Symptoms of Whooping Cough
Symptoms of Whooping Cough
The list of signs and symptoms mentioned in various sources
for Whooping Cough includes the 43
symptoms listed below:
Research symptoms & diagnosis of Whooping Cough:
Whooping Cough: Complications
Review medical complications possibly associated with Whooping Cough:
Whooping Cough Symptoms: Book Excerpts
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Do I have Whooping Cough?
Whooping Cough: Medical Mistakes
Whooping Cough: Undiagnosed Conditions
Diseases that may be commonly undiagnosed in related medical areas:
Home Diagnostic Testing
Home medical tests related to Whooping Cough:
- Child Behavior: Home Testing
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Wrongly Diagnosed with Whooping Cough?
The list of other diseases or medical conditions
that may be on the differential diagnosis list of alternative diagnoses
for Whooping Cough includes:
See the full list of 26
alternative diagnoses for Whooping Cough
Whooping Cough: Research Doctors & Specialists
- Baby & Newborn Health Specialists:
- Ear, Nose & Throat Specialists:
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More about symptoms of Whooping Cough:
More information about symptoms of Whooping Cough and related conditions:
Other Possible Causes of these Symptoms
Click on any of the symptoms below to see a full list
of other causes including diseases, medical conditions, toxins, drug interactions,
or drug side effect causes of that symptom.
Medical Books Online about Whooping Cough
Medical Books Excerpts
Excerpts of published medical book chapters related to Whooping Cough
are available from published medical books
for more detailed information about Whooping Cough.
Medical Books Excerpts
- COUGH
- "Algorithmic Diagnosis of Symptoms and Signs" (2003)
- [ read ]
- STRIDOR
- "Algorithmic Diagnosis of Symptoms and Signs" (2003)
- [ read ]
- WHEEZING
- "Algorithmic Diagnosis of Symptoms and Signs" (2003)
- [ read ]
- Stridor
- "In A Page: Pediatric Signs and Symptoms" (2007)
- [ read ]
- Wheezing
- "In A Page: Pediatric Signs and Symptoms" (2007)
- [ read ]
- COUGH
- "Differential Diagnosis in Primary Care" (2007)
- [ read ]
- Stridor
- "Handbook of Signs & Symptoms (Third Edition)" (2006)
- [ read ]
- Cough
- "A Pocket Manual of Differential Diagnosis" (1999)
- [ read ]
- Wheezing
- "A Pocket Manual of Differential Diagnosis" (1999)
- [ read ]
- Stridor
- "Professional Guide to Signs & Symptoms (Fifth Edition)" (2006)
- [ read ]
- Cough
- "The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter" (2000)
- [ read ]
- Stridor
- "The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter" (2000)
- [ read ]
- Wheezing
- "The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter" (2000)
- [ read ]
- Cough, barking
- "Alarming Signs and Symptoms: Lippincott Manual of Nursing Practice Series" (2007)
- [ read ]
- Cough, productive
- "Alarming Signs and Symptoms: Lippincott Manual of Nursing Practice Series" (2007)
- [ read ]
- Stridor
- "Alarming Signs and Symptoms: Lippincott Manual of Nursing Practice Series" (2007)
- [ read ]
- Wheezing
- "Alarming Signs and Symptoms: Lippincott Manual of Nursing Practice Series" (2007)
- [ read ]
- Stridor
- "Signs & Symptoms: A 2-in-1 Reference for Nurses" (2007)
- [ read ]
- Wheezing
- "Signs & Symptoms: A 2-in-1 Reference for Nurses" (2007)
- [ read ]
- Cough
- "The Diagnostic Approach to Symptoms and Signs in Pediatrics" (2006)
- [ read ]
- Wheezing
- "The Diagnostic Approach to Symptoms and Signs in Pediatrics" (2006)
- [ read ]
- Stridor
- "Nursing: Interpreting Signs and Symptoms" (2007)
- [ read ]
- COUGH
- "Differential Diagnosis in Primary Care" (2007)
- [ read ]
- Cough
- "Pediatric Complaints and Diagnostic Dilemmas" (2003)
- [ read ]
- Cough
- "The 5-Minute Pediatric Consult" (2008)
- [ read ]
Copyright notice for book excerpts: Copyright © 2008 Lippincott Williams & Wilkins. All rights reserved.
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Symptoms of Whooping Cough: Online Medical Books
16 MEDICAL BOOKS ONLINE!
Review excerpts from medical books online, free, without registration,
for more information about the symptoms of Whooping Cough.
Whooping cough:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
After an incubation period of about 7 to 10 days, B. pertussis enters the tracheobronchial mucosa, where it produces progressively tenacious mucus. Whooping cough follows a classic 6-week course that includes three stages, each of which lasts about 2 weeks.
First, the catarrhal stage characteristically produces an irritating hacking, nocturnal cough, anorexia, sneezing, listlessness, infected conjunctiva and, occasionally, a low-grade fever. This stage is highly communicable.
After a period of 7 to 14 days, the paroxysmal stage produces spasmodic and recurrent coughing that may expel tenacious mucus. Each cough characteristically ends in a loud, crowing inspiratory whoop; excessive coughing; and choking on mucus, causing vomiting. (Patients with persistent cough should be evaluated for whooping cough, because not every patient will develop paroxysms or the distinctive whooping sound.) Paroxysmal coughing may induce such complications as nosebleed, increased venous pressure, periorbital edema, conjunctival hemorrhage, hemorrhage of the anterior chamber of the eye, detached retina (and blindness), rectal prolapse, inguinal or umbilical hernia, seizures, atelectasis, and pneumonitis. In infants, choking spells may cause apnea, anoxia, and disturbed acid-base balance. During this stage, patients are highly vulnerable to fatal secondary bacterial or viral infections. Suspect such secondary infection (usually otitis media or pneumonia) in any whooping cough patient with a fever during this stage, because whooping cough itself seldom causes fever.
During the convalescent stage, paroxysmal coughing and vomiting gradually subside. However, for months afterward, even a mild upper respiratory tract infection may trigger paroxysmal coughing. (Paroxysmal coughing may not be present in partially immunized individuals.)
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Acute respiratory failure in COPD:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
In patients who have COPD with ARF, increased ventilation-perfusion mismatch and reduced alveolar ventilation decrease PaO2 (hypoxemia) and increase Paco2 (hypercapnia). This rise in carbon dioxide (CO2) lowers the pH. The resulting hypoxemia and acidemia affect all body organs, especially the CNS and the respiratory and cardiovascular systems.
Specific symptoms vary with the underlying cause of ARF but may include these systems:
❑ Respiratory — Rate may be increased, decreased, or normal depending on the cause; respirations may be shallow, deep, or alternate between the two; and air hunger may occur. Cyanosis may or may not be present, depending on the hemoglobin (Hb) level and arterial oxygenation. Auscultation of the chest may reveal crackles, rhonchi, wheezing, or diminished breath sounds.
❑ CNS — When hypoxemia and hypercapnia occur, the patient may show evidence of restlessness, confusion, loss of concentration, irritability, tremulousness, diminished tendon reflexes, and papilledema; he may slip into a coma.
❑ Cardiovascular — Tachycardia, with increased cardiac output and mildly elevated blood pressure secondary to adrenal release of catecholamine, occurs early in response to low PaO2. With myocardial hypoxia, arrhythmias may develop. Pulmonary hypertension, secondary to pulmonary capillary vasoconstriction, may cause increased pressures on the right side of the heart, elevated jugular veins, an enlarged liver, and peripheral edema. Stresses on the heart may precipitate cardiac failure.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Infant respiratory distress syndrome:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
Although a neonate with IRDS may breathe normally at first, he usually develops rapid, shallow respirations within minutes or hours of birth, with intercostal, subcostal, or sternal retractions, nasal flaring, and audible expiratory grunting. This grunting is a natural compensatory mechanism designed to produce positive end-expiratory pressure (PEEP) and prevent further alveolar collapse.
Severe disease is marked by apnea, bradycardia, and cyanosis (from hypoxemia, left-to-right shunting through the foramen ovale, or right-to-left intrapulmonary shunting through atelectatic regions of the lung). Other clinical features include pallor, frothy sputum, and low body temperature as a result of an immature nervous system and the absence of subcutaneous fat.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Acute respiratory distress syndrome:
Signs and Symptoms
(Professional Guide to Diseases (Eighth Edition))
Rapid, shallow breathing; dyspnea, crackles, rhonchi; hypoxemia; bilateral infiltrates on chest X-ray
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Acute respiratory failure:
Signs and Symptoms
(Professional Guide to Diseases (Eighth Edition))
Shallow or deep respirations (or both), air hunger, cyanosis, adventitious breath sounds, confusion, decreased level of consciousness, tachycardia, pulmonary hypertension, irritability, decreased reflexes
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Respiratory alkalosis:
Signs and Symptoms
(Professional Guide to Diseases (Eighth Edition))
Deep, rapid breathing; dizziness; agitation; circumoral and peripheral paresthesia; carpopedal spasms; twitching; muscle weakness; seizures; arrhythmias
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Respiratory syncytial virus infection:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
Clinical features of RSV infection vary in severity from mild, coldlike symptoms to bronchiolitis or bronchopneumonia and, in a few patients, severe, life-threatening lower respiratory tract infections. Symptoms usually include coughing, wheezing, malaise, pharyngitis, dyspnea, and inflamed mucous membranes in the nose and throat. Reinfection is common, producing milder symptoms than the primary infection.
Otitis media is a common complication of RSV in infants. RSV has also been identified in patients with a variety of central nervous system disorders, such as meningitis and myelitis.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Severe acute respiratory syndrome:
Signs and symptoms
(Professional Guide to Diseases (Eighth Edition))
The incubation period for SARS is typically 3 to 5 days but may last as long as 14 days. Initial signs and symptoms include fever, shortness of breath and other minor respiratory symptoms, general discomfort, headache, rigors, chills, myalgia, sore throat, and dry cough. Some individuals may develop diarrhea or a rash. Later complications include respiratory failure, liver failure, heart failure, myelodysplastic syndromes, and death.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Respiratory acidosis:
Signs and Symptoms
(Professional Guide to Diseases (Eighth Edition))
Confusion, apprehension, asterixis, coma, headache, dyspnea, tachypnea, papilledema, depressed reflexes, tachycardia, hypertension or hypotension, arrhythmias, vasodilation
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Acute respiratory failure in COPD:
Signs and symptoms
(Handbook of Diseases)
In COPD patients with ARF, increased ventilation-perfusion mismatching and reduced alveolar ventilation decrease Pao2 (hypoxemia) and increase Paco2 (hypercapnia). This rise in carbon dioxide tension lowers the pH. The resulting hypoxemia and acidemia affect all body organs, especially the central nervous, respiratory, and cardiovascular systems. Specific symptoms vary with the underlying cause of ARF but can include any of the following:
Respiratory symptoms. The respiratory rate may be increased, decreased, or normal, depending on the cause; respirations may be shallow or deep, or they may alternate between the two; and air hunger may occur. Cyanosis may or may not be present, depending on the hemoglobin (Hb) level and arterial oxygenation. Auscultation of the chest may reveal crackles, rhonchi, wheezes, or diminished breath sounds.
CNS symptoms. The patient may show evidence of restlessness, confusion, loss of concentration, irritability, tremulousness, diminished tendon reflexes, and papilledema; he may slip into a coma.
Cardiovascular symptoms. Tachycardia, with increased cardiac output and mildly elevated blood pressure secondary to adrenal release of catecholamines, occurs early in response to a low Pao2.With myocardial hypoxia, arrhythmias may develop. Pulmonary hypertension also occurs.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Respiratory alkalosis:
Signs and symptoms
(Handbook of Diseases)
The cardinal sign of respiratory alkalosis is deep, rapid breathing, possibly exceeding 40 breaths/minute and much like the Kussmaul’s respirations that characterize diabetic acidosis.
Such hyperventilation usually leads to CNS and neuromuscular disturbances, such as light-headedness or dizziness (from below-normal carbon dioxide levels that decrease cerebral blood flow), agitation, circumoral and peripheral paresthesia, carpopedal spasms, twitching (possibly progressing to tetany), and muscle weakness. Severe respiratory alkalosis may cause cardiac arrhythmias that fail to respond to conventional treatment, seizures, or both.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Respiratory distress syndrome:
Signs and symptoms
(Handbook of Diseases)
Although a neonate with respiratory distress syndrome may breathe normally at first, he usually develops rapid, shallow respirations within minutes or hours of birth, with intercostal, subcostal, or sternal retractions; nasal flaring; and audible expiratory grunting. This grunting is a natural compensatory mechanism designed to produce positive end-expiratory pressure (PEEP) and prevent further alveolar collapse.
The neonate may also display hypotension, peripheral edema, and oliguria; if he has severe disease, apnea, bradycardia, and cyanosis (from hypoxemia, left-to-right shunting through the foramen ovale, or right-to-left shunting through atelectatic regions of the lung) may be present. Other signs and symptoms include pallor, frothy sputum, and low body temperature as a result of an immature nervous system and the absence of subcutaneous fat.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Respiratory syncytial virus infection:
Signs and symptoms
(Handbook of Diseases)
Signs and symptoms of RSV infection vary in severity, ranging from mild coldlike symptoms to bronchiolitis or bronchopneumonia and, in a few patients, severe, life-threatening lower respiratory tract infections. Generally, signs and symptoms include coughing, wheezing, malaise, pharyngitis, dyspnea, and inflamed mucous membranes in the nose and throat.
Otitis media is a common complication of RSV in infants. RSV has also been identified in patients with various central nervous system disorders, such as meningitis and myelitis.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Respiratory acidosis:
Signs and symptoms
(Handbook of Diseases)
Acute respiratory acidosis produces CNS disturbances that reflect changes in the pH of cerebrospinal fluid rather than increased carbon dioxide levels in cerebral circulation.
Effects range from restlessness, confusion, and apprehension to somnolence, with a fine or flapping tremor (asterixis), or coma. The patient may complain of headaches and exhibit dyspnea and tachypnea with papilledema and depressed reflexes. Unless the patient is receiving oxygen, hypoxemia accompanies respiratory acidosis.
This disorder may also cause cardiovascular abnormalities, such as tachycardia, hypertension, atrial and ventricular arrhythmias and, in severe acidosis, hypotension with vasodilation (bounding pulses and warm periphery).
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Cough - Case 4-2: 7-Week-Old Boy:
IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)
Pulmonary vascular resistance determines the extent of the left-to-right shunt.
Pulmonary vascular resistance is elevated at birth and declines to adult levels
over the first week of life. Therefore, with small VSDs, usually no heart
murmur is heard at birth. Most often, the murmur is heard at about 1 to 6 weeks
of age; it is usually holosystolic, harsh, and located along the left sternal
border. Most infants with small VSDs have no significant symptoms and thrive.
In those infants with moderate or large VSDs, symptoms may develop at about 2
weeks of age and can include tachypnea, irritability, diaphoresis or fatigue
with feeding, and failure to thrive. These symptoms develop secondary to
progressive heart failure and pulmonary edema. Not uncommonly, symptoms come to
attention immediately after a respiratory infection, which stresses the infant
's small reserve. With large defects, infants often have a hyperactive precordium
with a palpable thrill. Large VSDs, like small ones, produce an associated
harsh, holosystolic murmur located along the left sternal border.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Complaints and Diagnostic Dilemmas, 2003
Cough - Case 4-3: 7-Month-Old Girl:
IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)
The prodromal phase begins with 3 to 5 days of malaise, fever, cough, coryza,
and conjunctivitis. These symptoms increase over the course of the prodromal
phase. Fevers vary from 39.4
° to 40.6°C and usually peak as the exanthem begins. Just before the development of the
exanthem, Koplik
's spots are noted. These are bluish spots on a red base that are found on the
buccal mucosa. Koplik
's spots are pathognomonic of measles. The exanthem begins as Koplik's spots begin to slough. The rash typically begins on the face and then moves in
a caudal direction. The rash is initially erythematous and maculopapular but
then becomes confluent. The rash usually lasts 5 to 7 days.
Other symptoms can include pharyngitis, lymphadenopathy, splenomegaly, diarrhea,
vomiting, and abdominal pain. With typical measles, patients are ill for 7 to
10 days. However, complications can occur and include pneumonia, encephalitis,
myocarditis, pericarditis, appendicitis, and corneal ulcerations. Subacute
sclerosing panencephalitis (SSPE) is an uncommon neurologic complication of
measles infection. SSPE consists of a degenerative central nervous system
process that is associated with a persistent measles infection.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Complaints and Diagnostic Dilemmas, 2003
Cough - Case 4-6: 4-Month-Old Boy:
IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)
The incubation period is 1 to 3 weeks. Infection is divided into three stages.
The catarrhal stage begins with symptoms of a mild upper respiratory tract
infection and lasts a few days to 1 week. The paroxysmal stage follows, with
the characteristic inspiratory whoop. Posttussive emesis is common, and fever
is infrequent. The whoop is typically absent in infants, because they are
unable to generate the force needed for this maneuver.
Increased intrathoracic and intraabdominal pressures during coughing may lead to
conjunctival and scleral hemorrhages, petechiae on the upper body, epistaxis,
and retinal hemorrhages. In infancy, apnea is a common complication of
B. pertussis infections. Even young adults can have episodes of laryngospasm. Seizures result
from either hypoxia or hyponatremia due to inappropriate secretion of
antidiuretic hormone.
In most cases, a pertussis infection lasts 6 to 10 weeks, but it is not uncommon
for infants and children to have persistent coughs for 3 to 4 months.
Respiratory distress between paroxysms of coughing suggests superinfection with
various viruses (adenovirus, respiratory syncytial virus, cytomegalovirus) or
bacteria (
S. pneumoniae, S. aureus). Other complications include pneumothorax, encephalopathy, and feeding
difficulties in infancy. The disease is most severe in infants younger than 1
year of age, especially premature infants.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Complaints and Diagnostic Dilemmas, 2003
Jaundice - Case 15-1: 14-Day-Old Boy:
IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)
Classic galactosemia is an autosomal recessive disease. If not recognized and
treated, it can be fatal in the neonatal period. Some of the more common
presenting clinical signs have already been discussed (jaundice, hepatomegaly,
vomiting, and encephalopathy); more fulminant clinical courses may represent
superimposed bacterial sepsis. Among the laboratory findings seen with classic
galactosemia are conjugated (or combined) hyperbilirubinemia, liver function
test and coagulation study abnormalities, elevations of serum and urine amino
acids, and a renal tubulopathy with galactosuria, glycosuria, proteinuria, and
metabolic acidosis. Not surprisingly, plasma galactose and erythrocyte
galactose-1-phosphate levels are also elevated.
Disappointingly, even galactosemic children whose diets were restricted very
early are at increased risk for developmental delays and learning disabilities,
compared with their healthy counterparts. Although many children have IQs in
the normal range, cognitive, speech, and motor impairments are, nevertheless,
more common. Longer delays before initial diagnosis and treatment of
galactosemia correlate with worse neurodevelopmental sequelae.
Hypergonadotropic hypogonadism is often observed in girls with galactosemia, and
most are infertile as adults. Galactosemic males demonstrate normal puberty and
fertility.
The gene for the galactose-1-phosphate uridyl transferase enzyme has been
localized to chromosome 9, and multiple variations at that locus have been
described. Some African-Americans with galactosemia have a milder clinical
course because of a different transferase variant. Still another variant, known
as the Duarte variant, is usually clinically insignificant. Prenatal diagnosis
of galactosemia is available.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Complaints and Diagnostic Dilemmas, 2003
Seizures - Case 19-1: 8-Day-Old Girl:
IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)
Neonatal L. monocytogenes infection, like group B streptococcal infection, manifests in both an early- and
a late-onset form. Clinical manifestations of
L. monocytogenes infection are similar to those of other neonatal bacterial infections. Signs of
infection include temperature instability, respiratory distress, irritability,
lethargy, and poor feeding. In early-onset disease, transplacental transmission
after maternal bacteremia or ascending spread from vaginal colonization leads
to intrauterine infection with
L. monocytogenes. Preterm labor is common among infants with early-onset L. monocytogenes infection; length of gestation is less than 35 weeks in approximately 70% of
cases. There is often evidence of an acute febrile maternal illness, with
symptoms of fatigue, arthralgias, and myalgias preceding delivery by 2 to 14
days. Blood cultures are positive for
L. monocytogenes in 35% of mothers of infants with early-onset listeriosis.
Early-onset infection classically develops within the first or second day of
life. Bacteremia (75%) and pneumonia (50%) are usually seen with early-onset
infection. Meningitis is seen in 25% of early-onset cases. In severe infection,
a granulomatous rash is associated with disseminated disease (granulomatosis
infantisepticum). The mortality rate, including stillbirths, is 40% for
early-onset infection. In late-onset infection, modes of transmission unrelated
to maternal carriage may be involved. Late-onset infection develops during the
second to eighth week of life. The most common form
of L. monocytogenes infection over this period is meningitis, which is present in approximately 95%
of cases. Bacteremia (20%) and pneumonia (10%) are less common. Mortality of
late-onset infection is generally low (15%) if the infection is diagnosed early
and treated appropriately.
A nosocomial outbreak occurred when nine newborn infants were bathed in mineral
oil contaminated with
L. monocytogenes. The affected infants developed bacteremia (two cases), meningitis (two cases),
or both (five cases); one infant died. Signs of infection developed within 1
week after exposure to the mineral oil.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Complaints and Diagnostic Dilemmas, 2003
Seizures - Case 19-2: 10-Day-Old Boy:
IV. Clinical Presentation
(Pediatric Complaints and Diagnostic Dilemmas)
Signs of hypocalcemia usually develop within the first 3 weeks of life. Signs of
neonatal hypocalcemia are often nonspecific and may be seen in a variety of
other conditions. Tremors and jitteriness are most commonly seen. Other signs
include irritability, hyperreflexia, facial twitching, carpopedal spasm,
seizures, cyanosis, and, rarely, laryngospasm. More importantly, other
disorders that can manifest with hypocalcemia should be considered. Features of
22q11 deletion syndromes include cleft palate, micrognathia, ear anomalies,
bulbous nasal tip, and conotruncal heart defects. Findings associated with
Albright hereditary osteodystrophy (pseudohypoparathyroidism type Ia) include
round face, short distal phalanges of the thumbs, subcutaneous calcifications,
and a family history of developmental delay and dental hypoplasia.
Sensorineural deafness, renal dysplasia, and mental retardation are also
associated with syndromes that include hypoparathyroidism.
» READ BOOK EXCERPT ONLINE »
Source: Pediatric Complaints and Diagnostic Dilemmas, 2003
Whooping Cough as a Cause of Symptoms or Medical Conditions
When considering symptoms of Whooping Cough, it is also important to consider Whooping Cough as a possible cause of other medical conditions.
The Disease Database lists the following medical conditions that Whooping Cough may cause:
- (Source - Diseases Database)
Whooping Cough: Onset and Incubation
Incubation period for Whooping Cough: 7 to 21 days
Medical articles and books on symptoms:
These general reference articles may be of interest
in relation to medical signs and symptoms of disease in general:
Full list of premium articles on symptoms and diagnosis
About signs and symptoms of Whooping Cough:
The symptom information on this page
attempts to provide a list of some possible signs and symptoms of Whooping Cough.
This signs and symptoms information for Whooping Cough has been gathered from various sources,
may not be fully accurate,
and may not be the full list of Whooping Cough signs or Whooping Cough symptoms.
Furthermore, signs and symptoms of Whooping Cough may vary on an individual basis for each patient.
Only your doctor can provide adequate diagnosis of any signs or symptoms and whether they
are indeed Whooping Cough symptoms.
Adults may already know the new ways to avoid catching and spreading the flu. But you may need to teach these behaviors to kids. Listen to an...
Our parents told us to cover our mouths when we cough. But that might not be the best strategy for flu prevention. Listen to an infection control...
It's inevitable that at some point in your child's life he or she will suffer vomiting and diarrhea. Most often they are nothing to get...
After nine months of doctor's visits, large clothing, physical discomfort and joyous expectation, a mother finally reaches the last stages of...
See full list of 4 related videos
» Next page: Diagnostic Tests for Whooping Cough
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