disorder characterized by thrombocytosis which is a high blood platelet count that occurs for no apparent reason. It is usually only discovered incidentally during routine blood tests.
• High Blood Iron: Where a patient has an elevated iron content of their blood
• Histiocytosis, Non-Langerhans-Cell: A group of disorders involving the excessive proliferation of histiocytes (macrophages) which are not Langerhans cells. Subgroups of this disorder includes hemophagocytic lymphohistiocytosis, sinus histiocytosis, xanthogranuloma and sea-blue histiocyte syndrome. Sometimes only the skin is involved and in other cases there may be systemic involvement which can cause serious problems.
• Histoplasmosis: Lung infection from fungus Histoplasma capsulatum
• Hodgkin's Disease: A form of cancer that affects the lymphatic system.
• Hodgkin's disease, adult: A type of cancer that affects the lymphatic system in adults. The lymphatic system forms part of the body's immune system. This type of cancer can also occur in children.
• Hodgkin's disease, childhood: A type of cancer that affects the lymphatic system in children. The lymphatic system forms part of the body's immune system. This type of cancer can also occur in children.
• Hunter-Carpenter-Macdonald syndrome: A very rare syndrome characterized by various abnormalities including small head, facial anomalies, mental retardation and hair, skin and nail anomalies. Fetal exposure to alcohol is believed to be a factor in the syndrome.
• Hydatidosis: A parasitic infection caused by a tapeworm larva called echinococci. The type and severity of symptoms is determined by the location of the infestation - the most common site for the larval cysts is the liver. The most common symptoms are due to compression of nearby organs or blood vessels due to increasing size of the cyst. The larva may incubate for months or even years.
• Hydrops ectrodactyly syndactyly: A very rare disorder characterized mainly be webbed fingers and toes, hand defect and hydrops (abnormal accumulation of fluid in the fetus).
• Hydrops fetalis: Abnormal accumulation of fluid in the fetus which can be fatal. Excessive fluid leaves the blood and enters the tissues. It can have non-immune and immune (mother's immune system destroys fetal red blood cells).
• Hyper IgM syndrome 1: Hyper IgM syndrome is a rare inherited immunodeficiency disorder which causes frequent infections involving the ears, eyes, sinuses, lungs, skin, respiratory tract and other areas of the body. Type 1 is caused by a defect on chromosome Xq26 and tends to affects males mostly.
• Hyper IgM syndrome 3: Hyper IgM syndrome is a rare inherited immunodeficiency disorder which causes frequent infections involving the ears, eyes, sinuses, lungs, skin, respiratory tract and other areas of the body. Type 3 is caused by a defect on chromosome 20q12-q13.2.
• Hyper-IgM Syndrome: A rare inherited immunodeficiency disorder which causes frequent infections involving the ears, eyes, sinuses, lungs, skin, respiratory tract and other areas of the body.
• Hyperbilirubinemia: Excessive bilirubin (a bile pigment) in the blood.
• Hyperchylomicronemia: A term used to describe the presence of too many chylomicrons in the blood.
• Hyperdibasic aminoaciduria type 2: A rare inborn urea cycle disorder characterized by an enzyme defect in the amino acid transporter gene SLC7A7 (positive amino acid transporter).
• Hypereosinophilic syndrome: A rare condition where too many eosinophils are produced over an extended period of time for no apparent reason. The eosinophils can infiltrate various organs and tissues and cause dysfunction or damage
• Hypergammaglobulinemia: An increase in the level of any of a number of gamma globulins. This can result from conditions such as chronic bacterial infections, multiple myeloma, lymphomas, dysproteinemias, liver disease and chronic granulomatous inflammations. The exact symptoms depend on the type of gamma globulins involved and the severity of the condition.
• Hyperimidodipeptiduria: A very rare genetic disorder characterized by an excessive level of imidodipeptides in the urine due to a deficiency of the enzyme prolidase.
• Hyperimmunoglobinemia D with recurrent fever: A very rare disorder involving a high immunoglobulin level associated with periods of fever which generally reoccur every month or two.
• Hyperlipoproteinemia type 3: A rare genetic disorder characterized by the body's impaired ability to break down certain lipids (triglycerides) which results in their buildup in the blood.
• Hyperlipoproteinemia type 5: An inherited disorder where the body's ability to break down lipids (triglycerides and cholesterol) is impaired resulting in increased blood lipid levels of chylomicrons and VLDL's.
• Hyperlysinemia, persistent: A rare genetic disorder where the body lacks enzymes (lysine ketoglutarate reductase and saccharopine dehydrogenase) to metabolize lysine which then causes a harmful builds up of lysine in the blood and urine and saccharopins in the urine. Some patients are asymptomatic while others have severe symptoms.
• Hyperparathyroidism, neonatal severe primary: A very rare disorder where high levels of parathyroid levels affects the body's use of calcium. The bones lack sufficiency calcification and become weak.
• Hyperpipecolatemia: A rare metabolic disorder characterized by high blood levels of pipecolic acid.
• Hypopigmented lesions in children:
• Hypoplasia hepatic ductular: A rare liver disorder where there are fewer than normal bile ducts in the liver - sometimes all the bile ducts are absent. Alagille syndrome is characterized by this liver abnormalitiy as well as a distinctive face, abnormal vertebrae and retarded physical, mental and sexual development.
• I cell disease: A rare inherited biochemical disorder characterized by the harmful accumulation of chemicals (glycoproteins and glycoplipids) due to the deficiency of an enzyme (UDP-N-acetylglucosamine-I-phosphotransferase).
• Ichthyohepatotoxication: Ichthyohepatotoxication is a condition caused by eating the liver of certain fish. It is believed that the high vitamin A content of the liver leads to vitamin A overdose and the resulting symptoms. Tropical shark livers are associated with this condition.
• Ichythosiform Erythroderma with Leukocyte Vacuolation: A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities. The body is unable to metabolize (break down) triglycerides so they build up in various tissues throughout the body. The severity of the symptoms is variable as the body tissues and organs affected varies amongst patients.
• Idiopathic hypereosinophilic syndrome: A rare blood disorder where the bone marrow produces too many eosinophils over a long period of time which can cause organ or tissue damage. The disorder can affect and part of the body but most often affects the skin, heart and nervous system. The increased eosinophil production continues for a long period of time (at least 6 months) and there is no apparent cause.
• Imerslünd-Grasbeck Syndrome: A rare genetic blood disorder where a defect in the vitamin B12 receptor means that it can't be absorbed from food during digestion. As a result of the low vitamin B12 levels, the body produces increased numbers of abnormal enlarged red blood cells (megaloblasts).
• Immune defect due to absence of thymus: A congenital disorder where the absence of the thymus results in immune system problems (deficiency of T-Lymphocytes). The thymus is involved in the production of mature functioning T-cells which fight infection.
• Immunoglobulinic amyloidosis: A disease characterized by the abnormal deposit of amyloid in various parts of the body, especially organs such as the kidneys, heart, liver, gastrointestinal tract and peripheral nerves. It occurs when plasma cells in the bone marrow produce too much of a protein portion of an antibody called the light chain. The exact symptoms are determined by the extent of the organ involvement.
• Immunoproliferative diseases: Diseases characterised by proliferation of the lymphoid cells
• Inborn amino acid metabolism disorder: A group of inherited disorders where the body is not able to metabolize amino acids consumed in the diet. Amino acids are a part of carbohydrates, fats and proteins and are metabolized in order to provide energy or to make other needed compounds. There are many steps involved in metabolism and the severity can be greatly variable depending on the exact nature of the disorder.
• India tick typhus: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Indian tick fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Infantile Refsum Disease: Genetic disease affecting nerve and muscle control.
• Infantile multisystem inflammatory disease: A rare autoinflammatory disease characterized by fever, rash, arthritic changes, eye problems and chronic meningitis.
• Infantile sialic acid storage disorder: A rare inherited biochemical disorder characterized by the accumulation of sialic acid in the tissues and excretion of sialic acid in the urine. The disorder results in death within the first few years of life - usually in infancy.
• Infectious myocarditis: Inflammation of the muscle of the heart (myocardium) due to an infection. It often occurs as a complication of various bacterial, viral or parasitic infections such as rubella, polio and rheumatic fever.
• Inherited spherocytic anemia: Inherited Spherocytic anemia is an inherited blood disorder characterized by red blood cells that are shaped like a sphere (spherocytes) rather than the normal doughnut shape. These abnormal cells are broken down prematurely resulting in a shortage of red blood cells (anemia).
• Interferon Beta - Teratogenic Agent: There is evidence to indicate that exposure to Interferon Beta during pregnancy may have a teratogenic effect on the fetus. A teratogen is a substance that can cause birth defects. The likelihood and severity of defects may be affected by the level of exposure and the stage of pregnancy that the exposure occurred at.
• Intrahepatic cholangiocarcinoma: A slow growing malignant cancer that occurs in the bile ducts in the liver.
• Israeli spotted fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Jequier-Kozlowski-skeletal dysplasia: A very rare syndrome characterized mainly by various skeletal abnormalities and facial anomalies.
• Juvenile idiopathic arthritis: A group of chronic inflammatory joint disorders that affects children. The condition generally involves periods of time where the condition is active followed by periods of abatement of symptoms. In some cases, the condition can be systemic and can cause symptoms such as fever and rash with organ involvement. There are three main types of juvenile idiopathic arthritis - oligoarticular, polyarticular and systemic (Still's disease).
• Juvenile myelomonocytic leukemia: A rare form of malignant bone marrow cancer that occurs in children and involves the proliferation of immature precursors of certain blood cells - myelocytes and monocytes. The proliferation is slower than in acute forms of the disease.
• Kartagener syndrome: A rare genetic disorder characterized by enlarged bronchial tubes, sinusitis and cross-positioning of body organs.
• Katayama fever: An acute disease due to infection with Schistosoma parasites. Transmission can occur through contact with infected waters.
• Kenya fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Kenya tick typhus: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Kenya tick-bite fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Korovnikov syndrome: A rare disorder characterized by enlarged spleen and increased blood platelets which leads to bleeding problems. It is considered to by a form of Banti's disease.
• Langerhans Cell Histiocytosis: A condition which is characterized by proliferation of Langerhans cells
• Langerhans cell sarcoma: A rare cancerous growth of Langerhans cells that can develop in the lymph nodes, skin, liver, spleen and bones.
• Large granular lymphocyte leukemia: A form of leukemia characterized by an increased number of circulating granular lymphocytes.
• Lathosterolosis: A very rare disorder where an enzyme (sterol C5-desaturase) deficiency prevents the normal synthesis of cholesterol in the body. The deficiency causes various malformations, mental retardation and liver disease.
• Lawrence-Seip syndrome: Lawrence-Seip syndrome is a relatively uncommon condition involving generalized loss of skin fat that is acquired rather than congenital as in Berardinelli-Seip Syndrome. Patients can also develop diabetes mellitus and other problems. The loss of skin fat tends to occur during childhood or adolescence and is often triggered by infections such as measles and hepatitis. The diabetes tends to occur after fat loss begins. The limbs and face tend to be the most affected.
• Leiomyosarcoma: A rare soft tissue cancer that arises from smooth muscle cells which form the involuntary muscles. Smooth muscle cells can occur in the skin, uterus, stomach, intestine and blood vessel walls. The main location for these tumors is in the uterus, retroperitoneum, pelvis and large blood vessels such as the inferior vena cava. Symptoms depend on the size and location of the tumor.
• Leishmaniasis: A rare infectious disease caused by any of a number of parasitic Leishmania species. Infection can cause any of three different manifestations: cutaneous leishmaniasis, mucosal leishmaniasis and visceral leishmaniasis.
• Leprosy: A chronic, progressive infectious disease caused by Mycobacterium leprae which causes skin sores and also affects the eyes, mucous membranes and peripheral nerves.
• Letterer-Siwe disease: A rare usually fatal condition characterized by skin lesions, bleeding tendency, enlarged liver and spleen, enlarged lymph nodes and progressive anemia. The condition is caused by excessive proliferation of histiocytes.
• Leukemia: Cancer of the blood cells, usually white blood cells.
• Leukemia, B-cell, chronic: A slow growing cancer of the immune system involving proliferation of B-cells.
• Leukemia, Lymphocytic, Acute, L1: Acute lymphocytic leukemia is a cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are three main subtypes (L1, L2 and L3) which are differentiated by the appearance of the cancerous cells: L1 is characterized by small, uniform cancer cells with a round nucleus and very little cytoplasm. L1 has a better prognosis than L2. L1 is the main form in children (about 85%) but is less common in adults (about 30%).
• Leukemia, Lymphocytic, Acute, L2: Acute lymphocytic leukemia is a cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are three main subtypes (L1, L2 and L3) which are differentiated by the appearance of the cancerous cells: L2 is characterized by larger cells, an irregular-shaped nucleus, more cytoplasm and significant variation between cells. L2 has a poorer prognosis than L1. L2 is the main form in adults (about 65%) but is less common in children (about 15%).
• Leukemia, Lymphocytic, Acute, L3: Acute lymphocytic leukemia is a relatively aggressive cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. There are three main subtypes (L1, L2 and L3) which are differentiated by the appearance of the cancerous cells. L3 is quite uncommon but is very similar to Burkitt's lymphoma - in fact, they may be considered the same disease with different manifestations.
• Leukemia, Monocytic, Acute: A cancer of the blood-forming tissues of the bone marrow involving the proliferation of a type of infection-fighting white blood cell called a monocyte. Acute leukemia involves a more rapid proliferation of cancer cells compared to chronic forms of leukemia.
• Leukemia, Myeloid: A form of blood cancer that causes a proliferation of the precursors or immature red blood cells, platelets and certain white blood cells such as granulocytes and monocytes.
• Leukemia, Myeloid, Aggressive-Phase: Myeloid leukemia is a form of cancer where the bone marrow makes too many myeloid cells (granulocytes and their precursors) in the bone marrow which accumulates in the blood and eventually invades various parts of the body. The aggressive phase of myeloid leukemia follows the chronic form and is a sign that the condition is progressing more rapidly to a blast crisis which is the final stage of leukemia.
• Leukemia, Myeloid, Chronic: A slow-growing cancer where the bone marrow makes too many myeloid cells (granulocytes and their precursors) in the bone marrow which accumulates in the blood and eventually invades various parts of the body.
• Leukemia, Myeloid, Chronic-Phase: Myeloid leukemia is a form of cancer where the bone marrow makes too many myeloid cells (granulocytes and their precursors) in the bone marrow which accumulates in the blood and eventually invades various parts of the body. The chronic phase of myeloid leukemia usually involves few if any symptoms. This is the early stage of the disease and without treatment it generally progresses to the accelerated phase of the condition. The majority of patients diagnosed with myeloid leukemia are diagnosed during the chronic phase.
• Leukemia, Myeloid, Philadelphia-Negative: Acute lymphocytic leukemia is a relatively aggressive cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. The Philadelphia negative form of the condition is not associated with a genetic mutation. It is distinguished from the positive form by the clinical course which is initially the same but progresses to eventual bone marrow failure without a distinct increase in blast cells. Other differences include poor response to chemotherapy, lower white blood cell counts, greater monocytosis, less basophilia, lower bone marrow myeloid to erythroid ratio and increased likelihood of developing thrombocytopenia. Philadelphia negative patients also tend to be older and median survival rates tend to be poorer.
• Leukemia, Myeloid, Philadelphia-Positive: Acute lymphocytic leukemia is a relatively aggressive cancer of the white blood cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets. The Philadelphia form of myeloid leukemia carries the poorest prognosis of all the different types. It involves an acquired genetic mutation which results in the production tyrosine kinase which causes too many white blood cells to be produced which results in a shortage of other blood cell types. Treatment is aimed at inhibiting the production of tyrosine kinase.
• Leukemia, T-Cell: A form of blood cancer characterized by the proliferation of cancerous T-cells which make up part of the body's immune system. The exact symptoms and progression vary depending on the subtype involved.
• Leukemia, T-Cell, Acute: A form of blood cancer characterized by the proliferation of cancerous T-cells which make up part of the body's immune system. The exact symptoms and progression vary depending on the subtype involved. The acute form involves a rapid proliferation of cancerous T-cells and hence a more rapid disease progression and increased severity of symptoms.
• Leukemia, T-cell, chronic: Cancer of blood cells called T-cells which form part of the immune system.
• Leukemia, mast-cell: A very aggressive form of leukemia - a subtype of acute myeloid leukemia. The cancer can in rare cases develop from chronic myeloid leukemia or systemic mastocytosis but generally develops on its own.
• Lipidosis with triglyceride storage disease: A very disorder involving abnormal storage of fat (triglycerides in parts of the body). The condition causes blood abnormalities as well as skin, eye and hearing problems.
• Lipoamide dehydrogenase deficiency: A very rare enzyme deficiency (dihydrolipoamide dehydrogenase) which can cause lactic acidosis. The age of onset and symptoms are variable.
• Lipodystrophy, familial partial, type 1 (FPLD1): An inherited disorder characterized by the loss of fatty tissue from the extremities while fatty tissue on the face, neck and trunk is normal or increased.
• Lipoproteine lipase deficiency: A rare inherited inborn error of metabolism involving the absence of the enzyme called lipoprotein lipase which results in increased blood triglyeride and chylomicron levels.
• Lissencephaly: A very rare disorder characterized by abnormal brain formation so that the brain surface appears smooth rather than convoluted.
• Lissencephaly syndrome type 1: A very rare form of the brain formation disorder called lissencephaly where the brain surface is abnormally smooth rather than convoluted resulting in mental retardation. Physical malformations are also present.
• Littoral cell angioma of the spleen: A usually benign type of tumor that develops in the spleen. The name arises from the fact that the tumor develops from the littoral cells which line the blood vessels in the spleen.
• Liver abscess: Pus in the liver
• Liver cancer: The occurrence of malignancy that occurs in the liver
• Liver conditions: Any condition that affects the liver
• Liver problems: Any problems that are associated with the livers structure and function
• Liver symptoms: Symptoms affecting the liver
• Lupus: Autoimmune disease with numerous effects on various organs and linings.
• Lymph node neoplasm: Lymph node tumor.
• Lymphadenopathy, angioimmunoblastic with dysproteinemia: A progressive immune system disorder that usually affects people over the age of 50 and is characterized by chills, sweating, weight loss, skin rashes.
• Lymphoblastic lymphoma: A cancer of the lymph system which usually involves immature T-lymphocytes and sometimes B-lymphocytes. The cancer usually affects the mediastinum (between the lungs), bone marrow, brain and spinal cord.
• Lymphogranuloma venereum: Type of chlamydia (sexually transmitted disease)
• Lymphoid interstitial pneumonia: A relatively rare form of lung disease characterized by the buildup of lymphocytes (a type of white blood cell) in the air spaces or alveoli of the lungs. The condition can affect children or adults and is frequently associated with conditions such as HIV and autoimmune conditions such as rheumatoid arthritis and Hashimoto's thyroiditis. Symptoms may develop gradually over a period of months or even years in some cases.
• Lymphoma: Any neoplastic disorder that occurs in lymphoid tissue
• MPS 3 C: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) due to deficiency of an enzyme called acetyl-CoA:alpha-glucosamide N-acetyltransferase. Mucopolysaccharide levels build up and are then deposited in various tissues.
• Macrocytic anemia: Macrocytic anemia is a blood disorder where the red blood cells are larger than normal but have low levels of haemoglobin which is needed to carry oxygen throughout the body. The condition usually results from a deficiency of vitamin B12 or folate, digestive problems, malabsorption and certain medications which affect folic acid levels. Various rare inherited disorders may also result in macrocytic anemia e.g. Lesch-Nyhan syndrome. Severity and range of symptom may vary depending on the underlying condition.
• Macrosomia with lethal microphthalmia: A very rare syndrome characterized mainly by very small eyes and a large body size at birth.
• Majeed syndrome: A rare syndrome characterized by blood abnormality and recurring bone infections.
• Malaria: A parasitic disease transmitted through mosquito bites.
• Mansonella ozzardi infection: Infection with a type of nematode (Mansonella ozzardi) found in Central America, South America and the Caribbean. Infection occurs through the bit of infected midges and blackflies.
• Marchiafava-Micheli disease: A rare disorder involving episodes of hemoglobinuria that usually occur at night.
• Mareck's disease - visceral: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Marek disease: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Marek's disease: A viral disease that affects poultry - it is very contagious and is a herpes virus. The virus tends to affect the nerves and cause tumors in internal organs. Poultry may be unable to lay eggs or may even die. Young birds are more susceptible than older birds with death most common between the ages of 8 and 20 weeks. The virus may attack the nervous system and result in paralysis or it may attack the visceral system and cause tumors in the internal organs. Some birds may die without any obvious symptoms.
• Marine turtle poisoning: Marine turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Green Sea Turtle: Green Sea turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Hawksbill Turtle: Hawksbill turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Leatherback Turtle: Leatherback turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Loggerhead Turtle: Loggerhead turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marine turtle poisoning - Soft-shelled Turtle: Soft-shelled turtles are found and eaten in the rivers and coastal waters of Southeast Asia. It is believed that sometimes these turtles become poisonous when the eat toxic algae which occur at certain times of the year. Symptoms vary in nature and severity amongst patients - obviously the more that is eaten, the more severe the symptoms are.
• Marseilles fever: An infectious disease that is caused by Rickettsia conorii which is transmitted by the brown dog tick (Rhipicephalus sanguineus). The disease occurs predominantly in Mediterranean areas such as India and Africa. The onset of symptoms is usually sudden and the incubation period is usually between 6 and 10 days.
• Mastocytosis: A disorder where excessive amounts of mast cells proliferate in organs such as the skin, liver, bone, spleen and gastrointestinal tract. Mast cells occur in connective tissue and defend the body against disease by releasing histamine to stimulate the immune system.
• Mauriac syndrome: A rare disorder characterized by diabetes, short stature and obesity.
• Mc Leod neuroacanthocytosis syndrome: A rare syndrome characterized by neuromuscular, blood and central nervous system symptoms. The disease is slowly progressive.
• Meadows syndrome: A rare condition that affects pregnant women during the last trimester or within two months after birth. It is characterized by breathing difficult, chest pain, congestive heart failure, heart rhythm abnormalities, high blood pressure, gastrointestinal symptoms and embolisms.
• Measles: Once common viral infection now rare due to vaccination.
• Megaloblastic Anemia 1: A rare genetic blood disorder where a defect in the vitamin B12 receptor means that it can't be absorbed from food during digestion. As a result of the low vitamin B12 levels, the body produces increased numbers of abnormal enlarged red blood cells (megaloblasts).
• Megaloblastic anemia: A rare blood disorder where insufficient vitamin B12 absorption results in reduced production of red blood cells and increased levels of abnormal, enlarged red blood cells (megaloblasts). Vitamin B12 insufficiency can result from absorption problems or lack of dietary intake of the vitamin or folic acid.
• Melioidosis: Bacterial infection from soil or water.
• Meningococcemia: A rare infectious disease whose main symptoms are upper respiratory tract infection, fever, rash and eye and ear problems.
• Mental retardation - macrocephaly - coarse facies - hypotonia: A very rare syndrome characterized mainly by mental retardation, large head, coarse face and reduced muscle tone.
• Metabolic disorder: occurs when abnormal chemical reactions occur in the body
• Metal-induced liver damage: Damage or injury to the liver caused by a exposure to a metal (usually ingestion). Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure.
• Metastatic cancer: Any cancer that has spread to other parts of the body.
• Methyldopa-induced Immune Hemolytic Anemia: Methyldopa-induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Methylmalonic acidemia, vitamin B12 responsive: A rare genetic disorder characterized by the impaired ability to make a chemical called adenosylcobalamin. Adenosylcobalamin is a derivative of vitamin B12. The defect results a biochemical abnormality which affects the body's normal biochemical functioning. The condition responds to the administration of vitamin B12.
• Methylmalonicaciduria, vitamin B12 unresponsive, mut 0: A metabolic disorder whose severity is somewhat variable - most patients die within months of birth with survivors having neurological problems. The condition involves abnormal metabolism of vitamin B12 which doesn't respond to treatment using vitamin B12 administration. This disorder is more severe than the mut (-) form
• Mevalonic aciduria: A rare disorder of amino acid metabolism characterized by a defect in the enzyme mevalonate kinase.
• Meyenburg-Altherr-Uehlinger syndrome: A rare disorder characterized by recurring inflammation of cartilage which results in deterioration of the cartilage. Any part of the body with cartilage may be affected - ears, nose bridge, larynx, trachea, aortic heart valve and around joints. Symptoms are determined by the part of the body that is affected. Breathing problems and even death can occur if the cartilage in the breathing airways or the aortic valve are affected.
• Microgastria short stature diabetes: A rare syndrome characterized by a very small stomach, diabetes and a growth hormone deficiency.
• Mononucleosis: Common infectious virus.
• Mosquito-borne diseases: Diseases that are carried by the mosquito
• Mosse syndrome: A condition involving the association of liver cirrhosis with polycythemia which is a chronic myeloproliferative disorder characterized by the excessive production of mainly red blood cells by the bone marrow.
• Mucopolysaccharidoses: A lysosomal storage disorder that is caused by a deficiency of the ability to metabolise glycosaminoglycans
• Mucopolysaccharidosis 1: A rare inherited condition caused by the absence of certain lysosomal enzymes which are needed to break down mucopolysaccharides. Without these enzymes, mucopolysaccharides build up in various body organs and tissues which causes harmful effects. There are three subtypes of the condition: Hurler, Scheie and Hurler-Scheie with Hurler being the most severe and Scheie the mildest.
• Mucopolysaccharidosis II: Disorder of mucopolysaccharide metabolism in juveniles.
• Mucopolysaccharidosis III: Disorder of mucopolysaccharide metabolism in children.
• Mucopolysaccharidosis Type III: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans). This occurs due to there being not enough of the enzyme called N-acetyl-alpha-glucosaminidase (B) which is needed to break down the mucopolysacharides. The mucopolysaccharides are then deposited in various tissues.
• Mucopolysaccharidosis VI: A lysosomal storage disorder that is caused by a deficiency of the ability to metabolise glycosaminoglycans
• Mucopolysaccharidosis VII: Disorder of mucopolysaccharide metabolism.
• Mucopolysaccharidosis type 2 Hunter syndrome- mild form: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (iduronidate 2-sulfatase) needed to break it down.
• Mucopolysaccharidosis type 2 Hunter syndrome- severe form: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (iduronidate 2-sulfatase) needed to break it down.
• Mucopolysaccharidosis type 6: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (Arylsulfatase ?) needed to break it down.
• Mucopolysaccharidosis type 7 Sly syndrome: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (beta-glucuronidase) needed to break it down.
• Mucopolysaccharidosis type I Hurler syndrome: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (?-L-diuronidase) needed to break it down. There are three main sub-types of the disorder: Hurler - severe form, Scheie - mild form and Hurler/Scheie form which is intermediate.
• Mucopolysaccharidosis type I Hurler/Scheie syndrome: A rare inherited biochemical disorder characterized by the accumulation of mucopolysaccharides (glycosaminoglycans) in various body tissues due to insufficient amounts of the enzyme (?-L-diuronidase) needed to break it down.
• Mulibrey Nanism syndrome: A very rare inherited malformation characterized by very small stature (dwarfism), pericardial constriction and yellow dots in fundus of the eye.
• Multi-centric Castleman's Disease: A rare disorder characterized by a generalized overgrowth of lymph node tissue which forms a tumor-like growth. This form of Castleman's disease is progressive and can be serious.
• Multiple myeloma: A rare malignant cancer that occurs in the bone marrow. More common in skull, spine, rib cage, pelvis and legs.
• Muscle phosphoglycerate kinase deficiency: An inherited genetic muscle disease where an enzyme deficiency (phosphoglycerate kinase) affects the normal processes that convert carbohydrates from food into energy.
• Muscular fibrosis, multifocal - obstructed vessels: A very rare syndrome characterized mainly by
• Mycobacterial infections: Any infection that is caused by a mycobacterial organisms
• Mycosis fungoides: Mycosis fungoides is a rare form of T-cell lymphoma of the skin. The disease is typically slowly progressive and chronic.
• Mycosis fungoides, familial: A rare form of lymphatic cancer (T-cell lymphoma) that primarily affects the skin and tends to occur with higher than normal frequency within a family. The skin is affected first, then the lymph nodes become inflamed and usually cancerous. The cancer can then spread to organs such as the liver, lungs and bone marrow. Survival depends on how early treatment starts. Patients diagnosed in the early stages can survive more than 12 years whereas once the cancer has spread to other organs, death usually occurs within three years.
• Mycotoxin-induced liver damage - Aflatoxin: Damage or injury to the liver caused by exposure to a mycotoxin called Aflatoxin which is a toxin produced by some Aspergillus fungus. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Cyclochlorotine: Damage or injury to the liver caused by exposure to a Mycotoxin called Cyclochlorotine which is a toxin produced by a particular fungus (Penicillium islandicum). Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Luteoskyrins: Damage or injury to the liver caused by exposure to a Mycotoxin called Luteoskyrin which is a toxin produced by a particular fungus (Penicillium islandicum). Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Ochratoxin: Damage or injury to the liver caused by exposure to a Mycotoxin called Ochratoxin. Ochratoxin is a toxin produced by a particular fungus (Aspergillus ochraceus and Penicillium verrucosum). It is a relatively common food contaminant and can be found in mouldy grain, pork, coffee and dried grapes. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Rubratoxin: Damage or injury to the liver caused by exposure to a Mycotoxin called Rubratoxin. Rubratoxin is a toxin produced by a particular fungus (Penicillium rubrum and purpurogenum) which is most commonly found on cereal grains. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Mycotoxin-induced liver damage - Sterigmatocystin: Damage or injury to the liver caused by exposure to a Mycotoxin called Sterigmatocystin. It is a toxin produced by a particular fungus (Aspergillus). Sterigmatocystin may also increase the risk of developing liver cancer. Most likely sources of exposure are foods such as wheat, maize, hard cheese and green coffee beans contaminated with mould. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. The toxin is also know to increase the risk of developing liver cancer.
• Myelodysplastic syndromes: A group of syndromes characterized by a disruption in the production of blood cells. Often the bone marrow increases production of various blood cells but because many of them are defective, they are destroyed before the reach the blood stream.
• Myelofibrosis: A rare condition where progressive scarring or fibrosis of the bone marrow impairs it's ability to make blood cells causing symptoms such as anemia and liver and spleen enlargement.
• Myelofibrosis-osteosclerosis: Formation of fibrous tissue in the bone marrow in response to bone marrow damage.
• Myeloid Sarcoma: A rare type of tumor that develops from myeloid cells (type of immature white blood cell) and tends to usually have a greenish color. The tumor is essentially a localized solid collection of leukemic cells that occurs outside the bone marrow. It can occur in many parts of the body such as the chest, vertebrae, pelvis, skin, lymph nodes and parts of the skull. On rare occasions, it can occur in various organs such as the heart and brain. The tumor can occur as a primary condition or may be associated with cancerous conditions such as acute leukemia and acute promyelocytic leukemia. Symptoms will vary depending on the location of the tumor.
• Myeloid splenomegaly: Spleen enlargement caused by myeloid cell (any leukocyte that isn't a lymphocyte) proliferation. The condition occurs with no obvious cause and is thus termed idiopathic.
• Myeloma: A primary malignancy of the plasma cells
• Myeloproliferative disease:
• Myelpathic anemia: Myelopathic anemia is a form of anemia resulting from the development of abnormal tissue in the bone marrow - usually metastatic cancers. It is characterized by abnormal number of immature blood cells in the blood.
• NISCH syndrome: A rare syndrome characterized by sparse scalp hair, scaly skin and inflammation and fibrosis of the bile ducts.
• NOMID syndrome: A rare autoinflammatory disease characterized by fever, rash, arthritic changes, eye problems and chronic meningitis.
• Nakajo-Nishimura syndrome: A rare disorder involving muscle degeneration, loss of skin fat and impaired immune functioning.
• Navajo neurohepatopathy: A rare genetic disease found in Navajo populations. It involves peripheral nerve degeneration, liver disease and corneal ulcers. The genetic disease is believed to be caused by maternal exposure to uranium from waters contaminated by old mines.
• Neonatal ALD: Progressive form of ALD in newborns.
• Neonatal hepatitis: A rare life-threatening condition where iron accumulates in the liver of the fetus and produces severe symptoms after birth. Death is common during the fetal or infant stage.
• Neonatal sepsis: Bacterial blood infection in an infant under 3 months of age.
• Nephroblastomatosis - fetal ascites - macrosomia - wilms tumor: A rare condition characterized by kidney abnormalities, macroxomia, endocrine pancreas abnormalities, large fetus and mental retardation.
• Nephroblastomatosis, fetal ascites, macrosomia and Wilms tumor: A rare condition characterized by kidney abnormalities, macroxomia, endocrine pancreas abnormalities, large fetus and mental retardation.
• Neuraminidase deficiency: A rare inherited disorder involving an enzyme (neuraminidase and beta-galactosidase) deficiency which results in a build up of glycoproteins (sialyloligosaccharides) in the urine.
• Niemann-Pick disease: A rare inherited biochemical disorder involving the deficiency of an enzyme (acid sphingomyelinase) needed to break down certain lipids which results in an accumulation of these lipids in the body.
• Niemann-Pick disease, type A: Niemann-Pick disease is an inherited metabolic disorder where lipids are not able to be metabolized adequately and hence build up and cause damage in various parts of the body such as the spleen, liver, lungs, bone marrow and brain. The different types of the disease are due to different genetic mutations. Type A is a severe infantile form in which death usually occurs by early childhood.
• Niemann-Pick disease, type B: Niemann-Pick disease is an inherited metabolic disorder where lipids are not able to be metabolized adequately and hence build up and cause damage in various parts of the body such as the spleen, liver, lungs, bone marrow and brain. The different types of the disease are due to different genetic mutations. Type B is a chronic form of the condition with no neurological involvement and patients can survive into adulthood.
• Niemann-Pick disease, type C1: Niemann-Pick disease is an inherited metabolic disorder where lipids are not able to be metabolized adequately and hence build up and cause damage in various parts of the body such as the spleen, liver, lungs, bone marrow and brain. The different types of the disease are due to different genetic mutations. Type C is a juvenile or subacute form of the condition which usually starts during childhood and survival into adulthood is possible.
• Non-Hodgkin's Lymphoma: A condition which is any neoplastic disorder of the lymphoid tissue
• Non-Hodgkin's lymphoma: A condition which is any neoplastic disorder of the lymphoid tissue
• Non-Hodgkin's lymphoma, during pregnancy: A cancer that originates in the lymphatic system and occurs during pregnancy. The greatest problem is the fact that the cancer is usually quite aggressive and delays in delivery often results in delayed treatment and a poor prognosis.
• Non-hereditary spherocytic anemia: Non-inherited spherocytic anemia is an acquired blood disorder characterized by red blood cells that are shaped like a sphere (spherocytes) rather than the normal doughnut shape. These abnormal cells are broken down prematurely resulting in a shortage of red blood cells (anemia). The blood condition may result from such things as acute, widespread burns to the body, low blood phosphate levels, Zieve's syndrome and toxins from poisonous snakes, spiders and insects.
• Obliterative portal venopathy: A rare condition characterized by abnormal hardening of liver blood vessels (portal veins), portal hypertension and an enlarged spleen. The condition usually occurs in adults but can rarely occur in children. The exact cause of the condition is unknown but various factors have been implicated e.g. genetic predisposition, workplace exposure to inorganic arsenicals, thorium compounds and vinyl chloride monomer.
• Occupational liver damage - 1,1,1-Tetrachloroethane: Damage or injury to the liver caused by exposure to 1,1,1-Tetrachloroethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 1,1,2-Tetrachloroethane: Damage or injury to the liver caused by exposure to 1,1,2-Tetrachloroethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 1,2-Dibromoethane: Damage or injury to the liver caused by exposure to 1,2-Dibromoethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 1,2-Dichloroethane: Damage or injury to the liver caused by exposure to 1,2-Dichloroethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 2-Nitropropane: Damage or injury to the liver caused by exposure to 2-Nitropropane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 2-acetylamino-fluorene: Damage or injury to the liver caused by exposure to 2-acetylamino-fluorene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 3,3-Dichlorobenzidine: Damage or injury to the liver caused by exposure to 3,3-Dichlorobenzidine and all its salts in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - 4-Dimethylaminoazobenzene: Damage or injury to the liver caused by exposure to 4-Dimethylaminoazobenzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Acetates: Damage or injury to the liver caused by exposure to various Acetates in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Acetonitrile: Damage or injury to the liver caused by exposure to Acetonitrile in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Acrylonitrile: Damage or injury to the liver caused by exposure to Acrylonitrile in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Alcohol: Damage or injury to the liver caused by exposure to alcohol in an occupational (industrial) setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Alicyclic Hydrocarbons: Damage or injury to the liver caused by exposure to alicyclic hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aliphatic Amines: Damage or injury to the liver caused by exposure to Aliphatic Amines in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aliphatic Hydrocarbons: Damage or injury to the liver caused by exposure to certain aliphatic hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aliphatic hydrogenated hydrocarbons: Damage or injury to the liver caused by exposure to aliphatic hydrogenated hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Allyl alcohol: Damage or injury to the liver caused by exposure to allyl alcohol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Amyl acetate: Damage or injury to the liver caused by exposure to Amyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aromatic Hydrocarbons: Damage or injury to the liver caused by exposure to certain Aromatic Hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aromatic amines: Damage or injury to the liver caused by exposure to certain aromatic amines (e.g. 2-acetylamino-fluorene) in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Aromatic halogenated hydrocarbons: Damage or injury to the liver caused by exposure to certain aromatic halogenated hydrocarbons in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Arsenic: Damage or injury to the liver caused by exposure to arsenic in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Arsine: Damage or injury to the liver caused by exposure to Arsine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Benzene: Damage or injury to the liver caused by exposure to Benzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Benzyl chloride: Damage or injury to the liver caused by exposure to Benzyl chloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Beryllium: Damage or injury to the liver caused by exposure to beryllium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Beta-Propiolactone: Damage or injury to the liver caused by exposure to Beta-Propiolactone in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Bipyridyl pesticides: Damage or injury to the liver caused by exposure to Bipyridyl pesticides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Bismuth: Damage or injury to the liver caused by exposure to bismuth and bismuth compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Boron: Damage or injury to the liver caused by exposure to boron and boron compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Boron hydrides: Damage or injury to the liver caused by exposure to Boron hydride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Bromide: Damage or injury to the liver caused by exposure to Bromide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Cadmium: Damage or injury to the liver caused by exposure to Cadmium and Cadmium compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Carbolic Acids and Anhydrides: Damage or injury to the liver caused by exposure to carbolic acids and anhydrides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Carbon Disulfide: Damage or injury to the liver caused by exposure to Carbon Disulfide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Carbon tetrachloride: Damage or injury to the liver caused by exposure to Carbon tetrachloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Carbonyls (metal): Damage or injury to the liver caused by exposure to Carbonyls (metal) in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chlorinated benzenes: Damage or injury to the liver caused by exposure to Chlorinated benzenes in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chlorinated naphthalenes: Damage or injury to the liver caused by exposure to Chlorinated naphthalenes in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chlorodiphenyls and derivatives: Damage or injury to the liver caused by exposure to Chlorodiphenyls and derivatives in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chloroform: Damage or injury to the liver caused by exposure to chloroform in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chloroprene: Damage or injury to the liver caused by exposure to chloroprene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Chromium: Damage or injury to the liver caused by exposure to chromium and chromium compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Copper: Damage or injury to the liver caused by exposure to copper in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Cresol: Damage or injury to the liver caused by exposure to Cresol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Cyclopropane: Damage or injury to the liver caused by exposure to cyclopropane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dibromochloropropane: Damage or injury to the liver caused by exposure to Dibromochloropropane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dimethyl sulfate: Damage or injury to the liver caused by exposure to Dimethyl sulfate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dimethylnitrosamine: Damage or injury to the liver caused by exposure to Dimethylnitrosamine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dinitrobenzene: Damage or injury to the liver caused by exposure to Dinitrobenzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dinitrophenol: Damage or injury to the liver caused by exposure to Dinitrophenol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Dinitrotoluene: Damage or injury to the liver caused by exposure to Dinitrotoluene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Diphenyl: Damage or injury to the liver caused by exposure to Diphenyl in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethanolamines: Damage or injury to the liver caused by exposure to Ethanolamines in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethyl Acetate: Damage or injury to the liver caused by exposure to Ethyl Acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethyl Ether: Damage or injury to the liver caused by exposure to ethyl ether in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethyl Salicylate: Damage or injury to the liver caused by exposure to Ethyl Salicylate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethyl alcohol: Damage or injury to the liver caused by exposure to ethyl alcohol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylene Dibromide: Damage or injury to the liver caused by exposure to Ethylene Dibromide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylene chlorohydrin: Damage or injury to the liver caused by exposure to ethylene chlorohydrin in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylene dichloride: Damage or injury to the liver caused by exposure to Ethylene dichloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylene oxide: Damage or injury to the liver caused by exposure to Ethylene oxide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ethylenediamine: Damage or injury to the liver caused by exposure to Ethylenediamine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Germanium: Damage or injury to the liver caused by exposure to Germanium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Hydrazine and derivatives: Damage or injury to the liver caused by exposure to Hydrazine and its derivatives in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Hydrogen Cyanide: Damage or injury to the liver caused by exposure to Hydrogen Cyanide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Hydrogen bromides: Damage or injury to the liver caused by exposure to Hydrogen bromides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Ionizing radiation: Damage or injury to the liver caused by exposure to Ionizing radiation in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Iron: Damage or injury to the liver caused by exposure to Iron in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Isopropyl acetate: Damage or injury to the liver caused by exposure to Isopropyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Kepone pesticides: Damage or injury to the liver caused by exposure to Kepone pesticides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Mercaptans: Damage or injury to the liver caused by exposure to Mercaptans in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methyl Bromide: Damage or injury to the liver caused by exposure to Methyl Bromide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methyl Chloride: Damage or injury to the liver caused by exposure to Methyl Chloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methyl acetate: Damage or injury to the liver caused by exposure to Methyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methylene chloride: Damage or injury to the liver caused by exposure to Methylene chloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Methylene dianiline: Damage or injury to the liver caused by exposure to Methylene dianiline in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N,N-Dimethylformamide: Damage or injury to the liver caused by exposure to N,N-Dimethylformamide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N-N-Dimethylacetamide: Damage or injury to the liver caused by exposure to N-N-Dimethylacetamide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N-Nitrosodimethylamine: Damage or injury to the liver caused by exposure to N-Nitrosodimethylamine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N-butyl acetate: Damage or injury to the liver caused by exposure to N-butyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - N-propyl acetate: Damage or injury to the liver caused by exposure to N-propyl acetate in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Naphthalene: Damage or injury to the liver caused by exposure to Naphthalene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Naphthol: Damage or injury to the liver caused by exposure to Naphthol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nickel: Damage or injury to the liver caused by exposure to nickel and nickel compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitriles: Damage or injury to the liver caused by exposure to certain nitriles in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitrobenzene: Damage or injury to the liver caused by exposure to Nitrobenzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitromethane: Damage or injury to the liver caused by exposure to Nitromethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitroparaffins: Damage or injury to the liver caused by exposure to Nitroparaffins in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Nitrophenol: Damage or injury to the liver caused by exposure to Nitrophenol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Phenol: Damage or injury to the liver caused by exposure to Phenol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Phosphine: Damage or injury to the liver caused by exposure to Phosphine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Phosphorus: Damage or injury to the liver caused by exposure to Phosphorus in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Phthalic Anhydride: Damage or injury to the liver caused by exposure to phthalic anhydride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Picric Acid: Damage or injury to the liver caused by exposure to Picric Acid in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Polybrominated biphenyls: Damage or injury to the liver caused by exposure to Polybrominated biphenyls in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Polychlorinated biphenyls: Damage or injury to the liver caused by exposure to Polychlorinated biphenyls in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Propylene dichloride: Damage or injury to the liver caused by exposure to Propylene dichloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Pyridine: Damage or injury to the liver caused by exposure to Pyridine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Pyrogallol: Damage or injury to the liver caused by exposure to Pyrogallol in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Selenium: Damage or injury to the liver caused by exposure to selenium and selenium compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Stibine: Damage or injury to the liver caused by exposure to Stibine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Styrene/ethyl benzene: Damage or injury to the liver caused by exposure to Styrene/ethyl benzene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tetrachloroethane: Damage or injury to the liver caused by exposure to Tetrachloroethane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tetrachloroethylene: Damage or injury to the liver caused by exposure to Tetrachloroethylene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tetramethylthiuram disulfide: Damage or injury to the liver caused by exposure to Tetramethylthiuram disulfide in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tetryl: Damage or injury to the liver caused by exposure to Tetryl in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Thallium: Damage or injury to the liver caused by exposure to Thallium and Thallium compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Thallium sulfate pesticides: Damage or injury to the liver caused by exposure to Thallium sulfate pesticides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Thorium dioxide: Damage or injury to the liver caused by exposure to Thorium dioxide (Thorotrast) in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Tin: Damage or injury to the liver caused by exposure to tin and tin compounds in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Toluene: Damage or injury to the liver caused by exposure to Toluene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Trichloroethylene: Damage or injury to the liver caused by exposure to Trichloroethylene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Trinitrotoluene: Damage or injury to the liver caused by exposure to Trinitrotoluene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Turpentine: Damage or injury to the liver caused by exposure to Turpentine in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Uranium: Damage or injury to the liver caused by exposure to Uranium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Vinyl Chloride: Damage or injury to the liver caused by exposure to Vinyl Chloride in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Whole body vibration: Damage or injury to the liver caused by whole body vibration in an occupational setting. This often occurs when operating equipment which cause constant physical vibration such as occurs when driving off-road vehicles or forklifts. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - Xylene: Damage or injury to the liver caused by exposure to Xylene in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational liver damage - n-Heptane: Damage or injury to the liver caused by exposure to n-Heptane in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms. Occupational liver damage is considered relatively uncommon due to current safe industrial practices.
• Occupational metal-induced liver damage - Antimony: Damage or injury to the liver caused by a exposure to antimony in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Arsenic: Damage or injury to the liver caused by a exposure to arsenic in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Barium: Damage or injury to the liver caused by a exposure to barium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Beryllium: Damage or injury to the liver caused by a exposure to beryllium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Bismuth: Damage or injury to the liver caused by a exposure to bismuth in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Boranes: Damage or injury to the liver caused by a exposure to boranes in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Boron: Damage or injury to the liver caused by exposure to boron in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Cadmium: Damage or injury to the liver caused by exposure to Cadmium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Chromium: Damage or injury to the liver caused by a exposure to chromium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Cobalt: Damage or injury to the liver caused by exposure to Cobalt in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Copper: Damage or injury to the liver caused by exposure to copper in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Germanium: Damage or injury to the liver caused by a exposure to germanium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Gold: Damage or injury to the liver caused by exposure to gold in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Hafnium: Damage or injury to the liver caused by a exposure to hafnium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Halides: Damage or injury to the liver caused by a exposure to halides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Hydrazines: Damage or injury to the liver caused by a exposure to hydrazines in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Iron: Damage or injury to the liver caused by a exposure to iron in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Lanthanides: Damage or injury to the liver caused by a exposure to Lanthanides in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Lead: Damage or injury to the liver caused by a exposure to lead in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Manganese: Damage or injury to the liver caused by a exposure to manganese in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Mercury: Damage or injury to the liver caused by exposure to mercury in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Occupational metal-induced liver damage - Molybdenum: Damage or injury to the liver caused by a exposure to molybdenum in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Nickel: Damage or injury to the liver caused by a exposure to nickel in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Niobium: Damage or injury to the liver caused by a exposure to niobium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Phosphorus: Damage or injury to the liver caused by a exposure to phosphorus in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Selenium: Damage or injury to the liver caused by a exposure to selenium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Tellurium: Damage or injury to the liver caused by a exposure to tellurium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Thallium: Damage or injury to the liver caused by a exposure to thallium in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Occupational metal-induced liver damage - Tin: Damage or injury to the liver caused by a exposure to tin in an occupational setting. Often other organs and tissues are also affected but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure.
• Oesophagostomiasis: A parasitic intestinal infection caused by a nematode called Oesophagostomum bifurcum. The parasite commonly infects monkeys, goats, cattle, sheep and pigs. The infection is relatively rare in humans but is most commonly found in northern Togo and Ghana. Transmission usually occurs through oral contact with contaminated soil.
• Okamuto Satomura syndrome: A very rare syndrome characterized mainly by kidney, heart and facial abnormalities as well as reduced muscle tone and mental retardation.
• Omenn syndrome: A rare inherited disorder of the immune system involving B and T lymphocytes and characterized by skin rash and frequent infections.
• Omsk hemorrhagic fever: A hemorrhagic fever caused by a virus. A serious outbreak occurred in Omsk and hence the name. Transmission occurs through tick bites. The infection has two phases: the first acute phase involves symptoms such as fever, rash and muscle pain and the second phase occurs after a week or two and involves the central nervous system (e.g. delirium, convulsions).
• Opisthorchiasis: Infection with a type of fluke (Southeast Asian liver fluke or cat liver fluke). Infection usually occurs by consuming infected undercooked fish. Acute infection may cause fever, joint pain, rash, eosinophilia and lymphadenopathy where as chronic infections may cause enlarged liver, malnutrition. Mild cases can cause constipation, diarrhea and abdominal pain. Occasionally, the infection may be asymptomatic and in the other extreme, severe cases may result in complications such as cholangiocarcinoma.
• Organic acidemia: High blood levels of organic acids which is caused by abnormal protein metabolism. Maple syrup urine disease and propionic academia are examples of organic acidemias. Deficiency of certain metabolic enzymes one of the main causes of organic academia.
• Ornithine Transcarbamylase Deficiency: A rare metabolic disorder where a deficiency of the enzyme ornithine transcarbamylase means that nitrogen isn't removed from the body and builds up in the blood in the form of ammonia.
• Ornithine transcarbamylase (OTC) Deficiency: A rare metabolic disorder where a deficiency of the enzyme ornithine transcarbamylase means that nitrogen isn't removed from the body and builds up in the blood in the form of ammonia.
• Orotic aciduria hereditary: A rare metabolic disorder where an inherited enzyme deficiency (lack of orotidylic pyrophosphorylase and orotidylic decarboxylase or a lack of orotidylic decarboxylase only) impairs the body's ability to break down orotic acid which then builds up in the body and causes damage.
• Osler-Vaquez disease: An uncommon chronic blood disease involving an increased red blood cell count.
• Osteopetrosis lethal: A severe, lethal form of the brittle bone condition called osteopetrosis.
• Osteopetrosis, autosomal recessive 2: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. This form is relatively mild with survival possible for a couple of decades in some cases.
• Osteopetrosis, autosomal recessive 4: A rare disorder characterized primarily by increased bone density as old bone is not resorbed and replaced with new bone - is also known as marble bone disease. The malignant infantile form is the most severe form of this disorder and death usually occurs in the first decade of life.
• Osteopetrosis, intermediate form: A recessively inherited bone disease characterized bybrittle bones with increased density. The intermediate form is less severe than the infantile form but more severe than the adult form.
• Osteopetrosis, mild autosomal recessive form: A rare disorder characterized by abnormally increased bone density as old bone is not resorbed and replaced with new bone. This form is relatively mild.
• Overhydrated hereditary stomatocytosis: A rare disorder where abnormal red blood cell membranes fail to regulate movement of sodium and potassium into and out of the red blood cell. The red blood cell accumulates too much salt and water which causes it to burst. Destruction of red blood cells leads to anemia. The red blood cells are abnormally shaped and resemble a stoma or mouth.
• PFIC: A rare inherited condition where bile is unable to drain from the liver where it builds up and causes progressive liver damage. End-stage liver disease usually occurs before adulthood. There are three different subtypes of the disorder, each with a different genetic origin of the defect.
• POEMS syndrome: A very rare disorder that has widespread effects on the body: P - polyneuropathy, O - organopathy, E - endocrinopathy, M - monoclonal gammopathy, S - skin changes.
• Pancreatic cancer: Pancreatic cancer is a malignant neoplasm of the pancreas
• Paragonimiasis: Infection by a parasitic worm, Paragonimus westermani, which are a type of lung fluke which invade the lungs, and sometimes other organs, where they cause problems. Occasionally the parasites infect the brain which can occasionally result in death. Infection occurs through eating freshwater crabs and crayfish which have not been cooked sufficiently.
• Pearson's anemia: Pearson's anemia is a rare progressive condition characterized by abnormal pancreatic functioning and sideroblastic anemia. Diagnosis usually occurs within the first 7 years of life and death in infancy is common.
• Penicillin-induced Immune Hemolytic Anemia: Penicillin-induced immune hemolytic anemia is a condition where use of a medication called Penicillin triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Perinatal-lethal Gaucher disease: A disorder caused by a deficiency of an enzyme called glucocerebrosidase. The foetal form is one of several forms of Gaucher disease and is the rarest. The fetal form causes death before birth or soon after.
• Periodic hyperlysinemia: A rare genetic disorder where the body lacks enzymes to metabolize lysine which then causes a harmful builds up of lysine and ammonia in the body.
• Pernicious anemia: A megaloblastic anaemia due to malabsorption of the vitamin B12
• Peroxisomal bifunctional enzyme deficiency: A rare disorder involving abnormal steroid metabolism due to an enzyme 17-beta-hydroxysteroid dehydrogenase 4) deficiency. The symptoms which make the condition appear very similar to another condition called neonatal adrenoleukodystrophy.
• Peroxisome biogenesis disorders: A group of inherited disorders involving the absence or dysfunction of one or more peroxisomal enzymes. Peroxisomes are numerous tiny organs within the cell which are involved in a large number of the body's biochemical reactions.
• Pfeiffer-Weber-Christian syndrome: A rare skin disorder characterized by recurring inflammation of the fatty layer of the skin and the development of nodules which can vary in size from 1 to 2 cm. The face and hands are not usually affected and the legs and feet tend to be the most commonly affected. The condition tends to reoccur within weeks or months.
• Philadelphia-negative chronic myeloid leukemia: A form of leukemia.
• Phocomelia - thrombocytopenia - encephalocele - urogenital malformation: An early congenital condition that is characterized by multiple congenital anomalies. The severity and range of abnormalities may vary amongst patients.
• Pinta: A tropical American skin disease that only affects dark-skinned races. It is caused by an organism the causes thickening and loss of pigmentation of the skin.
• Plague: Any epidemic disease with a high death rate.
• Plant toxin-induced liver damage - Albitocin: Damage or injury to the liver caused by exposure to a toxin called albitocin found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Cycasin: Damage or injury to the liver caused by exposure to a toxin called cycasin found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Icterogenin: Damage or injury to the liver caused by exposure to a toxin called Icterogenin found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Indospicine: Damage or injury to the liver caused by exposure to a toxin called Indospicine found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Lanthana: Damage or injury to the liver caused by exposure to a toxin called Lanthana found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Ngaione: Damage or injury to the liver caused by exposure to a toxin called Ngaione found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Nutmeg: Damage or injury to the liver caused by exposure to a toxin called Nutmeg found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Pyrrolidizine: Damage or injury to the liver caused by exposure to a toxin called Pyrrolidizine found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Safrole: Damage or injury to the liver caused by exposure to a toxin called Safrole found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plant toxin-induced liver damage - Tannic Acid: Damage or injury to the liver caused by exposure to a toxin called Tannic Acid found in certain plants. Often other organs and tissues are also affected by this toxin but only the liver toxicity symptoms are listed below. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Plasma cell leukemia: A form of leukemia characterized by the proliferation of plasma cells in the peripheral blood system. The prognosis is generally poor for this usually aggressive condition.
• Polar bear poisoning: Polar bears are often used as a food source by the arctic inhabitants. Eating the liver and kidneys of the polar bear is believed to result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases. Eating more than 200 grams of Polar bear liver can result in human death however death is considered rare.
• Polyarthritis, systemic: A chronic inflammatory disease (usually autoimmune) that causes inflammation in multiple parts of the body and causes arthritis in five or more joints.
• Polycystic kidney disease type 1: A rare condition where cysts in the kidney grow and cause the kidney to become larger and reduce it's ability to function. PKD 1 is an autosomal dominant form of the disease and differs from PKD 2 in that it is more severe and is caused by a mutation in a different gene.
• Polycystic kidney disease, adult type: A rare condition where cysts in the kidney grow and cause the kidney to become larger and reduce it's ability to function. PKD 2 is an autosomal dominant form of the disease and differs from PKD 1 in that it is less severe and is caused by a mutation in a different gene.
• Polycystic kidney disease, type 2: A rare condition where cysts in the kidney grow and cause the kidney to become larger and reduce it's ability to function. This adult form of the condition is milder than the infantile form.
• Polycythemia: increase in the total number of red blood cells in the circulation. It can be primary or secondary
• Polycythemia rubra: An uncommon chronic blood disease involving an increased production of red blood cells by the bone marrow. The production of platelets and white blood cells may also be increased.
• Polycythemia vera: A condition which is characterized by myeloproliferation of unknown etiology
• Polydactyly visceral anomalies cleft lip palate: A rare syndrome characterized mainly by the association of extra digits, cleft lip and/or palate and visceral anomalies. Various other abnormalities may also be present.
• Porphyria, congenital erythropoietic: An extremely rare inherited metabolic disorder involving a deficiency of the enzyme uroporphyrinogen III cosynthase which results in increased deposits of porphyrin in the bone marrow, blood, urine, feces and sometimes teeth and bones.
• Portal hypertension: Portal hypertension may be defined as a portal pressure gradient of 12 mm Hg or greater and is often associated with varices and ascites.
• Portal hypertension due to intrahepatic block: A blockage that forms in the portal vein (caries blood from the digestive tract to the liver) due to liver problems.
• Portal thrombosis: Clotting or obstruction of blood flow along the veins from the intestines and spleen and into the liver. This causes blood to back up and results various problems such as enlarged spleen and abdominal pain. The obstruction can occur acutely (over a short space of time) or chronically (over a longer period of time).
• Portal vein thrombosis: Clotting or obstruction of blood flow along the portal vein and into the liver. This causes blood to back up and results various problems such as enlarged spleen and abdominal pain. The obstruction can occur acutely (over a short space of time) or chronically (over a longer period of time). The portal vein collects blood from the intestines and spleen and carries it to the liver.
• Postperfusion syndrome: A complication that can occasionally occur following blood transfusion. The condition is believed to be caused by Ebstein-Barr virus or cytomegalovirus present in the transfused blood.
• Pregnancy symptoms: Symptoms related to pregnancy.
• Primary Autoimmune Hemolytic Anemia: Primary autoimmune hemolytic anemia is characterized by an abnormal immune system response. The body's immune system produces antibodies against the body's own red blood cells which leads to the destruction of red blood cells and hence anemia. The secondary form of the condition is the result of an underlying condition such as lupus or hepatitis whereas the primary form has no underlying condition.
• Primary Cold Autoimmune Hemolytic Anemia: Primary cold autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The process occurs only when the patient is exposed to temperatures colder the average body temperature. The red cells are destroyed at an abnormally rapid rate which leads to anemia. The primary form of the condition means that there is no underlying condition. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Primary biliary cirrhosis: Primary biliary cirrhosis is a chronic and progressive cholestatic disease of the liver.
• Primary granulocytic sarcoma: A malignant tumor derived from immature white blood cells called myeloblasts. It can occur anywhere in the body but is most commonly found in bone, soft tissue, lymph nodes and skin. Symptoms will vary according to the location of the tumor.
• Primary malignant lymphoma: The excessive proliferation of lymphocytes which forms part of the immune system. Primary cancers refer to the fact that the cancer originated in the lymph cells rather than having metastasized.
• Primary sclerosing cholangitis: Primary sclerosing cholangitis (PSC) is a chronic cholestatic liver disease of unknown etiology that is recognized increasingly in children.
• Progeria short stature pigmented nevi: A very rare inherited disorder characterized by premature aging, short stature, and immune system deficiency. The type and severity of symptoms is variable.
• Prolidase deficiency: A rare metabolic disorder where an enzyme (prolidase) deficiency impairs the breakdown of certain proteins which causes a harmful buildup in the body. The type and severity of symptoms are variable and may include skin disorders, retarded motor development, impaired cognitive development, frequent infections and skeletal abnormalities.
• Prolymphocytic leukemia: A very rare type of leukemia involving a proliferation of immature white blood cells (prolymphocytes - T-cells).
• Protozoan Conditions: Any condition caused by the infection of the human being by a protozoan organism
• Proximal tubulopathy - diabetes mellitus - cerebellar ataxia: A very rare syndrome characterized mainly by diabetes, movement disorder and kidney abnormality. Death often occurs during childhood.
• Pseudo-torch syndrome: A rare genetic syndrome characterized by a small head, eye disease and brain abnormalities. The symptoms are often very similar to another disorder caused by certain infections during pregnancy.
• Pseudoadrenoleukodystrophy: A rare disorder where an enzyme deficiency (Acyl-CoA Oxidase) results in symptoms such as seizures, apnea, delayed psychomotor retardation and neurological deterioration.
• Psittacosis: An infectious disease caused by Chlamydia psittaci and transmitted mainly by infected birds but also by some mammals.
• Psychotropic agent-induced liver damage: Damage or injury to the liver caused by exposure to psychotropic agents. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - Benzodiazepine: Damage or injury to the liver caused by exposure to a psychotropic agent called benzodiazepine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - Butyrophenone: Damage or injury to the liver caused by exposure to a psychotropic agent called butyrophenone. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - Phenothiazines: Damage or injury to the liver caused by exposure to a psychotropic agent called phenothiazine. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - Thioxanthene: Damage or injury to the liver caused by exposure to a psychotropic agent called Thioxanthene. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - monoamine oxidase inhibitors: Damage or injury to the liver caused by exposure to psychotropic agents called monoamine oxidase inhibitors. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Psychotropic agent-induced liver damage - tricyclic antidepressant: Damage or injury to the liver caused by exposure to a psychotropic agent called tricyclic antidepressant. Psychotropic agents are a relatively uncommon cause of liver damage. Symptoms vary depending on the degree of exposure and hence extent of the liver damage or injury. Mild liver damage may cause few if any symptoms whereas severe damage can ultimately result in liver failure. Symptoms may be acute, subacute or chronic depending on the severity of the exposure. Factors such as age, race, gender, overall health and underlying liver problems may also influence a person's risk of developing liver problems and the severity of the symptoms.
• Pulmonary valve stenosis: Often a congenital defect but may be caused by such things as rheumatic fever or bacterial endocarditis. Severity depends on the degree of narrowing of the pulmonary valve.
• Pulmonary venous return anomaly: A rare disorder where one or more of the four veins that carry oxygenated blood from the lungs drain to the right atrium of the heart instead of the left atrium. Symptom severity is determined by the number of veins involved and the exact location of the heart that the veins drain into.
• Pylephlebitis: A pus-producing inflammation of the wall of the portal vein that drains blood from the abdominal part of the gastrointestinal tract. The infection is often fatal. It usually occurs as a complication of abdominal or pelvic infections such as diverticulitis and appendicitis.
• Pyruvate Kinase Deficiency: A rare inherited blood disorder involving a deficiency of the enzyme pyruvate kinase.
• Pyruvate carboxylase deficiency, Group A: A rare inherited disorder characterized by a deficiency of pyruvate carboxylase which leads to accumulation of lactic acid and other compounds which can have a toxic effect on the nervous system and body organs. Type A has moderately severe symptoms that usually start during infancy.
• Pyruvate kinase deficiency, hemolytic anemia: A rare inherited blood disorder where the deficiency of an enzyme (pyruvate kinase) needed by red blood cells causes them to be destroyed prematurely and hence anemia occurs.
• Pyruvate kinase deficiency, liver type: An inherited deficiency of pyruvate kinase which causes varying degrees of hemolytic anemia.
• Pyruvate kinase deficiency, muscle type: An inherited deficiency of pyruvate kinase which causes varying degrees of hemolytic anemia.
• Q fever: A disease caused by Coxiella burnetti which causes fever, headache and muscle pain.
• Quinidine-induced Immune Hemolytic Anemia: Quinidine-induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Refsum disease, infantile form: A rare inherited biochemical disorder characterized by normal development initially followed by degeneration, facial anomalies and enlarged liver.
• Relapsing fever: Tick-borne disease with symptoms that resolve and then relapse
• Renal tubulopathy - diabetes mellitus - cerebellar ataxia: A very rare condition characterized by rapidly progressive diabetes, kidney disease and cerebellar ataxia. Symptoms develop during the first five years.
• Reticuloendotheliosis: An inherited disorder of the reticuloendothelial system which is involved in destroying bacteria and other unwanted substances that enter the body.
• Retinal telangiectasia and hypogammaglobulinemia: A rare disorder characterized by the association of poor immunity with dilated retinal blood vessels.
• Reye's syndrome: is a potentially fatal disease that causes numerous detrimental effects to many organs, especially the brain and liver
• Rheumatoid arthritis: An autoimmune inflammatory condition which primarily affects the joints
• Richter syndrome: A rare but serious form of acute adult leukemia. The disease occurs when chronic lymphocytic leukemia transforms into diffuse large cell lymphoma which is a fast-growing form of lymphoma. The condition tends to be more common in older adults.
• Rietti-Greppi-Micheli syndrome: A type of genetic blood disorder involving reduced hemoglobin production.
• Ringed seal poisoning: The Ringed seal is often used as a food source by the arctic inhabitants. Eating the liver of the ringed seal can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases.
• Roifman syndrome: A rare syndrome characterized by abnormal bone growth, immune deficiency and an eye disorder.
• Rosai-Dorfman disease: A rare condition characterized by excessive production and accumulation of a particular white blood cell (histiocyte). Accumulation primarily occurs in the lymph nodes, especially in the neck, but may also occur in the skin, central nervous system, digestive tract and kidneys.
• Rubella: A contagious viral infection caused by the Rubella virus which produces a rash and lymph node swelling. It can have serious implication in pregnant women as the virus can be transmitted through the placenta and cause serious fetal defects or even fetal death.
• SCARF syndrome: A rare syndrome characterized mainly by skeletal abnormalities, loose skin, premature fusion of skull bones, ambiguous genitalia, psychomotor retardation and facial abnormalities.
• Sandhoff Disease: An inherited biochemical disorder involving a deficiency of the enzyme called hexosaminidase A&B which results in the harmful accumulation of chemicals in the central nervous system and other body tissues.
• Sarcoid: A fleshy tumor.
• Sarcoidosis: Rare autoimmune disease usually affecting the lungs.
• Sarrouy disease: A disease characterized mainly by anemia, poor growth and enlarged liver and spleen. The disease usually ends with death.
• Scheie syndrome:
• Schistosomiasis: Parasitic fluke infection in developing countries
• Schnitzler syndrome: A rare syndrome characterized by the association of chronic urticaria as well as a blood abnormality called macroglobulinemia.
• Schridde syndrome: A rare disorder involving a fetal blood abnormality which usually results in death.
• Sea-Blue histiocytosis: A rare condition involving the presence of too many histiocytes in the spleen and bone marrow. These histiocytes stain a sea-blue color during testing. These abnormal staining histiocytes have been noted in several diseases such as Niemann-Pick disease, lipid metabolism disorders and Norum disease.
• Secondary Autoimmune Hemolytic Anemia: Secondary autoimmune hemolytic anemia is characterized by an abnormal immune system response. The body's immune system produces antibodies against the body's own red blood cells which leads to the destruction of red blood cells and hence anemia. The secondary form of the condition is the result of an underlying condition such as lupus or hepatitis whereas the primary form has no underlying condition. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Secondary Cold Autoimmune Hemolytic Anemia: Secondary cold autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The process occurs only when the patient is exposed to temperatures colder the average body temperature. The red cells are destroyed at an abnormally rapid rate which leads to anemia. The secondary form of the condition means that it is the result of an underlying condition - it usually occurs after some sort of infection. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Secondary syphilis: A condition which is characterized by fever, multiform skin eruptions, iritis, alopecia, mucous patches and severe pain in the head and joints
• Selective Vitamin B12 malabsorption with Proteinuria: A rare genetic blood disorder where a defect in the vitamin B12 receptor means that it can't be absorbed from food during digestion. As a result of the low vitamin B12 levels, the body produces increased numbers of abnormal enlarged red blood cells (megaloblasts).
• Septicemia: A systemic inflammatory response to an infection.
• Serum sickness: Type of allergic reaction to certain medications or serums
• Short stature - cranial hyperostosis - hepatomegaly: A very rare syndrome characterized mainly by a thickened skull, short stature and an enlarged liver.
• Short stature cranial hyperostosis hepatomegaly diabetes: A very rare syndrome characterized mainly by enlarged liver, diabetes, short stature and a thickened skull.
• Short stature, cranial hyperostosis, hepatomegaly and diabetes: A very rare syndrome characterized mainly by enlarged liver, diabetes, short stature and a thickened skull.
• Sialidosis type 2: A very rare inherited metabolic disorder where a defect in the enzyme alpha-neuraminidase prevents glycoproteins being metabolized - a severe form of Sialidosis type I.
• Sialidosis type II: A very rare inherited metabolic disorder where a defect in the enzyme alpha-neuraminidase prevents glycoproteins being metabolized - a severe form of Sialidosis type I.
• Sialidosis type II, infantile: A very rare inherited metabolic disorder where a defect in the enzyme alpha-neuraminidase prevents glycoproteins being metabolized. The infantile form is characterized by structural or functional kidney problems.
• Sialuria syndrome: A rare metabolic disorder characterized by excessive levels of sialic acid in the urine as well as liver and spleen enlargement and coarse facial features.
• Sialuria, Finnish type: A rare inherited biochemical disorder characterized by the accumulation of sialic acid in the tissues and excretion of sialic acid in the urine. This condition is an adult form of sialuria.
• Sialuria, French type: A rare disorder characterized by the excretion of large amounts of sialic acid in the urine due to a metabolic error.
• Siccardi syndrome: A rare disorder characterized by impaired immunity (due to defective bacterial destruction by neutrophils), lymphadenopathy and abnormal skin and hair color.
• Sickle Cell Anemia: Sickle cell anemia is an inherited blood disorder characterized by red blood cells which are crescent-shaped rather than the normal doughnut shape. These abnormally shaped red blood cells are unable to function normally and tend to undergo premature destruction which leads to anemia. If the genetic defect which causes the condition is inherited from both parents the condition can be quite severe whereas if it is inherited from only one parent, often there are no symptoms. The abnormally shaped red blood cells can cause problems when they clump together and block blood vessels.
• Sickle cell anemia: Sickle cell anemia is an inherited blood disorder characterized by red blood cells which are crescent-shaped rather than the normal doughnut shape. These abnormally shaped red blood cells are unable to function normally and tend to undergo premature destruction which leads to anemia. If the genetic defect which causes the condition is inherited from both parents the condition can be quite severe whereas if it is inherited from only one parent, often there are no symptoms. The abnormally shaped red blood cells can cause problems when they clump together and block blood vessels.
• Sideroblastic anemia, autosomal: An inherited form of anemia where an enzyme deficiency prevents red blood cells from using iron properly. The red blood cells become overloaded with iron which impairs the blood's ability to carry oxygen.
• Sideroblastic anemia, hereditary: An inherited form of anemia where an enzyme deficiency prevents red blood cells from using iron properly. The red blood cells become overloaded with iron which impairs the blood's ability to carry oxygen.
• Sideroblastic anemia, pyridoxine-responsive, autosomal recessive: Pyridoxine-responsive sideroblastic anemia is a rare inherited blood disorder characterized by abnormal deposits of iron in the red blood cells which makes them unable to function properly. The condition is inherited in an autosomal recessive manner and responds to treatment with pyridoxine.
• Simpson-Golabi-Behmel syndrome, type 1 (SGBS1): A rare genetic disorder characterized by accelerated growth, a peculiar face and other defects.
• Sitosterolemia: A very rare inherited disorder of lipid metabolism where excessive amounts of sterols are absorbed and not removed from the body resulting in a build up in the blood.
• Smith disease: A harmless condition involving increased lymphocyte levels which may manifest as a variety of symptoms or may be asymptomatic. Diseases such as whooping cough and German measles are believed to be possible causes. The disease most commonly occurs in children and young adults.
• Smoldering adult T-Cell leukemia: A form of blood cancer affecting the T-cells which make up the body's immune system. The disease is caused by the HTLV-1 virus (human T-cell leukemia virus) which causes the proliferation of abnormal T-cells. The virus can be transmitted sexually and may lay dormant for decades. There are four subtypes: acute, chronic, lymphoma and smoldering. The acute and lymphoma subtypes have the poorest prognosis. The smoldering form tends to progress even slower than the chronic form and responds the best to treatment.
• Soft Tissue Sarcoma: A rare type of cancerous tumor that originates in soft tissues of the body which includes muscles, nerves, tendons, blood vessels and fat. They can occur anywhere in the body and are often asymptomatic during the early stages and symptoms in the later stages depend on the location and size of the tumor.
• Speech symptoms: Problems with speech or voice.
• Spherocytic anemia: Spherocytic anemia is a blood disorder characterized by red blood cells that are shaped like a sphere (spherocytes) rather than the normal doughnut shape. These abnormal cells are broken down prematurely resulting in a shortage of red blood cells (anemia). The condition may be inherited or acquired.
• Spherocytic hereditary elliptocytosis: A rare inherited blood disorder characterized by the presence of spherocytes and oval-shaped red blood cells rather than the normal doughnut-shape. This form of hereditary elliptocytosis can cause hemolytic anemia.
• Spherocytosis: A rare blood disorder characterized by a defect of the red blood cell membrane which gives it a spherical shape rather than the normal doughnut shape.
• Sphingolipidosis: A group of diseases involving the abnormal metabolism and storage of a substance called sphingolipid. Symptoms will vary depending on the disease. Examples of diseases from this group include gangliosidosis, Gaucher's disease and Niemann-Pick disease.
• Spirochetes disease: Infection with a type of bacteria which is often found in mud, sewage and polluted water. Symptoms are determined by the species involved. Diseases caused by this bacteria include Treponema infection and borreliosis.
• Spleen conditions: Any condition that affects the spleen
• Spleen symptoms: Symptoms affecting the spleen
• Spondyloepimetaphyseal dysplasia congenita, Iraqi: A very rare syndrome characterized mainly by abnormal bone growth. Only a few cases have been reported.
• Spondyloepimetaphyseal dysplasia, hypotrichosis: An inherited bone growth disorder characterized by short stature, bone abnormalities and vision problems. An additional feature of this condition is the reduced amount of hair (hypotrichosis). The bone abnormalities are due to a genetic mutation that affects the development of bone and connective tissue.
• Spondylometaphyseal dysplasia with combined immunodeficiency: A rare syndrome characterized by skeletal abnormalities as well as a poor immune system.
• Spotted fevers: An acute condition caused by the bacteria Rickettsia rickettsii
• Spranger syndrome: A rare genetic disorder characterized by dwarfism and an unusually pleasant, happy face as well as other abnormalities involving the hands and feet. The condition involves localized accumulations of mucopolysaccharides in the liver, trachea and cardiovascular system cartilage.
• Stauffer syndrome: Spleen and liver problems associated with a kidney tumor.
• Still's Disease, Adult-Onset: A form of arthritic inflammation characterized by fever, rash and joint pain that occurs in adults. The cause is unknown.
• Strudwick syndrome: An inherited bone growth disorder characterized by short stature, bone abnormalities and vision problems. The bone abnormalities are due to a genetic mutation that affects the development of bone and connective tissue.
• Stuart-Bras disease: A type of liver disease that is seen in children (usually aged 1-6 years) in the West Indies and is possibly caused by ingestion of plant toxins from plants such as Senico.
• Subacute granulomatous thyroiditis: Subacute thyroiditis is a self-limited thyroid condition associated with a triphasic clinical course of hyperthyroidism, hypothyroidism, and return to normal thyroid function.
• Sulfatidosis juvenile, Austin type: A very rare metabolic disorder where all the sulfatase enzymes are impaired resulting in a variety of physical and developmental abnormalities.
• Sulphonamide -induced Immune Hemolytic Anemia: Sulphonamide-induced immune hemolytic anemia is a condition where use of a medication called Methyldopa triggers the body's immune system to destroy it's own red blood cells which results in anemia.
• Summerskill-Walshe-Tygstrup syndrome: A rare liver disorder characterized by intermittent episodes of cholestasis (impaired bile flow within the liver) that doesn't involve obstruction of bile ducts outside the liver. The jaundice tends to disappear in weeks or months without treatment and the condition does not lead to liver failure. The two subtypes of the disorder have a different origin of the genetic mutation.
• Swelling symptoms: Symptoms causing swelling or enlargement.
• Syphilis: A sexually transmitted disease caused by a bacteria (Treponema pallidum). The condition is often asymptomatic in the early stages but one or more sores may be present in the early stages. Untreated syphilis usually results in remission of visible symptoms but further severe damage may occur to internal organs and other body tissues which can result in death.
• Systemic Juvenile Rheumatoid Arthritis: Onset of JRA with fevers and systemic symptoms
• Systemic lupus erythematosus: chronic autoimmune disease that can be fatal, though with recent medical advances, fatalities are becoming increasingly rare.
• Systemic mastocytosis: A condition which is characterized by an accumulation of mast cells in the tissues of the body
• T-cell acute lymphoblastic leukemia: Cancer of particular white blood cells called T-cells. Precursors to white blood cells are called blasts and are made by the bone marrow but in ALL the blasts are abnormal and do not develop into lymphocytes. Instead, the abnormal blasts or leukemic cells multiply rapidly and reduce the level of other types of blood cells such as red blood cells and platelets.
• T-cell lymphoma 1A: T-cell lymphoma 1A is a type of cancer that has genetic origins. The genetic defect is located on chromosome 14q32.1. The anomaly is usually implicated in t-cell prolymphocytic leukemia. The cancer is usually aggressive and tends to target the blood, bone marrow, lymph nodes, liver, spleen and skin. The cancer is generally quite rare and tends to occur in people over the age of 30.
• T-cell prolymphocytic leukemia: A form of cancer which causes proliferation of T-cells in the intermediate stage of development.
• TORCH Syndrome: Infection of a fetus by any of a group of infectious agents which have been transmitted from the mother through the placenta. The infections include toxoplasmosis, rubella, cytomegalovirus, herpes virus, hepatitis and syphilis. The severity and nature of symptoms is determined by the type of infection.
• TRAPS (TNF-receptor-associated periodic syndrome): A rare syndrome involving periods of fever and chills along with gastrointestinal symptoms and muscle pain. Symptoms last for two or three weeks.
• Tang Hsi Ryu syndrome: A rare syndrome characterized by enlarged liver and spleen, increased pigmentation and abnormal peripheral nerve functioning.
• Tangier disease: A rare disorder characterized by a very low level of HDL (high-density lipoprotein or "good cholesterol") in the blood. The condition occurs because the body lacks the gene to make a certain protein (Apolipoprotein A1) which normally transports fats from tissues to where it is needed.
• Tertiary syphilis: A condition which is characterized by late generalized syphilis with involvement of many organs and tissues
• Thalassemia: Thalassemia is an inherited blood disorder characterized by abnormal synthesis of hemoglobin. Hemoglobin consists of two main protein chains called ? and ?. There are two subtypes of the disorder (? and ?) depending on what portion of the hemoglobin is abnormally synthesized. Each of these subtypes can be further classified depending on the number of thalassemia genes involved. A total of four genes is needed to make each ? protein chains and two genes are needed to make each ? chain. The main symptom is anemia, the severity of which can vary amongst patients depending on how many defective genes are involved.
• Thiamine responsive megaloblastic anemia syndrome: A rare genetic disorder characterized by megaloblastic anemia, hearing loss and diabetes. The condition is caused by a deficiency of a thiamine (vitamin B1) transporter protein which means that the body is unable to effectively utilize thiamine from the diet.
• Thoraco abdominal enteric duplication: A very rare syndrome characterized by chest and abdominal abnormalities as well as intestinal duplication.
• Thoracopelvic dysostosis: A rare syndrome characterized mainly by abnormalities involving the chest, larynx and pelvis. The small chest cavity affects breathing and can affect survival, especially during infancy.
• Thrombocythemia: A haematological disorder in which there is excessive proliferation of platelet precursor cells, resulting in increased numbers of platelets in the blood.
• Thrombocytopenia: Decreased concentration of platelets in the blood.
• Thrombocytopenic purpura, autoimmune: A rare blood disorder where a low number of platelets impairs the bloods ability to clot and results in bleeding into the skin and mucous membranes.
• Thrombotic thrombocytopenic purpura, acquired: A rare blood condition where small blood clots form in blood vessels which reduces the number of blood platelets and results in kidney failure, neurological symptoms and anemia. The condition may be familial or acquired - symptoms tend to recur regularly in the familial form.
• Tollner-Horst-Manzke syndrome: A very rare syndrome characterized by extra fingers, cleft lip, cleft palate and abdominal organ anomalies.
• Toxic mushrooms - Monomethylhydrazine: Some mushrooms contain a toxic chemical called gyromitrin which is converted to monomethylhydrazine after digestion. Mushroom species from this group include certain species of Gyromitra, Helvella, Sarcosphaera and Peziza. Poisoning may occur from inhaling fumes from cooking mushrooms. The amount of toxin varies amongst and within species but some are toxic enough to cause death. Urgent medical attention should be sought if mushroom poisoning is suspected.
• Toxocariasis: A parasitic roundworm (Toxocara canis or Toxocara cati) infection that normally occurs in cats and dogs but can be transmitted to humans by ingesting the larvae or eggs. The infection may be asymptomatic or severe and symptoms depend on where the larvae travel to when they migrate through the body.
• Toxoplasmosis: Infection often caught from cats and their feces.
• Transposition of great arteries: A congenital malformation where the aorta and pulmonary artery are transposed which causes oxygenated blood from the lungs to be sent back to the lungs and de-oxygenated blood to be sent to body tissues. Often there is some other defect such as an opening in the heart chambers which allows mixing of the blood and hence survival is possible for a short while at least.
• Treponema infection: A rare infectious diseases which is transmitted through sexual contact and caused by Treponema pallidum (a spirochete bacterium). Untreated cases can result in severe complications and even death.
• Trichomegaly cataract hereditary spherocytosis: A rare syndrome characterized mainly by cataracts, excessive eyelash growth and a blood abnormality (the red blood cells are spherical instead of doughnut shaped which makes them fragile).
• Tricuspid valve diseases: Any disease that affects the tricuspid valve
• Triglyceride storage disease with impaired long-chain fatty acid oxidation: A rare inherited disorder involving the metabolism of fat which causes skin, muscle and blood abnormalities.
• Triosephosphate isomerase 1: A rare inherited blood disorder involving a deficiency of red cell triosephosphate isomerase which is involved in the glycolysis (glucose metabolism) process to produce energy. The disorder is characterized by anemia, recurring infection and often progressive neurological degeneration. The severity of the disorder is variable with some patients having central nervous system involvement while others don't.
• Trypanosomiasis:
• Tuberculosis: Bacterial infection causing nodules forming, most commonly in the lung.
• Tularemia: A rare infections disease caused by the bacterium Francisella tularensis (a gram-negative pleomorphic coccobacillus). Transmission occurs through contact with infected animals or there habitats e.g. bites from infected insects or other animals, eating infected wild animals, contact with contaminated water and soil. Symptoms can vary greatly depending on the method of infection. For example infection through inhalation can cause symptoms similar to pneumonia, eating infected animals can cause a sore throat and abdominal symptoms and transmission through the skin can cause result in a painful skin ulcer.
• Type I Hyperlipoproteinemia: Type I Hyperlipoproteinemia is a rare inherited condition characterized by high levels of chylomicrons (a type of lipoprotein) in the blood. This condition is the result of low levels of lipoprotein lipase which is responsible for lowering lipoprotein levels. Cholesterol levels are usually normal and the risk of heart disease is low.
• Type III Hyperlipoproteinemia: Type III hyperlipoproteinemia is a rare genetic condition characterized by increased blood levels of intermediate density lipoproteins (IDL) which in turn causes high cholesterol levels. The high IDL levels are caused by an abnormality in apolipoprotein E. Apolipoprotein E is a protein found on IDLs which allows the IDLs to bind to liver cells. Without this binding process, the level of IDLs in the blood increase. Lipoproteins are responsible for transporting cholesterol in the bloodstream and high levels of them in the bloodstream means that there are high levels of cholesterol in the bloodstream.
• Type Ia Hyperlipoproteinemia: Type Ia Hyperlipoproteinemia is a rare inherited condition characterized by high levels of chylomicrons (a type of lipoprotein) in the blood. This condition is the result of low levels of lipoprotein lipase which is responsible for lowering lipoprotein levels. Cholesterol levels are usually normal and the risk of heart disease is low.
• Type Ib Hyperlipoproteinemia: Type Ib Hyperlipoproteinemia is a rare inherited condition characterized by high levels of chylomicrons (a type of lipoprotein) in the blood. This condition is the result of low levels of apolipoprotein C-II which is responsible for lowering lipoprotein levels. Cholesterol levels are usually normal and the risk of heart disease is low.
• Type Ic Hyperlipoproteinemia: Type Ic Hyperlipoproteinemia is a rare inherited condition characterized by high levels of chylomicrons (a type of lipoprotein) in the blood. This condition is the result of inhibited lipoprotein lipase activity. Lipoprotein lipase is responsible for lowering lipoprotein levels. Cholesterol levels are usually normal and the risk of heart disease is low.
• Typhoid fever: Fever from bacterial food poisoning.
• Typhus fever: A general name for various arthropod-borne rickettsial infections
• Tyrosinemia: A rare genetic metabolic disorder characterized by a deficiency of particular enzymes which prevents the breakdown of tyrosine which then builds up in the liver.
• UDP-Galactose-4-epimerase deficiency: A condition which is characterised by a mutation in the UDP-galactose-4-epimerase gene.
• VLCAD deficiency: A rare condition that is characterised by cardiomyopathy, fatty liver, skeletal myopathy, pericardial effusions, ventricular arrhythmias, and sudden death
• Valvular dysplasia of the child: A condition which is characterised by the dysplasia of one or more of the valves to a childs heart
• Vaquez disease: An uncommon chronic blood disease involving an increased red blood cell count.
• Ventricular septal defect: An abnormal connection between the 2 lower chambers (ventricles) of the heart.
• Very Long Chain Acyl CoA Dehydrogenase Deficiency - Early onset: Very Long Chain Acyl CoA Dehydrogenase Deficiency is a rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form. The early-onset form is the most severe and can readily lead to death if undiagnosed.
• Very Long Chain Acyl CoA Dehydrogenase Deficiency - adult-onset: Very Long Chain Acyl CoA Dehydrogenase Deficiency is a rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form. The adult-onset form is relatively mild and tends to be characterized mainly by muscle problems.
• Very Long Chain Acyl CoA Dehydrogenase Deficiency - intermediate: Very Long Chain Acyl CoA Dehydrogenase Deficiency is a rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form. The intermediate form is relatively mild and tends to be characterized by periods of low blood sugar - symptoms tend to develop during childhood.
• Very-Long-Chain Acyl-CoA Dehydrogenase Deficiency: A rare inherited genetic condition where the body is unable to convert certain fats to energy i.e. there is not enough of a certain enzyme which is needed to metabolize a type of fat called long-chain fatty acids. The build-up of these fatty acids in the body causes damage. There are three subtypes of the disorder each with varying severity: severe early-onset form, an intermediate form and an adult-onset form.
• Virus associated hemophagocytic syndrome: A fulminant condition characterised by viral infection causing multiple organ infiltrates of haemophagocytic histiocytes.
• Visceral larva migrans: A parasitic roundworm (Toxocara canis or Toxocara cati) infection that normally occurs in cats and dogs but can be transmitted to humans by ingesting the larvae or eggs. The infection may be asymptomatic or severe and symptoms depend on where the larvae travel to when they migrate through the body.
• Visceral leishmaniasis: A tropical disease caused by a protozoan organism and transmitted to humans through sand fly bites. Also called Assam fever, black fever, dumdum fever, ponos or kala-azar.
• Visceral leishmaniasis (kala-azar): A rare infectious disease caused by any of a number of parasitic Leishmania species. Infection can cause any of three different manifestations: cutaneous leishmaniasis, mucosal leishmaniasis and visceral leishmaniasis. Visceral leishmaniasis infection involves the spleen, liver and bone marrow and can be fatal if untreated.
• Vitamin A overdose: Overdose of Vitamin A usually due to Vitamin A supplement overuse or poisoning.
• Vitamin B12 Deficiency: A deficiency of Vitamin B12 primarily causes anemias the body is unable to make sufficient quantities of normal red blood cells. Severe cases can lead to permanent nervous system problems. The vitamin B12 deficiency can result from absorption problems, insufficient dietary intake, certain medications (e.g. metformin), inherited conditions (e.g. transcobalamin deficiency) and certain chronic parasitic intestinal infestations.
• Von Gierke Disease: An inherited metabolic disorder where a deficiency of the enzyme glucose-6-phosphatase prevents glycogen being turned into glucose leading to a buildup of glycogen in the liver and kidneys.
• Von Gierke disease IA: A genetic metabolic disorder involving a deficiency of the enzyme glucose-6-phosphatase which results in the accumulation of glycogen in various tissues. G6P is stored as glycogen until the body needs to convert it to a sugar and use it to create energy. The enzyme deficiency prevents the conversion and hence low blood sugar levels result.
• Von Gierke disease IB: A metabolic disorder involving a deficiency of glucose-6-phosphatase (due to a G6P transporter defect) which results in the accumulation of glycogen in various tissues and a reduced blood sugar level. The condition is characterized primarily by an increased risk of bacterial infections due to impaired neutrophil activity which is needed to fight bacterial invasions.
• WAGR Syndrome: A syndrome that is due to the deletion of chromosome 11.
• WT limb blood syndrome: A rare genetic disorder characterized by blood and limb abnormalities.
• Waldenstrom macroglobulinemia: A plasma cell dyscrasia that resembles leukaemia
• Waldenström Macroglobulinemia: A rare disorder involving malignancy of the lymph and blood cells.
• Walrus poisoning: The walrus is used as a food source in some parts of the world. Eating the liver of the walrus can result in a Vitamin A overdose which can cause serious symptoms and even death in extreme cases.
• Wandering spleen: A very rare birth defect where the structures (ligaments) that hold the spleen in position are absent or not developed properly which allows the spleen to move around in the abdominal cavity.
• Warm Autoimmune Hemolytic Anemia: Warm autoimmune hemolytic anemia is a condition where the body's immune system triggers the production of antibodies against the body's own red blood cells. The red cells are destroyed at an abnormally rapid rate which leads to anemia. Warm haemolytic anemia is characterized by the fact that the abnormal destruction of red blood cells is more active when the patient is exposed to warm temperatures. The severity of the condition varies depending on the underlying cause e.g. cytomegalovirus, hepatitis, HIV and lupus. The condition may develop gradually or occur suddenly and cause serious symptoms.
• Warm-reacting-antibody haemolytic anemia: An autoimmune disorder characterised by the premature destruction of red blood cells
• Warm-reacting-antibody hemolytic anemia: A rare autoimmune condition where the body's defense system attacks and destroys red blood cells. The onset of the condition is triggered by temperatures 37ºC or lower. The severity of the disorder is variable.
• Weber-Christian disease: A form of panniculitis involving fever and liver and spleen enlargement.
• Weil syndrome: A rare infectious disorder which affects liver and kidney function and also causes hemorrhaging. It is a severe form of the second phase of leptospirosis which is an infection caused by the spiral shaped bacteria Leptospira interrogans which is transmitted from animals to humans.
• Weil's syndrome: Severe form of Leptospirosis
• Weinstein Kliman Scully syndrome: A syndrome that is characterised by cardiomyopathy, hypogonadism and metablic anomalies
• Whipple's Disease: Rare malabsorption disease from bacterial digestive infection
• Wilms tumor - aniridia - genitourinary anomalies - mental retardation: A syndrome resulting from deletion of genetic material from chromosome the short arm of chromosome 11 (11p13). The characteristic symptoms are partial or complete absence of iris, genitourinary anomalies, mental retardation and Wilms' tumor. The specific range and severity of symptoms is variable depending on the size and exact location of the genetic material that is missing.
• Wilson's Disease: Wilson disease, or hepatolenticular degeneration, is a neurodegenerative disease of copper metabolism.
• Wilson's disease: Wilson disease, or hepatolenticular degeneration, is a neurodegenerative disease of copper metabolism.
• Wolcott-Rallison syndrome: A very rare genetic condition characterized primarily by the onset of diabetes mellitus during infancy and epiphyseal dysplasia (abnormal joint development).
• Wolman disease: A rare inherited lipid storage disease. The condition usually results in death early in life.
• Wolman syndrome: A form of lipoidosis where acid cholesteryl ester hydrolase deficiency causes an accumulation of lipids (particularly cholesterol esters and triglycerides) in tissues and organs.
• Wyatt disease: A cytomegalus virus infection that occurs before birth.
• X-linked alpha thalassemia mental retardation syndrome (ATR-X): An x-linked condition that features mental retardation, dysmorphic features, and alpha thalassemia
• X-linked lymphoproliferative syndrome: A rare inherited immunodeficiency disorder where the body's immune systm is unable to respond appropriately to certain viral infections (Epstein Barr virus).
• X-linked sideroblastic anemia: X-linked sideroblastic anemia is a rare inherited blood disorder characterized by abnormal deposits of iron in the red blood cells which makes them unable to function properly. The body continues to absorb more iron from digesting food in order to try and correct the problem but this simply leads to excessive iron being deposited in body tissues as the it can't use the iron properly. The severity of the condition is variable.
• Xerocytosis, heriditary: A form of hereditary stomatocytosis syndrome
• Yaws: A rare infections disease caused by the spiral-shaped bacteria Treponema pertenue. The disease consists of three phases: skin lesions are followed by bone, joint and widespread skin symptoms and finally by inflammation and destruction of cartilage in the nose, pharynx and palate. Transmission can be through direct contact with infected skin, insect bites or sex.
• ZAP70 deficiency: A condition characterised by a deficiency of the Zap70 tyrosine kinase which is a component
• Zellweger Syndrome: Zellweger spectrum disorders are a group of rare, genetic, multisystem disorders that were once thought to be separate entities. These disorders are now classified as different expressions (variants) of one disease process. Collectively, they form a spectrum or continuum of disease. Zellweger syndrome is the most severe form; neonatal adrenoleukodystrophy is the intermediate form; and infantile Refsum disease is the mildest form.
• Zellweger-like syndrome, without peroxisomal anomalies: A rare syndrome characterized by unusual facial appearance, very low muscle tone, mental retardation and metabolic defects.
• Zimmerman-Laband syndrome: A rare inherited condition characterized by fibrosis of the gums and ear, nose, nail and finger abnormalities.

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