The list of treatments mentioned in various sources for X-linked sideroblastic anemia includes the following list. Always seek professional medical advice about any treatment or change in treatment plans.
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Treatment of sideroblastic anemias depends on the underlying cause. The hereditary form usually responds to several weeks of treatment with high doses of pyridoxine (vitamin B6). The acquired secondary form generally subsides after the causative drug or toxin is removed, or the underlying condition is adequately treated. Folic acid supplements may also be beneficial when concomitant megaloblastic nuclear changes in RBC precursors are present. Elderly patients with sideroblastic anemia (usually the primary acquired form) are less likely to improve quickly and are more likely to develop serious complications. Deferoxamine may be used to treat chronic iron overload in selected patients.
Carefully crossmatched transfusions (providing needed Hb) or high doses of androgens are effective palliative measures for some patients with the primary acquired form of sideroblastic anemia. However, this form is essentially refractory to treatment and usually leads to death from acute leukemia or from respiratory or cardiac complications.
Some patients with sideroblastic anemia may benefit from phlebotomy to prevent hemochromatosis (the accumulation of iron in body tissues). Phlebotomy steps up the rate of erythropoiesis and uses up excess iron stores; thus, it reduces serum and total-body iron levels.
Source: Professional Guide to Diseases (Eighth Edition), 2005
The underlying cause determines the type of treatment.
Hereditary sideroblastic anemia usually responds to several weeks of treatment with high doses of pyridoxine (vitamin B6).
Elderly patients with sideroblastic anemia — most commonly the primary acquired form — are less likely to improve quickly and are more likely to develop serious complications. Deferoxamine may be used to treat chronic iron overload in selected patients.
Carefully cross-matched transfusions (providing needed Hb) or high doses of androgens are effective palliative measures for some patients with the primary acquired form of sideroblastic anemia. However, this form is essentially refractory to treatment and usually leads to death from acute leukemia or from respiratory or cardiac complications.
CLINICAL TIP: Some patients with sideroblastic anemia may benefit from phlebotomy to prevent hemochromatosis. Phlebotomy steps up the rate of erythropoiesis and uses up excess iron stores; thus, it reduces serum and total-body iron levels.
The secondary acquired form generally subsides after the causative drug or toxin is removed or the underlying condition is adequately treated. Folic acid supplements may also be beneficial when concomitant megaloblastic nuclear changes in RBC precursors are present.
Source: Handbook of Diseases, 2003
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