Causes of Von Willebrand disease
Von Willebrand disease Causes: Book Excerpts
Von Willebrand disease as a symptom:
Conditions listing Von Willebrand disease
as a symptom may also be potential underlying causes of Von Willebrand disease.
Our database lists the following as having
Von Willebrand disease as a symptom of that condition:
Related information on causes of Von Willebrand disease:
As with all medical conditions,
there may be many causal factors.
Further relevant information on causes of Von Willebrand disease may be found in:
Causes of Von Willebrand disease: Online Medical Books
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Von Willebrand's disease:
Causes and incidence
(Professional Guide to Diseases (Eighth Edition))
Unlike hemophilia, von Willebrand’s disease is inherited as an autosomal dominant trait that affects males and females equally. One theory of pathophysiology holds that mild to moderate deficiency of factor VIII and defective platelet adhesion prolong coagulation time. Specifically, this results from a deficiency of the vWF, which stabilizes the factor VIII molecule and is needed for proper platelet function.
Defective platelet function is characterized by:
❑ decreased agglutination and adhesion at the bleeding site
❑ reduced platelet retention when filtered through a column of packed glass beads
❑ diminished ristocetin-induced platelet aggregation.
Recently, an acquired form has been identified in patients with cancer and immune disorders.
Von Willebrand’s disease, which doesn’t have any racial or ethnic associations, affects about 1% of the population.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Von Willebrand's disease:
Causes
(Handbook of Diseases)
Unlike hemophilia, von Willebrand’s disease is inherited as an autosomal dominant trait and occurs more often in females. One theory of pathophysiology holds that mild to moderate deficiency of Factor VIII and defective platelet adhesion prolong coagulation time. Specifically, this results from a deficiency of the von Willebrand factor (VWF), which stabilizes the Factor VIII molecule and is needed for proper platelet function.
Defective platelet function is characterized by:
❑ decreased agglutination and adhesion at the bleeding site
❑ reduced platelet retention when filtered through a column of packed glass beads
❑ diminished ristocetin-induced platelet aggregation.
Recently, an acquired form has been identified in patients with cancer and immune disorders.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
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